ライフサイエンスコーパス: Salmonella

1 and two gram negative pathogens (E. coli and Salmonella).

2 ling of the tagged amplicon from E. coli and Salmonella.

3 with host adaptation and systemic disease in Salmonella.

4 mples that have been achieved up to diagnose salmonella.

5 ween antimicrobial susceptible and resistant Salmonella.

6 n of zoonotic bacterial pathogens especially Salmonella.

7 d post-translational modification studies in Salmonella.

8 tion and promoting sustained colonization by Salmonella.

9 ion (LOD) down to 10CFUmL(-1) of E. coli and Salmonella.

10 ive technologies for real-time monitoring of salmonella.

11 umber in the infected cells, is derived from Salmonella 5'-leader of the ribosomal RNA transcript and

12 We now report that the Salmonella adaptor ClpS binds to the N terminus of the r

13 3% and 10 to 16% of mice with Klebsiella and Salmonella administrations, respectively.

14 PhoPQ-dependent Mg(2+) homeostasis protects Salmonella against nitrooxidative stress.

15 Traditional methodologies for salmonella analysis provide high reliability and very lo

16 f FlgK differs from that of its orthologs in Salmonella and Burkholderia, whose structures have previ

17 found in only 16 to 29% and 0% of mice with Salmonella and Klebsiella administrations, respectively.

18 ompete for the preferred nutrient sources of Salmonella and thus prevent or treat infection.

19 ntial pathogens (e.g. Clostridium difficile, Salmonella, and Escherichia coli) that do not produce si

20 an efficient carrier for nasal delivery of a Salmonella antigen that results in protection upon activ

21 hus, this is the first to demonstrate use of Salmonella aptamers for development of the colorimetric

22 Non-typhoidal Salmonella are associated with gastrointestinal disease

23 We found that wild-type Salmonella are capable of replicating within infected ho

24 stone genes from the entire GH complement of Salmonella are required to degrade glycans to change inf

25 patients infected with an invasive pathogen, Salmonella, are considered, based on recent studies in a

26 Here, we use the duplicate tuf genes in Salmonella as a quantitatively tractable model system fo

27 The host glycans were altered during Salmonella association via the induction of N-glycan bio

28 Salmonella bacteremia was found in only 16 to 29% and 0%

29 epleting cellular Sal-1 strongly renders the Salmonella bacteria less resistant to the host defenses

30 The reduction in the amount of fecal Salmonella bacteria with Lactobacillus treatment was dem

31 Salmonella behaved similarly.

32 gainst Salmonella colitis and showed reduced Salmonella burdens in viscera, suggesting that adenosine

33 n against systemic infections by E. coli and Salmonella by directly coating bacteria to promote killi

34 oduce this technique into the human pathogen Salmonella by incorporating p-azido-phenylalanine, benzo

35 abrogated the fitness advantage conferred on Salmonella by lactate utilization.

36 on of potentially pathogenic Escherichia and Salmonella by reducing the bioavailability of respirator

37 wo-component systems of Escherichia coli and Salmonella can also perceive an osmotic upshift, another

38 In the present study, we show that Salmonella can exploit mammalian cell non-classical micr

39 Salmonella can hijack host atypical miRNA processing mac

40 Anaerobic bacteria, such as Clostridium and Salmonella, can selectively invade and colonize in tumor

41 Approximately 97% lower Salmonella carriage is measured in a treated group, 14 d

42 Salmonella causes over a million foodborne illnesses per

43 espite the increased luminal colonization by Salmonella, CD73(f/f)Villin(Cre) mice were protected aga

44 is measured in a treated group, 14 days post-Salmonella challenge.

45 tigen, which mediated significant killing of Salmonella Choleraesuis and provided full protection aga

46 n 83% or 50% heterologous protection against Salmonella Choleraesuis challenge, respectively.

47 t expression of heterologous O-antigens from Salmonella Choleraesuis in Salmonella Typhimurium.

48 ombinant Asd(+) plasmid pCZ1 with the cloned Salmonella Choleraesuis O-antigen gene cluster was intro

49 IMS anti-Salmonella coated magnetic beads were applied to capture

50 (f/f)Villin(Cre) mice were protected against Salmonella colitis and showed reduced Salmonella burdens

51 Analysis of Salmonella colonization in IEC-specific CD73 knockout mi

52 ment inside infected host cells known as the Salmonella-containing vacuole.

