ライフサイエンスコーパス: S. enterica serovar Typhimurium

1 each of S. enterica serovar Enteritidis and S. enterica serovar Typhimurium.

2 wild-type but not for attenuated strains of S. enterica serovar Typhimurium.

3 sceptibility of caveolin-1-deficient mice to S. enterica serovar Typhimurium.

4 encoding PagL, PagP, and LpxR into wild-type S. enterica serovar Typhimurium.

5 , there was significant biofilm formation by S. enterica serovar Typhimurium.

6 (28)/FlgM interactions were also isolated in S. enterica serovar Typhimurium.

7 in the formation of an extensive biofilm of S. enterica serovar Typhimurium.

8 ty did not reach the level of wild-type (WT) S. enterica serovar Typhimurium.

9 ppears to have been acquired horizontally in S. enterica serovar Typhimurium.

10 ted on an island integrated at tRNA(PheU) in S. enterica serovar Typhimurium.

11 rechallenge of immunized mice with virulent S. enterica serovar Typhimurium.

12 In order to identify SoxS-regulated genes in S. enterica serovar Typhimurium, a lacI-regulated expres

13 tive specimens identical to the hisJ gene of S. enterica serovar Typhimurium and a second group of am

14 Inoculations of S. enterica serovar Typhimurium and E. coli resulted in

15 Six genetic loci from S. enterica serovar Typhimurium and four from S. enteric

16 ductase) reduces the growth of intracellular S. enterica serovar Typhimurium and has no effect on ext

17 In contrast, S. enterica serovar Typhimurium and S. enterica serovar

18 urther into this pathway, we also found that S. enterica serovar Typhimurium and S. flexneri activate

19 S. enterica serovar Typhimurium and S. flexneri cell ent

20 ing 12/15-lipoxygenase (12/15-LOX), and that S. enterica serovar Typhimurium and S. flexneri share ce

21 Thus, although S. enterica serovar Typhimurium and S. flexneri utilize

22 Microscopy studies indicated that both S. enterica serovar Typhimurium and S. flexneri were loc

23 r stability of antigen-expressing plasmid in S. enterica serovar Typhimurium and/or prolonged intesti

24 trast, other pathogenic salmonellae, such as S. enterica serovars Typhimurium and Dublin (S. typhimur

25 ci were useful in distinguishing isolates of S. enterica serovars Typhimurium and Newport that had di

26 Isolates of S. enterica serovars Typhimurium and Newport that were r

27 d used to amplify PCR targets in isolates of S. enterica serovars Typhimurium and Newport.

28 In this study we have compared in S. enterica serovars Typhimurium and Typhi the effect of

29 (by single and double mutations) strains of S. enterica serovars Typhimurium and Typhi were recovere

30 om Vibrio cholerae, Yersinia enterocolitica, S. enterica serovar Typhimurium, and Klebsiella pneumoni

31 n of inflammatory responses by intracellular S. enterica serovar Typhimurium, and perhaps Shigella fl

32 rom Salmonella enterica serovar Enteritidis, S. enterica serovar Typhimurium, and Pseudomonas aerugin

33 binantly expressed in the insoluble phase in S. enterica serovar Typhimurium, and the immunogenicity

34 Five of the six volunteers seroconverted to S. enterica serovar Typhimurium antigens and had strong

35 Attenuated S. enterica serovar Typhimurium appears to be more effec

36 In this study, we used S. enterica serovar Typhimurium as an in vivo heterologo

37 constructed and characterized a recombinant S. enterica serovar Typhimurium avirulent vaccine strain

38 nonoxidative early intracellular killing of S. enterica serovar Typhimurium by human macrophages and

39 The results of this study indicate that S. enterica serovar Typhimurium can outgrow E. coli in h

40 he cecum and large intestine with 10x LD(50) S. enterica serovar Typhimurium challenge at 7 days post

41 onal and if it was evolutionarily similar to S. enterica serovar Typhimurium CorA.

42 var Typhimurium or pulsed with proteins from S. enterica serovar Typhimurium culture supernatants.

