ライフサイエンスコーパス: EF

1 EF and DB measures displayed criterion and construct val

2 EF and DB procedures can conjointly provide useful diagn

3 EF and DB were associated with the rostral dorsolateral

4 EF and DB were distinct but related (r=-0.48).

5 EF data for many PAHs from the eucalypt forest fire were

6 EF global longitudinal strain ratio showed the best perf

7 EF had recovered to > 35% in 72 of 253 (28%) patients at

8 EF was lowest and DB was highest in bvFTD participants.

9 EF was measured with the Delis-Kaplan Executive Function

10 EF was uniquely associated with caudal left dorsolateral

11 EF-Tu is required to support this tRNase activity in vit

12 EFs for other SVOCs including polychlorinated biphenyl (

13 Significantly higher (t test, P < 0.01) EFs (mug kg(-1) dry fuel, gas + particle-associated) for

14 nd the translation elongation factor 1alpha (EF-1alpha) gene.

15 maximum wall thickness </=16 mm and EF>55%), EF global longitudinal strain ratio remained the best pr

16 caused by thermally excited electrons above EF.

17 Moreover, symmetric alternating current (AC) EF directs cell migration in a frequency-dependent manne

18 in-ricin loop of the 50S subunit, activating EF-Tu for GTP hydrolysis and enabling accommodation of t

19 of IHD events and all other outcomes in all EF categories except all-cause mortality in HFpEF.

20 Although EF-Ts is required for toxicity in vivo, our results indi

21 of YmfI, accumulation of 5-aminopentanonated EF-P is inhibitory to swarming motility.

22 compared to placebo treatment, adefovir, an EF inhibitor, decreased urine cAMP levels, water and sod

23 xt showed that coordination of calcium by an EF-hand in ELC1 and prebinding of MLC1 to the MyoA neck

24 ded to test ICD efficacy in patients with an EF >35%.

25 sudden cardiac death among patients with an EF </=35% at baseline, with or without an improvement in

26 tolic dysfunction following acute MI with an EF </=35%, 57% had EF recovery to >35%.

27 /=40% in 50 of the 56 HF cases (89%) with an EF measurement.

28 -insured patients >/=65 years of age with an EF</=35% during the index MI admission from January 2007

29 . population, HF patients across all age and EF groups had markedly lower median survival.

30 iovascular events, and all-cause death), and EF change during a median follow-up of 2.2 years.

31 ial interaction between treatment effect and EF (EF </=40% versus >40%) on the ROSE AHF end points.

32 Study researchers (TS, NP, EK, and EF) rated the adherence.

33 In patients with HF and EF >/=40%, IASD treatment reduces PCWP during exercise.

34 ETHODS AND In 86 stable patients with HF and EF >/=45% in the Karolinska Rennes (KaRen) biomarker sub

35 n load, and age were used to explain IPS and EF in regression models.

36 , PA, translocates the large enzymes, LF and EF, across the endosomal membrane into the host cell's c

37 channel catalyze the translocation of LF and EF.

38 atio of N2 O emission rate and DIN load) and EF(b) (i.e., the ratio of N2 O and DIN concentrations) v

39 bgroups (maximum wall thickness </=16 mm and EF>55%), EF global longitudinal strain ratio remained th

40 Volumes and EFs derived by the area-length method showed correlated

41 rns of biodiversity-ecosystem functioning (B-EF) remains challenging, in part because the importance

42 potential drivers of context dependency of B-EF relations.

43 rtance of environmental factors in shaping B-EF relations is poorly understood.

44 ontinent, we found a tendency for stronger B-EF relations in drier climates as well as in areas with

45 oal showed significant fuel and energy based EF differences for CO, but no significant PM changes wit

46 Subsequently, throughput-based EFs for both the NGPPs and refineries were used to estim

47 dy to understand the inconsistencies between EF and strains.

