ライフサイエンスコーパス: SAF

1 SAF also significantly lowered fasting glucose (P = 0.03

2 SAF is a potentially valuable clinical screening tool fo

3 SAF partially mediated the association between T2DM and

4 SAF was classified into two types: type 1, which was fil

5 SAF-1 activity was detected in the chondrocytes of OA ca

6 SAF-1 contains several negative and positively functioni

7 SAF-1 DNA-binding activity is increased in both cytokine

8 SAF-1 is a 477-amino acid protein with six zinc fingers.

9 SAF-1, a zinc finger transcription factor, is activated

10 SAF-1-overexpressing mice spontaneously developed AA amy

11 SAF-A oligomerization decompacts large-scale chromatin s

12 SAFs for photons were generated for mildly and severely

13 SAFs of W-CIN cells were remarkably similar to those ind

14 SAFs studied between the obese phantoms and the 50th per

15 role of serum amyloid A-activating factor 1 (SAF-1) in MMP-1 expression was assessed by transient tra

16 factor, serum amyloid A-activating factor 1 (SAF-1), has been shown to regulate several genes, includ

17 factor serum amyloid A-activating factor-1 (SAF-1) has been identified as a regulator of a number of

18 Serum amyloid A-activating factor-1 (SAF-1) is a zinc finger transcription factor that is act

19 Serum amyloid A (SAA) activating factor-1 (SAF-1) is an inducible transcription factor that plays a

20 amyloid A-activating transcription factor-1 (SAF-1) plays a major role in regulating transcription of

21 factor, serum amyloid A activating factor-1 (SAF-1), leading to markedly higher levels of angiogenesi

22 Of the three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of

23 ulated with various adjuvants: QS-21, IL-12, SAF-1, CD40L, and GM-CSF were compared.

24 These include NF-kappaB, C/EBP, YY1, AP-2, SAF and Sp1.

25 cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of homology to MAZ/ZF

26 nesis of AA amyloidosis, we have developed a SAF-1 transgenic mouse model.

27 ly higher DNA binding activity than either a SAF-1 homodimer or a c-Fos/c-Jun heterodimer.

28 Scaffold attachment factor A (SAF-A), also called heterogenous nuclear ribonuclear pro

29 we report that scaffold attachment factor A (SAF-A), originally identified as a structural nuclear pr

30 oantigens, and scaffold attachment factor A (SAF-A).

31 hosphorylation sites created a highly active SAF-1 protein with high DNA-binding ability.

32 that reentry is important to maintain acute SAF.

33 important in maintaining stretch-induced AF (SAF).

34 Although SAF is expressed in all lymphocytes, immunofluorescence

35 Although SAF-1 transgenic mice do not spontaneously develop arthr

36 e SAA promoter in association with SAF as an SAF.Sp1 heteromeric complex.

37 To develop an SAF teledermatology workflow within the Epic system, the

38 transfected cells increased expression of an SAF-regulated promoter.

39 her levels of matrix metalloproteinase-1 and SAF-1 in the inflamed joints of SAF-1 transgenic mice co

40 on responsive transcription factors AP-1 and SAF-1 synergistically regulate transcriptional induction

41 QS-21, IL-12, and SAF-1 biased the humoral immune response toward Th1-type

42 gy to MAZ/ZF87/Pur-1 protein while SAF-5 and SAF-8 isoforms are unique and are related to SAF-1/MAZ/Z

43 he three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of homology t

44 same slides by using the FLIP algorithm and SAF score, blinded to their first evaluation.

45 tains the tumor suppressors SAFB1, BRG1, and SAF-A.

46 MP-dependent protein kinase (PKA-Calpha) and SAF-1.

47 ctural component of articular cartilage, and SAF-1 in both SAF-1 transgenic and nontransgenic mice.

48 Supplementation with CLA and SAF exerted different effects on BMI, total and trunk ad

49 benefits of beam spinning EW excitation and SAF detection and provides the conditions for quantitati

50 P-1 family of proteins, c-Fos and c-Jun, and SAF-1 form a ternary protein complex, which has markedly

51 The combination of SP-PCR and SAF microlens array allows for on-chip highly sensitive

52 ed transactivation potential of both Sp1 and SAF as a consequence of a phosphorylation event.

53 own to regulate microtubule organization and SAFs known to promote microtubule assembly such as Maski

54 ntaining either a functional or an antisense SAF-1 complementary DNA.