43 S. enterica serovar Typhimurium Dam mutant strains exhib

44 Also, S. enterica serovar Typhimurium Dam(-) vaccines were not

45 To improve sensitivity of S. enterica serovar Typhimurium detection, multiwalled c

46 um chloride (DPI), but infection of MDM with S. enterica serovar Typhimurium did not cause an increas

47 A colanic acid/cellulose S. enterica serovar Typhimurium double mutant formed a m

48 ritidis, whose overexpression conferred upon S. enterica serovar Typhimurium enhanced resistance to e

49 S. enterica serovar Typhimurium entry requires a functio

50 ions occurring in this locus in FQ-resistant S. enterica serovar Typhimurium epidemic clones resulted

51 kappa B p50 and p52 subunits, was induced by S. enterica serovar Typhimurium even in the absence of f

52 10(7) to 8 x 10(7) CFU of phoP/phoQ-deleted S. enterica serovar Typhimurium expressing the same anti

53 The half-saturation affinity of S. enterica serovar Typhimurium for H2 is only 2.1 micro

54 on, we screened a transposon library made in S. enterica serovar Typhimurium for the ability to persi

55 Additional experiments with wild-type S. enterica serovar Typhimurium, fully capable of switch

56 which is necessary for the full virulence of S. enterica serovar Typhimurium Gifsy-2 lysogens.

57 Nevertheless, the strain complemented with S. enterica serovar Typhimurium GS grew as well as the w

58 ype strain because of poor expression of the S. enterica serovar Typhimurium GS in the heterologous M

59 While S. enterica serovar Typhimurium has been shown to kill i

60 The S. enterica serovar Typhimurium hybrid strains showed si

61 e inducible genes by the insertions rendered S. enterica serovar Typhimurium hypersensitive to millim

62 on the concentrations, inhibits E. coli and S. enterica serovar Typhimurium in an additive or antago

63 CD8 T cells contribute to protection against S. enterica serovar Typhimurium in mice, but little is k

64 B signaling pathway and promote virulence of S. enterica serovar Typhimurium in mice.

65 imple method for identifying new variants of S. enterica serovar Typhimurium in the field.

66 (CD11b+), and dendritic cells (CD11c+) with S. enterica serovar Typhimurium induced an up-regulation

67 port here that CD8 T-cell lines derived from S. enterica serovar Typhimurium-infected BALB/c mice lys

68 ings indicate that c-Abl is activated during S. enterica serovar Typhimurium infection and that its p

69 ce factor known to be upregulated in vivo in S. enterica serovar Typhimurium infection of mice.

70 e of two different haplotypes following oral S. enterica serovar Typhimurium infection.

71 r alpha (TNF-alpha) secretion in response to S. enterica serovar Typhimurium infection.

72 ibitor, Co(III) hexaammine, had no effect on S. enterica serovar Typhimurium invasion of Caco-2 epith

73 These results indicate that S. enterica serovar Typhimurium is an excellent delivery

74 contained class I integrons, with 71% of the S. enterica serovar Typhimurium isolates and 6.9% of iso

75 s that could be used to discriminate between S. enterica serovar Typhimurium isolates from the same g

76 multiple methods is needed to differentiate S. enterica serovar Typhimurium isolates that geneticall

77 fications were absent in the closely related S. enterica serovar Typhimurium LT2 and from a mutant of

78 serB hsdM, S and R, that in E. coli K-12 and S. enterica serovar typhimurium LT2 is serB hsdR, M and

79 It is proposed that S. enterica serovar Typhimurium LT2 may not have a cob(I

80 to the sequence reported in GenBank for the S. enterica serovar Typhimurium LT2 strain.

81 S. enterica serovar Typhimurium MntH contributes to H(2)

82 S. enterica serovar Typhimurium MntH expression is impor

83 s also reduce bacterial proliferation in the S. enterica serovar Typhimurium mouse model.

84 An S. enterica serovar Typhimurium mutant carrying a loss-o

85 rgJ and the needle protein PrgI in different S. enterica serovar Typhimurium mutant strains.

86 S. enterica serovar Typhimurium oligonucleotide microarr

87 lysed bone marrow macrophages infected with S. enterica serovar Typhimurium or pulsed with proteins

88 ved in other Salmonella strains, i.e., other S. enterica serovar Typhimurium phage types and other S.

89 with the serovar Typhi plasmid pHCM1 and an S. enterica serovar Typhimurium plasmid pR27.

90 than the hyperactive phenotype seen with the S. enterica serovar Typhimurium protein.

