ライフサイエンスコーパス: SCS

1 SCS and SCL were visible in 15 eyes.

2 SCS applied with the three frequencies produced similar

3 SCS at 500 Hz significantly increased cutaneous blood fl

4 SCS at 500 Hz significantly increased SCS-induced vasodi

5 SCS at frequencies of 50, 200, or 500 Hz was applied at

6 SCS converge to form local gaps that close based on both

7 SCS disrupted this pathological circuit behavior in a ma

8 SCS injection of low (0.2 mg) and high doses (2.0 mg) of

9 SCS is a dominant disorder with mutations reported for a

10 SCS is an under-recognized cause of longitudinally exten

11 SCS is used to improve peripheral circulation in selecte

12 SCS patients had constitutional symptoms, cerebrospinal

13 SCS significantly improved cardiac contractile function

14 SCS was delivered at one of 3 different frequencies (4 H

15 l and reversed polarities for samples PO-01, SCS-01 and SCS-02, indicating a primary record of the Ea

16 .48 mm/Ma and 11.28 mm/Ma for samples PO-01, SCS-01, SCS-02 and IO-01, respectively.

17 a and 11.28 mm/Ma for samples PO-01, SCS-01, SCS-02 and IO-01, respectively.

18 ific Ocean (PO-01), South China Sea (SCS-01, SCS-02) and Indian Ocean (IO-01).

19 We studied 71 patients (NMOSD, 37; SCS, 34).

20 Sixteen (47%) SCS cases were initially diagnosed as NMOSD or idiopathi

21 (LarA) of Lactobacillus plantarum harbors a (SCS)Ni(II) pincer complex derived from nicotinic acid.

22 onal theory optimizations, the high-accuracy SCS-MP2/cc-pVinfinityZ energies, and PCM solvation corre

23 However, how infection affects SCS macrophages remains largely unexplored.

24 rs of oxygenated HMP (at 12 degrees C) after SCS and before NMP.

25 mals were perfused either 5 min or 2 h after SCS and c-fos protein examined immunohistochemically.

26 s underwent NMP without oxygenated HMP after SCS.

27 significantly increased 5 min (35 min after SCS began) after 4 Hz SCS in the NRM, but not PAG in ani

28 sed polarities for samples PO-01, SCS-01 and SCS-02, indicating a primary record of the Earth's magne

29 fraction was noted in the SCS (52+/-5%) and SCS+MEDS (46+/-4%) groups compared with the MEDS (38+/-2

30 ere explored by means of B3LYP, B3LYP-D, and SCS-MP2 calculations.

31 DFT and SCS-ADC(2) calculations showed that contaminant-contamin

32 We have employed hybrid DFT and SCS-MP2 calculations at the SMD-PCM-6-311++G(2d,2p)//6-3

33 high level quantum chemical methods (DFT and SCS-MP2-calculations).

34 several DFT methods as well as with MP2 and SCS-MP2.

35 were carried out with M06-2X/6-311G(d,p) and SCS-MP2/6-311G(d,p)//M06-2X/6-311G(d,p) methods.

36 ding the neural circuits related to pain and SCS in the dorsal horn, supraspinal structures, and the

37 nts for cell movement between parenchyma and SCS in what we speculate is a program of immune surveill

38 for the lowly heritable traits DPR, PL, and SCS.

39 e authors' hypothesis that a single anterior SCS injection can reach the ocular posterior segment was

40 The anterior SCS of adult porcine and canine ex vivo eyes was cannula

41 ection site to the opposite ventral anterior SCS and the posterior SCS.

42 Calculations at SCS-MP2/CBS-level of theory and experimental data of the

43 Calculations at SCS-MP2/CBS//TPSS-D3/def2-SVP-level of theory provided p

44 a barrier of 22.7 kcal/mol (22.2 kcal/mol at SCS-MP2/cc-PVTZ).

45 ntly higher, 34.6 kcal/mol (41.9 kcal/mol at SCS-MP2/cc-PVTZ).

46 LY294002 at 100 microM also attenuated SCS-induced vasodilation at 60% and 90% of MT (P<0.05).

47 itochondrial extracts and purified bacterial SCS (BSCS), we showed that this enhanced 32P labeling re

48 racemase, which features a pyridinium-based SCS pincer ligand framework bound to nickel.

