ライフサイエンスコーパス: S. enterica

1 S. enterica cleanly converted all of the acid to acetyl-

2 S. enterica serotype Agona was isolated concurrently fro

3 S. enterica serovar Typhi carrying pTETlpp induced Frag

4 S. enterica serovar Typhimurium and S. flexneri cell ent

5 S. enterica serovar Typhimurium entry requires a functio

6 S. enterica serovar Typhimurium oligonucleotide microarr

7 S. enterica serovar Typhimurium strain 14028 produces tw

8 S. enterica subsp. salamae encodes the Salmonella pathog

9 (9 genovars) in 12 multiplex PCR mixes on 11 S. enterica strains.

10 oquinolone resistance in a collection of 136 S. enterica isolates, including 111 with intermediate or

11 sylvania and nearby states during July 2004: S. enterica serotypes Javiana, Anatum, Thompson, Typhimu

12 lly identify 12 serovars (16 genovars) in 24 S. enterica strains.

13 ta on 291 Salmonella isolates, including 250 S. enterica subspecies I strains from 32 serovars (52 ge

14 he cecum and large intestine with 10x LD(50) S. enterica serovar Typhimurium challenge at 7 days post

15 A total of 52 S. enterica serotype Enteritidis isolates representing 1

16 owth on 10 mM acetate, acs(+) induction in a S. enterica strain that cannot acetylate (i.e. inactivat

17 Our data suggest that the 12-2 antigen is a S. enterica subspecies I-specific LPS modification that

18 mented the flagella-deficient phenotype of a S. enterica acnB mutant, and the isolated AcnB5-4 polype

19 e corrected the pantothenate auxotrophy of a S. enterica yhhK strain, supporting in vitro evidence ob

20 We show that STM0557, a S. enterica subspecies I-specific gene encoding an inner

21 We also show that a S. enterica cobT strain that synthesizes GkCblS ectopica

22 f phenotypic similarity and diversity across S. enterica subspecies I and shown how the core genome o

23 differentiate the two invasive avian-adapted S. enterica serovar Gallinarum biotypes Gallinarum and P

24 or the disease promoted by the human-adapted S. enterica serovar Typhi.

25 be associated with antibody production after S. enterica serovar Enteritidis vaccination.

26 uced colonization resistance of mice against S. enterica serotype Typhimurium.

27 CD8 T cells contribute to protection against S. enterica serovar Typhimurium in mice, but little is k

28 tory proteins and is highly conserved in all S. enterica serovars.

29 fim, bcf, and stb) were conserved within all S. enterica strains included in this study.

30 Thus, although S. enterica serovar Typhimurium and S. flexneri utilize

31 I, BlnI, and SpeI were most concordant among S. enterica serovar Typhimurium strains.

32 with the serovar Typhi plasmid pHCM1 and an S. enterica serovar Typhimurium plasmid pR27.

33 utant retained intact LPS, we constructed an S. enterica serovar Typhimurium triple-knockout (TKO) mu

34 rect the tricarballylate growth defect of an S. enterica apbC mutant.

35 as necessary and sufficient for growth of an S. enterica cobA eutT strain on ethanolamine as a carbon

36 MSX and MSO inhibited the growth of an S. enterica DeltamddA strain unless glutamine or methion

37 The EutD protein restored growth of an S. enterica pta strain on acetate as the source of carbo

38 the diminished pathogenicity phenotype of an S. enterica sptP mutant.

39 romoter and was used to support growth of an S. enterica strain under conditions that demanded CobC-l

40 positive S. enterica serovar Paratyphi C and S. enterica serovar Dublin isolates revealed the presenc

41 A comparison of the E. coli and S. enterica AraC regulons, coupled with a bioinformatic

42 We conclude that E. coli and S. enterica derive these unique genes from a common sour

43 The E. coli and S. enterica proteins were the most similar, although the

44 preciably more strongly than the E. coli and S. enterica proteins.

