ライフサイエンスコーパス: NADPH diaphorase

1 the nodose also were stained positively for NADPH-diaphorase.

2 immunoreactive cells were also positive for NADPH-diaphorase.

3 f this study was to determine the changes in NADPH-diaphorase (a commonly used marker for neuronal NO

4 ere used for dual localization of Phox2a and NADPH diaphorase, a marker of nitric oxide-containing ne

5 e Fos-immunoreactive neurons did not contain NADPH-diaphorase, a marker for nitric oxide synthase.

6 is study demonstrates the co-localisation of NADPH diaphorase activity and GFAP immunoreactivity in n

7 kocytes contained little iNOS antigen and no NADPH diaphorase activity and were minimally able to con

8 n nNOS to basal and injury-induced levels of NADPH diaphorase activity in MNs.

9 on of cyclic guanosine monophosphate (cGMP), NADPH diaphorase activity, and nitrotyrosine occurred 3

10 nthase (eNOS)--these cells also co-localised NADPH diaphorase activity.

11 hydroxylase, somatostatin, substance P, and NADPH diaphorase activity.

12 nicotinamide adenine dinucleotide phosphate (NADPH) diaphorase activity.

13 cally, the development of neurons expressing NADPH-diaphorase activity (an early marker found in inhi

14 NADPH-diaphorase activity also was observed in the later

15 Similar to iNOSFL, iNOS8(-)9(-) exhibited NADPH-diaphorase activity and contained tightly bound ca

16 Throughout this period, the optic lobes show NADPH-diaphorase activity and stain with an antibody to

17 n the literature relating to the presence of NADPH-diaphorase activity in hippocampal principal cells

18 The intensity of NADPH-diaphorase activity in pyramidal cells located in

19 If NADPH-diaphorase activity is an indicator of NOS, then o

20 critical determinants of CA1 pyramidal cell NADPH-diaphorase activity is shown to be incubation of b

21 The restrained-induced NADPH-diaphorase activity was significantly higher in th

22 d reductions in ischemia-induced BH4 levels, NADPH-diaphorase activity, and caspase-3 gene expression

23 rom guinea pig hearts stained positively for NADPH-diaphorase activity, suggesting that these cells d

24 ere also found to contain NOS and to possess NADPH-diaphorase activity.

25 4% of LES-projecting neurons also contained NADPH-diaphorase activity; however, TH immunoreactivity

26 n of the ventromedial nucleus contained both NADPH diaphorase and brain nitric oxide synthase as demo

27 That NADPH diaphorase and brain nitric oxide synthase were fo

28 inding cells was shown by co-localization of NADPH diaphorase and estrogen receptor and brain nitric

29 Mitochondria in mSOD1 mouse MNs accumulate NADPH diaphorase and inducible nitric oxide synthase (iN

30 The distribution of the enzymes NADPH diaphorase and nitric oxide synthase in the ventro

31 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase and Fos.

32 anglia were extremely rich in VIP, nNOS, and NADPH-diaphorase and moderate in CHAT.

33 d plates were also demonstrated in SM, using NADPH-diaphorase and NOS immunoreactivity, indicating ni

34 small vessels exhibited strong staining for NADPH-diaphorase but no nNOS immunoreactivity.

35 the entire ventromedial nucleus showed that NADPH diaphorase cellular staining was localized primari

36 ed a high density of nNOS immunopositive and NADPH-diaphorase containing neurons and fibers at the ro

37 Combined techniques for NADPH-diaphorase enzyme histochemistry and huntingtin im

38 tive, affecting principally parvalbumin- and NADPH diaphorase-expressing interneurons.

39 n influences neuronal process projection for NADPH diaphorase-expressing, but not acetylcholinesteras

40 NADPH diaphorase histochemical and NOS I immunohistochem

41 ine acetyltransferase immunocytochemical and NADPH diaphorase histochemical preparations at ages (pos

42 We used a modified form of NADPH diaphorase histochemistry to compare the neuroanat

43 cholinergic (NANC) relaxation, NOS activity, NADPH diaphorase histochemistry, NOS immunohistochemistr

44 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry and in situ hybridizati

45 udies used nicotinamide adenine diphosphate (NADPH)-diaphorase histochemistry as an indicator of nitr

46 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry to identify populations

47 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry to investigate nitric o

48 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry.