55 ce of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibited interleukin-1-mediated MMP

56 is regulated by a posttranslational event as SAF DNA-binding and transactivation abilities are increa

57 ng the function of spindle matrix-associated SAFs.

58 Non-invasive skin autofluorescence (SAF) measurement serves as a proxy for tissue accumulati

59 ests, and noninvasive skin autofluorescence (SAF; a measure of tissue AGE levels) on people aged >55

60 Epic-based SAF teledermatology can improve access to dermatologic c

61 The physical interaction between SAF-1 and AP-1 was demonstrated both in vitro by Far-Wes

62 ealed colocalization and interaction between SAF-1 and PKA-Calpha.

63 efficiently promotes transcription from both SAF-1 and AP-1 sites of human MMP-1 promoter.

64 nt of articular cartilage, and SAF-1 in both SAF-1 transgenic and nontransgenic mice.

65 omised the transactivation potential of both SAF-1 and AP-1.

66 es additional layers of regulation, and both SAFs are only degraded after being released from their i

67 or protein p21 was significantly affected by SAF-1; its expression level was highly induced by cellul

68 Of the three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high de

69 In this report, using a cloned SAF gene in transient transfection assay, we provide evi

70 rvation, increased DNA binding of the cloned SAF and its hyperphosphorylation, in response to okadaic

71 Of the three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showe

72 CMV-SAF-1 transgenic mice, upon B. burgdorferi infection, sh

73 Conceivably, SAF-1 could be a potential therapeutic target to control

74 In contrast, SAF had no effect on BMI or total adipose mass but reduc

75 health care organizations wanting to create SAF teledermatology workflows within the Epic EHR system

76 ion of SAF-1 protein from either end creates SAF-1 isoforms that are highly transcriptionally active.

77 he most influential variables in determining SAF values in men, whilst in female participants, SR was

78 NUSAP1 contains a conserved SAP domain (SAF-A/B, Acinus, and PIAS).

79 whose expression levels were altered during SAF-1 overexpression.

80 This result suggested that C/EBP, SAF, and NF-kappa B are required for transient acute pha

81 Photon and electron SAFs were then calculated for relevant organs in all mod

82 sentative plots were made of photon electron SAFs, evaluating the traditional assumption that all ele

83 The DNA binding activity of endogenous SAF was increased by agonists of PKC.

84 MP1 promoter, and interference of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibite

85 Phosphorylation of endogenous SAF-1 in response to IL-1 and IL-6 was markedly inhibite

86 promoter as well as knockdown of endogenous SAF-1 markedly inhibited IL-1beta- and TGF-beta-mediated

87 Moreover, cells expressing SAF-A in which serine 59 is mutated to alanine have mult

88 , in addition to tissue-specific expression, SAFs, a family of zinc finger transcription factors, und

89 These data show that transcription factor SAF-1 is involved in the regulation of IL-6-mediated ind

90 n that the inflammatory transcription factor SAF-1 is, at least in part, responsible for the marked i

91 inflammation-responsive transcription factor SAF-1 was found to interact with it.

92 Increased SAA-activating factor (SAF) activity was detected in the liver, lung, and brain

93 tor serum amyloid A (SAA)-activating factor (SAF), a family of zinc finger proteins, plays a signific

94 show that serum amyloid A-activating factor (SAF)-1, a novel transcription factor, and the AP-1 famil

95 n factor, serum amyloid A activating factor (SAF)-1, was demonstrated by EMSA.

96 , referred to as silencer-associated factor (SAF), is a member of the helix-turn-helix factor family

97 n, and a nuclear factor, SAS-binding factor (SAF), that interacts with the SAS element has been ident

98 and SAA activating sequence binding factor (SAF).

99 nflammation-responsive transcription factor, SAF (for SAA activating factor), has been implicated in

100 le matrix contains spindle assembly factors (SAFs) such as Eg5 and dynein which are known to regulate

101 of importin-bound spindle assembly factors (SAFs), which stimulate microtubule (MT) nucleation and o

102 id cells and senescence-associated features (SAFs).

