ライフサイエンスコーパス: be protected from

1 pts associated with arrested RNA polymerases are protected from 3'-5' degradation and thus, unstable

2 In addition, Gsdmd(-/-) mice are protected from a lethal dose of lipopolysaccharide.

3 eceptor (TLR) adaptor Mal (encoded by TIRAP) are protected from a number of infectious diseases, incl

4 because we show that vaccinated animals can be protected from a mucosal challenge with a heterologou

5 with plasmids or given nonhuman-primate sera were protected from a lethal challenge with purified Tcd

6 irus A/California/7/2009 (H1N1) strain (Cal) were protected from a lethal challenge with the heterolo

7 ina using a prime-boost vaccination strategy were protected from a subsequent lung challenge with P.

8 The 3' U-tract of scR1 is protected from aberrant processing by the La homologu

9 t manner while the conjugated-ubiquitin (Ub) is protected from active deubiquitination.

10 These mice were protected from acute podocyte foot process effaceme

11 wild-type littermates, hCD39 transgenic mice were protected from acute renal injury at 24 hours, but

12 male) mice deficient in Arg-II (Arg-II(-/-)) are protected from age-associated glucose intolerance an

13 necrosis factor (TNF)-deficient mice, which are protected from age-associated inflammation, age-rela

14 sulin levels following glucose challenge and were protected from age-related reductions in GSIS and g

15 Mislocalized proteins (MLPs) are protected from aggregation by the Bag6 complex and d

16 ar fat mass and energy balance, M(IL10) mice were protected from aging-associated insulin resistance

17 WT but not Ido1(-/-) mice were protected from AIA by IFN-alpha, and Kyn, the main

18 ted that integrin alphavbeta6-deficient mice are protected from airway hyperresponsiveness, due in pa

19 inflammasome components NLRP3 and caspase-1 were protected from aldosterone-induced vascular damage.

20 In contrast, NT4(-/-) mice were protected from allergen-induced mucus overproductio

21 All animals were protected from an intrarectal SIVmac239 challenge,

22 We hypothesized that host cells would be protected from anthrax toxins if anthrax toxin recept

23 e conserved HIV-1 site of coreceptor binding is protected from antibody-directed neutralization by co

24 Within DCs A. terreus conidia were protected from antifungals, whereas A. fumigatus co

25 immunized with a single dose of this vector were protected from any signs of disease following letha

26 report that active spindle assembly factors are protected from APC/C-dependent degradation by microt

27 cells that received Tax-containing exosomes were protected from apoptosis through activation of AKT.

28 that 13-lined ground squirrel tubular cells are protected from apoptotic cell death during IBA.

29 k-out mice that lacked PEMT showed that they were protected from atherosclerosis, diet-induced obesit

30 Such critical functions need to be protected from attack by pests and pathogens or from

31 NOD mice treated with AZD1480 were protected from autoimmune diabetes, and diabetes wa

32 Transport is protected from back-scattering, possibly reflecting g

33 in response to different learning tasks and are protected from being eliminated when multiple tasks

34 ZDF rats with global deletion of CB1R are protected from beta-cell loss, hyperglycemia, and ne

35 n-G, amoxicillin, ampicillin, and cefazolin, are protected from beta-lactamase hydrolysis via the for

36 tic Ppp1r3b upon long-term fasting (12-36 h) were protected from blood ketone-body accumulation, unli

37 cient in the double-stranded DNA sensor AIM2 are protected from both subtotal body irradiation-induce

38 Interestingly, mitochondria are protected from both the deficiency in NAD(+) biosynt

39 p to 1,200 microg/mL in serum, and up to 70% were protected from both i.v. and mosquito bite challeng

40 absence of target, aptamer coated particles are protected from capture on the test line and are inst

41 PGC-1beta knockout mice are protected from carcinogenesis.

42 eletal muscle-specific EcSOD transgenic mice are protected from cardiac hypertrophy, fibrosis, and dy

43 v integrin is depleted in PDGFRbeta(+) cells are protected from cardiotoxin and laceration-induced sk

44 us for the ancestral C allele (EE genotype), are protected from cardiovascular disease (CVD), showing

45 nvestigated how glutamate in a neural tissue is protected from catabolism.