49 rther research into the interactions between SCS and pain pathways in the nervous system using animal

50 e both ATP and GTP fit into the Blastocystis SCS active site, GTP is destabilized by electrostatic di

51 ate the risk of SCS-related complications by SCS exposure and quantify the associated health care cos

52 To elucidate potential sites modulated by SCS we examined distribution of c-fos in Sprague-Dawley

53 ls that received healthy livers preserved by SCS (n=9) or NELP (n=11) for 6 hr.

54 d from heart-beating donors and preserved by SCS served as controls.

55 mechanisms underlying the pain reduction by SCS and how different frequencies of SCS produce the ana

56 Given that mtGTP synthesized by SCS-GTP is an indicator of TCA flux that is crucial for

57 Thus, we characterize SCS macrophages as specialized antigen-presenting cells

58 elationship was demonstrated between chronic SCS use and risk of SCS-related complications in patient

59 ent serious acute exacerbations, but chronic SCS use is associated with complications.

60 ses and had more than 6 months of continuous SCS use.

61 tent or continuous systemic corticosteroids (SCS) or high-dose ICS.

62 nce treatment with systemic corticosteroids (SCSs) to control daily symptoms and prevent serious acut

63 Defected SCS ADP-forming beta subunit (SCS A-beta) is linked to l

64 l lines established from CHL and RLN-derived SCSs remain cytokine-secretory.

65 ures that, taken together, help discriminate SCS from NMOSD.

66 In this review, we will discuss SCS technologies and applications, as well as translatio

67 A disrupted SCS macrophage barrier enables tEVs to enter the lymph n

68 rages of 1.36 mg/dL (HMP) versus 1.40 mg/dL (SCS).

69 Low-dose SCS TA was also effective in reducing inflammation; howe

70 Effective SCS-sparing strategies might reduce the burden associate

71 medium occupied 39.0% to 52.1% of the entire SCS.

72 and persistent enhancement >2 months favored SCS, and ringlike enhancement favored NMOSD (p < 0.05).

73 Median delay to diagnosis was longer for SCS than NMOSD (5 vs 1.5 months, p < 0.01).

74 points examined in this model, low frequency SCS likely activates supraspinal and spinal mechanisms t

75 one) versus those with low (reference group) SCS exposure.

76 was shown that P(i) binds the porcine heart SCS alpha-subunit (SCSalpha) in a noncovalent manner and

77 HF-SCS may provide a more physiological method of inspirato

78 terspace during spontaneous breathing and HF-SCS following C2 spinal section.

79 gher and rib cage movement greater during HF-SCS compared to spontaneous breathing, stimulus amplitud

80 f the external intercostal muscles during HF-SCS is similar to that occurring during spontaneous brea

81 Stimulus amplitude during HF-SCS was adjusted such that inspired volumes matched spon

82 eous breathing, stimulus amplitude during HF-SCS was adjusted such that rib cage movement matched (Pr

83 dorsoventral gradients of activity during HF-SCS were not significantly different compared to spontan

84 During HF-SCS, mean peak SMU firing frequency was highest in the 3

85 ce during either spontaneous breathing or HF-SCS.

86 , high frequency spinal cord stimulation (HF-SCS) results in more physiological activation of these m

87 tients' follow-up into low, medium, and high SCS exposure (</= 6, >6-12, and >12 mg/d, respectively).

88 Patients with medium and high SCS exposure had significantly higher risks of SCS-relat

89 Medium and high SCS exposure were also associated with significantly mor

90 eam spacing of 375 microm and larger hinders SCS advancement, thus providing abilities to engineer cl

91 100 Hz SCS had no effect on c-fos expression.

92 ed 5 min (35 min after SCS began) after 4 Hz SCS in the NRM, but not PAG in animals with nerve injury

93 In uninjured animals 4 Hz SCS increased c-fos expression at the electrode site and

94 d bilaterally 2 h after either 4 Hz or 60 Hz SCS in the spinal cord dorsal horn in the cervical enlar

95 Previously we showed both 4 Hz and 60 Hz SCS reduces hyperalgesia in an animal model of neuropath

96 These results identify SCS macrophages as crucial gatekeepers to the CNS that p

97 Our results identify SCS macrophages as primary mediators of NK cell activati

98 In SCS, the new index accounts for as high as 75% of the to

99 iologic IOP had no significant difference in SCS distension.

100 s unfavorable for typhoon intensification in SCS, which therefore serves as a natural buffer that shi

101 st that ERK and AKT pathways are involved in SCS-induced vasodilation.

102 ficient LNs VSV replicated preferentially in SCS macrophages but not in adjacent nerves.

