ライフサイエンスコーパス: augmentation index

1 ts were change in flow-mediated dilation and augmentation index.

2 ere seen in either flow-mediated dilation or augmentation index.

3 n independent predictor of higher hs-CRP and augmentation index.

4 o influence on augmentation index (change in augmentation index, -0.4%; 95% CI, -1.7 to 0.8; P=0.5) o

5 NO3(-) reduced aortic augmentation index (132.2+/-16.7% versus 141.4+/-21.9%;

6 ry flow reserve, (3) pulse wave velocity and augmentation index, (4) circulating NT-proBNP (N-termina

7 +/- 1.0 m/s vs. -0.1 +/- 0.9 m/s, p < 0.01), augmentation index (-5.2 +/- 6.1% vs. -1.4 +/- 5.9%, p <

8 orearm mitochondrial oxidative function, and augmentation index (a marker of arterial wave reflection

9 sterone, and endothelin (ET)-1 together with augmentation index, a measure of arterial stiffness, wer

10 The peak reductions in peripheral augmentation index after the WA, AO, and CO meals (-9.5%

11 01), AI d (r = -0.17, P = 0.06), the central augmentation index (AI c ) (r = 0.61, P < 0.001) or AI c

12 AI rd , a combination of AI r and diastolic augmentation index (AI d ) with a weight alpha, to achie

13 Radial augmentation index (AI r ) can be more conveniently meas

14 Augmentation index (AI(a)) and timing of the reflected p

15 tid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI) at a steady state.

16 emoral pulse wave velocity (PWV) and carotid augmentation index (AI)].

17 t the hypothesis that age-related changes in augmentation index (AIx) are more prominent in younger i

18 Augmentation index (AIx) is a measure of systemic arteri

19 Augmentation index (AIx) is widely used as a measure of

20 pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) were assessed in 130 subjects (

21 Pulse wave velocity (PWV) and the augmentation index (AIx) were assessed noninvasively and

22 and dose on the change in blood pressure and augmentation index (AIx) were determined.

23 s, LV wall thickness and dimensions, central augmentation index (AIx), aortic pulse wave velocity (aP

24 Pulse wave analysis was used to determine augmentation index (AIx), which provides a measure of sy

25 using pulse wave velocity (PWV) and central augmentation index (AIx).

26 emoral pulse wave velocity (PWV), and aortic augmentation index (AIx).

27 % vs. placebo -0.69 +/- 2.8%; p = 0.017) and augmentation index (allopurinol -2.8 +/- 5.1% vs. placeb

28 hanges in air pollution were associated with augmentation index and augmentation pressure at several

29 t-term exposure to air pollution and central augmentation index and augmentation pressure, correlates

30 ate capabilities for measuring radial artery augmentation index and pulse pressure velocity.

31 elocity and radial tonometry-derived central augmentation index and subendocardial viability ratio we

32 arterial stiffness (pulse wave velocity and augmentation index) and blood pressure were also not sig

33 ntral pulse pressure, augmentation pressure, augmentation index, and mean arterial pressure.

34 tic modulus, impedance, pulse wave velocity, augmentation index, and pulse pressure amplification) ar

35 6 mm Hg (95% confidence interval, 2.4-20.7), augmentation index, and pulse wave velocity without chan

36 Brachial stiffness, augmentation index, and systemic arterial compliance wer

37 -femoral pulse wave velocity (cfPWV), aortic augmentation index, and systemic arterial compliance.

38 Augmentation index (as %) and augmentation pressure (in

39 n of either meal significantly decreased the augmentation index at 2 and 4 h (P < 0.002) and signific

40 a and augments pressure in late systole [ie, augmentation index = (augmented pressure/pulse pressure)

41 mpliance estimates but may underestimate the augmentation index because the latter requires greater f

42 ulse wave velocity (beta = -0.09, p = 0.04), augmentation index (beta = -0.11, p = 0.03), and subendo

43 artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pressure (BP), and vascular bi

44 arkedly change wave reflection amplitude and augmentation index by altering stiffness of the muscular

45 tic impedance [Zc]) and late-systolic loads (augmentation index [cAI]; late pressure-time integral [P

46 y arterial pulse-wave velocity (Doppler) and augmentation index (carotid tonometry) declined with ver

47 Calculations included the carotid augmentation index, carotid artery compliance and the di

48 pulse wave velocity [PWV]), wave reflection (augmentation index, carotid-brachial pressure amplificat

49 tion, carotid artery intima-media thickness, augmentation index, central blood pressure, subendocardi

50 In contrast, augmentation index, central pulse pressure, and pulse pr

51 -1.9 to 1.0], P=0.6) and had no influence on augmentation index (change in augmentation index, -0.4%;

52 HLS increased the augmentation index compared with the other test meals (P

53 ial stiffness (pulse wave velocity [PWV] and augmentation index corrected for heart rate [AI@75]) wer

54 The secondary outcomes were CFPWV, FWA, and augmentation index during examination cycle 8.

55 ex, height, weight, end-diastolic LV volume, augmentation index, end-systolic pressure, and cardiovas

56 oCor Mx system was used to derive the aortic augmentation index from radial artery pulse pressure wav

57 Central augmentation index improved significantly with allopurin

58 sodilatation was calculated as the change in augmentation index in response to an endothelium-depende

59 essure augmentation was determined using the augmentation index in the ascending aorta (AIaa) and dis

60 arterial stiffness (pulse wave velocity and augmentation index) in 20 adult patients with hypertensi

61 Augmentation index increased after transplant and was gr

62 a strong inverse relationship between HR and augmentation index, indicative of increased wave reflect

63 levels and 3 measures of vascular function (augmentation index, mean arterial pressure, and pulse pr

64 , 1.6 [95% CI, 1.3-2.0] per 1 SD; P < .001), augmentation index (OR, 1.7 [95% CI, 1.4-2.0] per 1 SD;

65 ention did not significantly change CCA-IMT, augmentation index, or BP, but pulse pressure variabilit

66 eactive protein (P(interaction) < 0.001) and augmentation index (P = 0.06) values at or above the 75t

67 had higher hs-CRP (P=0.014), higher central augmentation index (P=0.015), and lower glutathione leve

68 function (P=0.009), and improved the central augmentation index (P=0.015).

69 lood pressure, mean arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intim

70 = [Reflected/Forward wave amplitude] x 100), augmentation index ([Second/First systolic peak] x 100)

71 rterial compliance to 6 +/- 7% accuracy, and augmentation index to within -7% points (30 +/- 45% accu

72 a decrease in pulse wave velocity (PWV) and augmentation index up to 26 h after the walk.

73 nts/cm(3) IQR increase) were associated with augmentation index values that were 0.8% (95% confidence

74 In healthy volunteers, PWV and augmentation index were associated both with black carbo

75 is cohort, higher aortic stiffness, FWA, and augmentation index were associated with higher risk of i

76 Pulse wave velocity and augmentation index were improved only after anti-IL-12/2

77 Aortic pulse wave velocity (PWV) and carotid augmentation index were reduced only with SR (p < 0.05).

78 Six months after RD aortic augmentation and augmentation index were significantly reduced by -11 mm

79 y [CFPWV], forward wave amplitude [FWA], and augmentation index) were examined over a 7-year period i

80 ticles and PM2.5, and an increase in PWV and augmentation index with NO2 and ultrafine particles.