53 -component regulatory system to exist inside Salmonella-containing vacuoles in the macrophage, as wel

54 Salmonella contains 47 glycosyl hydrolases (GHs) that ma

55 demonstrated that the 5' leader mRNA of the Salmonella corA gene can adopt two mutually exclusive co

56 approaches in electrochemical biosensors for Salmonella detection are presented and a critical analys

57 t challenges towards a complete solution for Salmonella detection in microbial food control based on

58 the commercially available rapid methods for Salmonella detection is provided along with a critical d

59 Available incidence data for invasive salmonella disease in sub-Saharan Africa are scarce.

60 it is a major cause of invasive nontyphoidal Salmonella disease, associated with high case fatality.

61 ed plasma membrane cholesterol, facilitating Salmonella docking and invasion.

62 to monitor recruitment of endogenous LC3C to Salmonella during xenophagy, as well as to mitochondria

63 By reconstituting the activities of the Salmonella effector SopE, we recapitulated Rho GTPase-dr

64 Ag(+) resistance was initially found on the Salmonella enetrica serovar Typhimurium multi-resistance

65 -propanediol utilization microcompartment of Salmonella enterica and use it to analyze the function o

66 Salmonella enterica are invasive intracellular pathogens

67 at viable Francisella tularensis, as well as Salmonella enterica bacteria transferred from infected c

68 Serovars of Salmonella enterica cause both gastrointestinal and syst

69 Recent papers have shown that the Salmonella enterica FinO-domain protein ProQ binds a lar

70 infection with Burkholderia pseudomallei and Salmonella enterica HMBA treatment was also associated w

71 tructures of GusRs from Escherichia coli and Salmonella enterica in complexes with a glucuronide liga

72 d in the EnvZ-OmpR two-component system from Salmonella enterica in vitro and in vivo, which directly

73 EutT from Salmonella enterica is a member of a class of enzymes te

74 Between March 1, 2010, and Jan 31, 2014, 135 Salmonella enterica serotype Typhi (S Typhi) and 94 iNTS

75 o develop the first human challenge model of Salmonella enterica serovar Paratyphi A infection.

76 ting protein, to influence susceptibility to Salmonella enterica serovar Typhi (S Typhi) infection.

77 Salmonella enterica serovar Typhi (S Typhi) is responsib

78 Salmonella enterica serovar Typhi causes the systemic di

79 Salmonella enterica serovar Typhi is a human-restricted

80 We report a typhoid fever case with a Salmonella enterica serovar Typhi isolate showing extend

81 Salmonella enterica serovar Typhi, the causative agent o

82 ly showed that l-asparaginase II produced by Salmonella enterica serovar Typhimurium (S Typhimurium)

83 BA would be more resistant to infection with Salmonella enterica serovar Typhimurium (S Typhimurium).

84 wo intracellular [Listeria monocytogenes and Salmonella enterica serovar Typhimurium (S.

85 Salmonella enterica serovar Typhimurium (S.

86 Salmonella enterica serovar Typhimurium can inject effec

87 Conversely, Salmonella enterica serovar Typhimurium causes gastroent

88 l compartments and a reduced ability to kill Salmonella enterica serovar Typhimurium compared to that

89 The ST313 pathovar of Salmonella enterica serovar Typhimurium contributes to a

90 We also demonstrate that the Salmonella enterica serovar Typhimurium core promoter is

91 toxified outer membrane vesicles (OMVs) from Salmonella enterica serovar Typhimurium displaying the v

92 Salmonella enterica serovar Typhimurium exploits the hos

93 We found that intracellular Salmonella enterica serovar Typhimurium induced the binu

94 n simultaneously in pathogen and host during Salmonella enterica serovar Typhimurium infection and re

95 alis, Escherichia coli K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococ

96 ed a metabolically competent, but avirulent, Salmonella enterica serovar Typhimurium mutant for its a

97 we present the structure of the prototypical Salmonella enterica serovar Typhimurium pathogenicity is

98 antigen from Mycobacterium tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.

