ライフサイエンスコーパス: PSVT

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1 After the first or second bolus, PSVT converted to sustained sinus rhythm for > or =5 min

2 ophysiologic procedure because of documented PSVT and were found to have dual AV node physiology or i

3 en required for an accurate diagnosis during PSVT.

4 cing maneuvers that are commonly used during PSVT in the electrophysiology laboratory.

5 n 37 vs. 69 years, p = 0.0002), had a faster PSVT heart rate (mean 186 vs. 155 beats/min, p = 0.0006)

6 The second bolus was administered only if PSVT persisted for 1 minute after the first bolus.

7 ermination of electrophysiologically induced PSVT.

8 a history of symptomatic PSVT and inducible PSVT at the time of a clinically indicated electrophysio

9 story of PSVT at times do not have inducible PSVT in the electrophysiology laboratory.

10 single AV node echo beats, but no inducible PSVT despite the administration of isoproterenol and atr

11 e-entrant tachycardia, n = 8) with inducible PSVT sustained for > or =1 min during an electrophysiolo

12 ascular disease were labeled as having "lone PSVT."

13 ildbearing years in 58% of females with lone PSVT versus 9% of females with other cardiovascular dise

14 ther cardiovascular disease, those with lone PSVT were younger (mean 37 vs. 69 years, p = 0.0002), ha

15 r cardiovascular disease and those with lone PSVT.

16 in patients with documented but noinducible PSVT who have evidence of dual AV node pathways.

17 vely and rapidly converted 90% (28 of 31) of PSVT patients to normal sinus rhythm with no significant

18 ases as of July 1, 1991 and all new cases of PSVT diagnosed from that day until June 30, 1993.

19 The frequency of the episodes of PSVT ranged from > or = 1/day to 1/month.

20 Patients with a documented history of PSVT at times do not have inducible PSVT in the electrop

21 Current knowledge of PSVT has been derived primarily from otherwise healthy p

22 iologic heterogeneity in the pathogenesis of PSVT and the need for more population-based research on

23 dy, tecadenoson rapidly terminated sustained PSVT by depressing AV nodal conduction without causing h

24 Patients with a history of symptomatic PSVT and inducible PSVT at the time of a clinically indi

25 ith paroxysmal supraventricular tachycardia (PSVT) in the electrophysiology laboratory.

26 of paroxysmal supraventricular tachycardia (PSVT) in the general population.

27 of paroxysmal supraventricular tachycardia (PSVT) to sinus rhythm.

28 ble paroxysmal supraventricular tachycardia (PSVT) who have evidence of dual atrioventricular (AV) no

29 ced paroxysmal supraventricular tachycardia (PSVT) without the clinically significant side effects ca

30 stic tools in a large group of patients with PSVT.

31 re are two distinct subsets of patients with PSVT: those with other cardiovascular disease and those

32 ,000 new cases/year and 570,000 persons with PSVT in the United States.

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