49 al nucleus of the trapezoid body (MNTB) with NADPH-diaphorase histochemistry and in situ hybridizatio

50 fied with either ChAT immunocytochemistry or NADPH-diaphorase histochemistry and they appeared to be

51 NADPH-diaphorase histochemistry has been shown to stain

52 + neurons and fibers were also identified by NADPH-diaphorase histochemistry in sections and whole-mo

53 ic oxide synthase activity (visualized using NADPH-diaphorase histochemistry) was undetectable in the

54 The brainstem nuclei were also visualized by NADPH-diaphorase histochemistry, a marker of nNOS activi

55 Using NADPH-diaphorase histochemistry, neuronal nitric oxide s

56 Furthermore, NADPH-diaphorase immunohistochemical staining of neurons

57 Generally, the staining pattern of nNOS and NADPH-diaphorase in the NTS was similar.

58 The localization of NADPH-diaphorase in these efferents indicated that they

59 iety of identified neurons were positive for NADPH-diaphorase in various central ganglia, including t

60 NADPH diaphorase is also present in a subpopulation of t

61 We conclude that (i) fixative-resistant NADPH-diaphorase is a characteristic marker of 12-15% of

62 NOS I, as defined mainly by the presence of NADPH diaphorase, is present in a subpopulation of both

63 At the cellular level the NADPH-diaphorase marker for nNOS revealed a significant

64 the presence and subcellular distribution of NADPH diaphorase (NADPH-d)/nitric oxide synthase (NOS) i

65 Expression of specific, fixative-resistant NADPH-diaphorase (NADPH-d) activity, characteristic of N

66 Recent evidence indicates that NADPH-diaphorase (NADPH-d) and nitric oxide synthase (NO

67 usc Aplysia californica was studied by using NADPH-diaphorase (NADPH-d) histochemistry in the CNS and

68 means of specific antibodies against NOS and NADPH-diaphorase (NADPH-d) histochemistry, which, with t

69 eech, Hirudo medicinalis, were studied using NADPH-diaphorase (NADPH-d) histochemistry.

70 californica was studied histochemically via NADPH-diaphorase (NADPH-d) reduction of Nitro Blue Tetra

71 ined the normal distribution of constitutive NADPH-diaphorase (NADPH-d), a marker for nitric oxide sy

72 Formaldehyde-insensitive NADPH diaphorase (NADPHd) activity is used widely as a h

73 e synthase (nNOS) and enzymatic activity for NADPH diaphorase (NADPHd) are extensively colocalized in

74 labeling (IFL) methods, and each followed by NADPH diaphorase (NADPHd) histochemical staining in the

75 n parvalbumin (PV) and nitric oxide synthase NADPH diaphorase (NADPHd) is well documented within neur

76 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase (NADPHd) and parvalbumin (PV)-positive

77 NADPH-diaphorase (NADPHd) has been determined biochemica

78 NADPH-diaphorase (NADPHd) histochemistry or NOS-immunost

79 gregates were present in almost all cortical NADPH-diaphorase neurons and in approximately 50% of the

80 were more prominent as nuclear inclusions in NADPH-diaphorase neurons, with less perikaryal and neuro

81 myenteric and submucosal plexus stained with NADPH diaphorase (neurons and neurites), anti-TuJ1 (neur

82 A few VIP+ and NADPH- diaphorase+ neurons were also observed in the cho

83 increase in the number of cells labeled for NADPH diaphorase or neuronal NOS in the lumbosacral spin

84 es, with little or no colocalization between NADPH-diaphorase or nitric oxide synthase neurons and hu

85 Axons labeled for VIP, nNOS, NADPH-diaphorase, or the 3A10 antigen could be traced fr

86 ic striatal neurones and the degeneration of NADPH-diaphorase positive interneurones within 24 h.