45 on the concentrations, inhibits E. coli and S. enterica serovar Typhimurium in an additive or antago

46 dentified consensus sequence for E. coli and S. enterica.

47 ion of rpoS is conserved between E. coli and S. enterica.

48 ties for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains when a minimum o

49 PFGE for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains, respectively.

50 each of S. enterica serovar Enteritidis and S. enterica serovar Typhimurium.

51 f S. enterica serovar Typhi CVD 908-htrA and S. enterica serovar Typhimurium SL3261 carrying plasmid

52 on the roles of their aligned Y. pestis and S. enterica partners and showed that up to 73% of the pr

53 S. bongori, S. enterica subsp. salamae, and S. enterica subsp. arizonae share features of the infect

54 Salmonella enterica serotype Typhi (ST) and S. enterica serotype Paratyphi A (SPA) isolates were inc

55 rica in the liver, subsequently confirmed as S. enterica serotype I 4,5,12:-:1,2.

56 form from E. amylovora restored growth of as S. enterica cobB mutant strain on low acetate.

57 h nontyphoidal Salmonella serotypes, such as S. enterica serotype Typhimurium, are characterized by a

58 In contrast, a highly modified attenuated S. enterica serovar Typhimurium strain was not present i

59 udy used transcriptional differences between S. enterica wild-type and ridA strains to explore the br

60 s that could be used to discriminate between S. enterica serovar Typhimurium isolates from the same g

61 Matches between S. enterica clustered regularly interspaced short palind

62 These results show that S. bongori, S. enterica subsp. salamae, and S. enterica subsp. arizo

63 we identified EspJ homologues in S. bongori, S. enterica subsp. salamae, and Salmonella enterica subs

64 Microscopy studies indicated that both S. enterica serovar Typhimurium and S. flexneri were loc

65 xt of the selective pressures encountered by S. enterica in vivo.

66 , there was significant biofilm formation by S. enterica serovar Typhimurium.

67 dent PRA formation in vitro was inhibited by S. enterica YjgF and the human homolog UK114.

68 almonella enterica infections represented by S. enterica serovar Newport has increased markedly among

69 In summary, MddA is the mechanism used by S. enterica to respond to oxidized forms of methionine,

70 A colanic acid/cellulose S. enterica serovar Typhimurium double mutant formed a m

71 that the MLST scheme employed here clustered S. enterica serovar Newport isolates in distinct molecul

72 gy searches to determine how far the E. coli/S. enterica paradigm can be generalised to other flagell

73 Concurrently, S. enterica subsp. salamae infection of J774.A1 macropha

74 Under aerobic conditions, S. enterica performs the corrinoid-dependent degradation

75 2 proteins restored growth of SIR2-deficient S. enterica on acetate and propionate, suggesting that e

76 of SpvD is highly conserved across different S. enterica serovars, but residue 161, located close to

77 multiple methods is needed to differentiate S. enterica serovar Typhimurium isolates that geneticall

78 ings indicate that c-Abl is activated during S. enterica serovar Typhimurium infection and that its p

79 One hundred twenty-eight S. enterica serovar Typhimurium strains isolated from ca

80 ndings indicate that the sensor PhoQ enables S. enterica to respond to both host- and bacterial-deriv

81 population structure of commonly encountered S. enterica serotype Enteritidis outbreak isolates in th

82 om Vibrio cholerae, Yersinia enterocolitica, S. enterica serovar Typhimurium, and Klebsiella pneumoni

83 for immune responses and survival following S. enterica infection.

84 uorescein, biotin and digoxigenin coding for S. enterica, L. monocytogenes and E. coli, respectively.

85 BlnI, SfiI, and PacI as most concordant for S. enterica serovar Enteritidis, while XbaI, BlnI, and S

86 able sequence and microarray information for S. enterica subspecies I strains.