99 sine harbors bacteriostatic activity against Salmonella enterica serovar Typhimurium that is not shar

100 egulatory system coordinates the response of Salmonella enterica serovar Typhimurium to diverse envir

101 he current study, we examined the ability of Salmonella enterica serovar Typhimurium to infect the ce

102 bound in vivo by the CspA family members of Salmonella enterica serovar Typhimurium to link the cons

103 Listeria monocytogenes V7 and Salmonella enterica serovar Typhimurium were used as mod

104 ic bacteria, either Klebsiella pneumoniae or Salmonella enterica serovar Typhimurium, enhanced transl

105 nterohemorrhagic Escherichia coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogat

106 create FLIM-phasor maps of Escherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aer

107 To examine individual functions, strains of Salmonella enterica serovar Typhimurium, the murine mode

108 that TcpB protein can efficiently attenuate Salmonella enterica serovar Typhimurium-induced pyroptos

109 t intracellular states of the model pathogen Salmonella enterica serovar Typhimurium.

110 helial oxygenation, and aerobic expansion of Salmonella enterica serovar Typhimurium.

111 en Vibrio cholerae and the invasive pathogen Salmonella enterica serovar Typhimurium.

112 hlortetracycline on the temporal dynamics of Salmonella enterica spp. enterica in feedlot cattle.

113 (EPEC), and Citrobacter rodentium Moreover, Salmonella enterica strains encode up to three NleB orth

114 S. bongori, S. enterica subsp. salamae, and Salmonella enterica subsp. arizonae The beta-lactamase T

115 aced this domain with a nuclease domain from Salmonella enterica subsp. arizonae This modified V. cho

116 Human infections with Salmonella enterica subspecies enterica serovar Senftenb

117 We report a traveler who acquired a Salmonella enterica subspecies enterica serovar Typhi st

118 Within host cells such as macrophages, Salmonella enterica translocates virulence (effector) pr

119 onlethal gastric infections of Gram-negative Salmonella enterica Typhimurium (ST), a major source of

120 in bacterial cells, particularly E. coli and Salmonella enterica Typhimurium.

121 bserved PPI, including Bacillus subtilis and Salmonella enterica which are predicted to have up to 18

122 rimers specific for E. coli eaeA (151bp) and Salmonella enterica yfiR (375bp) genes.

123 aMN):DMB phosphoribosyltransferases (CobT in Salmonella enterica), in a reaction that is considered t

124 Strains of Salmonella enterica, and other organisms lacking RidA, h

125 the foodborne pathogens E. coli O157:H7 and Salmonella enterica, in detail a nucleic acid lateral fl

126 ccus pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, Klebsiella pneumoniae, and Escheric

127 ted microorganisms such as Escherichia coli, Salmonella enterica, Listeria innocua, Mycobacterium par

128 Two major serovars of Salmonella enterica, Typhi and Typhimurium, have evolved

129 leic acid, a major lipid in E. coli Last, in Salmonella enterica, ubiK was required for proliferation

130 of the essential endoribonuclease RNase E in Salmonella enterica.

131 m impacted by the metabolic stress of 2AA in Salmonella enterica.

132 ilament among B. subtilis, P. aeruginosa and Salmonella enterica.

133 We report on a technology to reduce Salmonella enteritidis in poultry.

134 ons of people each year and among pathogens, Salmonella Enteritidis is most widely found bacteria cau

135 domonas aeruginosa, Pseudomonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escheric

136 ria monocytogenes, Staphylococcus aureus and Salmonella enteritidis.

137 The proportions of non-Salmonella Enterobacteriaceae with extended spectrum bet

138 Thus, Nramp1 is not essential for Salmonella entry into the CNS or neuroinflammation, but

139 There was no occurrence of Salmonella, Escherichia coli or psychotropic bacteria.

140 this study provides an explanation as to how Salmonella evades activation of autophagy mechanisms as

141 clusion, we demonstrate a novel strategy for Salmonella evading the host immune clearance, in which S

142 Taken together, these data indicate that Salmonella flagellin has unique adjuvant properties that

143 g of human TLR5 and a secreted derivative of Salmonella flagellin structurally analogous to a clinica

144 At a site early in the Salmonella flgM gene, the effects on translation of repl

145 ner to manipulate host cells into becoming a Salmonella-friendly zone.

146 the proportion of multi-drug resistant (MDR) Salmonella from day 4 through day 26, which was the last

147 results reveal a means whereby intracellular Salmonella gain access to the host cell cytosol from wit

148 trols iron export from vacuoles and inhibits Salmonella growth in macrophages.

149 reseeable future trends for onsite detecting salmonella have been summarized.

