ライフサイエンスコーパス: Streptococcus mutans

1 events it from forming protein aggregates in Streptococcus mutans.

2 mensal Streptococcus gordonii and pathogenic Streptococcus mutans.

3 conditions to inhibit competing species like Streptococcus mutans.

4 were assayed by 74 DNA probes and by PCR to Streptococcus mutans.

5 commensal S. gordonii and the oral pathogen Streptococcus mutans.

6 ression of important virulence attributes of Streptococcus mutans.

7 r the production of the alarmone (p)ppGpp in Streptococcus mutans.

8 e of repressing mutacin I gene expression in Streptococcus mutans.

9 y Actinobacillus actinomycetemcomitans), and Streptococcus mutans.

10 nii, Streptococcus sanguinis, and cariogenic Streptococcus mutans.

11 d functional maturation of the adhesin P1 of Streptococcus mutans.

12 in Gram-positive bacteria does not apply to Streptococcus mutans.

13 bstrata of Streptococcus gordonii but not on Streptococcus mutans.

14 siologic functions and virulence factors for Streptococcus mutans.

15 peratively on the expression of virulence of Streptococcus mutans.

16 ce development for genetic transformation in Streptococcus mutans.

17 sensing is involved in biofilm formation of Streptococcus mutans.

18 ding for a PDE promoted biofilm formation in Streptococcus mutans.

19 eptococcal species Streptococcus sanguis and Streptococcus mutans.

20 d is produced by the Gram-positive bacterium Streptococcus mutans.

21 strong antigenic relationship with GBPs from Streptococcus mutans.

22 rmus thermophilus, Synechocystis PCC6803 and Streptococcus mutans.

23 is closely associated with the virulence of Streptococcus mutans.

24 e, mediated by the cariogenic oral bacterium Streptococcus mutans.

25 t of genetic competence in the oral pathogen Streptococcus mutans.

26 ence factors of the cavity-causing bacterium Streptococcus mutans.

27 P1 is an adhesin on the surface of Streptococcus mutans.

28 l for biofilm formation by the oral pathogen Streptococcus mutans.

29 Composites were inoculated with Streptococcus mutans.

30 ains), Streptococcus oralis (4 strains), and Streptococcus mutans (1 strain).

31 dherence and glucan-induced agglutination of Streptococcus mutans 3209 and S. sobrinus B13.

32 ells versus 8.0 x 10(5) cells; P <0.05), and Streptococcus mutans (6.2 x 10(5) cells versus 2.0 x 10(

33 significant antibiofilm bioactivity against Streptococcus mutans, a causative agent of human dental

34 timated timing of a demographic expansion in Streptococcus mutans, a causative agent of human dental

35 Streptococcus mutans, a dental caries pathogen, also cau

36 Streptococcus mutans, a dental pathogen, secretes differ

37 modulation of CtsR activity is different in Streptococcus mutans, a dental pathogen.

38 Streptococcus mutans, a gram-positive pathogen considere

39 Streptococcus mutans, a major etiological agent of human

40 Streptococcus mutans, a major oral pathogen responsible

41 There are suggestions that the phylogeny of Streptococcus mutans, a member of the human indigenous b

42 Streptococcus mutans, a member of the human oral flora,

43 prevalent infectious disease associated with Streptococcus mutans, a pathogen also linked to endocard

44 Streptococcus mutans, a primary agent of dental caries,

45 Streptococcus mutans, a principal causative agent of den

46 Streptococcus mutans, a principal causative agent of den

47 at inhibits the glucosyltransferase (GTF) of Streptococcus mutans, a virulence enzyme involved in ora

48 Deletion of comR or comS in Streptococcus mutans abolished transformability, as did

49 inked properties of oral pathogens including Streptococcus mutans, Actinomyces naeslundii and Prevote

50 ecies-specific adherence/aggregation between Streptococcus mutans AgI/II and Streptococcus gordonii S

51 e with the surface adhesin protein AgI/II of Streptococcus mutans alone or in combination with LT-IIa

52 e with the surface adhesin protein AgI/II of Streptococcus mutans alone or in combination with LT-IIa

53 y by gastric intubation or intranasally with Streptococcus mutans alone or S. mutans complexed with a

54 e with the surface protein adhesin AgI/II of Streptococcus mutans alone or supplemented with an adjuv

55 ion rich in glucosyltransferase (C-GTF) from Streptococcus mutans, alone or in liposomes.