87 9% and 96% for five-enzyme combined PFGE for S. enterica serovar Enteritidis and S. enterica serovar

88 Here we show that apbC is required for S. enterica to use tricarballylate as a carbon and energ

89 scriminatory PFGE-based subtyping scheme for S. enterica serovar Enteritidis that relies on a single

90 100% correlation among Dice similarities for S. enterica serovar Enteritidis and S. enterica serovar

91 port here that CD8 T-cell lines derived from S. enterica serovar Typhimurium-infected BALB/c mice lys

92 cterization of the lytic domain of FlgJ from S. enterica as the model enzyme.

93 parameters of K(m) and k(cat) for FlgJ from S. enterica were determined to be 0.64 +/- 0.18 mg ml(-1

94 . enterica serovar Typhimurium and four from S. enterica serovar Typhi were used to create an assay c

95 Six genetic loci from S. enterica serovar Typhimurium and four from S. enteric

96 var Typhimurium or pulsed with proteins from S. enterica serovar Typhimurium culture supernatants.

97 he His6-tagged PduS cobalamin reductase from S. enterica was produced at high levels in Escherichia c

98 We identified a gene, yafD from S. enterica serovar Enteritidis, whose overexpression co

99 d in the prediction and prevention of future S. enterica outbreaks.

100 to Escherichia coli phages lambda and HK97, S. enterica phage ST64T, or a Shigella flexneri prophage

101 as measured after treatment with homogeneous S. enterica SIR2 protein.

102 However, S. enterica serotype Typhimurium strains carrying deleti

103 In S. enterica serovar Typhi, viaB is encoded on a 134-kb p

104 In S. enterica serovar Typhimurium strains that had the abi

105 In S. enterica serovar Typhimurium, the PmrA/PmrB two-compo

106 In S. enterica serovar Typhimurium, the reduction in activi

107 In S. enterica, 2AA inactivates a number of pyridoxal 5'-ph

108 In S. enterica, the Ser-derived enamine/imine inactivates a

109 In S. enterica, this carboxysome-like structure (hereafter

110 ere, we demonstrate that Dap accumulation in S. enterica elicits a proline requirement for growth and

111 ling the genetic basis of host adaptation in S. enterica.

112 A model for tricarballylate catabolism in S. enterica is proposed.

113 esponse to osmotic challenge is conserved in S. enterica, dependence on these two sRNA regulators is

114 H, the predominant cysteine desulfhydrase in S. enterica.

115 , and have studied this phenotype further in S. enterica.

116 gg albumen, while disruption of this gene in S. enterica serovar Enteritidis rendered the organism mo

117 ppears to have been acquired horizontally in S. enterica serovar Typhimurium.

118 tative OmpR binding sites were identified in S. enterica serovar Typhi, 22 of which were associated w

119 (28)/FlgM interactions were also isolated in S. enterica serovar Typhimurium.

120 We further demonstrate that, like in S. enterica, the structural genes required for the flage

121 on, we screened a transposon library made in S. enterica serovar Typhimurium for the ability to persi

122 ding of the effects Dap has on metabolism in S. enterica, and likely other organisms, and highlight t

123 pproximately half the pseudogenes present in S. enterica serovar Typhi were conserved.

124 ng with ratA, sivI, and sivH were present in S. enterica subsp. II and S. bongori in addition to S. e

125 bial drug resistance is a growing problem in S. enterica that threatens to further compromise patient

126 uencing (RNA-seq) to map the AraC regulon in S. enterica.

127 The same residues in S. enterica ArnT are also needed for function.

128 CblS proteins restore alpha-RP synthesis in S. enterica lacking the CobT enzyme.

129 Here, we show that in S. enterica serotype Typhi, the causative agent of typho

130 We show that in S. enterica, BcsE is not essential for cellulose synthes

131 is of the determinants of thermotolerance in S. enterica serovar Typhimurium, we isolated the chr-1 m

132 onia-lyase (DpaL) alleviated Dap toxicity in S. enterica by catalyzing the degradation of Dap to pyru

133 und that SP control of rpoS transcription in S. enterica involves repression of the major rpoS promot

134 een in the absence of glucose and, unlike in S. enterica, causes a substantial growth defect.