150 Salmonella have developed a sophisticated machinery to e

151 antimicrobials reduced overall prevalence of Salmonella; however, these treatments increased the prop

152 ated to determine the periodic prevalence of Salmonella in a population of dogs and cats in the Unite

153 The overall study prevalence of Salmonella in cats (3 of 542) was <1%.

154 s defective transepithelial translocation by Salmonella In conclusion, we define a novel antimicrobia

155 cipitation was developed for the analysis of Salmonella in dairy products.

156 xpression promotes intracellular survival of Salmonella in macrophages, and contributes to the resist

157 re virulent and less able to protect against Salmonella in some instances.

158 e deletion of cspC and cspE fully attenuates Salmonella in systemic mouse infection.

159 Additionally, growth of Salmonella in the presence of histone deacetylases inhib

160 helminth infection increased colonization by Salmonella independently of T regulatory or Th2 cells.

161 During Salmonella-induced gastroenteritis, mucosal inflammation

162 Within the gut, Salmonella-infected enterocytes are expelled into the lu

163 First, depletion of Sal-1 in Salmonella-infected epithelial cells significantly incre

164 stinct microbiota members prevent intestinal Salmonella infection by enhancing antibacterial IFNgamma

165 n of specific gut bacteria that protect from Salmonella infection by priming host IFN-gamma responses

166 espond to bacterial and helminth infections: Salmonella infection caused an increase in the abundance

167 e show that a chronic systemic non-typhoidal Salmonella infection in an immunocompromised human patie

168 We found that T cell clonotypes in a mouse Salmonella infection model span early activated CD4(+) T

169 regulate a maximal innate immune response to Salmonella infection, allowing a sustained inflammatory

170 ption of raw food as a major risk factor for Salmonella infection.

171 erform lactate fermentation, thus supporting Salmonella infection.

172 lular proliferation within host cells during Salmonella infections, although none have been found to

173 Salmonella infectious diseases spreading every day throu

174 To establish infections, Salmonella injects virulence effectors that hijack the h

175 of typhoid fever and invasive non-typhoidal salmonella (iNTS) disease in sub-Saharan Africa, and the

176 stic dissection revealed how VAC14 regulates Salmonella invasion and typhoid fever susceptibility and

177 Salmonella invasion protein A (SipA) is a dual-function

178 revealed a key role for myosin VI (MYO6) in Salmonella invasion.

179 his regulator is required for Pat to control Salmonella invasion.

180 Salmonella is an intracellular pathogen infecting a wide

181 l mechanism used by macrophages to eradicate Salmonella is production of reactive oxygen species.

182 he Escherichia coli genome, and reveals that Salmonella is remarkably amenable to genome-scale modifi

183 According to the recent statistics, Salmonella is still an important public health issue in

184 By screening 73 clinical and environmental Salmonella isolates, we identified EspJ homologues in S.

185 ite its potential importance in Escherichia, Salmonella, Klebsiella, Shigella, and Yersinia opportuni

186 entation, increasing both lactate levels and Salmonella lactate utilization.

187 und the protective efficacy of broad-ranging Salmonella lipopolysaccharide conjugate vaccines.

188 Salmonella more rapidly clear the ceca of birds administ

189 samples, and urinary mutagenicity using the Salmonella mutagenicity assay (Ames test).

190 Furthermore, by visualizing this machine in Salmonella mutants we obtained major insights into the m

191 , rotavirus (n = 15), sapovirus (n = 9), and Salmonella (n = 8).

192 ifferentially regulates the E. coli K-12 and Salmonella nrf operons.

193 s employed to control foodborne nontyphoidal Salmonella (NTS), infections have not declined in decade

194 How Salmonella obtains nutrients for growth within this intr

195 oactive compounds which inhibit migration of Salmonella on wet surfaces.

196 Risk estimates for Salmonella or EHEC-related AGI were most sensitive to th

197 of the protein LC3B following engulfment of Salmonella or treatment with autophagy-inducing rapamyci

198 6 (10%) positive cultures for Campylobacter, Salmonella, or Shigella entero-pathogens in traditional

199 nteric fever, caused by Salmonella Typhi and Salmonella Paratyphi A, is the leading cause of bacteria

200 d aptamers based biosensors for detection of salmonella pathogen.

201 are sufficient to protect C. elegans against Salmonella pathogenesis in a tol-1-dependent manner.