56 Streptococcus mutans, along with many other gram-positiv

57 Since Streptococcus mutans also releases Gtf enzymes that coul

58 the LraI family gene (designated sloC) from Streptococcus mutans, an agent of dental caries and endo

59 ar mass component of human saliva that binds Streptococcus mutans, an oral bacterium implicated in de

60 showed a significant microbicidal effect for Streptococcus mutans and an unencapsulated strain of Por

61 ractions of the response regulator ComE from Streptococcus mutans and DNA binding sites through DNase

62 ocalized on the surface of the oral pathogen Streptococcus mutans and facilitates an interaction with

63 independent of total bacteria and levels of Streptococcus mutans and Lactobacilli.

64 tro wear, and antibacterial activity against Streptococcus mutans and Lactobacillus casei (in both pl

65 firmed the association of the acid producers Streptococcus mutans and Lactobacillus spp. with childho

66 e complexes (IC) of the cariogenic bacterium Streptococcus mutans and mAbs against its surface adhesi

67 the oral cavity with the cariogenic pathogen Streptococcus mutans and other bacteria.

68 evaluated the ability of the oral pathogens Streptococcus mutans and Porphyromonas endodontalis to i

69 mechanisms of AgD from H. pylori as well as Streptococcus mutans and Porphyromonas gingivalis.

70 against the oral specific bacterial species, Streptococcus mutans and Porphyromonas gingivalis.

71 us actinomycetemcomitans) and dental caries (Streptococcus mutans and S. sobrinus).

72 t defect observed for orthologous mutants of Streptococcus mutans and Streptococcus agalactiae.

73 hen tested against the gram-positive species Streptococcus mutans and Streptococcus mitis, however, l

74 led a high degree of synteny with operons in Streptococcus mutans and Streptococcus pneumoniae.

75 Oral bacteria, including Streptococcus mutans and Streptococcus salivarius, contr

76 s study, we used two oral bacterial species, Streptococcus mutans and Streptococcus sanguinis (former

77 In contrast, Streptococcus mutans and Streptococcus sanguinis, caries

78 n reaction (PCR)/quantitative PCR (qPCR) for Streptococcus mutans and Streptococcus sobrinus.

79 Streptococcus mutans and sucrose are key modulators asso

80 zing glucosyltransferase enzyme (GTF-I) from Streptococcus mutans and thioredoxin from Escherichia co

81 oxidase is involved in both competition with Streptococcus mutans and virulence for infective endocar

82 , adjusted for age, sex, and the presence of Streptococcus mutans) and SM surfaces (1,004 participant

83 terococcus faecalis, Actinomyces naeslundii, Streptococcus mutans, and Aggregatibacter actinomycetemc

84 cterium nucleatum, Porphyromonas gingivalis, Streptococcus mutans, and Campylobacter rectus are also

85 , including oral streptococci, lactobacilli, Streptococcus mutans, and Candida, in saliva than did HI

86 rial load was calculated for total bacteria, Streptococcus mutans, and Lactobacilli.

87 ive group were Actinomyces sp. strain B19SC, Streptococcus mutans, and Lactobacillus spp., which exhi

88 treptococcus oralis, the late oral colonizer Streptococcus mutans, and the pioneer enteric bacterium

89 s including those of Salmonella typhimurium, Streptococcus mutans, and Thermus aquaticus encode a ded

90 The Streptococcus mutans antigen I/II (AgI/II) is a cell sur

91 Streptococcus mutans antigen I/II (AgI/II) is a cell sur

92 Streptococcus mutans antigen I/II (AgI/II) protein was o

93 ssing the saliva-binding region (SBR) of the Streptococcus mutans antigen I/II adhesin, either alone

94 encoding the saliva-binding region (SBR) of Streptococcus mutans antigen I/II adhesin, either alone

95 cidal effect was observed against cariogenic Streptococcus mutans at pH 7.4, even when using NO-relea

96 ppear to synthesize it and others, including Streptococcus mutans ATCC 33402, import it from their gr

97 The Streptococcus mutans atlA gene encodes an autolysin requ

98 Streptococcus gordonii and Streptococcus mutans avidly colonize teeth.

99 Streptococcus mutans belongs to the viridans group of or

100 oups (N-G, N-H, A-G and A-H) were exposed to Streptococcus mutans biofilm for 4, 8, 15, 20 or 25 days

101 Streptococcus mutans biofilm was disrupted from sandblas

102 o mediate photodynamic therapy (PDT) against Streptococcus mutans biofilms.