135 ce factor known to be upregulated in vivo in S. enterica serovar Typhimurium infection of mice.

136 ause infection in different hosts, including S. enterica serovar Enteritidis (multiple hosts), S. Gal

137 H8 infected S. enterica serotypes Enteritidis and Typhimurium and Es

138 biosynthetic gene, wcaM, was introduced into S. enterica serovar Typhimurium strain BJ2710 and was fo

139 Introduction of tviA into S. enterica serotype Typhimurium rendered flhDC transcri

140 n of inflammatory responses by intracellular S. enterica serovar Typhimurium, and perhaps Shigella fl

141 ductase) reduces the growth of intracellular S. enterica serovar Typhimurium and has no effect on ext

142 ane permeability) upon infection by invasive S. enterica serovar Typhimurium than do infected control

143 context suggest that the evolution of known S. enterica sublineages is mediated mostly by two mechan

144 to identify both novel and previously known S. enterica virulence factors (HilA, HilD, InvH, SptP, R

145 e types and other S. enterica serovars, like S. enterica serovar Infantis, possessing SGI1, while DT1

146 We identified matching S. enterica serotype Typhimurium isolates from 28 patien

147 It appears that MDR S. enterica serotype Typhi has emerged as a predominant

148 NO during coincubation with N. meningitidis, S. enterica, or E. coli.

149 Among the methods, S. enterica subsp. enterica serovars 4,5,12:i:-, Typhimu

150 was demonstrated using different Vi-negative S. enterica derivatives.

151 nd phenotypically characterized nontyphoidal S. enterica strains to 11 previously sequenced S. enteri

152 G, or sthABCDE did not reduce the ability of S. enterica serotype Typhimurium to colonize the spleen

153 The half-saturation affinity of S. enterica serovar Typhimurium for H2 is only 2.1 micro

154 c phenotype caused by the rpoD1181 allele of S. enterica allows past in vitro results to be incorpora

155 ribute to the epidemiological association of S. enterica serovar Enteritidis with egg products.

156 in the formation of an extensive biofilm of S. enterica serovar Typhimurium.

157 ch treatment against established biofilms of S. enterica and P. aeruginosa, respectively.

158 ave been isolated from the culture broths of S. enterica and uropathogenic E. coli, but MGE and TGE h

159 ntribute to long-term intestinal carriage of S. enterica serotype Typhimurium in genetically resistan

160 e genes important for intestinal carriage of S. enterica serotype Typhimurium in vertebrate animals.

161 munization of newborn mice with 10(9) CFU of S. enterica serovar Typhi CVD 908-htrA and S. enterica s

162 A unique epidemiological characteristic of S. enterica serovar Enteritidis is its association with

163 e addition of propionaldehyde to cultures of S. enterica caused growth arrest from 8 to 20 mM, but no

164 ntitative, spatial, and temporal dynamics of S. enterica interactions are key to understanding how im

165 SfiI, PacI, and NotI) for 74 strains each of S. enterica serovar Enteritidis and S. enterica serovar

166 exchange, and loss play in the evolution of S. enterica sublineages, which to a certain extent are r

167 of an rpoE null mutant and the psp genes of S. enterica and Shigella flexneri are highly induced dur

168 alamae strain 3588/07 against the genomes of S. enterica subsp. enterica serovar Typhimurium strain L

169 hat EutQ is required during anoxic growth of S. enterica on ethanolamine and tetrathionate.

170 yoxylate shunt is not required for growth of S. enterica on tricarballylate.

171 gene is rarely found outside subspecies I of S. enterica and often present in nonfunctional allelic f

172 terica, and the dose-dependent inhibition of S. enterica by a soluble carbohydrate antiadhesive.

