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1 the surface of the human commensal bacterium Streptococcus gordonii.
2 re supernatants of Staphylococcus aureus and Streptococcus gordonii.
3 the presence of antecedent organisms such as Streptococcus gordonii.
4 s on rates of hydrogen peroxide synthesis by Streptococcus gordonii.
5 by adhering to early plaque bacteria such as Streptococcus gordonii.
6 commensal species Streptococcus sanguinis or Streptococcus gordonii.
7 with the antecedent oral biofilm constituent Streptococcus gordonii.
8  attaches and forms biofilms on substrata of Streptococcus gordonii.
9 ggregation receptor polysaccharides (RPS) of Streptococcus gordonii 38 and Streptococcus oralis J22 w
10                              Type 2Gn RPS of Streptococcus gordonii 38 and type 2G RPS of Streptococc
11              The cell wall polysaccharide of Streptococcus gordonii 38 functions as a coaggregation r
12 tion of this polysaccharide in transformable Streptococcus gordonii 38.
13 dominant salivary enzyme in humans, binds to Streptococcus gordonii, a primary colonizer of the tooth
14 oped in this study assessed a multi-species (Streptococcus gordonii, Actinobacillus actinomycetemcomi
15                   In contrast, nonpathogenic Streptococcus gordonii adhered poorly to keratinocytes r
16 ed as Streptococcus oralis (RPS bearing) and Streptococcus gordonii (adhesin bearing).
17  an important determinant of colonization by Streptococcus gordonii, an oral commensal and opportunis
18 nvestigated its role in biofilm formation by Streptococcus gordonii, an organism that colonizes human
19 erence of early colonizing organisms such as Streptococcus gordonii and Actinomyces naeslundii to the
20 en paired with two other initial colonizers, Streptococcus gordonii and Actinomyces oris, as well as
21 Structural studies of the type-C PPases from Streptococcus gordonii and Bacillus subtilis reveal a ho
22                                              Streptococcus gordonii and other viridans streptococci (
23 nge between two bacterial species, commensal Streptococcus gordonii and pathogenic Streptococcus muta
24 NAc-sensitive coaggregations with strains of Streptococcus gordonii and S. sanguis.
25 cause a similar activity is also secreted by Streptococcus gordonii and Staphylococcus aureus.
26                                              Streptococcus gordonii and Streptococcus mutans avidly c
27                                              Streptococcus gordonii and Streptococcus sanguinis are p
28  the human oral flora (Streptococcus mutans, Streptococcus gordonii and Streptococcus sanguinis) to d
29        Studies revealed that S. oralis, like Streptococcus gordonii and Streptococcus sanguinis, bind
30 igher, respectively, in the S-ECC group, and Streptococcus gordonii and Streptococcus sanguinis, whic
31 dulated by coculture with the oral commensal Streptococcus gordonii and the opportunistic commensal F
32 ction in vivo, mice were first infected with Streptococcus gordonii and then challenged with P. gingi
33  C, showed an increased ability to adhere to Streptococcus gordonii and to invade primary cultures of
34                                              Streptococcus gordonii and Veillonella atypica, two earl
35                                              Streptococcus gordonii and Veillonella atypica, two earl
36 l was synthesized, and Streptococcus mutans, Streptococcus gordonii, and Streptococcus sanguinis were
37                    Oral streptococci such as Streptococcus gordonii are facultative anaerobes that in
38 enes (asp4 and asp5) essential for export in Streptococcus gordonii are missing in S. aureus.
39         Viridans streptococci, which include Streptococcus gordonii, are pioneer oral bacteria that i
40                   Oral streptococci, such as Streptococcus gordonii, are the predominant early coloni
41                                     By using Streptococcus gordonii as a model organism for streptoco
42                                        Using Streptococcus gordonii as a model, we now show the mecha
43                                           In Streptococcus gordonii, Asp2 is required for the transpo
44       In contrast, heterotypic P. gingivalis-Streptococcus gordonii biofilm formation was enhanced in
45 lpXP also enhanced heterotypic P. gingivalis-Streptococcus gordonii biofilm formation.
46                                    Layers of Streptococcus gordonii Blackburn or Streptococcus oralis
47                           Strains used were: Streptococcus gordonii Blackburn, 10558, Streptococcus m
48 velops biofilm microcolonies on substrata of Streptococcus gordonii but not on Streptococcus mutans.
49 of PMNs or HL-60 cells abolished adhesion of Streptococcus gordonii but was required for adhesion of
50 pathogen Porphyromonas gingivalis adheres to Streptococcus gordonii by interacting with a specific re
51                                              Streptococcus gordonii can mediate its platelet attachme
52                                          The Streptococcus gordonii cell surface glycoprotein GspB me
53                       The alpha-hemolysin of Streptococcus gordonii CH1 caused characteristic shifts
54                                   Planktonic Streptococcus gordonii CH1 killed HUVEC over a 5-h perio
55 agenesis identified a new competence gene in Streptococcus gordonii Challis designated comYA.
