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1 terogeneity, and increased NAL resistance of S. enterica serovar Typhi.
2 han in previous studies of phoP/phoQ-deleted S. enterica serovar Typhi.
3 or the disease promoted by the human-adapted S. enterica serovar Typhi.
4 tative OmpR binding sites were identified in S. enterica serovar Typhi, 22 of which were associated w
6 munization of newborn mice with 10(9) CFU of S. enterica serovar Typhi CVD 908-htrA and S. enterica s
7 ella enterica serovar Typhimurium SL3261 and S. enterica serovar Typhi CVD 908-htrA by the Hly type I
9 Salmonella enterica serovar Typhimurium and S. enterica serovar Typhi expressing fragments of the si
10 previous clinical study of phoP/phoQ-deleted S. enterica serovar Typhi expressing Helicobacter pylori
11 vectors in animals, early clinical trials of S. enterica serovar Typhi expressing heterologous antige
12 yphimurium appears to be more effective than S. enterica serovar Typhi for engendering immune respons
13 tion, we identify shared genetic features of S. enterica serovars Typhi, Paratyphi A, and Sendai that
14 of the S. typhimurium LT2 genes missing from S. enterica serovar Typhi (S. typhi), and 29% missing fr
15 Sequence analysis of the SPI-7 region from S. enterica serovar Typhi strain CT18 revealed significa
16 ith the genome sequence of recently isolated S. enterica serovar Typhi strain CT18 showed that 29 of
17 a type IV pilus involved in the adherence of S. enterica serovar Typhi to host cells was missing.
20 . enterica serovar Typhimurium and four from S. enterica serovar Typhi were used to create an assay c
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