46 re deficient for both B cells and antibodies were protected from CAV.

47 Germ-free mice are protected from CCM formation, and a single course of

48 oth mucosal and serum IgA anti-toxin Abs and were protected from CDI upon rechallenge, with protectio

49 Differentiated keratinocytes are protected from cell death, whereas cells treated wit

50 ntial sH1N1 influenza virus-infected ferrets were protected from challenge with a novel H1N1 influenz

51 operties, are strikingly enriched in AT, and are protected from chemotherapy by the GAT microenvironm

52 Leukemia cells are protected from chemotherapy-induced apoptosis by the

53 contrast, the stability of the XRCC1 monomer is protected from CHIP-mediated ubiquitylation by intera

54 e we demonstrate that mice deficient in Irp2 were protected from cigarette smoke (CS)-induced experim

55 ed cattle), despite vaccinated cattle having been protected from clinical disease.

56 Mice with PU.1 deficiency in T cells were protected from colitis, whereas treatment with anti

57 TTPDeltaARE mice were protected from collagen antibody-induced arthritis,

58 intact complement negative regulatory system are protected from complement attack, whereas cells with

59 eficient mice, but not RIPK3-deficient mice, were protected from ConA-induced liver injury.

60 operties rivalling suspended graphene, while being protected from contamination and mechanical damage

61 ssemia trait carriers displayed lower TC and were protected from coronary artery disease (CAD); (ii)

62 ionic doping, and the LLMO cathode materials are protected from corrosion induced by organic electrol

63 CPE-sensitive enterocyte-like cell line) can be protected from CPE-induced cytotoxicity by preincubat

64 al iron chelator or mice fed a low-iron diet were protected from CS-induced COPD.

65 ytochrome c oxidase, which have reduced COX, were protected from CS-induced pulmonary inflammation an

66 Endothelial cells and isolated lungs were protected from cytotoxin-induced death by stimulati

67 del, we report here that TRPV4-knockout mice were protected from D. farinae-induced airway remodeling

68 dling development, the shoot apical meristem is protected from damage as the seedling emerges from so

69 type in the heart during EAM, IL-4(-/-) mice were protected from DCMi like DeltadblGATA1 mice, and eo

70 effect of IL-5, as IL-5TgDeltadblGATA1 mice were protected from DCMi, whereas IL-5(-/-) mice exhibit

71 The mechanisms by which CGIs are protected from de novo methylation remain elusive.

72 Until such foci were lost, cells were protected from death.

73 ly assumed that mRNAs undergoing translation are protected from decay.

74 Other FNWs were protected from decomposition with an amorphous carb

75 that viral genomes may in some circumstances be protected from degradation for centuries.

76 ergen extracts: The protein/DNA molecule can be protected from degradation, higher local concentratio

77 bstrate of the Lon-type protease and that it is protected from degradation by Nfs1, the sulfur donor

78 terious interaction with the Bam complex but was protected from degradation and eventually inserted i

79 subunit, the palmitoylated CaV2.2 I-II loop was protected from degradation, although oligoubiquitina

80 blueberry polyphenols complexed with protein were protected from degradation during 16weeks at 4 degr

81 of ultralow methylation at transposons that are protected from demethylation in the germline and ICM

82 Growing microtubules are protected from depolymerization by the presence of a

83 ry diseases, ICER/CREM-deficient B6.lpr mice are protected from developing autoimmunity.

84 Ormdl3 knockout mice were found to be protected from developing allergic airways disease an

85 did not occur in IL-17-deficient mice, which were protected from development of lupus autoantibodies

86 r, mice with liver-specific deletion of MPC2 were protected from development of NASH on this diet.

87 red with WT, we demonstrated that Parp1(-/-) were protected from dextran-sulfate sodium-induced colit

88 upregulated when type 1 diabetes-prone rats are protected from diabetes development.

