ライフサイエンスコーパス: N-formylated peptide

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1 ole in degrading bacterial and mitochondrial N-formylated peptides.

2 asin-deficient mice, even in the presence of N-formylated peptides.

3 s Ib molecule with a high propensity to bind N-formylated peptides.

4 The lack of endogenous N-formylated peptide allows discovery of novel pathogen-

5 in nociceptor neurons, in part via bacterial N-formylated peptides and the pore-forming toxin alpha-h

6 + T cells, which recognize short hydrophobic N-formylated peptides, appear to comprise a substantial

7 tide deformylase using a series of synthetic N-formylated peptides as substrates.

8 Although three N-formylated peptides derived from L. monocytogenes are

9 ned whether hPepT1 could transport the model n-formylated peptide fMLP and, if so, whether such cellu

10 ed by prior exposure to the chemoattractants N-formylated peptides (fMLP) or a complement cleavage pr

11 f the fifth component of complement (C5a) or n-formylated peptides (formylmethionylleucylphenylalanin

12 complex (MHC) class Ib molecule H2-M3 binds N-formylated peptides from mitochondria and bacteria.

13 he cell surface by addition of high-affinity N-formylated peptides from mitochondria and listeria.

14 Upon addition of exogenous N-formylated peptides, M3 trafficks rapidly to the cell

15 Pseudopod growth was stimulated using N-formylated peptide (N-formyl-methionyl-leucyl-phenylal

16 , whereas tapasin is critical for loading of N-formylated peptides onto the intracellular pool of M3.

17 e to the classic bacterial chemoattractants, n-formylated peptides, or other soluble bacterial factor

18 ein is catalytically active in deformylating N-formylated peptides, shares many of the properties of

19 Purified deformylase is highly active toward N-formylated peptide substrates.

20 ass Ib molecule that preferentially presents N-formylated peptides to CD8(+) T cells.

21 MHC class Ib molecule H2-M3 presents N-formylated peptides to CD8+ CTLs.

22 lity complex class Ib molecule that presents N-formylated peptides to cytotoxic T cells.

23 Addition of N-formylated peptides to TAP(o) cells stabilizes M3 in t

24 le of the mouse with a unique preference for N-formylated peptides, which may come from the N-termini

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