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1 milar before and after the injections of the 5-HT(3) receptor agonists.
2             Pretreatment of ganglia with the 5-HT3 receptor agonist 1-m-(chlorophenyl) biguanide (m-C
3 ione (DNQX) on the excitatory actions of the 5-HT3 receptor agonist 1-phenylbiguanide (PBG) were stud
4    This initial response was mimicked by the 5-HT3 receptor agonist, 2-methyl-5-HT, whereas 5-methoxy
5         The effect was mimicked by two other 5-HT3 receptor agonists, 2-methyl-5-HT and m-chloropheny
6 data are consistent with the hypothesis that 5-HT3 receptor agonists activate DVMN neurones partly by
7                                     Only the 5-HT3 receptor agonist altered the quality of the lordos
8                                            A 5-HT3 receptor agonist CPBG (1 microM), mimicked the unm
9                                 In contrast, 5-HT(3) receptor agonists increased sEPSCs on a minority
10                                              5-HT(3) receptor agonists increased the firing rate of T
11                                              5-HT(3) receptor agonists increased the frequency, but n
12 T(3B) with 5-HT(3A) modified the duration of 5-HT(3) receptor agonist-induced responses, linearized t
13 urrent by low concentrations of bath-applied 5-HT3 receptor agonists is compatible with the cyclic mo
14 Likewise, posterior IC administration of the 5-HT(3) receptor agonist m-chlorophenylbiguanide (mCPBG)
15 sed with the 5-HT1A receptor agonist and the 5-HT3 receptor agonist, m-chlorophenyl-biguanide (mCPBG;
16                                          The 5-HT3 receptor agonist, m-chlorophenylbiguanide (mCPBG),
17 These findings suggest that tachyphylaxis to 5-HT(3) receptor agonists may be due to the desensitizat
18                               The effects of 5-HT3 receptor agonists on cortical circuit response pro
19  pressure and cardiac output elicited by the 5-HT(3)-receptor agonists, phenylbiguanide (100 microg/k
20 retic application of PBG, a highly selective 5-HT3 receptor agonist, significantly increased activity
21 r antagonist ondansetron and mimicked by the 5-HT(3) receptor agonists SR5227 and mCPBG.
22 ptor by intrathecal injection of a selective 5-HT(3) receptor agonist, SR57227, induced spinal glial
23                                              5-HT(3) receptor agonists stimulate intestinal motility,
24  that phenyldiguanide (later recognized as a 5-HT3 receptor agonist) stimulated the firing of C-fibre
25 icrog kg(-1)) and PBG (100 microg kg(-1)), a 5-HT(3) receptor agonist, stimulated nine ischaemically
26 2-methylserotonin (100 microg kg-1, i.a.), a 5-HT3 receptor agonist, stimulated eleven of twelve affe
27         The rank order of potency of all the 5-HT3 receptor agonists tested in the PI assay as compar
28  by white matter stimulation were reduced by 5-HT3 receptor agonists, whereas the frequency of sponta

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