103 We use a self-assembling fiber (SAF) system to form structures tens of micrometers long.

104 rization of a self-assembling peptide fiber (SAF) system based on alpha-helical coiled-coil building

105 m fillets and the Sparus aurata fibroblasts (SAF-1) cell-line during an 8day storage period at +4 deg

106 oring system (steatosis, activity, fibrosis [SAF]) allowing the use of an algorithm (fatty liver inhi

107 ation with supercritical-angle fluorescence (SAF) detection efficiently rejects the fluorescence orig

108 -PCR with super critical angle fluorescence (SAF) microlens array embedded in a microchip.

109 To identify possible activation partners for SAF-1, we used a yeast two-hybrid system that detected i

110 where most of the cells stained positive for SAF-1.

111 sis suggests that L(pro) contains a SAP (for SAF-A/B, Acinus, and PIAS) domain, a protein structure a

112 which one serves as the DNA-binding site for SAF-1 transcription factor.

113 External store-and-forward (SAF) teledermatology systems operate separately from the

114 e of obesity on specific absorbed fractions (SAFs) and dose factors in adults.

115 ed to study how specific absorbed fractions (SAFs) vary with changes in adult body size, for persons

116 es to calculate specific absorbed fractions (SAFs) with internal photon and electron sources.

117 ta by first computing site allele frequency (SAF) likelihood for each site (i.e. the likelihood a sit

118 the task's speed-accuracy tradeoff function (SAF), which prevented us from falsely interpreting varia

119 Furthermore, SAF-1 transgenic mice rapidly developed severe AA amyloi

120 iotropic role of SAF-1 in vivo, we generated SAF-1 transgenic mice, in which CMV immediate-early prom

121 Greater SAF, T2DM, and cognitive impairment were each associated

122 T2DM was associated with greater SAF.

123 12 male patients; mean age, 20.1 years) had SAF type 1 (mean width, 5.2 x 4.5 mm).

124 46 male patients; mean age, 37.8 years) had SAF type 2 (mean width, 5.1 x 4.7 mm).

125 To evaluate how SAF is involved in SAA promoter activation, we have inve

126 sent in the atherosclerotic plaque implicate SAF-1 as a key regulator of MMP-14 gene induction in mac

127 sly unreported and significant difference in SAF values between men and women, with median (range) va

128 Two distinct but adjacent domains in SAF-1 are involved in protein-protein contact with c-Fos

129 vidence that VEGF expression is increased in SAF-1-transgenic mice and that SAF-1 induces VEGF transc

130 Differences in SAFs for organs irradiating themselves were between 0.5%

131 Differences in SAFs for organs outside the trunk were not greater than

132 ther splicing regulatory proteins, including SAF-B, hnRNP G, YB-1, and p72.

133 Also, during inflammation SAF-1 transgenic mice exhibited a prolonged acute phase

134 Development of internal SAF workflows within existing electronic health records

135 Although many SAFs are non-motile MT-associated proteins, such as NuMA

136 The mean SAF of the study cohort was 2.06 (SD = 0.57) arbitrary u

137 We fabricated miniaturized SAF microlens array as part of a microfluidic chamber in

138 MPN-SAF TSS results significantly differed among MPN disease

139 MPN-SAF TSS was calculable for 1,408 of 1,433 patients with

140 A total of 402 MPN-SAF surveys were administered (English [25%], Italian [4

141 Serial administration of the English MPN-SAF among 53 patients showed that most MPN-SAF items are

142 rative Neoplasm Symptom Assessment Form [MPN-SAF], coadministered with the Brief Fatigue Inventory) t

143 N-SAF among 53 patients showed that most MPN-SAF items are well correlated (r > 0.5, P < .001) and hi

144 tomatic elements represented on both the MPN-SAF and the European Organisation for Research and Treat

145 The MPN-SAF is a comprehensive and reliable instrument that is a

146 The MPN-SAF TSS had excellent internal consistency (Cronbach's a

147 The MPN-SAF TSS is a concise, valid, and accurate assessment of

148 nderlying construct, indicating that the MPN-SAF TSS is an appropriate, unified scoring method.

149 The MPN-SAF TSS strongly correlated with overall quality of life

150 h increase could be detected with the mutant SAF-1 proteins.

151 ograms (n = 6) or 250 micrograms (n = 20) of SAF-m.

152 Our findings identify serine 59 of SAF-A as a new target of both PLK1 and PP2A in mitosis a

153 ether, these results show that activation of SAF-1 in response to IL-1 and -6 is mediated via MAP kin

154 tribution of MAP kinase in the activation of SAF-1, we prepared two independent mutant proteins in wh

155 tion is involved in functional activation of SAF-1.