48 lysis characterised the relationship between EF and DB, binary logistic regression evaluated the incr

49 al equation showing the relationship between EF and the 4 parameters, global longitudinal strain (GLS

50 atory domain containing four calcium-binding EF-hands, a linker loop domain with an amphipathic alpha

51 ranslation elongation that are controlled by EF-P, with a particular focus on the purpose behind the

52 or near-cognate aminoacyl-tRNAs delivered by EF-Tu.

53 RNA(Lys) complex following GTP hydrolysis by EF-Tu.

54 We stratified these patients by EF </=35% and >35% based on the first repeated EF measur

55 RNA(Gly) 'misediting paradox' is resolved by EF-Tu in the cell.

56 Calculated EF had very good agreement with measured EF (r = 0.95).

57 Conduit EFs and total EFs showed gender differences.

58 onance energy transfer (smFRET) between (Cy5)EF-G and (Cy3)tRNALys, we studied the translational elon

59 ested the association between time-dependent EF reassessment and 1-year ICD implantation, stratified

60 to use an emission factor model to determine EFs for CO and then estimate dilution factors using meas

61 decreased in chronic ischemic hearts, as did EF (both p < 0.05).

62 the similarity of variance between different EF measures (verbal abstraction, verbal initiation, moto

63 parameters were not significantly different (EF: 49.3% +/- 11.9 with standard of reference, 48.8% +/-

64 This work focused on S100A11, a dimeric EF-hand protein with two hydrophobic target binding site

65 x binds RNA in vitro, consistent with direct EF-RNA interaction.

66 e resulting closure of the 30S subunit docks EF-Tu at the sarcin-ricin loop of the 50S subunit, activ

67 idyl-tRNA in the small subunit P site during EF-G-catalyzed translocation.

68 interaction between treatment effect and EF (EF </=40% versus >40%) on the ROSE AHF end points.

69 standardized methodology to study evergreen (EF), semi-deciduous (SDF), dry forests (DF) and woody sa

70 is not clear how cells perceive the external EF.

71 The extrafollicular (EF) plasmablast response to self-antigens that contain T

72 or host cell toxin uptake, and edema factor (EF), the toxic moiety which increases host cell cyclic A

73 (PA), lethal factor (LF), and edema factor (EF).

74 The emission factors EF(a) (i.e., the ratio of N2 O emission rate and DIN loa

75 alculated throughput-based emission factors (EF) derived from the NGPP measurements made in this stud

76 reasing particulate matter emission factors (EF).

77 mong household cookstoves, emission factors (EF; g (kg wood)(-1)) were lowest for the Philips, with s

78 (Pol) II associates with elongation factors (EFs).

79 Emission factors (EFs) for 39 individual SVOCs are determined from burning

80 The emission factors (EFs) for a broad range of semivolatile organic chemicals

81 Emission factors (EFs) were calculated for carbon monoxide (CO), carbon di

82 ated using Al-referenced enrichment factors (EFs) and source factors resolved from positive matrix fa

83 Endogenous electric fields (EFs) are involved in developmental regulation and wound

84 subtilis, and B. subtilis encodes the first EF-P ortholog that retains function in the absence of mo

85 Trials were in early-follicular (EF) and mid-luteal (ML) phases in dry (DRY) and humid (H

86 ation of EF-P is thought to be essential for EF-P activity, here we show that in some cases it can be

87 with standard of reference measurements for EF, volume, and mass.

88 In order for EF-P to associate with paused ribosomes, certain tRNAs w

89 or efficient proline-proline bond formation, EF-P, suppress Deltarep DeltauvrD lethality.

90 tionary time, focusing primarily on the four EF-hand motifs.

91 and midrange or preserved ejection fraction (EF >/=40%).