103 e and well tolerated therapeutic strategy in SCS and other conditions of excitotoxicity or Ca(2+) ove

104 SCS at 500 Hz significantly increased SCS-induced vasodilation without influencing MT.

105 M1/CHARMM22 level) and high-level ab initio (SCS-MP2) QM/MM calculations to analyze the reactions, an

106 On the other hand, animals from ischemia/SCS control group all died within 12 hr postoperatively

107 [P < .0001] and 1.59 [P < .0001]) versus low SCS exposure.

108 ultiple, single, or zero complex spike (MCS, SCS, ZCS) cells.

109 The zebrafish mutant line, twister, models SCS in terms of a dominant mutation in the alpha subunit

110 blockade of ERK and AKT activation modulated SCS-induced vasodilation.

111 and optB88-vdW), wave function theory (MP2, SCS(MI)-MP2, MP2.5, MP2.X, and CCSD(T)), and empirical c

112 ls (n = 52) with a minimal form of SCS or no SCS.

113 However, on Dongsha Atoll, in the northern SCS, unusually weak winds created low-flow conditions th

114 Distinguishing FLS from NS/SCS is essential in assessing LSG biopsies, before deter

115 mpared to FLS with a focus score of <1 or NS/SCS, is strongly associated with the ocular and serologi

116 cific or sclerosing chronic sialadenitis (NS/SCS).

117 s also revealed a P(i)-induced activation of SCS activity by more than 2-fold when mitochondrial extr

118 ignificantly with frequent administration of SCS, and patients on such medications should undergo pre

119 The association of SCS with leukoaraiosis suggests that it is part of a mor

120 srupted organization reduces the capacity of SCS macrophages to retain and present antigen in a subse

121 approach, several application categories of SCS produced materials, such as for energy conversion an

122 Moreover, depletion of SCS macrophages using clodronate liposomes abolished NK

123 Further development of SCS injections for treatment of the ocular posterior seg

124 Furthermore, effects of SCS at 500 Hz are mediated via activation of TRPV1-conta

125 This study investigated effects of SCS at both a normal frequency (as used clinically) and

126 Cases with a severe form of SCS (n = 27) were randomly selected for comparison with

127 re found between those with a severe form of SCS and controls in rates of parkinsonism (19% vs 17%; o

128 hed controls (n = 52) with a minimal form of SCS or no SCS.

129 atures with the presence of a severe form of SCS.

130 lcium-dependent myopathy in certain forms of SCS.

131 tion by SCS and how different frequencies of SCS produce the analgesic effect are unclear.

132 , non-ischemic livers preserved with 6 hr of SCS or NELP and unpreserved ischemic livers were transpl

133 of warm ischemia and preserved with 5 hr of SCS or NELP, and transplanted into syngeneic recipients.

134 However, the impacts of SCS A-beta deficiency on mitochondria specifically in ne

135 derlying chronic pain and the interaction of SCS with these circuits may underlie this plateau in cli

136 by down-regulating the expression levels of SCS A-beta in cultured mouse neurons, we have found that

137 extent, validity, and historical meaning of SCS findings, as well as their influence and the influen

138 7)) was also used to elucidate mechanisms of SCS vasodilation at these higher frequencies.

139 this study suggest that 0.2 mg and 2.0 mg of SCS TA was as effective in reducing inflammation as 2.0

140 composition, structure, and nanostructure of SCS products, and routes to regulate the size and morpho

141 and inflammation disrupt the organization of SCS macrophages in a manner that involves the migration

142 rnished our knowledge on the pathogenesis of SCS A-beta deficiency-related mitochondrial diseases and

143 Given the negative emission potential of SCS and biochar and their potential advantages compared

144 However, the properties of SCS macrophages are poorly defined.

145 Removal of SCS macrophages did not compromise adaptive immune respo

146 We sought to evaluate the risk of SCS-related complications by SCS exposure and quantify t

147 s were used to estimate the adjusted risk of SCS-related complications for patients with medium and h

148 nstrated between chronic SCS use and risk of SCS-related complications in patients with severe asthma

149 S exposure had significantly higher risks of SCS-related complications, including infections and card

150 rial diseases and revealed the vital role of SCS A-beta in maintaining neuronal mitochondrial quality

151 edical records and MRI reports suggestive of SCS.

152 slowly decaying synaptic currents typical of SCS that transit to a much faster decay upon the appeara

153 ital-optimized spin-component scaled MP2 [OO-SCS-MP2]) methods.

154 onal models used to investigate and optimize SCS.

