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1 childhood bacteremia, with a predominance of Salmonella enterica serovar Typhi.
2 e systemic infection caused by the bacterium Salmonella enterica serovar Typhi.
3 le to typhoid fever caused by infection with Salmonella enterica serovar Typhi.
4 ical isolate of the human-specific pathogen, Salmonella enterica serovar Typhi.
5 ified that target the Vi capsular antigen of Salmonella enterica serovar Typhi.
6 exhibit specificity for Vi capsule producing Salmonella enterica serovar Typhi.
7 nts likely explains the monophasic nature of Salmonella enterica serovar Typhi.
8 Y. pestis and the exclusively human pathogen Salmonella enterica serovar Typhi.
9 uncharacterized proteins encoded by genes of Salmonella enterica serovar Typhi and E. coli O157:H7 (i
10 of a chromosomal region designated SPI-10 in Salmonella enterica serovar Typhi and located adjacent t
11 man monocyte-derived macrophages (MDMs) with Salmonella enterica serovar Typhi and S. Typhimurium wil
12 a human-specific serovar that, together with Salmonella enterica serovar Typhi and Salmonella enteric
16 tion caused by ingestion of human-restricted Salmonella enterica serovars Typhi and Paratyphi predomi
18 p., Rickettsia spp., Salmonella enterica and Salmonella enterica serovar Typhi, and Yersinia pestis),
19 e used a protein microarray containing 2,724 Salmonella enterica serovar Typhi antigens (>63% of prot
22 n humans, including Mycobacterium leprae and Salmonella enterica serovar Typhi, but the function of p
23 ay an important role in host defense against Salmonella enterica serovar Typhi by several effector me
26 serovars, which cause self-limiting disease, Salmonella enterica serovar Typhi can infect only humans
28 e that the causative agent of typhoid fever, Salmonella enterica serovar Typhi, can partially subvert
31 ally encoded 34-kDa cytolysin A hemolysin of Salmonella enterica serovar Typhi (ClyA) as a novel expo
33 e applied the IVOM approach to the genome of Salmonella enterica serovar Typhi CT18, a well-studied p
34 li O157:H7 Sakai, Haemophilus influenzae Rd, Salmonella enterica serovar Typhi CT18, and two strains
35 fective in young infants, we used attenuated Salmonella enterica serovar Typhi CVD 908-htrA and Shige
36 d in mice immunized intranasally either with Salmonella enterica serovar Typhi CVD 908-htrA or Shigel
41 tic treatments are often ineffectual against Salmonella enterica serovar Typhi in carriers with galls
42 ent in the Indian subcontinent, with chronic Salmonella enterica serovar Typhi infection reported as
43 city, even though their role in clearance of Salmonella enterica serovar Typhi infections is poorly d
44 ptor for entry of Pseudomonas aeruginosa and Salmonella enterica serovar Typhi into epithelial cells.
53 The asymptomatic, chronic carrier state of Salmonella enterica serovar Typhi occurs in the bile-ric
54 lly and results from systemic infection with Salmonella enterica serovar Typhi or Paratyphi pathovars
56 ll-characterized structures: SPI-7, found in Salmonella enterica serovar Typhi; PAP1 or pKLC102, foun
57 PCR methodology was developed to identify Salmonella enterica serovars Typhi, Paratyphi A, and Par
58 ivalent to fever (39 degrees C-42 degrees C) Salmonella enterica serovars Typhi, Paratyphi A, and Sen
60 ting protein, to influence susceptibility to Salmonella enterica serovar Typhi (S Typhi) infection.
64 Here whole-genome sequence analysis of 1,832 Salmonella enterica serovar Typhi (S. Typhi) identifies
71 is exclusively encoded by the human-specific Salmonella enterica serovars Typhi (S. Typhi) and Paraty
74 roteins of the Vi capsular polysaccharide of Salmonella enterica serovar Typhi (serovar Typhi) are en
77 esent the 4.8-Mb complete genome sequence of Salmonella enterica serovar Typhi strain Ty2, a human-sp
79 ed into the licensed typhoid vaccine strain, Salmonella enterica serovar Typhi strain Ty21a, and was
80 esized that adequately engineered attenuated Salmonella enterica serovar Typhi strains can serve as m
81 investigated the ability of live attenuated Salmonella enterica serovar Typhi strains delivered to m
85 oteins expressed during human infection with Salmonella enterica serovar Typhi, the cause of typhoid
87 tor on gastrointestinal epithelial cells for Salmonella enterica serovar Typhi, the etiologic agent o
88 Salmonella enterica serovar Paratyphi A and Salmonella enterica serovar Typhi to induce protective i
89 lial receptor mediating the translocation of Salmonella enterica serovar Typhi to the gastric submuco
94 ive of this study was to enhance survival of Salmonella enterica serovar Typhi vaccine strains at pHs
96 ded-spectrum-beta-lactamase (ESBL)-producing Salmonella enterica serovar Typhi was identified, whole-
97 the serotyped salmonellae, 14% (21/152) were Salmonella enterica serovar Typhi, whereas 86% (131/152)
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