173 Inoculations of S. enterica serovar Typhimurium and E. coli resulted in

174 ay complementary roles in the interaction of S. enterica serovar Typhimurium with the host intestinal

175 ci were useful in distinguishing isolates of S. enterica serovars Typhimurium and Newport that had di

176 Isolates of S. enterica serovars Typhimurium and Newport that were r

177 d used to amplify PCR targets in isolates of S. enterica serovars Typhimurium and Newport.

178 present the most common clinical isolates of S. enterica subsp. enterica.

179 nonoxidative early intracellular killing of S. enterica serovar Typhimurium by human macrophages and

180 A 50,000-CFU transposon library of S. enterica serovar Typhimurium strain SL1344 was serial

181 select the fliC transcript from a library of S. enterica transcripts; thus, the effect of AcnB on Fli

182 Toward this goal, a random library of S. enterica typhimurium 14028 genomic DNA was cloned ups

183 the previously uncharacterized aer locus of S. enterica serovar Typhimurium revealed them to be cont

184 A fis mutant of S. enterica serovar Typhimurium showed a ninefold increa

185 A library of 960 signature-tagged mutants of S. enterica serovar Choleraesuis was constructed and scr

186 Random transposon mutants of S. enterica serovar Typhimurium were screened for impair

187 re that transcription of the vapBC operon of S. enterica is controlled by a recently discovered regul

188 ace major lineages and ecological origins of S. enterica serotype Enteritidis.

189 wn or potential roles in the pathogenesis of S. enterica.

190 was used to understand the pathogenicity of S. enterica serovar Choleraesuis in its natural host and

191 xamined the distribution of PFGE patterns of S. enterica serotype Typhi isolates from patients with a

192 aecal shedding and intestinal persistence of S. enterica serotype Typhimurium ATCC14028 in Salmonella

193 l in assessing the evolutionary potential of S. enterica sublineages and aid in the prediction and pr

194 he GTPase and ribosome-binding properties of S. enterica BipA.

195 wo organisms showed this to be a property of S. enterica rather than of the FadR proteins per se.

196 Here we show that the PduQ protein of S. enterica is an iron-dependent alcohol dehydrogenase u

197 echanisms that determine net growth rates of S. enterica within the host.

198 r infection, suggesting that the recovery of S. enterica serotype Typhimurium from fecal samples clos

199 ACDEFG did not result in reduced recovery of S. enterica serotype Typhimurium from fecal samples coll

200 terogeneity, and increased NAL resistance of S. enterica serovar Typhi.

201 To improve sensitivity of S. enterica serovar Typhimurium detection, multiwalled c

202 ost common clinical and nonhuman serovars of S. enterica in the United States.

203 h five strains each of the target species of S. enterica and L. monocytogenes, along with five strain

204 complicate efforts to control the spread of S. enterica serovar Heidelberg in food animal and human

205 ype protein, supported growth of a strain of S. enterica devoid of Acs (acetyl-CoA synthetase; AMP-fo

206 The outbreak strain of S. enterica serotype Typhimurium was cultured from a pat

207 We report that wild-type strains of S. enterica grow on decanoic acid, whereas wild-type E.

208 of the growth behavior of mutant strains of S. enterica lacking specific functions encoded by the 17

209 wild-type but not for attenuated strains of S. enterica serovar Typhimurium.

210 (by single and double mutations) strains of S. enterica serovars Typhimurium and Typhi were recovere

211 Phenotypically, strains of S. enterica that lack RidA accumulated significantly mor

212 cherichia coli is similar overall to that of S. enterica but is seen in the absence of glucose and, u

213 ubtilis also enhanced the thermotolerance of S. enterica.

214 imple method for identifying new variants of S. enterica serovar Typhimurium in the field.

215 B signaling pathway and promote virulence of S. enterica serovar Typhimurium in mice.

216 ibitor, Co(III) hexaammine, had no effect on S. enterica serovar Typhimurium invasion of Caco-2 epith

217 pecies in all samples, with detection of one S. enterica and two Listeria TRFs in all cases, and dete

218 e of two different haplotypes following oral S. enterica serovar Typhimurium infection.