155 P) has been suggested as an improvement over SCS.

156 s were carried out with M06-2X/6-311+G(d,p), SCS-MP2/6-311+G(d,p)//M06-2X/6-311+G(d,p), and B97D/6-31

157 Lithospheric storage of OC in peripheral SCS basins potentially exceeded 4,000 Gt (equivalent to

158 ntrast arrived at the opposite and posterior SCS 7.8 (4.6) and 7.7 (4.6) seconds after injection, res

159 osite ventral anterior SCS and the posterior SCS.

160 In the relatively pristine SCS sediments, the most prevalent and abundant ARGs are

161 EDS (carvedilol plus ramipril 2.5 mg PO QD), SCS plus MEDS (concurrent therapy), or CTRL group.

162 anted with the electrode who did not receive SCS.

163 37) (2.37 mg/kg, i.v.) significantly reduced SCS-induced vasodilation at 500 Hz (P<0.05, n=6) as comp

164 f currents by salicylidine salycylhydrazide (SCS) indicate expression of the beta2 or beta3 subunit,

165 ional theory (DFT) and spin component scaled SCS-MP2 methods.

166 al currents (significant current scattering, SCS) could reliably discriminate between consciousness a

167 fat, and protein yield; somatic cell score (SCS); productive life (PL); and daughter pregnancy rate

168 the Zhongsha Islands in the South China Sea (SCS) deep basin during the period from August 2012 to Ja

169 re observed in the southern South China Sea (SCS) from 5 years of direct measurements.

170 ce temperature (SST) of the South China Sea (SCS) increased by 2 degrees C in response to the develop

171 The deep ocean bed of the South China Sea (SCS) is considered to be largely devoid of anthropogenic

172 in the Oligo-Miocene of the South China Sea (SCS) is evaluated herein.

173 River Estuary (PRE) and the South China Sea (SCS) were analyzed.

174 etected in the northwestern South China Sea (SCS) with its intensity, size, polarity and structure be

175 rough the Luzon Strait into South China Sea (SCS).

176 the Pacific Ocean (PO-01), South China Sea (SCS-01, SCS-02) and Indian Ocean (IO-01).

177 Single-cell sequencing (SCS) has emerged as a powerful new set of technologies f

178 Single-cell sequencing (SCS) is a powerful new tool for investigating evolution

179 treams, which support suspended cell sheets (SCS) of various sizes and curvatures.

180 Here we show SCS macrophage development depended on lymphotoxin-alpha

181 and that we called 'Sertoli Cell Signature' (SCS).

182 mphocytes that survey the subcapsular sinus (SCS) and associated macrophages for pathogen entry.

183 yer of macrophages at the subcapsular sinus (SCS) captures pathogens entering the lymph node, prevent

184 s and preferentially bind subcapsular sinus (SCS) CD169(+) macrophages in tumor-draining lymph nodes

185 h maintained a protective subcapsular sinus (SCS) macrophage phenotype within virus draining lymph no

186 ed with CD169(+)-positive subcapsular sinus (SCS) macrophages and collagen fibers.

187 Subcapsular sinus (SCS) macrophages capture antigens from lymph and present

188 s, relocalized toward the subcapsular sinus (SCS) near invaded macrophages, and engaged in prolonged

189 hages, which populate the subcapsular sinus (SCS) of LNs, are critical for the clearance of viruses f

190 structures with a single formal charge site (SCS), whereas ArgArg x H(+) has a salt-bridge (SB) struc

191 histidine-tryptophan-ketoglutarate solution (SCS), followed by kidney autotransplantation.

192 rces for the persistent TRWs in the southern SCS.

193 ward thickening of the suprachoroidal space (SCS) compared with monotherapy alone (13.4 mum vs 5.3 mu

194 roidal layer (SCL) and suprachoroidal space (SCS).

195 is unclear whether spinal cord stimulation (SCS) at higher frequencies induces further increases in

196 Spinal cord stimulation (SCS) is an alternative approach for treatment of neuropa

197 Spinal cord stimulation (SCS) is an established and cost-effective therapy for tr

198 Spinal cord stimulation (SCS) reduces the incidence of ventricular tachyarrhythmi

199 ve method--epidural spinal cord stimulation (SCS)--has been suggested as an alternative approach for

200 fiber (ZCS) input during visual stimulation; SCS cells fired complex spikes associated with learned s

201 We investigated whether static cold storage (SCS) can be safely replaced with a novel technique of pr

202 perfusion (NEVKP) with static cold storage (SCS) in a porcine model of DCD autotransplantation.