202 ch directly enhanced bacterial expression of Salmonella pathogenicity island 1 (SPI-1) genes and incr

203 his TRAF6 ubiquitination is triggered by the Salmonella pathogenicity island 1 (SPI-1) T3SS effectors

204 S. enterica subsp. salamae encodes the Salmonella pathogenicity island 1 (SPI-1), SPI-2, and th

205 Here, starting with the 35 kb Salmonella pathogenicity island 1 (SPI-1), we eliminated

206 polymorphonuclear leukocytes, SipA or other Salmonella pathogenicity island 1 effectors had no effec

207 d that the effect was dependent on an intact Salmonella pathogenicity island 2 (SPI-2) type 3 secreti

208 hagocytosis, a significant proportion of the Salmonella population forms non-growing persisters throu

209 Rural dogs were also more likely to be Salmonella positive than urban (P = 0.002) or suburban (

210 Of note is that almost half of the Salmonella-positive animals were clinically nondiarrheic

211 suggests an overall decline in prevalence of Salmonella-positive dogs and cats over the last decades

212 Salmonella-positive dogs were significantly more likely

213 brogate the cytotoxicity of RNS against phoQ Salmonella, presumably by limiting the formation of pero

214 On day 0, mean Salmonella prevalence was 75.0% and the vast majority of

215 ate count (APC) and Campylobacter as well as Salmonella prevalence.

216 evading the host immune clearance, in which Salmonella produce microRNA-like functional RNA fragment

217 etyl-lysine, and phosphoserine into selected Salmonella proteins including a microcompartment shell p

218 Here, we show that intracellular Salmonella recruit the host proteins LAMP-2A and Hsc73,

219 Preventing intracellular acidification of Salmonella renders it avirulent, suggesting that acid st

220 himurium yfgA mutant lost the characteristic Salmonella rod-shaped appearance, exhibited increased se

221 Salmonella serotype Reading isolates were extensively MD

222 S. Enteritidis is a further example of a Salmonella serotype that displays niche plasticity, with

223 Only six Salmonella serotypes were detected.

224 The mucosal inflammatory response induced by Salmonella serovar Typhimurium creates a favorable niche

225 To investigate the distribution of Salmonella serovars in MCL and their products, a total o

226 produced by both nontyphoidal and typhoidal Salmonella serovars.

227 biopsies from patients with IBS, addition of Salmonella significantly reduced levels of occludin; sub

228 te the simultaneous detection of E. coli and Salmonella sp. in hamburger sample using a multichannel

229 specificity was low ( approximately 60%) for Salmonella species.

230 mation about antigenic targets of protective Salmonella-specific T and B cells.

231 Salmonella spp accounted for 33% or more of all bacteria

232 ns of pathogenic bacteria (Escherichia coli, Salmonella spp and Vibrio cholerae), with 8 strains of e

233 le for community-acquired infections such as Salmonella spp, Campylobacter spp, N gonorrhoeae, and H

234 nterohemorrhagic Escherichia coli (EHEC), or Salmonella spp.

235 ons in P. ruminicola 23, whereas E. coli and Salmonella spp. responses to excess nitrogen involve onl

236 studies were conducted by testing Shigella, Salmonella spp., Salmonella typhimurium and Staphylococc

237 his article, we report that simply combining Salmonella SseB with flagellin substantially enhances pr

238 ikely to explain the additive phenotype of a Salmonella strain lacking both SseK1 and SseK3.

239 We report here that Salmonella strains with mutations of phoPQ are hypersens

240 22-nt RNA fragment, Sal-1, which facilitates Salmonella survival in the infected host.

241 Finally, Sal-1 facilitating Salmonella survival through suppressing iNOS induction w

242 The mechanism through which Sal-1 promotes Salmonella survival, however, remains unknown.

243 ntimutagenic activity than zerumbone against Salmonella tester strains.

244 much greater effect to inhibit Listeria and Salmonella than non-emulsion, aqueous formulations.

245 nt for its ability to compete with wild-type Salmonella The modified Nissle strain became more virule

246 tribution toward the intracellular growth of Salmonella The results reveal a means whereby intracellu

247 toxin-producing Escherichia coli (STEC) and Salmonella" The document says CDC and its public health

248 s response was sufficient to protect against Salmonella tissue invasion and involved a previously rep

249 an salmonellosis, yet the strategies used by Salmonella to colonize chickens are poorly understood.