103 In Streptococcus mutans, both competence and bacteriocin pr

104 lood mononuclear cells (PBMC) after engaging Streptococcus mutans, but monocytes in developing endoca

105 Carbohydrate catabolite repression (CCR) in Streptococcus mutans can be independent of catabolite co

106 Streptococcus mutans can produce exopolysaccharides (EPS

107 Actinomyces israelii, Streptococcus sanguis, Streptococcus mutans, Candida tropicalis, Candida paraps

108 Addition of tears to late-exponential-phase Streptococcus mutans cells resulted in time- and dose-de

109 saccharides (EPS) by a cariogenic bacterium, Streptococcus mutans (Cheng et al., 2016).

110 The Streptococcus mutans Cid/Lrg system represents an ideal

111 that the underlying mechanisms by which the Streptococcus mutans ClpXP protease affects virulence tr

112 ously that mucosal immunization of mice with Streptococcus mutans coated with the monoclonal antibody

113 In subjects positive for Streptococcus mutans, collagen binding activity was posi

114 that examines whether HIV infection affects Streptococcus mutans colonization in the oral cavity.

115 ns, all Caucasians had significantly greater Streptococcus mutans colonization, but only Db-negative

116 The ciaRH operon in Streptococcus mutans contains 3 contiguous genes, ciaXRH

117 en salivary agglutinin and the adhesin P1 of Streptococcus mutans contribute to bacterial aggregation

118 with a bacterial protein antigen (AgI/II of Streptococcus mutans) coupled to the B subunit of choler

119 d with 50 to 100 microg of free or liposomal Streptococcus mutans crude glucosyltransferase (C-GTF) w

120 In this study, we found that in Streptococcus mutans CSP induced co-ordinated expression

121 eractions between sucrose- (and starch-) and Streptococcus mutans-derived exoenzymes present in the p

122 Streptococcus mutans develops competence for genetic tra

123 mRNA (irvA) from the dental caries pathogen Streptococcus mutans directly modulates target mRNA (gbp

124 Streptococcus mutans displays complex regulation of gene

125 parable genotyping results for investigating Streptococcus mutans diversity and transmission.

126 An insertionally inactivated fabM strain of Streptococcus mutans does not produce unsaturated membra

127 Streptococcus mutans employs a key virulence factor, thr

128 The oral pathogen Streptococcus mutans employs a variety of mechanisms to

129 The cariogenic bacterium Streptococcus mutans employs so-called competence-stimul

130 Streptococcus mutans employs the secreted peptides CSP a

131 The fruA gene of Streptococcus mutans encodes for a secreted fructan hydr

132 s in humans, and the primary caries pathogen Streptococcus mutans encodes multiple enzymes involved i

133 In a Streptococcus mutans endocarditis rat model, we identifi

134 ce TLR2 was associated with poor response to Streptococcus mutans, Enterococcus faecalis, and Lactoba

135 In Streptococcus mutans, enzyme II(scr) and sucrose-6-phosp

136 (for Streptococcus GTP-binding protein) is a Streptococcus mutans essential GTPase which has signific

137 We previously demonstrated that Streptococcus mutans expresses a functional agmatine dei

138 Streptococcus mutans F-ATPase, the major component of th

139 frame (ORF) encoding a protein homologous to Streptococcus mutans FruK.

140 Active immunization with Streptococcus mutans glucan binding protein B (GBP-B) ha

141 Of these, Streptococcus mutans glucan binding protein B (GbpB) is

142 Streptococcus mutans glucan-binding protein A (GbpA) has

143 ted (beta,alpha)8 catalytic barrel domain of Streptococcus mutans glucosyltransferase (GTF).

144 which are derived from functional domains of Streptococcus mutans glucosyltransferases (GTF) have bee

145 Streptococcus mutans glucosyltransferases form extracell

146 bstrates for the product of the fruA gene of Streptococcus mutans GS-5.