203 livers by conventional static cold storage (SCS) is associated with an unacceptable risk of primary

204 se preserved using only static cold storage (SCS).

205 compared with standard static cold storage (SCS).

206 of oxygenated HMP after static cold storage (SCS).

207 ith HMP were compared to static cold stored (SCS) transplant controls.

208 iatum is known as the Swiss cheese striatum (SCS); however, its clinical impact is unknown.

209 rom 15 cohorts of the Seven Countries Study (SCS), the first systematic study of diet, risk character

210 ieved using a structured chimeric substrate (SCS) that releases a downstream activator after cleavage

211 Defected SCS ADP-forming beta subunit (SCS A-beta) is linked to lethal infantile Leigh or leigh

212 from 18 CHL-derived single-cell suspensions (SCSs) compared to reactive lymph nodes (RLNs).

213 e ACh receptors cause slow-channel syndrome (SCS) and Escobar syndrome, two forms of congenital myast

214 Slow-channel syndrome (SCS) is an autosomal-dominant disease resulting from mut

215 ty and the slow-channel myasthenic syndrome (SCS), IP(3)R(1) knockdown eliminated NMJ Ca(2+) overload

216 the field of solution combustion synthesis (SCS) obtained during recent years.

217 Succinyl Coenzyme A synthetase (SCS) is a key mitochondrial enzyme.

218 Succinyl-CoA synthetase (SCS) is the only mitochondrial enzyme capable of ATP pro

219 ically performed by succinyl-CoA synthetase (SCS), has arisen in diverse bacterial groups, including

220 is of mtGTP made by succinyl-CoA synthetase (SCS-GTP) to an anaplerotic pathway producing phosphoenol

221 study investigated the effects of long-term SCS on ventricular function in a postinfarction canine h

222 on (average: 6.1 vs 7.4 days, P = .003) than SCS organs.

223 nd had systemic autoimmunity more often than SCS (p < 0.05).

224 bies virus transmitted by insect bites, that SCS macrophages perform a third vital function: they pre

225 concentration is increased, we conclude that SCS activation by P(i) binding represents another mitoch

226 Furthermore, we have found that SCS A-beta deficiency has detrimental influence on neuro

227 n cultured mouse neurons, we have found that SCS A-beta deficiency induces severe mitochondrial dysfu

228 Here, we show that SCS reproducibly alleviates motor deficits in a primate

229 These results suggest that SCS should be considered as an additional treatment opti

230 The SCS can be activated by various upstream DNAzymes, can b

231 The SCS findings included 30-fold cohort differences in rate

232 The SCS is characterized by a precise organization of bindin

233 The SCS was accessed using microneedles in a minimally invas

234 demonstrated a visible SCS at baseline, the SCS expanded significantly after suprachoroidal CLS-TA i

235 /6-31+G(d,p) structures and energies for the SCS and SB forms of these protonated dipeptides indicate

236 251 mug kg(-1)) and those collected from the SCS (12-83 mug kg(-1)).

237 f fluid and that it is possible to image the SCS with ultrasound contrast.

238 e 3 also were significantly decreased in the SCS (27+/-17 and 27%, respectively; P<0.03) and MED (58+

239 covery in ejection fraction was noted in the SCS (52+/-5%) and SCS+MEDS (46+/-4%) groups compared wit

240 g isotopic compositions were observed in the SCS (delta(202)Hg, from -2.82 to -2.10 per thousand; Del

241 n fraction was significantly improved in the SCS compared with the MED and CTRL groups (P<0.001 for b

242 In the mean, the DWBC in the SCS flows southwestward with core velocity of 2.0 cm/s a

243 RT-PCR of reperfusion biopsy samples in the SCS group showed high expression of inflammatory cytokin

244 also revealed ultrastructural damage in the SCS group that was not seen in the HMP group.

245 numbers of CD68 positive macrophages in the SCS group when compared to the HMP group.

246 In the SCS group, left ventricular ejection fraction was 65+/-5

247 The large positive Delta(199)Hg in the SCS indicated that a fraction of Hg has undergone Hg(2+)

248 nalysis in the PRE sediments, but not in the SCS sediments.

249 pe fractionation by natural processes in the SCS.

250 e and resistance mechanism than those in the SCS.

251 toxicity following injection of TA into the SCS in porcine eyes.

252 Migration into the SCS intrinsically requires S1pr1, whereas movement from

253 injection of 0.2 mg or 2.0 mg of TA into the SCS or IVT.