92 death in patients with an ejection fraction (EF) </= 35% and clinical heart failure is well establish

93 nd that patients with low ejection fraction (EF) after myocardial infarction (MI) have their EF reass

94 ression analysis, only LV ejection fraction (EF) and LAS independently indicated the combined end poi

95 ved left ventricular (LV) ejection fraction (EF) and reduced myocardial strain are reported in patien

96 is (LGSAS) with preserved ejection fraction (EF) is incompletely understood.

97 c and nonischemic) and an ejection fraction (EF) of 40% or less were screened; 86 individuals were en

98 ement in left ventricular ejection fraction (EF) to >35% occurs in many patients with reduced EF at b

99 with reduced or preserved ejection fraction (EF) to receive nesiritide or placebo in addition to stan

100 Mean (SD) RV ejection fraction (EF) was 44% (10%), and mean (SD) LV EF was 53% (8%).

101 of patients with reduced ejection fraction (EF) who were undergoing cardiac magnetic resonance (MR)

102 IR (r = 0.92-0.98) for LV ejection fraction (EF), volume, and mass (P < .0001 for all).

103 is and are categorized by ejection fraction (EF).

104 failure (HF) with reduced ejection fraction (EF).

105 kness </=16 mm) or normal ejection fraction (EF).

106 ved left ventricular (LV) ejection fraction (EF).

107 ot and RV dysfunction (RV ejection fraction [EF] <50%) but without severe valvular dysfunction were e

108 obal myocardial function (ejection fraction [EF] and left ventricular end-diastolic pressure) was ass

109 al dimensions, volumes, and empty fractions (EFs) were provided by short axis (SAX) and area-length m

110 the concentrations, enantiomeric fractions (EFs), compound-specific carbon isotope composition of DD

111 utive behaviour (DB) and executive function (EF) in patients with behavioural variant frontotemporal

112 the hairpin promotes dissociation of futile EF-G and thus causes multiple EF-G driven translocation

113 Elongation factor G (EF-G) is a universally conserved translational GTPase th

114 otic protein synthesis, elongation factor G (EF-G), a guanosine triphosphatase (GTPase), binds to the

115 a proper substrate for elongation factor G (EF-G), thus inhibiting translocation until the E-site tR

116 in the context of translationally active GTP.EF-Tu.tRNA ternary complexes.

117 ally cleaves substrate in the context of GTP.EF-Tu.aa-tRNA complexes.

118 ose that EF-Ts promotes the formation of GTP.EF-Tu.tRNA ternary complexes, thereby accelerating subst

119 rvations suggest that the toxin remodels GTP.EF-Tu.aa-tRNA complexes to free the 3'-end of aa-tRNA fo

120 the toxin domain onto previously solved GTP.EF-Tu.aa-tRNA structures reveals potential steric clashe

121 38 (64%) had EF>/=50% (HFpEF), 18 (31%) had EF<50% (HF with reduced EF), and 3 (5%) did not have EF

122 ollowing acute MI with an EF </=35%, 57% had EF recovery to >35%.

123 Of HF cases, 38 (64%) had EF>/=50% (HFpEF), 18 (31%) had EF<50% (HF with reduced E

124 95% confidence interval [CI], 65.9-67.8) had EF reassessment within the next year.

125 HF with reduced EF), and 3 (5%) did not have EF measured.

126 scularized patients were more likely to have EF reassessment (76.9% [95% CI, 75.8-78.0)] versus 53.7%

127 ated that in patients with thickened hearts, EF global longitudinal strain ratio has the best accurac

128 Secretagogin (SCGN), a hexa EF-hand calcium binding protein, plays key roles in insu

129 ciation class III or ambulatory class IV HF, EF >/=40%, exercise PCWP >/=25 mm Hg, and PCWP-right atr

130 e with borderline ejection fraction (HFbEF) (EF 41% to 49%), and heart failure with reduced ejection

131 ic significance in HF with midrange (HFmrEF; EF 40%-50%) and preserved EF (HFpEF; EF >/=50%) has been

132 included HFpEF (EF>/=50%), borderline HFpEF (EF 40%-49%), HFrEF (EF<40%), and HF of unknown type (EF