147 Recently, Streptococcus mutans has been found with high frequency

148 inding region (SBR) of the adhesin AgI/II of Streptococcus mutans has been shown to induce a mixed Th

149 Glucan-binding protein B (GbpB) from Streptococcus mutans has been shown to induce protective

150 The adhesin P1 of Streptococcus mutans has been studied as an anticaries v

151 Streptococcus mutans has two YidC paralogs.

152 The Streptococcus mutans hdrRM operon encodes a novel two-ge

153 y partially characterized the dnaK operon of Streptococcus mutans (hrcA-grpE-dnaK) and demonstrated t

154 We found that inactivation of Streptococcus mutans htrA by gene-replacement also resul

155 Streptococcus mutans in dental plaque biofilms play a ro

156 ndida albicans cells are often detected with Streptococcus mutans in plaque biofilms from children af

157 frequently detected with heavy infection of Streptococcus mutans in plaque-biofilms from children af

158 In this study, the role of luxS of Streptococcus mutans in the regulation of traits crucial

159 efore investigated the roles of cnm-positive Streptococcus mutans in this single hospital-based, obse

160 or age, education group, and the presence of Streptococcus mutans) in self-reported whites (ages 14 t

161 Inactivation of the gbpA gene of Streptococcus mutans increases virulence in a gnotobioti

162 Escherichia coli and Streptococcus mutans induced lesser effects.

163 e polymers which contribute significantly to Streptococcus mutans-induced cariogenesis.

164 expression of the fructanase gene (fruA) of Streptococcus mutans: induction by levan, inulin, or suc

165 Strategies aimed at the prevention of Streptococcus mutans infection and dental caries include

166 Streptococcus mutans is a biofilm-forming bacterium that

167 Streptococcus mutans is a biofilm-forming oral pathogen

168 Streptococcus mutans is a cariogenic oral pathogen whose

169 Streptococcus mutans is a commensal member of the health

170 Streptococcus mutans is a facultative member of the oral

171 The biofilm-forming Streptococcus mutans is a gram-positive bacterium that r

172 Streptococcus mutans is a key contributor to the formati

173 Streptococcus mutans is a major etiologic agent of denta

174 Streptococcus mutans is a primary pathogen for dental ca

175 The transport of carbohydrates by Streptococcus mutans is accomplished by the phosphoenolp

176 that the membrane fatty acid composition of Streptococcus mutans is affected by growth pH.

177 environmental stress, especially low pH, by Streptococcus mutans is central to the virulence of this

178 Streptococcus mutans is considered the primary etiologic

179 Streptococcus mutans is considered the principal carioge

180 Streptococcus mutans is considered to be the major etiol

181 urface Cnm protein expressed on cnm-positive Streptococcus mutans is involved in the development of C

182 The quorum-sensing system of Streptococcus mutans is mediated by the competence-stimu

183 Streptococcus mutans is often cited as the main bacteria

184 Streptococcus mutans is one of several members of the or

185 Dental caries induced by Streptococcus mutans is one of the most prevalent chroni

186 Streptococcus mutans is particularly well adapted for hi

187 Streptococcus mutans is prominently linked to dental car

188 Streptococcus mutans is the etiological agent of dental

189 Streptococcus mutans is the leading cause of dental cari

190 Streptococcus mutans is the leading cause of dental cari

191 Streptococcus mutans is the primary cariogen that produc

192 The gram-positive bacterium Streptococcus mutans is the primary causative agent in t

193 Streptococcus mutans is the primary etiological agent of

194 Streptococcus mutans is the principal acidogenic compone

195 The cnm gene in Streptococcus mutans isolated from saliva was screened u

196 Strains of Streptococcus mutans lacking DnaK or GroEL appear not to

197 3 bp region (igr66) between grpE and dnaK of Streptococcus mutans lacks a promoter but is required fo

198 The primary structure of the Streptococcus mutans lantibiotic mutacin 1140 was elucid

199 ith special emphasis on the immunobiology of Streptococcus mutans, leading to active and passive vacc

200 The cariogenic bacterial species Streptococcus mutans metabolizes sucrose to initiate bio

201 omologous with transcriptional regulators of Streptococcus mutans (MetR), Streptococcus iniae (CpsY),

202 ed a previously constructed stress-sensitive Streptococcus mutans mutant Tn-1 strain resulting from d

203 jor cell-surface adhesion protein SA I/II of Streptococcus mutans, one of the major causative agents

204 essential for in vitro biofilm formation by Streptococcus mutans or Streptococcus gordonii grown in

205 causative agent of dental caries in humans, Streptococcus mutans, outcompetes other bacterial specie

