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1 ICallo) to autologous transplantation alone (auto).
2 nsplants are more effective than autologous (auto).
3 ower, too, in the allo group (p = 0.0186 vs. auto).
4  regulatory loop (R-loop) by > 10(4)-fold (k(auto) = 2.6 +/- 0.3 s(-1)).
5              The DKM (1) underwent a facile (auto) acid-mediated methanolysis to yield seco-shornephi
6              Moreover, RECQ1 regulates PARP1 auto-(ADP-ribosyl)ation and the choice between long-patc
7 mpled with a conventional automatic sampler (Auto) and by grab (Grab) sampling.
8 otein complex is a favored target for allo-, auto-, and drug-dependent antibodies associated with imm
9             They prevent immune responses to auto- and alloantigens and are thus under close scrutiny
10 ymic and peripheral T-cell responses against auto- and alloantigens.
11  including marrow stimulation, osteochondral auto- and allografting, and autologous chondrocyte impla
12  FCGR genes has been associated with various auto- and alloimmune diseases.
13 nctions to inhibit aggressive T(H)1-mediated auto- and alloimmune responses.
14 ia time, diabetogenic immunosuppression, and auto- and alloimmunity.
15 een parental genomes, although comparison of auto- and allopolyploids suggests that intergenomic inco
16 only cotransplanted along with islets during auto- and allotransplantations.
17 -component peptide system that is capable of auto- and cross-catalysis and allows for the selective a
18 he search for chemical systems in which both auto- and cross-catalysis can occur has therefore attrac
19 les and used this information to predict the auto- and cross-catalysis pathways and the resulting pla
20 approach makes use of the sequence-dependent auto- and cross-catalytic functional characteristics of
21        Here we measure an extensive range of auto- and cross-correlated spin relaxation rates at mult
22 ) depends on the accurate measurement of the auto- and cross-correlation function (ACF and CCF) ampli
23 hm that simultaneously analyzes all distinct auto- and cross-correlation functions from 15 independen
24  effect of reaction kinetics on the shape of auto- and cross-correlation functions.
25                                              Auto- and cross-correlation of spectral data facilitates
26 cleotide interactions by means of a modified auto- and cross-covariance function, (iv) nucleotide the
27 ggestive of immediate-early genes capable of auto- and cross-induction through cis-acting regulatory
28 R) activation was also necessary for trefoil auto- and cross-induction, and both spasmolytic polypept
29 at the 3'UTRs of their own transcripts in an auto- and cross-regulated mechanism that limits their ex
30 ts revealed MYB31 and MYB42 participation in auto- and cross-regulation in all three species.
31 rovide evidence for a concerted role for DLX auto- and cross-regulation in the establishment of a nes
32              These loops can be generated by auto- and cross-regulation of expression of CREB protein
33 nd those of six other Dmrt genes, indicating auto- and cross-regulation of these genes.
34 mbryos and present evidence for existence of auto- and cross-regulatory control of expression among t
35                   We also discover extensive auto- and cross-regulatory interactions among the Hoxa1
36 ength to the emerging importance of positive auto- and cross-regulatory interactions between Hox gene
37 b2 and Hoxa1, is integrated into a series of auto- and cross-regulatory loops between Hox genes.
38 oduction, serum IgG1 and IgG2 levels of both auto- and heteroantibodies, and soluble CD44.
39 oduction, serum IgG1 and IgG2 levels of both auto- and heteroantibodies, and soluble CD44.
40 luding pro- and anti-inflammatory cytokines, auto- and heteroantibody productions) or antigen-specifi
41 excitability, and its control by presynaptic auto- and heteroreceptors on presynaptic terminals.
42 e chemistry of complex samples by decrypting auto- and heterospectral correlations that may exist bet
43 le autoregulatory models for E2EPF involving auto- and multiubiquitination.
44 lpha L29F, beta N108Q) is stabilized against auto- and NO-induced oxidation as compared to rHb (beta
45 se of the complex relationship between Syk's auto- and other internal phosphorylation sites, scaffold
46  of Hoxb1 expression in rhombomere 4 through auto- and para-regulatory interactions.