156 which PKA increases functional activities of SAF-1.

157 Activity of SAF is regulated by a phosphorylation event involving se

158 Functional activity of SAF is regulated by a posttranslational event as SAF DNA

159 lation increases transcriptional activity of SAF-1 by unmasking the DNA-binding domain.

160 mostly increases the DNA binding activity of SAF-1.

161 Increased binding of SAF-1 to the MMP-14 promoter correlated with the transcr

162 overexpression of SAF-1 and coexpression of SAF-1 and MMP-14 in the macrophages present in the ather

163 specific subcellular compartmentalization of SAF plays an important role in mediating the specificity

164 The data also demonstrate that control of SAF-1 activity can suppress induced expression of MMP-1.

165 Deletion of SAF-1 binding elements from the VEGF promoter as well as

166 thermore, we found that terminal deletion of SAF-1 protein from either end creates SAF-1 isoforms tha

167 oth phosphorylation and dephosphorylation of SAF-A serine 59 by PLK1 and PP2A, respectively, are requ

168 ed 293 patients with a lifetime diagnosis of SAF disorder, bipolar disorder and major depressive diso

169 n constructs, the core DNA-binding domain of SAF-1 is mapped between amino acids 282 and 361, which c

170 In vivo evidence of SAF-1 and PKA-Calpha interaction was further revealed by

171 Prolonged high level expression of SAF-1 in cultured cells failed to produce any stable cel

172 ys a major role in controlling expression of SAF-regulated genes by increasing the interaction betwee

173 To test the fate of SAF-1-overexpressing cells, the cells were monitored for

174 The high frequency of SAF on MR arthrograms (10.5%), the absence of subchondra

175 e cells is primarily due to the induction of SAF activity.

176 PKA) and presented evidence for induction of SAF-1-regulated genes by a PKA signaling pathway.

177 Previously, we showed in vivo interaction of SAF-1 and protein kinase A (PKA) and presented evidence

178 einase-1 and SAF-1 in the inflamed joints of SAF-1 transgenic mice compared with their levels in nont

179 We present evidence that knockdown of SAF-1 by small interfering RNAs inhibits AP-1-mediated i

180 on assay, in vivo, markedly higher levels of SAF-1 interaction with the VEGF promoter was detected in

181 processing activity during overexpression of SAF-1 and coexpression of SAF-1 and MMP-14 in the macrop

182 Although overexpression of SAF-1 could increase expression of the MMP-9 promoter an

183 Overexpression of SAF-1 in OA chondrocytes increased the expression of the

184 Our study shows that overexpression of SAF-1 predisposes animals to arthritis.

185 ical conditions that favor overexpression of SAF-1, such as an acute inflammatory condition, can trig

186 on of MMP-9 gene concurrent participation of SAF-1 and AP-1 is required.

187 markedly induces in vivo phosphorylation of SAF-1 and transcription of SAF-regulated reporter genes.

188 In vitro phosphorylation of SAF-1 by PKC-beta markedly increased its DNA binding abi

189 ably lowers the transactivation potential of SAF-1.

190 ng activity and transactivation potential of SAF-1.

191 ptional repression and active recruitment of SAF-1-mediated transcriptional activation.

192 chanism of synergy and the essential role of SAF-1 and AP-1 in up-regulating human MMP-1 expression u

193 Together these results suggest a role of SAF-1 in the pathogenesis of inflammation-induced arthri

194 To assess the pleiotropic role of SAF-1 in vivo, we generated SAF-1 transgenic mice, in wh

195 t under native condition, N and C termini of SAF-1 are engaged in an inhibitory intramolecular intera

196 hosphorylation of SAF-1 and transcription of SAF-regulated reporter genes.

197 gated linoleic acid (CLA) and safflower oil (SAF), on body weight and composition in obese postmenopa

198 g/d of ethyl esters of either safflower oil (SAF; control), eicosapentaenoic acid (EPA), or docosahex

199 Mechanistically, this is dependent on SAF-A's AAA(+) ATPase domain, which mediates cycles of p

200 bony fossa in the roof of the acetabulum, or SAF.

201 IR and steatosis, activity, and fibrosis (or SAF) scoring systems.

202 fluorescently labeled streptavidin (SAF) or SAF conjugated to biotinylated Cy3 adenoviral-vector (BC

203 duce any stable cell line that overexpresses SAF-1.

