ライフサイエンスコーパス: S-nitroso-N-acetylpenicillamine

1 vial tissues and incubated with the NO donor S-nitroso-N-acetylpenicillamine.

2 ented the relaxation induced by the NO donor S-nitroso-N-acetylpenicillamine.

3 be restored by the addition of the NO donor S-nitroso-N-acetylpenicillamine.

4 tonic conditions with the nitric oxide donor S-nitroso-N-acetylpenicillamine.

5 S-Nitroso-N-acetylpenicillamine, a compound that liberat

6 F) cultures was inhibited by the addition of S-nitroso-N-acetylpenicillamine, a nitric oxide (NO)-gen

7 Addition of S-nitroso-N-acetylpenicillamine, a nitric oxide donor, p

8 ly enhances M1 macrophage polarization while S-nitroso-N-acetylpenicillamine, a NO donor, suppresses

9 The NO donors, sodium nitroprusside and S-nitroso-N-acetylpenicillamine, alone did not increase

10 -diolate (NOC-18), S-nitrosoglutathione, and S-nitroso-N-acetylpenicillamine also suppressed CYP2B pr

11 tic antibody Jo2, a nitric oxide (NO) donor (S-nitroso-N-acetylpenicillamine), an NO inhibitor (N(G)-

12 ffects with those of the nitric oxide donor, S-nitroso-N-acetylpenicillamine, an agent we showed prev

13 duced response, the vasodilation elicited by S-nitroso-N-acetylpenicillamine, an NO donor that activa

14 posure of slices to the nitric oxide donors, S-nitroso-N-acetylpenicillamine and S-nitroso-glutathion

15 Using the NO releasing agents S-nitroso-N-acetylpenicillamine and sodium nitroprusside

16 Aorta treated with the nitric oxide donors, S-nitroso-N-acetylpenicillamine and sodium nitroprusside

17 However, treatment with the NO donors S-nitroso-N-acetylpenicillamine and spermine NONOate [N-

18 oxidized by NO donors, sodium nitroprusside, S-nitroso-N-acetylpenicillamine, and S-nitrosoglutathion

19 ionophore ionomycin, the nitric oxide donor S-nitroso-N-acetylpenicillamine, and the protein kinase

20 Similarly, sodium nitroprusside and S-nitroso-N-acetylpenicillamine by themselves did not in

21 enerated by the addition of 400 microM SNAP (S-nitroso-N-acetylpenicillamine) caused a 10-fold increa

22 m nitroprusside inhibited HIV-PR by 70%, and S-nitroso-N-acetylpenicillamine completely inhibited the

23 The NO donor S-nitroso-N-acetylpenicillamine decreased dialysate GABA

24 The NO donor S-nitroso-N-acetylpenicillamine decreases ceramide-induc

25 nterestingly, treatment of EC with NO donor, S-nitroso-N-acetylpenicillamine, did not activate SREBP,

26 Moreover, S-nitrosoglutathione and S-nitroso-N-acetylpenicillamine directly blocked the act

27 We found that the NO donor S-nitroso-N-acetylpenicillamine failed to block DNA synt

28 xyimino]-5-nitro-6-methoxy-3-hexeneamide and S-nitroso-N-acetylpenicillamine greatly enhanced the for

29 HeLa and COS-7 cells, the nitric oxide donor S-nitroso-N-acetylpenicillamine increased the activity o

30 S-nitroso-N-acetylpenicillamine inhibited Bid cleavage,

31 , and the NO donors S-nitrosoglutathione and S-nitroso-N-acetylpenicillamine mimicked CYP2B1 protein

32 S-nitroso-N-acetylpenicillamine or (Z)-1-[N-(2-aminoethy

33 cells were exposed to an exogenous NO donor, S-nitroso-N-acetylpenicillamine or a substrate inhibitor

34 nt of N18 neuroblastoma cells with NO donors S-nitroso-N-acetylpenicillamine or sodium nitroprusside

35 nted that the addition of NO donor molecules S-nitroso-N-acetylpenicillamine or sodium nitroprusside

36 -A promoter, in which the nitric oxide donor S-nitroso-N-acetylpenicillamine reduces urea transporter

37 Increasing levels of nitric oxide using S-nitroso-N-acetylpenicillamine resulted in a decrease i

38 a-dependent sensitization whereas NO donors (S-nitroso-N-acetylpenicillamine) sensitized these cells

39 We report that 2 NO donors, S-nitroso-N-acetylpenicillamine (SNAP) and 2, 2-(hydroxy

40 Exogenous and endogenous sources of NO, from S-nitroso-N-acetylpenicillamine (SNAP) and bradykinin or

41 t cGMP-elevating agents such as the NO donor S-nitroso-N-acetylpenicillamine (SNAP) and C-type natriu

42 e phosphorylated in response to the NO donor S-nitroso-N-acetylpenicillamine (SNAP) and the activatio

43 Exposure to the NO donor (+/-)-s-nitroso-n-acetylpenicillamine (SNAP) blocked increases

44 nor compounds sodium nitroprusside (SNP) and S-nitroso-N-acetylpenicillamine (SNAP) both inhibited th

45 t not higher (100-500 mum), concentration of S-nitroso-N-acetylpenicillamine (SNAP) can reverse the e

46 The NO donor S-nitroso-N-acetylpenicillamine (SNAP) induced apoptosis

47 ultures of rat hepatocytes with the NO donor S-nitroso-N-acetylpenicillamine (SNAP) induced the expre

