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1 n of bacterial flagellins that interact with Toll-like receptor 5.
2 ediated by the interaction of flagellin with Toll-like receptor 5.
3 independently of flagellin identification by Toll-like receptor 5.
4 nal effectively through both human and mouse Toll-like receptor 5.
5 e to flagellin is likely driven, in part, by Toll-like receptor 5.
6 triggers innate immune responses mediated by Toll-like receptor 5.
10 innate immune responses by signaling through Toll-like receptor 5 and is a potential vaccine adjuvant
12 n some genetic backgrounds dominant-negative Toll-like receptor 5 associated negatively with Crohn's
13 ablated in DCs from NLRC4(-/-) mice but not Toll-like receptor 5-deficient (TLR5(-/-)) mice, indicat
14 gellate Salmonella were highly attenuated in Toll-like receptor 5-deficient (TLR5(-/-)) mice, indicat
17 nduces caspase-1 activation independently of Toll-like receptor 5 in salmonella-infected and lipopoly
18 ellin:allergen fusion protein containing the Toll-like receptor 5 ligand flagellin A from Listeria mo
20 ave designed a membrane-anchored form of the Toll-like receptor 5 ligand flagellin, the major proinfl
21 into the portal circulation, with flagellin (Toll-like receptor 5 ligand) being the most plentiful an
22 rane of the intestinal epithelium, activates Toll-like receptor 5-mediated innate immune signaling pa
24 nduction was also noted after stimulation of Toll-like receptor 5 or 7, but not of other pattern reco
26 ivation of caspase-1 that was independent of Toll-like receptor 5, required for recognition of extrac
30 cognition site of the innate immune receptor Toll-like receptor 5, three of four FliC epitopes recogn
31 ther such antibodies might negatively impact Toll-like receptor 5 (TLR5) activation, an important com
33 of purified Salmonella-derived flagellin, a Toll-like receptor 5 (TLR5) agonist, protects mice from
36 ived from Salmonella flagellin that binds to Toll-like receptor 5 (TLR5) and activates nuclear factor
37 e I and II) and required flagellin receptors Toll-like receptor 5 (TLR5) and NOD-like receptor C4 (NL
38 rized one candidate nonsynonymous variant in Toll-like receptor 5 (TLR5) and show that it leads to al
39 in the sensing of flagellins; these involve toll-like receptor 5 (TLR5) and the cytosolic proteins B
42 lin revealed that cell surface expression of Toll-like receptor 5 (TLR5) conferred NF-kappaB gene exp
44 ind that mice lacking the flagellin receptor Toll-like receptor 5 (TLR5) exhibit a profound loss of f
50 t of a decrease in the steady-state level of toll-like receptor 5 (TLR5) or interleukin-1 receptor as
56 bacterial flagella, is a potent activator of toll-like receptor 5 (TLR5) signaling and is a major pro
59 , we show that mice genetically deficient in Toll-like receptor 5 (TLR5), a component of the innate i
60 ecules known to activate innate immunity via Toll-like receptor 5 (TLR5), and critical targets of the
62 agellin, a bacterial protein that stimulates Toll-like receptor 5 (TLR5), induces epithelial expressi
66 d not elicit antibodies that neutralized the Toll-like receptor 5 (TLR5)-activating activity of flage
67 ve bacteria activate inflammatory cells by a toll-like receptor 5 (TLR5)-dependent signaling pathway.
75 tigate whether flagellin, the sole ligand of Toll-like receptor-5 (TLR5), induces an innate defense t
76 ractions revealed flagellin, via ligation of Toll-like receptor 5, to be a major means of activating
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