204 he cells that were programmed to overproduce SAF-1 were found to undergo growth arrest and reduce DNA

205 dy we provide evidence that, similar to p53, SAF-1 is able to activate p21 gene expression by promoti

206 were made to the reference (50th) percentile SAF values.

207 were made to the reference (50th) percentile SAF values.

208 tions of radiation transport were performed; SAFs for photons were generated for 10th, 25th, 75th, an

209 ted that unphosphorylated and phosphorylated SAF-1 proteins are structurally distinct.

210 tion between promoter DNA and phosphorylated SAF.

211 is, we asked whether DNA-PKcs phosphorylates SAF-A in mitosis.

212 alone or pHrodo complexed to E. coli, pHrodo-SAFs report pH in both the cytoplasm and phagosomes, as

213 In the MMP-9 promoter, SAF-1 and AP-1 DNA-binding elements are present in close

214 In the human VEGF promoter, SAF-1- and KLF-4-binding elements are overlapping, where

215 ctor-1/c-Myc-associated zinc finger protein (SAF-1/MAZ), and mutation of the SAF-1RE had little effec

216 nopausal women with type 2 diabetes received SAF or CLA (8 g oil/d) during two 16-wk diet periods sep

217 We assessed reference SAF and skin reflectance (SR) values in a Saudi populati

218 microtubule assembly caused by the released SAFs would lead to excessive microtubule sliding that re

219 Myocardin is a SAP (SAF-A/B, Acinus, PIAS) domain transcription factor that

220 c Leukemia-1 (MKL1), a gene encoding an SAP (SAF-A/B, Acinus and PIAS) DNA-binding domain.

221 isorders (that is, bipolar, schizoaffective (SAF), major depression) based on contemporary diagnostic

222 or four known SR protein-binding motifs: SF2/SAF, SC35, SRp40, and SRp55.

223 e with Xist, three of these proteins--SHARP, SAF-A and LBR--are required for Xist-mediated transcript

224 MR arthrograms showing SAF were evaluated for subchondral reactions.

225 Since SAF-A, DNA-PK catalytic subunit (DNA-PKcs), and protein

226 ation of fluorescently labeled streptavidin (SAF) or SAF conjugated to biotinylated Cy3 adenoviral-ve

227 ited States, and development of a successful SAF workflow within it is needed.

228 used to study the relationships among T2DM, SAF, and gray matter volume (GMV).

229 olishing reentry with chloroquine terminates SAF more effectively than traditional Na+-channel blocka

230 ore effective than flecainide in terminating SAF in isolated sheep hearts by significantly increasing

231 increased in SAF-1-transgenic mice and that SAF-1 induces VEGF transcription by directly binding to

232 We demonstrate that SAF improves the surface selectivity of TIRF, even at sh

233 cription factor genes have demonstrated that SAF-Sp1 heteromer is a highly potent transactivator of S

234 transfection assay, we provide evidence that SAF potentiates SAA gene expression through SAS element.

235 , these results provide direct evidence that SAF-1 plays a key role in the development of AA amyloido

236 nsgenic mice, supporting the hypothesis that SAF is important in mediating silencer function.

237 Our studies indicate that SAF belongs to a family of transcription factors charact

238 es, immunofluorescence studies indicate that SAF is present primarily in the cytoplasm in T cells in

239 Together these data indicate that SAF-1 controls cell cycle progression via p21 induction,

240 The results indicate that SAF-1 is involved in the regulation of MMP1 gene express

241 e kinase 1 (PLK1) rather than DNA-PKcs, that SAF-A interacts with PLK1 in nocodazole-treated cells, a

242 green fluorescent protein reporter show that SAF-1 contains two independent nuclear localization sign

243 We further show that SAF-1 is phosphorylated in vitro by PKA-Calpha and that

244 Here, we show that SAF-1-mediated induction of VEGF is repressed by KLF-4 t

245 Our results show that SAF-A and caRNAs form a dynamic, transcriptionally respo

246 We show that SAF-A is phosphorylated on serine 59 in mitosis, that ph

247 ivation of SRp20 by SRrp86, we now show that SAF-B, hnRNP G, and 9G8 all antagonize the activity of S

248 In this paper we have shown that SAF-1, a major member of the SAF family, is abundantly p

249 distinct DNA-binding elements suggests that SAF-1 and AP-1 function in a mutually beneficial manner

250 The SAF results were compared with values from similar studi

251 The SAF-1.c-Fos.c-Jun ternary complex efficiently promotes t

252 No subchondral changes were found around the SAF.

253 For each case, the SAF score was created including the semiquantitative sco

254 near in the number of genomes to compute the SAF likelihood.