56  a 585-bp gene was cloned and sequenced from Streptococcus gordonii Challis encoding a 20.5-kDa amyla
57                                          The Streptococcus gordonii (Challis) glucosyltransferase-enc
58                                              Streptococcus gordonii colonizes multiple sites within t
59 tococcus pyogenes) exposed on the surface of Streptococcus gordonii commensal bacterial vectors: (i)
60 produced by the glucosyltransferase (GTF) of Streptococcus gordonii confer a hard, cohesive phenotype
61 tion with P. gingivalis, whereas noninvasive Streptococcus gordonii did not have a significant effect
62 acid-containing receptors is associated with Streptococcus gordonii DL1 (Challis) but not with a spon
63 dentified by transposon Tn916 mutagenesis of Streptococcus gordonii DL1 (Challis).
64 inactivate a coaggregation-relevant locus of Streptococcus gordonii DL1 (Challis).
65                  The accessory Sec system in Streptococcus gordonii DL1 is a specialized export syste
66 ageneric coaggregation, streptococci such as Streptococcus gordonii DL1 recognize receptor polysaccha
67 ne cluster and used to transform E. coli and Streptococcus gordonii DL1, a nonureolytic, dental plaqu
68                                           In Streptococcus gordonii DL1, inactivation of the ccpA gen
69 ating early colonizers of the tooth surface (Streptococcus gordonii DL1, Streptococcus oralis 34, and
70 he scaCBA operon in the human oral bacterium Streptococcus gordonii encodes the components of an ABC-
71                                              Streptococcus gordonii expresses two related adhesins, S
72 pV, a dipeptidase found in culture fluids of Streptococcus gordonii FSS2, was purified and characteri
73  polypeptide (259 kDa) in the oral bacterium Streptococcus gordonii, generates mutants that are marke
74                                              Streptococcus gordonii genes involved in beta-glucoside
75                  A DNA microarray identified Streptococcus gordonii genes regulated in response to co
76                                          The Streptococcus gordonii glucosyltransferase gene, gtfG, i
77 biofilm formation by Streptococcus mutans or Streptococcus gordonii grown in human plasma.
78  parasanguinis, Streptococcus sanguinis, and Streptococcus gordonii, inhibit the growth of P. aerugin
79             Pioneer oral bacteria, including Streptococcus gordonii, initiate the formation of oral b
80 omonas gingivalis and the accessory pathogen Streptococcus gordonii interact to form communities in v
81                 The oral commensal bacterium Streptococcus gordonii interacts with salivary amylase v
82 ed by early dental plaque colonizers such as Streptococcus gordonii interfere with the subsequent col
83                                              Streptococcus gordonii is a frequent cause of infective
84 eptococcal coaggregation regulator (ScaR) of Streptococcus gordonii is a manganese-dependent transcri
85                          ScaA lipoprotein in Streptococcus gordonii is a member of the LraI family of
86                                              Streptococcus gordonii is a pioneer colonizer of the tee
87                                              Streptococcus gordonii is a primary colonizer of the mul
88                                              Streptococcus gordonii is a primary etiological agent in
89                                              Streptococcus gordonii is an oral commensal and an early
90                  The accessory Sec system of Streptococcus gordonii is comprised of SecY2, SecA2, and
91                  The accessory Sec system of Streptococcus gordonii is essential for transport of the
92                                              Streptococcus gordonii is generally considered a benign
93                          Platelet binding by Streptococcus gordonii is mediated in large part by GspB
94                                              Streptococcus gordonii is shown to express, on the cell
95 ort, we show that the A regions from the two Streptococcus gordonii M5 antigen I/II proteins (SspA an
96 large cell-surface glycoprotein expressed by Streptococcus gordonii M99 that mediates binding of this
97 he cytosolic O-glycosyltransferase GtfA/B of Streptococcus gordonii modifies the Ser/Thr-rich repeats
98                           The oral commensal Streptococcus gordonii must adapt to constantly fluctuat
99 ) was started 2 days before inoculation with Streptococcus gordonii or Staphylococcus aureus.