219 eement with results obtained in the original S. enterica serovar Typhimurium STM screen, illustrating

220 ca serovar Typhimurium phage types and other S. enterica serovars, like S. enterica serovar Infantis,

221 ved in other Salmonella strains, i.e., other S. enterica serovar Typhimurium phage types and other S.

222 y following eradication of the fowl pathogen S. enterica serovar Gallinarum in the mid-20th century.

223 for bacterial colonization after pathogenic S. enterica serovar Enteritidis inoculation and for circ

224 ated by low Mg(2+) and C18G as in pathogenic S. enterica.

225 e serotypes impact the ecology of pathogenic S. enterica on-farm.

226 alysis of viaB-associated DNA in Vi-positive S. enterica serovar Paratyphi C and S. enterica serovar

227 Closely related S. enterica serotype Typhimurium isolates were cultured

228 fications were absent in the closely related S. enterica serovar Typhimurium LT2 and from a mutant of

229 Remarkably, S. enterica serovar Typhi does not encode apparent homol

230 ions occurring in this locus in FQ-resistant S. enterica serovar Typhimurium epidemic clones resulted

231 smid-associated genes in multidrug resistant S. enterica serovar Heidelberg, antimicrobial resistance

232 e transfer; different isolates from the same S. enterica serovar can exhibit significant variation in

233 TRFs in all cases, and detection of a second S. enterica TRF in 91% of cases.

234 enterica strains to 11 previously sequenced S. enterica genomes to carry out the most comprehensive

235 es are missing (e.g., avrA, sopB, and sseL), S. enterica subsp. salamae invades HeLa cells and contai

236 These results demonstrate that systemic S. enterica infection and diarrhea/colitis are distinct

237 These gene panels distinguish all tested S. enterica subspecies I serovars and their known genova

238 nterica ridA causes Ser sensitivity and that S. enterica RidA and its homologs from other organisms h

239 ing 12/15-lipoxygenase (12/15-LOX), and that S. enterica serovar Typhimurium and S. flexneri share ce

240 We further demonstrate that S. enterica LT2 retained the ability to grow on 1,2-prop

241 urther into this pathway, we also found that S. enterica serovar Typhimurium and S. flexneri activate

242 The results of this study indicate that S. enterica serovar Typhimurium can outgrow E. coli in h

243 Our results indicate that S. enterica synthesizes alpha-R, a metabolite that had n

244 oreover, the data raise the possibility that S. enterica DeltafrdABCD DeltasdhCDA double mutants and

245 We also show that S. enterica yhhK strains accumulate pro-PanD, and that n

246 The S. enterica fadR strains grow more rapidly than the wild

247 The S. enterica serovar Typhimurium hybrid strains showed si

248 As the S. enterica species comprises sublineages that differ gr

249 xpressed in C. elegans intestinal cells, the S. enterica TTSS-exported effector protein SptP inhibite

250 to the sequence reported in GenBank for the S. enterica serovar Typhimurium LT2 strain.

251 In the S. enterica ACS structure, the propyl group of adenosine

252 s also reduce bacterial proliferation in the S. enterica serovar Typhimurium mouse model.

253 The flagella-release phenotype of the S. enterica fliL mutant has a bearing on FliL-dependent

254 olog complemented the Ser sensitivity of the S. enterica ridA mutant.

255 The chromosomal copy of the S. enterica serovar Typhimurium sigma(28) structural gen

256 Most lineages of the S. enterica subspecies Typhimurium cause gastroenteritis

257 es between the two organisms showed that the S. enterica FadE and FadBA enzymes were responsible for

258 trophic CO(2) fixation), suggesting that the S. enterica organelles and carboxysomes have a related m

259 ons that exhibit significant homology to the S. enterica transducing phage ES18.