133 Outcomes included HFpEF (EF>/=50%), borderline HFpEF (EF 40%-49%), HFrEF (EF<40%)

134 the 8,873 hospitalized patients with HFpEF (EF >/=50%) in the Medicare-linked OPTIMIZE-HF (Organized

135 re with preserved ejection fraction (HFpEF) (EF >/=50%), heart failure with borderline ejection fract

136 HFmrEF; EF 40%-50%) and preserved EF (HFpEF; EF >/=50%) has been much less explored.

137 =50%), borderline HFpEF (EF 40%-49%), HFrEF (EF<40%), and HF of unknown type (EF missing).

138 lure with reduced ejection fraction (HFrEF) (EF </=40%).

139 (HF) with reduced ejection fraction (HFrEF; EF <40%) is well established, but its pathogenic and pro

140 tegory and less likely to change to a higher EF category over time.

141 10), due to the approximately 60-fold higher EFs for 3-ring PAHs from the former.

142 The model further demonstrated how EF can be maintained in ventricles with increased wall t

143 Most bacterial trGTPases, including IF2, EF-Tu, EF-G and RF3, play well-known roles in translatio

144 lt, rAAV.Tbeta4 but not rAAV.VEGF-A improved EF in db hearts (34.5 +/- 1.4%), but less so than in wt

145 the toxin induces conformational changes in EF-Tu, displacing a beta-hairpin loop that forms a criti

146 d rafts, are the primary response element in EF sensing.

147 f follow-up, those who had an improvement in EF to >35% accrued a similar relative reduction in morta

148 baseline, with or without an improvement in EF to >35% during follow-up.

149 effects of monthly high-dose IV pulse MTX in EF.

150 ell migration motility nor directionality in EFs.

151 site) of an open 30S subunit, while inactive EF-Tu is separated from the 50S subunit.

152 re-accommodation intermediates with inactive EF-Tu.

153 inical and cardiac MR risk factors-including EF and LGE.

154 Ca(2+) and Mg(2+) binding to the individual EF-hands, observed metal-induced conformational changes,

155 s, thus within the uncertainty range of IPCC-EF.

156 lex formation of the immune receptor kinases EF-TU RECEPTOR (EFR) and FLAGELLIN-SENSING 2 (FLS2) with

157 mes (all P < 0.001) and overestimated the LA EFs (all P < 0.001).

158 of electronic states below the Fermi level (EF) with increasing temperature, as well as Pauli blocki

159 iabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.051 per mm decrease; 95% confidenc

160 CD implantation in post-MI patients with low EF.

161 , were also more likely to change to a lower EF category and less likely to change to a higher EF cat

162 ng an MRA tended to be younger, with a lower EF, lower systolic blood pressure, and more advanced HF

163 The quantified thresholds (RV EF <30% and LV EF <45%) may be implemented in noninvasive risk stratifi

164 io, 3.90; 95% CI, 1.84-8.26; P < .001 and LV EF <45%: hazard ratio, 3.23; 95% CI, 1.57-6.65; P = .001

165 and offers incremental information beyond LV EF and the presence of myocardial fibrosis.

166 on and regression tree analysis, combined LV EF and LAS cutoff values were used to stratify patients

167 raction (EF) was 44% (10%), and mean (SD) LV EF was 53% (8%).

168 Calmodulin-like (CML) proteins are major EF-hand-containing, calcium (Ca(2+))-binding proteins wi

169 contributes to chromatin association of many EFs, including the Pol II serine 2 kinases Ctk1 and Bur1

170 Here we show that many EFs crosslink to RNA emerging from transcribing Pol II i

171 ted EF had very good agreement with measured EF (r = 0.95).

172 d comparing model-predicted EF with measured EF.

173 Most EFs crosslink preferentially to mRNAs, rather than unsta

174 tion of futile EF-G and thus causes multiple EF-G driven translocation attempts.