250 Therefore, we assessed the ability of Salmonella to disseminate to the central nervous system

251 The increased susceptibility of phoQ Salmonella to RNS requires molecular O2 and coincides wi

252 he visualization and characterization of the Salmonella type III secretion machine in live bacteria b

253 ctor of the human-adapted bacterial pathogen Salmonella Typhi (6,7) , the cause of typhoid fever in h

254 Typhoid toxin, a unique virulence factor of Salmonella Typhi (the cause of typhoid fever), recapitul

255 Enteric fever, caused by Salmonella Typhi and Salmonella Paratyphi A, is the lead

256 Shigella sonnei and Salmonella Typhi cause significant morbidity and mortali

257 gene and did not belong to the dominant H58 Salmonella Typhi clade.

258 ampylobacter spp, Neisseria gonorrhoeae, and Salmonella typhi were included in the high-priority tier

259 B. subtilis, Enterococcus, P. aeruginosa and Salmonella typhi) to antibiotics such as ampicillin and

260 related AB5 toxin encoded by the broad-host Salmonella Typhimurium (15) .

261 In Salmonella typhimurium and Escherichia coli, the virulen

262 ta and IL-18 in response to NLRC4 activators Salmonella Typhimurium and flagellin, canonical or non-c

263 ducted by testing Shigella, Salmonella spp., Salmonella typhimurium and Staphylococcus aureus on E. c

264 tremely susceptible to systemic infection by Salmonella Typhimurium because of loss-of-function mutat

265 Salmonella Typhimurium causes a self-limiting gastroente

266 luster was introduced into three constructed Salmonella Typhimurium Deltaasd mutants: SLT11 (Deltarfb

267 Salmonella typhimurium infection is reported to activate

268 that severity of disease induced by enteric Salmonella Typhimurium infection is strongly modulated b

269 Here, we show that Salmonella Typhimurium infection was accompanied by dysb

270 During Salmonella Typhimurium infection, intestinal CX3CR1(+) c

271 sub-ng/muL standard DNA and 10(1) copies of Salmonella typhimurium InvA gene sequences (cloned in E.

272 We recode 200 kb of the Salmonella typhimurium LT2 genome through a process we t

273 Flagellin isolated from the Salmonella Typhimurium SJW1660 strain, which differs by

274 able co-culture of metabolically competitive Salmonella typhimurium strains in microfluidic devices.

275 ere, we show that human NAIP also senses the Salmonella Typhimurium T3SS inner rod protein PrgJ and t

276 luorescently labeled Escherichia coli HS and Salmonella typhimurium that passed through from the muco

277 nced cytokine expression during infection by Salmonella typhimurium This occurred in the first 3 d of

278 t a high-resolution in situ structure of the Salmonella Typhimurium type III secretion machine obtain

279 Roseburia intestinalis, Ruminococcus obeum, Salmonella typhimurium, and Clostridium difficile) to qu

280 Three bacterial pathogens (Escherichia coli, Salmonella typhimurium, and methicillin-resistant Staphy

281 ts, including latex beads, Escherichia coli, Salmonella typhimurium, and Mycobacterium tuberculosis i

282 domonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escherichia coli).

283 Here, we report that an intestinal pathogen, Salmonella Typhimurium, inhibits anorexia by manipulatin

284 minths induce IgG1, whereas Th1 Ags, such as Salmonella Typhimurium, predominantly induce IgG2a.

285 Here, by studying Salmonella Typhimurium, we show that the E3 ligase LUBAC

286 chnique was used to capture a food pathogen, Salmonella typhimurium, with starting concentrations as

287 an animal model of sepsis, we observed that Salmonella typhimurium-infected mice exhibited simultane

288 s O-antigens from Salmonella Choleraesuis in Salmonella Typhimurium.

289 macrophages with the intracellular pathogen Salmonella typhimurium.

290 lla pneumophila, Pseudomonas aeruginosa, and Salmonella typhimurium.

291 l specificity of Zur, ZntR, RcnR and FrmR in Salmonella Typhimurium.

292 mmunomagnetic separation (IMS) for detecting Salmonella typhimurium.

293 The use of recombinant attenuated Salmonella vaccine (RASV) strains is a promising strateg

294 The development of a subunit Salmonella vaccine has been hindered by the absence of d

295 strategy for the development of multivalent Salmonella vaccines.

296 Salmonella virulence depends in part on its pathogenicit

297 effector activities work together to promote Salmonella virulence.

298 Salmonella was isolated from faecal samples and antimicr

299 and 4 controls were mounted in chambers and Salmonella were added; we studied passage routes through

300 Using SIRCAS, we create a Salmonella with 1557 synonymous leucine codon replacemen