87 in activity was in accordance with decreased NADPH-diaphorase-positive cells and decreased staining o

88 In contrast, a similar loss of NADPH-diaphorase-positive cells was observed in the stri

89 NADPH-diaphorase-positive cells were also increased in t

90 sed by efferent fibers and to localize these NADPH-diaphorase-positive efferent cell bodies in the tu

91 ound the locus coeruleus corresponded to the NADPH-diaphorase-positive efferent cells in the avian is

92 n which P-CREB-lir was induced by light were NADPH-diaphorase-positive neurons of the SCN's retinorec

93 nNOS immunostaining and NADPH-diaphorase-positive neurons were significantly inc

94 At both dorsal and ventral levels, NADPH-diaphorase-positive subicular pyramidal cells and

95 ric ganglia, total neurons per ganglion, and NADPH diaphorase presumptive inhibitory neurons per gang

96 nicotinamide-adenine dinucleotide phosphate (NADPH)- diaphorase reaction was used as a marker for nit

97 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase reaction, we determined that the organ

98 The NADPH-diaphorase reaction was also used as a marker for

99 r of studies have mapped the distribution of NADPH-diaphorase-reactive neurons in the hippocampal for

100 oint of controversy concerns the presence of NADPH-diaphorase-reactive pyramidal cells in the CA1 sub

101 results show that nNOS immunoreactivity and NADPH-diaphorase reactivity are consistently increased i

102 nNOS immunostaining and NADPH-diaphorase reactivity was neither altered in the g

103 rs exhibiting both nNOS immunoreactivity and NADPH-diaphorase reactivity was present in the central,

104 nNOS expression and NADPH-diaphorase reactivity were quantified by using a m

105 teric neurons, and whole-mount staining with NADPH-diaphorase showed that myenteric and submucosal ga

106 Diabetic mice had loss of NADPH diaphorase-stained myenteric neurons, delayed gast

107 NADPH diaphorase staining (NOS index) intensity was high

108 as demonstrated by in situ hybridization and NADPH diaphorase staining in rats treated with TGF-beta1

109 NADPH diaphorase staining was visible in both neuronal p

110 as 32 and 25, three defined bands of diffuse NADPH diaphorase staining were located in layer 2 and in

111 teral dorsal tegmental nucleus identified by NADPH diaphorase staining, as well as the cuneiform nucl

112 NADPH-diaphorase staining also was detected in blood ves

113 O-producing neurons using fixation-resistant NADPH-diaphorase staining and antisera that recognize a

114 ed, however, between nNOS immunostaining and NADPH-diaphorase staining in blood vessels in the brains

115 treated arteries showed a 2-fold increase in NADPH-diaphorase staining intensity relative to sham-inf

116 urons exhibiting NOS activity as assessed by NADPH-diaphorase staining was significantly greater in t

117 study, nitric oxide synthase (NOS) activity (NADPH-diaphorase staining), neuronal NOS (nNOS) protein,

118 independent of cell volume, correlates with NADPH-diaphorase staining, and appears to be a character

119 inhibitory neurotransmitter NO, as shown by NADPH-diaphorase staining, and the glial marker GFAP.

120 fibers exhibiting nNOS immunoreactivity and NADPH-diaphorase staining.

121 urons and in approximately 50% of the spared NADPH-diaphorase striatal neurons from early grade HD ca

122 n to be involved in learning and memory, the NADPH-diaphorase technique was used in conjunction with

123 rons was determined on sections stained with NADPH diaphorase to identify the cholinergic boundaries

124 cotinamide adenosine dinucleotide phosphate (NADPH)-diaphorase, tyrosine hydroxylase (TH), and dopami

125 n almost perfect co-localization of ChAT and NADPH-diaphorase was also observed.

126 ns and fibers that stained for both nNOS and NADPH-diaphorase was noted in the interstitial and ventr

127 containing choline acetyltransferase and/or NADPH diaphorase were studied in E12.5-E17.5 reeler and

128 Nerve terminals positive for NADPH- diaphorase were colocalized with SM alpha-actin-p

129 ioside (P-Path), and the granule cell marker NADPH-diaphorase, were disrupted.