ライフサイエンスコーパス: SHRSP

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1 ol x min(-1) x [20 microl packed cells](-1), SHRSP vs. WKY, respectively, P = 0.01) and inhibition of

2 All SHRSP received dexamethasone (12 micro g/kg per d, subcu

3 derived from SHRSP(HD)/WKY-0(HD) (n=115) and SHRSP(HD)/WKY-1(HD) (n=139) crosses (WKY-0(HD) and WKY-1

4 apoptosis in neural tissues of both WKY and SHRSP rats.

5 were also evident in myoblast cultures from SHRSP compared with WKY cultures.

6 is were performed on F2 hybrids derived from SHRSP(HD)/WKY-0(HD) (n=115) and SHRSP(HD)/WKY-1(HD) (n=1

7 ions of insulin) in adipocytes isolated from SHRSP.

8 We show that skeletal muscle from SHRSP animals exhibits a marked decrease in insulin-stim

9 e markedly elevated in skeletal muscles from SHRSP compared with WKY animals.

10 The stroke-prone spontaneously hypertensive (SHRSP) rat is a model of human insulin resistance and is

11 In SHRSP rats, SEH inhibition reduced wall-to-lumen ratio a

12 ease in response to insulin: 1.4 +/- 0.15 in SHRSP, 2.29 +/- 0.22 in WKY; n = 4, P = 0.02), but the s

13 We measured BP and HR in SHRSP(HD) and normotensive Wistar-Kyoto rats (WKY), as w

14 tributes directly to the regulation of HR in SHRSP(HD) but exhibits no effect on BP.

15 HR in SHRSP(HD) was significantly higher than in WKY-0(HD) bot

16 ence and expression are apparently normal in SHRSP, it is likely that the molecular mechanism for def

17 cerebral ischemia via vascular protection in SHRSP rats and neural protection in both the SHRSP and W

18 ase and plasma levels of NEFA are similar in SHRSP and WKY.

19 map was constructed in two F2 intercrosses (SHRSP x BN and FHH x ACI), containing a total of 4736 si

20 ts for defects in insulin action in the male SHRSP rat model compared with the normotensive, insulin-

21 ide screen in an F2 cross obtained by mating SHRSP and SHR, in which latency to stroke on Japanese ra

22 pathogenesis of thrombotic microangiopathy, SHRSP were adrenalectomized and infused with vehicle, An

23 ive effect against stroke in the presence of SHRSP alleles and STR-2 co-localized with the genes enco

24 in spontaneously hypertensive stroke-prone (SHRSP) rats protects against cerebral ischemia induced b

25 stroke-prone spontaneously hypertensive rat (SHRSP(HD)), is a primary, genetically determined trait a

26 stroke-prone spontaneously hypertensive rat (SHRSP) as a model organism, mated it with the stroke-res

27 stroke-prone spontaneously hypertensive rat (SHRSP) is a genetically determined model of "salt-sensit

28 spontaneously hypertensive stroke-prone rat (SHRSP) is an experimental model of stroke characterized

29 roke-prone, spontaneously hypertensive rats (SHRSP) without controlling hypertension.

30 were also observed in the insulin-resistant SHRSP strain.

31 tty acid metabolism in the stroke-prone SHR (SHRSP).

32 SHRSP rats and neural protection in both the SHRSP and WKY rats, indicating that SEH inhibition has b

33 chromosome 3: in animals homozygous for the SHRSP(HD) allele, HR was 414+/-49 compared with 383+/-44

34 genome scan in an F2 cross derived from the SHRSP and the normotensive reference strain, WKY rat.

35 In the SHRSP fed a normal NaCl diet, supplementing dietary K+ w

36 responsible for large infarct volumes in the SHRSP in response to a focal ischaemic insult by perform

37 that the insulin resistance observed in the SHRSP is manifest at the level of skeletal muscle, that

38 The observations suggest that in the SHRSP selectively supplemented with Cl- the likelihood o

39 y of stroke the phenotypic expression of the SHRSP is (i) either increased or decreased, depending on

40 determines the phenotypic expression of the SHRSP.

41 sulin action on 2-deoxy-D-glucose transport (SHRSP 3.3 +/- 1.5 vs. 21.0 +/- 7.4 pmol x min(-1) x [20

42 g 120 F(2) rats generated from an SR/JrHsd x SHRSP intercross.

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