175 ge that is most prominent around the mutated EF-hand IV, as well as throughout the C-domain.

176 Overall the mean N2O-EF was 1.2% for the tropics and sub-tropics, thus within

177 Our annual N2O-EFs, estimated for a range of fertiliser rates using the

178 ting annual N2O emissions and estimating N2O-EFs, particular attention should be paid in modelling th

179 ic-specific annual N2O emission factors (N2O-EFs) using a Generalized Additive Mixed Model (GAMM) whi

180 tudies hypothesising non-linear increase N2O-EFs as a function of applied N.

181 On a regional basis, mean N2O-EFs were 1.4% for Africa, 1.1%, for Asia, 0.9% for Austr

182 ps as those with mild hypertrophy and normal EF.

183 The toxin binds exclusively to domain 2 of EF-Tu, partially overlapping the site that interacts wit

184 Maximal activity of EF-P requires a posttranslational modification in Escher

185 of the SCD-HeFT had a repeated assessment of EF a mean (SD) of 13.5 (6) months after randomization.

186 tion destabilises the native conformation of EF-hand IV, leading to a transient unfolding and dissoci

187 process of translation through impairment of EF-Tu function.

188 0.005), concomitant with a distinct loss of EF (44.9% vs. 53.4% in nondiabetic controls; p < 0.05).

189 en RNA emerges from Pol II, and that loss of EF-RNA interactions upon RNA cleavage at the polyadenyla

190 Thus, while modification of EF-P is thought to be essential for EF-P activity, here

191 e behind the many different modifications of EF-P.

192 Ca(2+) binding domain encompassing a pair of EF-hand motifs (EF1 and EF2) in the skeletal muscle ryan

193 The physiological role of LepA, a paralog of EF-G found in all bacteria, has been a mystery for decad

194 This study examines rates of EF reassessment and their association with 1-year ICD im

195 Reassessment of EF was associated with a higher likelihood of ICD implan

196 the protein YmfI catalyzes the reduction of EF-P-5 aminopentanone to EF-P-5 aminopentanol.

197 with both aa-tRNA and the switch I region of EF-Tu.

198 as been as laboratory tools for the study of EF-Tu and the ribosome, as their poor pharmacokinetic pr

199 The effect of the different parameters on EF was explored in the model and compared with findings

200 sence of YmfI were found at two positions on EF-P, including one that changed the conserved modificat

201 Once EF-P is accommodated into the ribosome and bound to Pro-

202 tion factor eIF5A and its bacterial ortholog EF-P bind in the E site of the ribosome where they conta

203 Elongation factor P (EF-P) binds to ribosomes requiring assistance with the f

204 Translation elongation factor P (EF-P) in Bacillus subtilis is required for a form of sur

205 trikingly, we show that elongation factor P (EF-P), traditionally known to alleviate ribosome stallin

206 e performance did not differ between phases [EF: 257 (37), ML: 255 (43) kJ, P = 0.62], but was 7 (9)%

207 We examined rates of postdischarge EF reassessment and ICD implantation among 10 289 Medica

208 In US practice, EF reassessments are commonly performed among patients w

209 rom 16% to 72% and comparing model-predicted EF with measured EF.

210 midrange (HFmrEF; EF 40%-50%) and preserved EF (HFpEF; EF >/=50%) has been much less explored.

211 ymptomatic patients with LGSAS and preserved EF underwent cardiac catheterization with comparison of

212 essures in patients with LGSAS and preserved EF, enabling reclassification to moderate stenosis in 25

213 Reduced deformation despite preserved EF can be explained through geometric factors.

214 sodium nitroprusside in LGSAS with preserved EF.

215 systolic function in patients with preserved EF.

216 glycolipids are therefore the likely primary EF sensor driving polarization of membrane proteins and

217 sformed in 14 steps into the fully protected EF and GH disaccharide fragments.