48 As a consequence, the NO donor S-nitroso-N-acetylpenicillamine (SNAP) inhibits lipopoly

49 the NO donors sodium nitroprusside (SNP) and S-nitroso-N-acetylpenicillamine (SNAP) on cell cycle act

50 The effects of authentic NO and the NO donor S-nitroso-N-acetylpenicillamine (SNAP) on swelling-activ

51 roprusside (SNP), S-nitroglutathione (GSNO), S-nitroso-N-acetylpenicillamine (SNAP) or 3-morpholinosy

52 e of SMC in culture to the nitrovasodilators S-nitroso-N-acetylpenicillamine (SNAP) or sodium nitropr

53 Generation of ONOO- with the NO donor S-nitroso-N-acetylpenicillamine (SNAP) plus the superoxi

54 ine (CSNO), S-nitrosoglutathione (GSNO), and S-nitroso-N-acetylpenicillamine (SNAP) reversibly oxidiz

55 The vasodilation elicited by the NO donor S-nitroso-N-acetylpenicillamine (SNAP) was inhibited by

56 l death and nitrite levels at 24 h caused by S-nitroso-N-acetylpenicillamine (SNAP), a direct nitric

57 es or cytosol isolated from hepatocytes with S-nitroso-N-acetylpenicillamine (SNAP), a nitric oxide d

58 monophosphate (8-Br-cGMP), a cGMP analog or S-nitroso-N-acetylpenicillamine (SNAP), a NO donor essen

59 nors were used: spermine NONOate (SP), (+/-)-S-nitroso-N-acetylpenicillamine (SNAP), and S-nitrosoglu

60 O) mice, inducible NOS KO mice, the NO donor S-nitroso-N-acetylpenicillamine (SNAP), and the NOS inhi

61 treatment with the nitric oxide (NO) donor, S-nitroso-N-acetylpenicillamine (SNAP), and the phosphod

62 The NO donor S-nitroso-N-acetylpenicillamine (SNAP), at all concentra

63 donors, diethylenetriamine/NO (DETA/NO) and S-nitroso-N-acetylpenicillamine (SNAP), at doses previou

64 Three such agents, S-nitroso-N-acetylpenicillamine (SNAP), diethylenetriami

65 donors, S-nitroglutathione (GSNO) and (+/-)-S-nitroso-N-acetylpenicillamine (SNAP), directly inhibit

66 eurons (CGNs) by treatment with the NO donor S-nitroso-N-acetylpenicillamine (SNAP), for short (6 h)

67 chelatase incubated with NO or the NO donor, S-nitroso-N-acetylpenicillamine (SNAP), indicate a rapid

68 tigated by using the NO.-generating compound S-nitroso-N-acetylpenicillamine (SNAP), or by stimulatin

69 An NO donor, S-nitroso-N-acetylpenicillamine (SNAP), suppressed apopt

70 ntly enhanced TH17 cell differentiation, and S-nitroso-N-acetylpenicillamine (SNAP), the NO donor, do

71 mo-cAMP, nitric oxide (NO), and the NO donor S-nitroso-N-acetylpenicillamine (SNAP), which circumvent

72 er NTG or the spontaneous nitric oxide donor S-nitroso-N-acetylpenicillamine (SNAP).

73 for 24 h with sodium nitroprusside (SNP) or S-nitroso-N-acetylpenicillamine (SNAP).

74 ssed by either 8-Br-PET-cGMP or the NO donor S-nitroso-N-acetylpenicillamine (SNAP).

75 Slow oscillations were also evoked with S-nitroso-N-acetylpenicillamine (SNAP, 1 microM).

76 NAME, the addition of an exogenous NO donor, S-nitroso-N-acetylpenicillamine (SNAP, 10(-8) M) or an a

77 Whereas a high concentration of the NO donor S-nitroso-N-acetylpenicillamine (SNAP, 100 micromol/L) s

78 0.1 mg/kg i.v., 4 times [group II, n=5]) or S-nitroso-N-acetylpenicillamine (SNAP, 2.5 microg x kg(-

79 The RSNO compounds we used were S-nitroso-N-acetylpenicillamine (SNAP, 5 to 10 nmol/L or

80 S-nitroso-N-acetylpenicillamine (SNAP, 9 +/- 3% to 50 +/

81 S-Nitroso-N-acetylpenicillamine (SNAP; 10 microM), an NO

82 -dextran-10K) during suffusion with vehicle, S-nitroso-N-acetylpenicillamine (SNAP; 100 microM) and 3

83 In the resting state, NO donors S-nitroso-N-acetylpenicillamine (SNAP; 50 microM) and so

84 dazole (7NI; 1 mM) or the nitric oxide donor S-nitroso-N-acetylpenicillamine (SNAP; 500 microM-5 mM)

85 of primary cultured RCECs with an NO donor, S-nitroso-N-acetylpenicillamine (SNAP; N-acetyl-3-(nitro

86 s were exposed to combinations of NO donors (S-nitroso-N-acetylpenicillamine [SNAP] and others), a cG

87 nitrosothiol donors S-nitrosoglutathione and S-nitroso-N-acetylpenicillamine suppressed caspase-9 and

88 When FLS were cultured with S-nitroso-N-acetylpenicillamine, the pattern of MMR expr

89 Through the use of S-nitroso-N-acetylpenicillamine, which releases NO upon

90 S-nitroso-N-acetylpenicillamine, (Z)-1-[N-(2-aminoethyl)