254 n of microbubble contrast injection into the SCS were assessed by 2D and 3D ultrasound.

255 is required for their accumulation near the SCS and for efficient IL17 induction.

256 eys, near the end of his long career, of the SCS design, conduct, and findings, with his discussion a

257 Mean (SD) distension of the SCS with PBS increased from 1.57 (0.48) mm after injecti

258 In the deep ocean bed of the SCS, source-related signatures of Hg isotopes may have b

259 ed to image the effect and distension of the SCS.

260 e to RVO, but may result in expansion of the SCS.

261 dently associated with the visibility of the SCS: disarrangement of plexiform layers, CRT, and multip

262 contrast was visible in 24.0%to 27.2% of the SCS; in 10 of 12 eyes, contrast reached the posterior po

263 ermore, quantum chemical calculations on the SCS-MP2 and DFT levels gave insights into the reaction m

264 These results suggest that the SCS can expand, in a dose-dependent manner, to accommoda

265 Thus, the SCS macrophage layer may act as a sensor or valve during

266 2, 28 surviving animals were assigned to the SCS (delivered at the T4/T5 spinal region for 2 hours 3

267 roducing plasmacytoid dendritic cells to the SCS and in addition were a major source of IFN-I themsel

268 hat stability of the SB form relative to the SCS form generally increases with increasing gas-phase b

269 Delivery of TA to the SCS using microneedles was simple, effective, and not as

270 e 1 survivors were equally randomized to the SCS, MEDS (carvedilol plus ramipril 2.5 mg PO QD), SCS p

271 Macrophages within the SCS of B cell-deficient LNs, or of mice that lack LTalph

272 ctions to form the organic component of the (SCS)Ni(II) pincer cofactor of LarA.

273 We report a theoretical (SCS-MP2//B3LYP) and experimental study of the uncatalyze

274 ed only with the highest level of QM theory (SCS-MP2) used.

275 anisms underlying motor improvements through SCS are unknown.

276 neutrophils and macrophages when compared to SCS controls.

277 rial DNA pol gamma and Twinkle contribute to SCS A-beta deficiency-mediated mtDNA instability.

278 ing enzyme (18%, p = 0.30) were exclusive to SCS.

279 SNI) and those without injury in response to SCS.

280 C) machine perfusion (NMP) after traditional SCS (0 degrees C-4 degrees C) for 7 to 9 hours.

281 wo hours of oxygenated HMP after traditional SCS restores hepatic ATP levels and improves hepatobilia

282 MP grafts were matched 1:3 with transplanted SCS livers.

283 tion, animals transplanted with NEVKP versus SCS grafts demonstrated similar serum creatinine peak le

284 In the 15 eyes that demonstrated a visible SCS at baseline, the SCS expanded significantly after su

285 EVKP-preserved kidneys (NEVKP, 39 +/- 6.4 vs SCS, 18 +/- 10.6 mL/min; P = 0.012).

286 reatinine peak levels (NEVKP, 2.0 +/- 0.5 vs SCS 2.7 +/- 0.7 mg/dL; P = 0.11) and creatinine clearanc

287 wer peak values (NEVKP, 5.5 +/- 1.7 mg/dL vs SCS, 11.1 +/- 2.1 mg/dL, P = 0.002).

288 ce on day 10 (NEVKP, 65.9 +/- 18.8 mL/min vs SCS 61.2 +/- 15.6 mL/min; P = 0.74).

289 All recipients in WI-SCS group died within 6 hours after transplantation.

290 preserved either by simple cold storage (WI-SCS group) or HMP (WI-HMP group) using Belzer-MPS soluti

291 ndrome was observed in all animals of the WI-SCS group but none of the control or WI-HMP groups.

292 ter-preserved liver histology relative to WI-SCS group.

293 atients evaluated between 1996 and 2015 with SCS or NMOSD whose first myelitis episode was accompanie

294 gies might reduce the burden associated with SCS-related complications in patients with severe asthma

295 e were less elevated after HMP compared with SCS preservation alone.

296 rved with HMP than for organs preserved with SCS (34.2% vs 42.0%, P < .001), despite a slightly longe

297 ar factors that bind to genomic regions with SCS could functionally interact with SOX9, we identified

298 P = 0.59), whereas animals transplanted with SCS grafts had persistently elevated serum creatinine an

299 Over the past 5 years, SCS methods for DNA and RNA have had a broad impact on m

300 In the coming years, SCS will greatly improve our understanding of invasion,