100 ed with PAAP(+) S. sanguis than with PAAP(-) Streptococcus gordonii or type II collagen, suggesting a
101                   Actinomyces naeslundii and Streptococcus gordonii, oral bacteria that possess Gal/G
102  p38 and then stimulated with oral commensal Streptococcus gordonii, oral pathogens Porphyromonas gin
103 r example, more Streptococcus sanguinis than Streptococcus gordonii organisms are consistently isolat
104                The ADS of the oral bacterium Streptococcus gordonii plays major roles in physiologic
105  oral commensals Streptococcus sanguinis and Streptococcus gordonii release DNA in a process induced
106 ory region with high homology to that of the Streptococcus gordonii ScaR binding domain.
107 nalling between Porphyromonas gingivalis and Streptococcus gordonii serves to constrain development o
108 ion of hydrogen peroxide in solution above a Streptococcus gordonii (Sg) bacterial biofilm was studie
109  could detect 65 +/- 10 muM H2O2 produced by Streptococcus gordonii (Sg) in a simulated biofilm at 50
110 es, including commensal streptococci such as Streptococcus gordonii (Sg).
111                                              Streptococcus gordonii (Sg)/S. oralis (So)/S. sanguinis
112                                              Streptococcus gordonii shows promise as a live mucosal v
113 uence motif within the C-terminal portion of Streptococcus gordonii SspB (AgI/II) is bound by Porphyr
114 tion between Streptococcus mutans AgI/II and Streptococcus gordonii SspB in their interaction with th
115 icroorganisms, e.g., Veillonella parvula and Streptococcus gordonii, stimulated higher levels of ROS
116                                     However, Streptococcus gordonii strain M99 encodes SecA and SecY
117                    The gspB-secY2A2 locus of Streptococcus gordonii strain M99 encodes the platelet-b
118                          Platelet binding by Streptococcus gordonii strain M99 is dependent on expres
119                          Platelet binding by Streptococcus gordonii strain M99 is mediated predominan
120                          Platelet binding by Streptococcus gordonii strain M99 is predominantly media
121                          Platelet binding by Streptococcus gordonii strain M99 is strongly correlated
122 ologous serine-rich surface glycoproteins of Streptococcus gordonii strains M99 and Challis, respecti
123 nces in virulence among seven representative Streptococcus gordonii strains were observed by using th
124 infantis group, Corynebacterium matruchotii, Streptococcus gordonii, Streptococcus cristatus, Capnocy
125  (Streptococcus mitis, Streptococcus oralis, Streptococcus gordonii, Streptococcus cristatus, Strepto
126 t AgI/II family polypeptides from strains of Streptococcus gordonii, Streptococcus intermedius and St
127 omologous enzymes in Streptococcus pyogenes, Streptococcus gordonii, Streptococcus mutans, Staphyloco
128 phylococcus aureus, Streptococcus sanguinis, Streptococcus gordonii, Streptococcus oralis, and Strept
129 nt understanding of accessory Sec systems in Streptococcus gordonii, Streptococcus parasanguinis, Myc
130 racterized and closely related oral species, Streptococcus gordonii, Streptococcus sanguinis, and car
131 lizes lactate produced by the oral bacterium Streptococcus gordonii, suggesting the potential for cro
132  Hsa are homologous surface glycoproteins of Streptococcus gordonii that bind sialic acid moieties on
133  (30-day) colonization of smooth surfaces by Streptococcus gordonii that incorporates the nutrient fl
134 spB is a serine-rich glycoprotein adhesin of Streptococcus gordonii that is exported to the bacterial
135                         In the gram-positive Streptococcus gordonii, the ability to form disulfide bo
136                                           In Streptococcus gordonii, the SRR glycoprotein GspB has a
137 ies have shown that P. gingivalis adheres to Streptococcus gordonii through interaction of the minor
138 SspB (antigen I/II family proteins) can bind Streptococcus gordonii to other oral bacteria and also t
139                                Attachment of Streptococcus gordonii to the acquired pellicle of the t
140 the attachment of pioneer organisms, such as Streptococcus gordonii, to tooth surfaces.
141                                           In Streptococcus gordonii, transport of the serine-rich gly
142 erobes, including aerotolerant ones, such as Streptococcus gordonii, use pyruvate dehydrogenase to de
143 heterologous bacteria Lactococcus lactis and Streptococcus gordonii was demonstrated.
144      The amylase-binding protein A (AbpA) of Streptococcus gordonii was found to be undetectable in s
145 tA gene in the human oral commensal organism Streptococcus gordonii was insertionally inactivated.
146 tococcus oralis, Streptococcus sanguinis, or Streptococcus gordonii was investigated using flow cell
147 gnated atlS, encoding a major autolysin from Streptococcus gordonii, was identified and characterized
148   To investigate disulfide bond formation in Streptococcus gordonii, we identified five putative TDOR
149 n homologous to the platelet adhesin GspB in Streptococcus gordonii, were identified.
150                                              Streptococcus gordonii Wicky becomes competent only afte

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