89 arp contrast, TLR3- and MyD88-deficient mice were protected from diabetes following the same treatmen

90 lar to those of WT STZ mice, TLR4KO STZ mice were protected from diabetes-induced bladder hypertrophy

91 Glomeruli from G4 KO mice were protected from diabetes-induced hypertrophy, mesang

92 mice and mice treated with a P2X7 inhibitor were protected from diabetes-induced TNF-alpha, IL-1beta

93 DBim(-/-)) mice developed less insulitis and were protected from diabetes.

94 p66Shc that is not acetylatable on lysine 81 are protected from diabetic oxidative stress and vascula

95 deficient in small heterodimer partner (SHP) are protected from diet-induced hepatic steatosis result

96 more, adipose-specific Dnmt3a knock-out mice are protected from diet-induced insulin resistance and g

97 Mice lacking Tlr4s are protected from diet-induced insulin resistance and i

98 Mice lacking LR11 are protected from diet-induced obesity associated with

99 d and that Mfge8-deficient (Mfge8(-/-)) mice are protected from diet-induced obesity, steatohepatitis

100 functional gene encoding MGAT2 (Mogat2(-/-)) are protected from diet-induced obesity.

101 Stk25(-/-) mice were protected from diet-induced liver steatosis accompa

102 , we found that mice lacking hepatic ZFP36L1 were protected from diet-induced obesity and steatosis.

103 PDZ-RhoGEF-deficient mice were protected from diet-induced obesity and T2D.

104 heir severe fatty liver, the transgenic mice were protected from diet-induced obesity and type 2 diab

105 Notably, Il5 (Tg) /CD300f (-/-) were protected from diet-induced weight gain and glucose

106 th wild type, whereas AdipoR2-deficient mice were protected from diet-induced weight gain and metabol

107 e a limited elastic range of 3-4% strain but are protected from direct stretch during physiological l

108 In contrast to humans, rodents are protected from disease on infection with ebolaviruse

109 vial fluids after injury and Cxcr3(-/-) mice being protected from disease development.

110 tis, because Nfil3/Rag1 double-knockout mice were protected from disease.

111 These precursors must be protected from DNA damage-induced cell death, however

112 Furthermore, Sh2d1a(-/-) mice were protected from EAE and exhibited greatly decreased

113 Nevertheless, TPL-2-deficient mice were protected from EAE, which correlated with reduced i

114 tatic and dynamic disorder, but how carriers are protected from efficient scattering with charged def

115 By 6 months, Mmp28(-/-) mice were protected from emphysema.

116 n of IL-37 (IL-37tg mice) with intact IL-1R8 were protected from endotoxemia, IL-1R8-deficient IL-37t

117 ting becomes viable when a quantum state can be protected from environment-induced error.

118 Eventually, ERK inhibitor treated cells are protected from ETO-induced nuclear envelope (NE) rup

119 ic core of cyclodextrins and therefore, they are protected from exogenous stress.

120 e have decreased Th17 cell numbers, and they are protected from experimental autoimmune encephalitis,

121 Kv1.3-knockout (KO) mice are protected from experimental autoimmune encephalomyel

122 ve defect in Ag-specific alphabetaTh17 cells were protected from experimental ARDS induced by a singl

123 Furthermore, CD43(-/-) mice were protected from experimental autoimmune encephalomye

124 pe-specific deletions of PTEN-targeting APCs were protected from experimental autoimmune encephalomye

125 Both talin1(L325R) and talin1(W359A) mice were protected from experimental thrombosis.

126 ng facilities with large elderly populations are protected from extreme heat (for example through bac

127 genetic or pharmacological inhibition of BID are protected from Fas-mediated impairment of mitochondr

128 ogen synthesis, and as a result, CD36Tg mice were protected from fasting hypoglycemia.

129 les cannot sing to attract females, but they are protected from fatal attack by an acoustically orien

130 Mice deficient in IRAK-M were protected from fibrosis and displayed a diminished

131 Moreover, TRPV4-deficient mice were protected from fibrosis.

132 less nitrogen after 64 years than plots that were protected from fire.

133 Cyclic, unbranched structures are protected from fragmentation during the first oxidat

134 Our results suggest that certain tissues are protected from functional deleterious effects of pro

135 tive Staphylococcus aureus cells appeared to be protected from GA by an increased formation of nm-sca

136 mice, the tumor suppressor isoform of CUGBP1 is protected from Gank-mediated degradation.

137 proliferating cells in plant meristems must be protected from genome damage.