255 thod produces an accurate SFS, computing the SAF likelihood is quadratic in the number of samples seq

256 ity score but to report it separately in the SAF score.

257 orylation analyses revealed two sites in the SAF-1 protein, serine 187 and threonine 386, as the targ

258 Introduction of a specific mutation into the SAF binding site in the CD4 silencer abrogates silencer

259 cartilage defect was seen at the site of the SAF at arthroscopy.

260 have shown that SAF-1, a major member of the SAF family, is abundantly present in human AA amyloidosi

261 algorithm, all non-negligible values of the SAF likelihood are concentrated on a few cells around th

262 gh fluorescence collection efficiency of the SAF microlens array, the SP-PCR assay on the LOC platfor

263 ed multiplexed SP-PCR directly on top of the SAF microlens array.

264 tudy was to determine whether the use of the SAF score and FLIP algorithm can decrease interobserver

265 m 42% to 75% in Group 2 after the use of the SAF score and FLIP algorithm.

266 The width of the SAF was measured on coronal and sagittal MR images.

267 romoter was used to direct expression of the SAF-1 transgene in multiple organs.

268 reduced ability to promote expression of the SAF-1-regulated promoter.

269 ger protein (SAF-1/MAZ), and mutation of the SAF-1RE had little effect on IL-6 induction of gammaFBG

270 The FLIP algorithm based on the SAF score should decrease interobserver variations among

271 of the terminal negative modules renders the SAF-1 protein functionally very active.

272 age defects at arthroscopy indicate that the SAF of the acetabulum likely represents a variant.

273 With regard to the SAF score, concordance was substantial in Group 1 for st

274 Myocardin belongs to the SAF-A/B, Acinus, PIAS (SAP) domain family of transcripti

275 e suggest describing liver lesions using the SAF score.

276 Also, the SAFs were used with standardized decay data to develop d

277 With preceding designs, the SAFs are thickened, highly ordered structures in which m

278 f self-assembly and self-organization in the SAFs is unprecedented for a designed peptide-based mater

279 with C-terminal thioester moieties into the SAFs.

280 Here, we describe the structure of the SAFs determined to approximately 8 A resolution using cr

281 designed a self-assembling fiber system, the SAFs, in which two small alpha-helical peptides are prog

282 SAF-8 isoforms are unique and are related to SAF-1/MAZ/ZF87/Pur-1 at the zinc finger domains but diff

283 Relative to SAF, EPA and DHA supplementation resulted in higher MVs

284 Furthermore, overexpression of wild-type SAF-1 in transfected liver cells can increase transcript

285 While the DNA-binding activity of wild-type SAF-1 protein was markedly increased upon phosphorylatio

286 ng variations in position along an unchanged SAF as a change in skill.

287 We surmised that reentry underlies SAF, and that abolishing reentry with chloroquine termin

288 highly induced by cellular conditions where SAF-1 is abundant.

289 -4-binding elements are overlapping, whereas SAF-1 induces and KLF-4 suppresses VEGF expression.

290 ansient acute phase induction of SAA whereas SAF and NF-kappa B activities are necessary for persiste

291 To assess whether SAF-1 is directly linked to the pathogenesis of AA amylo

292 of homology to MAZ/ZF87/Pur-1 protein while SAF-5 and SAF-8 isoforms are unique and are related to S

293 mpacts large-scale chromatin structure while SAF-A loss or monomerization promotes aberrant chromosom

294 four hip joints with SAF type 1 and 13 with SAF type 2.

295 ts with the SAA promoter in association with SAF as an SAF.Sp1 heteromeric complex.

296 d obesity all showed strong association with SAF, particularly when gender differences were taken int

297 SAA expression by acting in conjunction with SAF.

298 icipants, SR was also highly correlated with SAF.

299 ose from arthroscopy in four hip joints with SAF type 1 and 13 with SAF type 2.

300 AP kinase phosphorylation site, PPTP, within SAF-1 could be phosphorylated by MAP kinase in vitro.