91 intestinal stage of infection but that once S. enterica serovar Typhimurium reaches the spleens of s

92 the previously uncharacterized aer locus of S. enterica serovar Typhimurium revealed them to be cont

93 In this study, PoxA- mutants of S. enterica serovar Typhimurium (S. typhimurium) were fo

94 several distinct pathways that can modulate S. enterica serovar Typhimurium's ability to express hil

95 n of a substrate of this secretion system in S. enterica serovar typhimurium (Salmonella typhimurium)

96 A fis mutant of S. enterica serovar Typhimurium showed a ninefold increa

97 The chromosomal copy of the S. enterica serovar Typhimurium sigma(28) structural gen

98 f S. enterica serovar Typhi CVD 908-htrA and S. enterica serovar Typhimurium SL3261 carrying plasmid

99 H2-M3-transfected fibroblasts infected with S. enterica serovar Typhimurium SL3261 or treated with S

100 of the four GTPases known to be activated by S. enterica serovar Typhimurium SopE are individually re

101 The S. enterica serovar Typhimurium soxRS system also mediat

102 , a lacI-regulated expression system for the S. enterica serovar Typhimurium soxS gene was developed.

103 S. enterica serovar Typhimurium ssrA mutants fail to sup

104 eement with results obtained in the original S. enterica serovar Typhimurium STM screen, illustrating

105 S. enterica serovar Typhimurium strain 14028 produces tw

106 biosynthetic gene, wcaM, was introduced into S. enterica serovar Typhimurium strain BJ2710 and was fo

107 SL2361 and then challenged with the virulent S. enterica serovar Typhimurium strain C5, 100% of the I

108 Serovars Gallinarum and Pullorum expressing S. enterica serovar Typhimurium strain LT2 type 1 fimbri

109 A 50,000-CFU transposon library of S. enterica serovar Typhimurium strain SL1344 was serial

110 In contrast, a highly modified attenuated S. enterica serovar Typhimurium strain was not present i

111 One hundred twenty-eight S. enterica serovar Typhimurium strains isolated from ca

112 et al. that oral inoculation with wild-type S. enterica serovar Typhimurium strains lead to bacteria

113 In S. enterica serovar Typhimurium strains that had the abi

114 ties for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains when a minimum o

115 PFGE for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains, respectively.

116 I, BlnI, and SpeI were most concordant among S. enterica serovar Typhimurium strains.

117 ane permeability) upon infection by invasive S. enterica serovar Typhimurium than do infected control

118 ns that were more closely related to hisJ of S. enterica serovar Typhimurium than to other hisJ seque

119 reaching the level in cells infected with WT S. enterica serovar Typhimurium, than the level in host

120 , we constructed phase-locked derivatives of S. enterica serovar Typhimurium that expressed only FljB

121 In S. enterica serovar Typhimurium, the PmrA/PmrB two-compo

122 In S. enterica serovar Typhimurium, the reduction in activi

123 was first formed followed by challenge with S. enterica serovar Typhimurium, there was significant b

124 cate that during infection of macrophages by S. enterica serovar Typhimurium, TLR4 signals are requir

125 e that mig-14 is necessary for resistance of S. enterica serovar Typhimurium to both polymyxin B and

126 have a significant effect on the ability of S. enterica serovar Typhimurium to establish a systemic

127 egulator (prpR)) were evaluated in wild-type S. enterica serovar Typhimurium TR6583 and prpB(-) or pr

128 utant retained intact LPS, we constructed an S. enterica serovar Typhimurium triple-knockout (TKO) mu

129 ve now characterized transport by SitABCD in S. enterica serovar Typhimurium using (54)Mn(2+) and (55

130 onstrated in both Escherichia coli JM109 and S. enterica serovar Typhimurium vaccine strain chi4072.

131 When mice were immunized with the S. enterica serovar Typhimurium vaccine strain SL2361 an

132 The S. enterica serovar Typhimurium vaccine strain was const

133 ognized by sera from mice immunized with the S. enterica serovar Typhimurium vaccine strain.

134 Despite success with attenuated S. enterica serovar Typhimurium vectors in animals, earl

135 I account for the contribution of Gifsy-2 to S. enterica serovar Typhimurium virulence in the murine

136 ociated with the needle complex in wild-type S. enterica serovar Typhimurium, was absent from needle

137 is of the determinants of thermotolerance in S. enterica serovar Typhimurium, we isolated the chr-1 m

138 Although excess Fe2+ was slightly toxic to S. enterica serovar Typhimurium, we were unable to elici

139 Random transposon mutants of S. enterica serovar Typhimurium were screened for impair

140 When S. enterica serovar Typhimurium with the chromosomally i

141 ay complementary roles in the interaction of S. enterica serovar Typhimurium with the host intestinal