138 to wild-type mice, Cd74-deficient mice also were protected from glomerular injury and ensuing activa

139 A9 express reduced neutrophil chemokines and are protected from HCC.

140 female neuron-specific SOCS3 knock-out mice were protected from HCD-induced obesity.

141 stimulation, whereas Bex1 gene-deleted mice are protected from heart failure-promoting insults.

142 te-specific Shp1 knockout mice (Ptpn6(H-KO)) are protected from hepatic insulin resistance evoked by

143 notype, adipose-specific 11beta-HSD1 KO mice were protected from hepatic steatosis and circulating fa

144 Finally, HFD-fed AdSod2 KO mice were protected from hepatic steatosis, adipose tissue in

145 lation and preserved brown adipose mass; and were protected from hepatic steatosis.

146 MPO knockout (MPO(-/-)) mice were protected from HFD-enhanced body weight gain and in

147 pe littermate control (M-JAK2(+/+)) mice and were protected from HFD-induced systemic insulin resista

148 le of binding leukocyte alphaMbeta2-integrin were protected from HFD-induced weight gain and elevated

149 homologous residue Thr1150 (InsrT1150A mice) were protected from high fat diet-induced hepatic insuli

150 y during the suckling period and, as adults, were protected from high fat diet-induced obesity.

151 e United States in 2004-2006, when consumers were protected from high WLLs.

152 Furthermore we found that male AhRR Tg mice were protected from high-dose TCDD-induced lethality ass

153 pontaneous NASH whereas AEG-1(DeltaHEP) mice were protected from high-fat diet (HFD)-induced NASH.

154 AKO/cTg mice were protected from high-fat diet (HFD)-induced obesity

155 show that GPR30 knockout (GPRKO) female mice were protected from high-fat diet (HFD)-induced obesity,

156 lacking both LDL receptor (LDLR) and Arhgef1 were protected from high-fat diet-induced atherosclerosi

157 note, adipocyte-specific gp130 knockout mice were protected from high-fat diet-induced hepatic steato

158 Plxnd1-deficient zebrafish were protected from high-fat-diet-induced insulin resist

159 CreERT mice on different genetic backgrounds were protected from high-fat/ streptozotocin (STZ)-induc

160 In accordance, LRAT-null mice are protected from holo-RBP-induced suppression of insul

161 nces of viral replication, and animals could be protected from HSE by acyclovir treatment provided 4

162 The data show that telomeres are protected from hyper-resection through the repressio

163 Panx1 knockout mice (Panx1(-/-)) were protected from hypersensitivity in two sciatic nerv

164 ike-overexpressing transgenic (Sike-TG) mice are protected from hypertrophic stimuli.

165 mmune privilege, in which immunogenic tissue is protected from immune attack.

166 Ptpn22(-/-) mice were protected from immune complex-mediated arthritis, i

167 Here, we show that eosinophil-deficient mice were protected from induction of Th2-mediated peanut foo

168 These findings help explain why CF mice are protected from infection and nominate ATP12A as a po

169 carrying neutralizing antibodies against MuV are protected from infection by batMuV.

170 factors that determine why some individuals are protected from infection while others go on to devel

171 tive cells engraft and traffic normally, and are protected from infection with CCR5- and CXCR4-using

172 ve neutralizing antibodies against MuV might be protected from infection by BMV.

173 e infected, while the 6 PrEP-treated animals were protected from infection.

174 igh-dose UV B radiation, IR/IGF-1R(MKO) mice were protected from inflammation, whereas controls devel

175 nism by which hosts with allergic asthma may be protected from influenza morbidity.

176 e one or more means by which transplants can be protected from injury.

177 silon knockout mice on a high-fat diet (HFD) were protected from insulin resistance and showed altere

178 aRIIB globally or selectively in endothelium were protected from insulin resistance as a result of th

179 TNF-receptor I (TNFRI) mutant mice were protected from intestinal barrier dysfunction and A

180 ing the fetal troponin I isoform, (ssTnI) to be protected from ischemia by increased glycolysis.