206 Streptococcus mutans (p < 0.005), Streptococcus sobrinus

207 bacterium Cluster 1 (p = 0.11), and by qPCR, Streptococcus mutans (p = 0.008) and Scardovia wiggsiae

208 In Streptococcus mutans, (p)ppGpp synthesis is catalyzed by

209 The presence of bacterial DNA from Streptococcus mutans, Porphyromonas gingivalis, Tannerel

210 The oral pathogen, Streptococcus mutans, possesses inducible DNA repair def

211 Many clinical isolates of Streptococcus mutans produce peptide antibiotics called

212 cal enterotoxin B, splenocytes cultured with Streptococcus mutans produced significantly greater amou

213 Streptococcus mutans proliferates as a biofilm on the to

214 Streptococcus mutans regulates genetic competence throug

215 many gram-positive bacteria, but its role in Streptococcus mutans remains enigmatic.

216 ed evidence that the oral cariogenic species Streptococcus mutans remains viable but physiologically

217 n of the agmatine deiminase system (AgDS) of Streptococcus mutans requires agmatine and is optimal at

218 ormerly designated salivary agglutinin) with Streptococcus mutans requires an alanine-rich repetitive

219 demonstrated that competence development in Streptococcus mutans requires the type II ComRS quorum-s

220 ccus faecalis, Cardiobacterium valvarum, and Streptococcus mutans, respectively.

221 secretion and acid tolerance (sat) operon of Streptococcus mutans resulted in an acid-sensitive pheno

222 Inactivation of the Smu0630 gene of Streptococcus mutans resulted in dramatic decreases in b

223 t shows homology to the N-terminal domain of Streptococcus mutans SagA protein (42% similarity), prev

224 ll-known acidogenic/aciduric species such as Streptococcus mutans, Scardovia wiggsiae, Parascardovia

225 species associated with severe ECC included Streptococcus mutans, Scardovia wiggsiae, Veillonella pa

226 source of CO(2), buffers acid production by Streptococcus mutans (Sm), a key organism associated wit

227 ated, infected with Actinomyces viscosus and Streptococcus mutans (sobrinus) 6715, and fed a cariogen

228 ported identification of two Spx proteins in Streptococcus mutans - SpxA1 was the primary activator o

229 eptococcus pyogenes, Streptococcus gordonii, Streptococcus mutans, Staphylococcus aureus, and Enteroc

230 tococcus pyogenes, Streptococcus pneumoniae, Streptococcus mutans, Staphylococcus aureus, and Lactoco

231 Streptococcus mutans strain GS-5 produces a two-peptide

232 T MAP constructs, followed by infection with Streptococcus mutans strain SJr.

233 Using Streptococcus mutans strain UA159, we demonstrate that t

234 Eleven cases showed Streptococcus mutans strains positive for cnm.

235 Streptococcus mutans strains were isolated from cohorts

236 colonizing species of the human oral flora (Streptococcus mutans, Streptococcus gordonii and Strepto

237 Antibacterial material was synthesized, and Streptococcus mutans, Streptococcus gordonii, and Strept

238 The salivary bacterial levels evaluated were Streptococcus mutans, Streptococcus sobrinus, Streptococ

239 In Streptococcus mutans, Streptococcus suis, and species of

240 ecombinant DNA methods were used to make the Streptococcus mutans supercolonizing strain, JH1140, lac

241 clonal antibody (MAb) 6-11A directed against Streptococcus mutans surface adhesin P1 was shown previo

242 an N-terminal saliva-binding region (SBR) on Streptococcus mutans surface antigen I/II (AgI/II) and s

243 Allelic replacement of the C terminus of a Streptococcus mutans surface protein affects murein hydr

244 lly modified lantibiotic peptide secreted by Streptococcus mutans T8, which inhibits the energy metab

245 ne segments were identified from a strain of Streptococcus mutans that was isolated from a patient wi

246 Among these is Streptococcus mutans, the causative agent of dental cari

247 ccus gordonii, Streptococcus intermedius and Streptococcus mutans, the genes were cloned and expresse

248 In Streptococcus mutans, the global response regulator CovR

249 d the metabolism of lactose and galactose by Streptococcus mutans, the major etiological agent of hum

250 he facultative anaerobic human oral pathogen Streptococcus mutans, the mechanisms used to protect aga

251 hment to the dental surfaces was studied for Streptococcus mutans, the most abundant cariogenic bacte

