ライフサイエンスコーパス: S. marcescens

1 S. marcescens does not normally colonize human skin, but

2 S. marcescens expresses prodigiosin, a bright red and ce

3 S. marcescens induced neutrophil recruitment to the corn

4 S. marcescens induces corneal inflammation by activation

5 S. marcescens isolates were compared using restriction-e

6 S. marcescens oxyR mutants were severely impaired in bio

7 S. marcescens was identified from a pharmacy water fauce

8 isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and

9 ene knockout mice was abraded, and 1 x 10(7) S. marcescens were added in the presence of a silicone h

10 It has been reported that a S. marcescens chimera, SM : rS5'ec, in which five diverg

11 The activity of the fraction against S. marcescens was explained by (R)-(-)-mellein alone, an

12 tic (R)-(-)-mellein and micromolide, against S. marcescens and a Gram-positive bacterium, Staphylococ

13 tion of bronchoscopes with P. aeruginosa and S. marcescens and possible infection of patients at a co

14 ducted an investigation of P. aeruginosa and S. marcescens isolates related to bronchoscopy at a comm

15 aerogenes, K. pneumoniae, P. aeruginosa, and S. marcescens) became more susceptible.

16 with Acinetobacter spp., P. aeruginosa, and S. marcescens, 5/6 with Citrobacter spp., 13/14 with Ent

17 seven of these had Enterobacter cloacae and S. marcescens in the same culture.

18 nce birth, but gut colonization with GBS and S. marcescens occurred closer to time of bloodstream inf

19 Cases were defined as S. marcescens BSIs in patients receiving PN from the pha

20 its transporter ShlB resulted in attenuated S. marcescens strains that failed to cause profound weig

21 eement was observed, excluding A. baumannii, S. marcescens, and S. pneumoniae, for which >/=4-fold di

22 n foreskin fibroblasts was also inhibited by S. marcescens secretomes indicating that the effect is n

23 , that is located upstream of NucC-dependent S. marcescens promoters and the late promoters of P2-rel

24 P, and prodigiosin concentration changes for S. marcescens during cultivation in batch culture.

25 Several risk factors for S. marcescens infection were identified, but hospital an

26 Purified LPS from S. marcescens, but not from Escherichia coli or S. marce

27 roduce SlpB as a new cytotoxic protease from S. marcescens.

28 nd interleukin-1 receptor type 1 (IL-1R1) in S. marcescens-induced corneal inflammation and infection

29 itro cytotoxic activity commonly observed in S. marcescens culture filtrates.

30 nergy-dependent and Sec-dependent pathway in S. marcescens.

31 echanism for the second step of secretion in S. marcescens.

32 Tobramycin-killed S. marcescens induced corneal inflammation in C57BL/6 mi

33 encia burhodogranaria, is gram negative like S. marcescens.

34 R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. muenchen) do

35 less, the temperature response of the native S. marcescens ATCase suggests a strong entropic effect t

36 ase treatments abrogated the cytotoxicity of S. marcescens culture filtrates towards HeLa cells, sugg

37 LPS depletion of S. marcescens secretomes with polymyxin B agarose render

38 Despite multiple clinical descriptions of S. marcescens nosocomial pneumonia, little is known rega

39 ective means to control the dissemination of S. marcescens, an in-depth analysis of the population st

40 ential therapeutic targets for inhibition of S. marcescens-induced corneal inflammation.

41 This study describes a model of S. marcescens pneumonia that mimics known clinical featu

42 A case was defined as the occurrence of S. marcescens bacteremia in any patient in the surgical

43 An outbreak of S. marcescens and E. cloacae bacteremia in a surgical in

44 ding standards contributed to an outbreak of S. marcescens BSIs.

45 tify any environmental or staff reservoir of S. marcescens.

46 in which OxyR contributes to early stages of S. marcescens biofilm formation by influencing fimbrial

47 first reports applying PFGE to the study of S. marcescens, and this method was a useful marker of st

48 istant clones suggests that the treatment of S. marcescens infections will become increasingly diffic

49 marcescens, but not from Escherichia coli or S. marcescens strains with mutations in the waaG and waa

50 colonized with provisionally matching GBS or S. marcescens.

51 for Fis are 100% identical in K. pneumoniae, S. marcescens, E. coli, and S. typhimurium and 96 to 98%

52 ematic collection of antimicrobial-resistant S. marcescens associated with bloodstream infections in

53 what is observed in other bacterial species, S. marcescens OxyR is required for oxidative stress resi

54 eal aspiration model of lethal and sublethal S. marcescens pneumonia in BALB/c mice and extensively c

55 ability of the tigecycline Etest for testing S. marcescens, Acinetobacter spp., and S. pneumoniae is

56 or in vitro cytotoxic activity revealed that S. marcescens mutant strains that are deficient in produ

57 Our results show that S. marcescens is a diverse species with a high level of

58 Together these data suggest that S. marcescens LPS is sufficient for inhibition of epithe

59 Finally, recombinant expression of the S. marcescens 56-kDa metalloprotease conferred a cytotox

60 CTP stimulates the catalytic activity of the S. marcescens ATCase and CTP/UTP inhibitory synergism ha

61 measured differences in processivity of the S. marcescens chitinases.

62 Bioinformatic analysis of the S. marcescens Db11 genome revealed three additional open

63 One crystal form of the S. marcescens enzyme displays a bound pyruvate as well a

64 93-r97) of the regulatory polypeptide of the S. marcescens enzyme have been replaced with their E. co

65 ) retained 455 out of 460 amino acids of the S. marcescens enzyme, it possessed characteristics simil

66 n E. coli for extracellular secretion of the S. marcescens nuclease.

67 proteins are the original substrates of the S. marcescens T6SS, before horizontal acquisition of oth

68 yperthermophile Sulfolobus solfataricus, the S. marcescens structure shows similar subunit structures

69 NucC binding to the S. marcescens nuclease promoter P(nucA) and to the seque

70 ues of E. coli have been replaced with their S. marcescens counterpart, lost both heterotrophic and h

71 Literature pertinent to S. marcescens-mediated necrotizing fasciitis is also rev

72 (-/-), and TLR4/5(-/-) corneas infected with S. marcescens had significantly increased CFU, indicatin

73 itis of the chest wall due to infection with S. marcescens that initially manifested as bilateral bre

74 ed insect survival after oral infection with S. marcescens.

75 ording to univariate analysis, patients with S. marcescens bacteremia stayed in the surgical intensiv

76 Twenty-six patients with S. marcescens bacteremia were identified; eight (31 perc

77 tify risk factors, we compared patients with S. marcescens bacteremia with randomly selected controls

78 were positive for P. aeruginosa also yielded S. marcescens.

79 nyl infusions from two case patients yielded S. marcescens and E. cloacae.