47 ivation of T lymphocytes and functions as an auto- and paracrine growth factor.
48  and a dramatically increased sensitivity to auto- and paracrine Wnts.
49 ferences between the contraction patterns of auto- and photo-cure were minimal.
50 oad neutralization and its relationship with auto- and polyreactivity and may aid design of vaccine i
51 tutions in the SCD and the FHA domain impair auto- and trans-kinase activities of Chk2.
52                                      Slik is auto- and trans-phosphorylated in vivo.
53            Moreover, they support a role for auto- and trans-regulation of Gfi1 by GFI1 and GFI1B in
54 ne/threonine kinase activity, impairing both auto- and transkinase activities of Bcr.
55 for immunomodulation to achieve tolerance to auto- and transplantation Ags.
56 n of immunoglobulin M, which then serves as (auto-) antigen for a prosurvival BCR signal.
57                                         Such auto- antigen specificity and/or cross-reactivity may di
58 for example tonic B-cell signaling, chronic (auto)-antigen exposure, and self-binding properties of t
59                                         Both auto- as well as photo-curing composites were analyzed.
60                                              Auto-, cross- and para-regulatory interactions help esta
61             Allogeneic (ALLO) or autologous (AUTO) HSCT was employed, respectively, in 141 and 102 HR
62 to our understanding of normal and abnormal (auto) immune responses.
63     The physiological relevance for humoral (auto-)immunity was corroborated by exacerbated lupuslike
64 o and in vivo assays, as well as 2 relevant (auto-) inflammatory murine models.
65 ationship of AE with Th1- and Th17-mediated (auto-)inflammatory conditions such as diabetes mellitus
66  infection or tissue injury, likely to curb (auto)-inflammatory responses.
67 comes for patients who underwent autologous (auto, n = 128) or HLA-identical sibling (allo, n = 76) S
68            The oligotrophic ocean is neither auto- nor heterotrophic, but functionally diverse.
69  selectivity of action to facilitate intra-, auto-, or paracrine mechanisms that define and influence
70  DCs cultured in the laboratory can suppress auto- or alloimmunity.
71 lucagon secretion was observed in either the auto- or allotransplant recipients, whereas healthy cont
72 el was evaluated in19 monkeys that underwent auto- or allotransplantation, with or without subtherape
73 ons did not indicate any underlying positive auto- or cross-regulation of Mash1.
74 i-stage epithelial carcinogenesis, including auto- or paracrine growth stimulation, upregulation of a
75 ene amplification, activating mutations, and auto- or paracrine mechanisms.
76   Thus, shed soluble chemokines can generate auto- or paracrine signals by binding and activating the
77              The domain does not support the auto- or trans-kinase activity of p210 BCR/ABL or its ab
78 is hypothesized to occur as a consequence of auto- or transphosphorylation on tyrosine residues assoc
79 eptor dependent and independent, act through auto-, para- or intracrine mechanisms and can be modifie
80                  We also provide evidence of auto-, para-, and feedback regulation among these factor
81  levels in Hyp mice, its putative role as an auto-/paracrine osteomalacia-causing factor has not been
82                          Here we describe an auto-/paracrine physiologic circuit that controls quiesc
83  the signaling components, the logic of this auto-/paracrine signaling module in growth control remai
84 es the accumulation of pyrophosphate through auto-/paracrine suppression of TNAP.
85                           Here we report an "auto"-regulatory feedback mechanism between plasmacytoid
86 se bifurcation theory and numerical methods (AUTO) to characterize the codimension-one and -two bifur
87                                        Thus, auto-/transphosphorylation of S379 is required for Chk2
88 on by ATM, followed by Chk2 oligomerization, auto-/transphosphorylation, and activation.
89 e direct enumeration and characterization of auto-, tumor-, or neo-Ag-reactive T cells within the nai
90 arison of safety and efficacy of autologous (auto) versus allogeneic (allo) bone marrow-derived hMSCs
91                 In Experiment 2, we used an "auto- vs. self-play" manipulation to eliminate outcome d

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