ライフサイエンスコーパス: 5-HT(1D) receptor

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1 xhibit high binding affinities for the human 5-HT1D receptor.

2 VA Ca2+ currents were mediated by 5-HT1A and 5-HT1D receptors.

3 d selective over the 5-HT(1A), 5-HT(1B), and 5-HT(1D) receptors.

4 It has been observed that reported 5-HT1D receptor agonists have at least one heteroatom (N

5 y)alkylamines may represent a novel class of 5-HT1D receptor agonists.

6 Here, we also report the ability of a 5-HT1D receptor antagonist, GR127935, to antagonize 5-HT

7 Our finding, that 5-HT1D receptors are distributed in nociceptors througho

8 ible explanation for this difference is that 5-HT1D receptors are preferentially expressed by cranial

9 d G protein subunits to membranes containing 5-HT1D receptors caused a small increase in affinity for

10 lves the activation of serotonin subtype 1D (5-HT1D) receptors expressed on "pain-responsive" trigemi

11 5-HT(1B) and 5-HT(1D) receptor expression in the fetal forebrain over

12 uggest that a "pain"-triggered regulation of 5-HT1D-receptor expression underlies the effectiveness o

13 g at least in part through the regulation of 5-HT1D-receptor gene expression.

14 We compared the distribution of 5-HT1D receptor-immunoreactive (5-HT1D-IR) peripheral af

15 Moreover, the widespread expression of 5-HT1D receptor in somatic nociceptive afferents suggest

16 At the ultrastructural level, 5-HT1D receptors in the spinal cord dorsal horn are loca

17 00-fold selective over both the 5-HT(1B) and 5-HT(1D) receptors, it exhibited appreciable 5-HT(1A) re

18 In contrast, the 5-HT1D receptor produced both voltage-dependent and -ind

19 Agonists at serotonin 1D (5-HT1D) receptors relieve migraine headache but are not

20 ptan (1 nM-1 microM), agonists at 5-HT1B and 5-HT1D receptors, respectively.

21 of EPSCs caused by 5-HT was mediated by the 5-HT1D receptor subtype, since the 5-HT1D agonist, sumat

22 There was a good agreement for 5-HT(1B) and 5-HT(1D) receptors to that previously demonstrated in Vg

23 e (8) binds with high affinity at calf brain 5-HT1D receptors, we explored the structure-affinity rel

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