218 g patients with MI with an initially reduced EF.

219 to >35% occurs in many patients with reduced EF at baseline.

220 HFpEF), 18 (31%) had EF<50% (HF with reduced EF), and 3 (5%) did not have EF measured.

221 pEF, and 3.17 (0.8-13.0) for HF with reduced EF.

222 Repeated EF was >35% in 186 participants (29.8%) randomized to IC

223 (95% CI, 0.29-1.30) in those with a repeated EF >35%.

224 ticipants in the SCD-HeFT who had a repeated EF assessment during the course of follow-up, those who

225 % CI, 0.48-0.85) in patients with a repeated EF of </=35% and 0.62 (95% CI, 0.29-1.30) in those with

226 on between treatment assignment and repeated EF for predicting mortality.

227 </=35% and >35% based on the first repeated EF measurement after randomization and compared all-caus

228 Follow-up started with the repeated EF assessment.

229 is distinct and ligated onto its respective EF-P through entirely convergent means.

230 eta-galactosidase, gamma-secretase, ribosome-EF-Tu complex, 20S proteasome and RNA polymerase III, we

231 lows down the rearrangements in the ribosome-EF-Tu-GDP-Pi-Lys-tRNA(Lys) complex following GTP hydroly

232 Here we asked which role EF hand domain containing 2 (EFhd2; Swiprosin-1) plays i

233 imal thresholds for ventricular function (RV EF <30%: hazard ratio, 3.90; 95% CI, 1.84-8.26; P < .001

234 Losartan did not significantly improve RV EF in comparison with placebo (+0.51%; 95% confidence in

235 is, losartan was associated with improved RV EF in a subgroup (n=30) with nonrestrictive RV and incom

236 ave a statistically significant impact on RV EF in subgroups with symptoms, restrictive RV, RV EF<40%

237 subgroups with symptoms, restrictive RV, RV EF<40%, pulmonary valve replacement, or QRS fragmentatio

238 The quantified thresholds (RV EF <30% and LV EF <45%) may be implemented in noninvasiv

239 The primary outcome was RV EF change, determined by cardiovascular MRI in intention

240 This was consistent with similar EF in 20 control subjects and 20 hypertrophic cardiomyop

241 Furthermore, B. subtilis EF-P is post-translationally modified with a 5-aminopent

242 ol moiety attached to Lys(32) of B. subtilis EF-P that is required for swarming motility.

243 pproximately 0.2 degrees C higher in ML than EF (P < 0.01) independent of environment (P = 0.66).

244 on the bacterial ribosome and indicate that EF-P can aid in resuming translation elongation stalled

245 We propose that EF-RNA interactions facilitate assembly of the elongatio

246 We propose that EF-Ts promotes the formation of GTP.EF-Tu.tRNA ternary c

247 Overall results showed that EF measures were clustered separately from the ToM measu

248 The EF was >/=40% in 50 of the 56 HF cases (89%) with an EF

249 we found that in the presence of Ca(2+) the EF-hands of human plastins bound to an immediately adjac

250 ts hospitalized with HF, patients across the EF spectrum have a similarly poor 5-year survival with a

251 These results indicate that, although the EF-hand domain is not required for RyR2 activation by cy

252 vanced synthetic intermediate containing the EF and G rings of the target.

253 depletion amplified rather than dampened the EF response in Fas-intact but not Fas-deficient mice.

254 unit) with a total yield of 24% (36% for the EF fragment).

255 revealed well-ordered conformations for the EF loop of VP2, the GH loop of VP3, and the N-terminal e

256 ameshift promoting signals mostly impair the EF-G-catalyzed translocation step of the two tRNALys and

257 efficient protocol for the synthesis of the EF fragment of idraparinux and its C5'-epi analogue (GH

258 pery sequence also helps dissociation of the EF-G by providing alternative base-pairing options.