260 y was not enhanced by Mg(2+) and, unlike the S. enterica CobA enzyme, it was >50% inhibited by Mn(2+)

261 Unlike the S. enterica enzyme, CobA(Mm) used cobalamin (Cbl) as a s

262 rica subsp. II and S. bongori in addition to S. enterica subsp. I.

263 e that YafD provides a survival advantage to S. enterica serovar Enteritidis in eggs by repairing DNA

264 tial DNA and glucuronide binding affinity to S. enterica GusR.

265 This difference in growth was attributed to S. enterica having higher cytosolic levels of the induci

266 glycerol dehydratase family, in contrast to S. enterica, which relies on a B12-dependent enzyme.

267 A, SopA, SopB, SopD, and SopE2 contribute to S. enterica serotype Typhimurium invasion of epithelial

268 sceptibility of caveolin-1-deficient mice to S. enterica serovar Typhimurium.

269 NPs as molecular markers for the response to S. enterica serovar Enteritidis may result in the enhanc

270 inacin genes with the phenotypic response to S. enterica serovar Enteritidis, an F1 population of chi

271 ons of sire alleles with progeny response to S. enterica serovar Enteritidis.

272 onal and if it was evolutionarily similar to S. enterica serovar Typhimurium CorA.

273 Although excess Fe2+ was slightly toxic to S. enterica serovar Typhimurium, we were unable to elici

274 TcuABC proteins were the only ones unique to S. enterica needed to catabolize tricarballylate.

275 et al. that oral inoculation with wild-type S. enterica serovar Typhimurium strains lead to bacteria

276 egulator (prpR)) were evaluated in wild-type S. enterica serovar Typhimurium TR6583 and prpB(-) or pr

277 encoding PagL, PagP, and LpxR into wild-type S. enterica serovar Typhimurium.

278 and invasive infections due to non-typhoidal S. enterica infections resulted in the highest burden, c

279 Some hazards, such as non-typhoidal S. enterica, were important causes of FBD in all regions

280 Unlike S. enterica serotype Typhimurium, serotype Typhi or a se

281 We show that the previously unsequenced S. enterica serovar 9,12:l,v:- belongs to the B clade of

282 ritidis, whose overexpression conferred upon S. enterica serovar Typhimurium enhanced resistance to e

283 In this study, we used S. enterica serovar Typhimurium as an in vivo heterologo

284 In vitro methylation analyses utilizing S. enterica and Thermotoga maritima CheR proteins and MC

285 conditions, E. coli failed to grow, whereas S. enterica grew well.

286 While S. enterica serovar Typhimurium has been shown to kill i

287 (CD11b+), and dendritic cells (CD11c+) with S. enterica serovar Typhimurium induced an up-regulation

288 was first formed followed by challenge with S. enterica serovar Typhimurium, there was significant b

289 lysed bone marrow macrophages infected with S. enterica serovar Typhimurium or pulsed with proteins

290 H2-M3-transfected fibroblasts infected with S. enterica serovar Typhimurium SL3261 or treated with S

291 oducing EcN to mice previously infected with S. enterica substantially reduced intestinal colonizatio

292 l vaccine, rabbits were orally infected with S. enterica Typhimurium strain chi3987 harboring phagemi

293 the first 1 or 2 weeks after infection with S. enterica serotype Typhi.

294 To identify cases of human infection with S. enterica serotype Typhimurium potentially related to

295 tion model, mice were orally inoculated with S. enterica 24 h post-initiation of abrupt withdrawal fr

296 ating antibody levels after inoculation with S. enterica serovar Enteritidis bacterin vaccine.

297 um chloride (DPI), but infection of MDM with S. enterica serovar Typhimurium did not cause an increas

298 These operons are widely distributed within S. enterica but absent from the closely related Escheric

299 reaching the level in cells infected with WT S. enterica serovar Typhimurium, than the level in host

300 ty did not reach the level of wild-type (WT) S. enterica serovar Typhimurium.