252 However, the function of this molecule in Streptococcus mutans, the primary aetiological agent of

253 Streptococcus mutans, the primary causative agent of hum

254 Streptococcus mutans, the primary etiological agent of d

255 Streptococcus mutans, the primary etiological agent of h

256 Streptococcus mutans, the primary etiological agent of h

257 trigger factor homologue, was identified in Streptococcus mutans, the primary etiological agent of h

258 h-coverage genome sequence of 57 isolates of Streptococcus mutans, the primary etiological agent of h

259 Streptococcus mutans, the principal causative agent of d

260 Streptococcus mutans, the principal etiologic agent of d

261 sives and the DOX-containing eluates against Streptococcus mutans through agar diffusion assays.

262 films by an important opportunistic pathogen Streptococcus mutans through the action of a family of 3

263 licated in the ability of certain strains of Streptococcus mutans to bind to collagen and to invade h

264 ugh substantial epidemiologic evidence links Streptococcus mutans to caries, the pathobiology of cari

265 The ability of Streptococcus mutans to catabolize cellobiose, a beta-li

266 ace and competes with oral pathogens such as Streptococcus mutans to maintain oral health.

267 Recently, we showed that exposure of Streptococcus mutans to oxygen strongly inhibits biofilm

268 The ability of Streptococcus mutans to produce and tolerate organic aci

269 -containing enzyme used by the oral pathogen Streptococcus mutans to reduce diatomic oxygen to water

270 hibited binding of a cell surface adhesin of Streptococcus mutans to salivary receptors in vitro, as

271 he tooth by recruiting the pioneer bacterium Streptococcus mutans to the surface.

272 elocity water microdrop on the detachment of Streptococcus mutans UA159 biofilms from the interproxim

273 The production of (p)ppGpp by Streptococcus mutans UA159 is catalyzed by three gene pr

274 o remove biofilms of the cariogenic pathogen Streptococcus mutans UA159, as well as Actinomyces naesl

275 been identified in the cariogenic bacterium Streptococcus mutans UA159.

276 being upregulated in a proteome analysis of Streptococcus mutans under acid stress conditions.

277 t was found to fail to inhibit the growth of Streptococcus mutans under microaerobic conditions.

278 The oral microbe Streptococcus mutans uses adaptive mechanisms to withsta

279 The cariogenic bacterium Streptococcus mutans uses adhesin P1 to adhere to tooth

280 caries (ECC), while strongly associated with Streptococcus mutans using selective detection (culture,

281 e human oral cavity plays a putative role in Streptococcus mutans virulence gene expression and in ap

282 The presence of cnm-positive Streptococcus mutans was significantly associated with I

283 ), which is an important virulence factor of Streptococcus mutans, was recombinantly expressed in the

284 In order to examine gene function in Streptococcus mutans, we have recently initiated an anti

285 al caries, recombinant, ureolytic strains of Streptococcus mutans were constructed.

286 Extremely high levels of Streptococcus mutans were detected in the S-ECC group.

287 implicated in the acid tolerance response of Streptococcus mutans when a mutation in its gene resulte

288 lular polysaccharide (IPS) is accumulated by Streptococcus mutans when the bacteria are grown in exce

289 reptococcus mitis, Streptococcus oralis, and Streptococcus mutans, whereas dentate individuals had hi

290 However, it does not adhere to Streptococcus mutans, which expresses SpaP, a highly con

291 sis that cytokines elicited by antigens from Streptococcus mutans, which frequently dominates shallow

292 the basis of these findings, we propose that Streptococcus mutans, which resides in a multispecies or

293 The mannose-PTS of Streptococcus mutans, which transports mannose and gluco

294 ified enolase as a cell surface component of Streptococcus mutans, which was confirmed by enzyme-link

295 en I/II) is a sucrose-independent adhesin of Streptococcus mutans whose functional architecture on th

296 (p < 0.001), Scardovia wiggsiae (p = 0.003), Streptococcus mutans with bifidobacteria (p < 0.001), an

297 able to selectively kill cariogenic pathogen Streptococcus mutans with high efficacy within a human s

298 h for noninvasive treatment based on killing Streptococcus mutans with high-frequency microwave energ

299 We previously showed that oral Streptococcus mutans with the cnm gene encoding a collag

300 r the function of the Escherichia coli YidC, Streptococcus mutans YidC2, and the chloroplast Arabidop