259 titution (ParvE101Q) at amino acid 12 of the EF-hand Ca(2+)/Mg(2+) binding loop disrupts bidentate Ca

260 n protein export is centrin, a member of the EF-hand superfamily of Ca(2+)-binding proteins.

261 tal NPP is highest in the SDF site, then the EF followed by the DF and WS and that (iii) different fo

262 studies revealed that Ca(2+) binding to the EF-hands of hSCGN induces significant structural changes

263 On the basis of the EFs determined in this work, estimates of the annual emi

264 after myocardial infarction (MI) have their EF reassessed 40 days after MI for implantable cardiover

265 very few deaths, regardless of whether their EF was above or below 35%.

266 nal transmission from Ca(2+) to ABD2 through EF-hands remains to be established.

267 Thus, EF values derived from both methods were below the curre

268 es the reduction of EF-P-5 aminopentanone to EF-P-5 aminopentanol.

269 ngly suggest that nascent RNA contributes to EF recruitment to transcribing Pol II.

270 protein identified in the pathway leading to EF-P modification in B. subtilis, and B. subtilis encode

271 t GCS contributes more than twice as much to EF than GLS.

272 2.2-55.2]; P<0.001) and had shorter times to EF reassessment (median, 67 versus 84 days; P<0.001) tha

273 Conduit EFs and total EFs showed gender differences.

274 nd to the ribosome and induce translocation, EF-G*GDP in complex with phosphate group analogs BeF3(-)

275 coiled-coil domain of elongation factor Ts (EF-Ts) and confer resistance to the CdiA-CT(EC869) tRNas

276 diA-CT(EC869) binds to elongation factor Tu (EF-Tu) with high affinity and this interaction is critic

277 e immunity protein and elongation factor Tu (EF-Tu).

278 cyl-tRNAs delivered by elongation factor Tu (EF-Tu).

279 t bacterial trGTPases, including IF2, EF-Tu, EF-G and RF3, play well-known roles in translation.

280 9%), HFrEF (EF<40%), and HF of unknown type (EF missing).

281 tra low background electroformed copper (ULB EF-Cu) foil cut and folded into boats, (2) dissolving bo

282 The use of low mass ULB EF-Cu boats for dry ashing successfully overcame the pro

283 7 (facility-scale) larger than industry-used EFs.

284 ss II to IV symptoms, and a left ventricular EF of 40% or less to treatment with enalapril 10 mg twic

285 ound on secondary outcomes: left ventricular EF, peak aerobic exercise capacity, and N-terminal pro-b

286 t ventricular dysfunction (right ventricular EF 45+/-12 versus 53+/-28%, P=0.04).

287 The underlying mechanism by which EF-P facilitates this reaction seems to have entropic or

288 Here, we show that while EF-G*GDP does not stably bind to the ribosome and induce

289 ICD vs placebo group, both in patients whose EF remained </=35% (7.7 vs 10.7 per 100 person-year foll

290 00 person-year follow-up) and in those whose EF improved to >35% (2.6 vs 4.5 per 100 person-year foll

291 in mortality with ICD therapy as those whose EF remained </=35%.

292 somal conformational changes associated with EF-G dissociation upon unsuccessful translocation attemp

293 maging for assessment of LV dysfunction with EF less than 50%.

294 rolled trial to evaluate the IASD in HF with EF >/=40%.

295 Among patients with EF </=35% during the index MI admission, 66.8% (95% conf

296 Among patients with EF reassessment, only 11% received an ICD within 1 year.

297 tes remain very low even among patients with EF reassessment, regardless of revascularization status.

298 ers by echocardiography in 100 subjects with EF ranging from 16% to 72% and comparing model-predicted

299 Although 1-year EF reassessment is associated with increased likelihood

300 eived Ad5.hAC6 (mean [SE] age, 63 [1] years; EF, 30% [1%]), and 14 individuals (25%) received placebo

301 s (25%) received placebo (age, 62 [1] years; EF, 30% [2%]).