181 A primary means by which the intestine is protected from its microbiota is via multi-layered mu

182 S1pr3(-/-)mice are protected from kidney IRI, because DCs do not mature

183 However, S1pr3(-/-) BMDC-pretreated mice were protected from kidney IRI.

184 r expressed on myeloid cells 2 (Trem2 (-/-)) were protected from LCMV-induced hepatitis and showed im

185 ally, mice vaccinated with the DeltatolC LVS are protected from lethal challenge and clear challenge

186 Vaccinated mice are protected from lethal challenge with diverse influen

187 ater column, seabirds, reptiles, and mammals are protected from lethal oiling at the surface, and mic

188 Gas6 null (-/-) mice are protected from lethal venous and arterial thromboemb

189 the CDV fusion and attachment glycoproteins were protected from lethal CDV challenge, whereas all an

190 usion protein-expressing recombinant viruses were protected from lethal CDV challenge.

191 myeloid cells and found that Blimp1 CKO mice were protected from lethal infection induced by Listeria

192 Nlrp12(-/-) mice are protected from lethality during IAV infection and sh

193 Fetal/neonatal progenitors may therefore be protected from leukemic transformation because they a

194 Plcbeta2-, Plcbeta3-, or Rac1-deficient mice were protected from lipopolysaccharide-induced lung inju

195 MRTF-A (Mkl1)-deficient mice are protected from lung fibrosis.

196 h VLPs and subsequently challenged with HMPV were protected from lung viral replication for at least

197 We show that aging mice lacking FGFR4 are protected from LVH.

198 malaria pathogenesis because EphB2(-/-) mice were protected from malaria-induced liver fibrosis despi

199 Target genes are protected from MazF activity by recoding the gene se

200 ommunities accumulate on teeth in areas that are protected from mechanical abrasion forces.

201 rgely confined to synaptic vesicles where it is protected from metabolic breakdown.

202 ith a specific 11betaHSD1 inhibitor (UE2316) were protected from metabolic disturbances despite simil

203 ce vaccinated with the group 1 HA mini-stems are protected from morbidity and mortality against letha

204 (H5N2), A(H5N8), A(H6N1), or A(H7N9) viruses were protected from mortality and showed drastically red

205 of contracting and transmitting the disease are protected from mosquito bites.

206 The CNS is protected from most virus infections by effective imm

207 treated with TAT2A during early adolescence were protected from MS-induced PVB loss and exhibited le

208 ut to reveal the mechanisms by which embryos are protected from mutant p53-induced transformation usi

209 n activating gene 1-deficient (Rag1-/-) mice were protected from NEC and transfer of intestinal lymph

210 iable region 1 yielded vector particles that were protected from neutralization by natural antibodies

211 he system was used to demonstrate that dsRNA is protected from nuclease digestion by virus-induced me

212 This DNA is protected from nucleases by a phage protein until Mu

213 nt biological catalysts of H2 oxidation, can be protected from O2 damage upon integration into a film

214 cantly reduced intestinal fat absorption and are protected from obesity, hepatic steatosis and insuli

215 sue inflammation, cadherin-11-deficient mice were protected from obesity-induced glucose intolerance

216 U L3 XAS indicates that this U(V) species is protected from oxidation likely incorporated into oct

217 is Mast cell-deficient mice (Kit(W-sh/W-sh)) were protected from P. gingivalis-induced alveolar bone

218 Within these autophagosomes, the bacteria are protected from phagocytic killing, thus providing an

219 sembly and how vulnerable assembly complexes are protected from photodamage are unknown.

220 cient mice made diabetic with streptozotocin were protected from physiological and structural indices

221 Conversely, areas exposed to high SS are protected from plaque development, but the mechanism

222 ked as the most upregulated in vaccinees who were protected from Plasmodium falciparum infection.

223 KEX1-immunized macaques were protected from Pneumocystis pneumonia, compared wit

224 and IL-1beta before intestinal manipulation were protected from POI.

225 nitrosylation, cyclic GMP, NO formation, and were protected from postinfarct and pressure overload HF

226 ct with cortactin, and Nox2-deficient hearts were protected from pressure overload-induced adverse my

227 V infection or SVR after antiviral treatment were protected from progressive liver disease and showed

228 Finally, coexposed mice were protected from prolonged bacterial infection by adm

229 eptum to the adjacent cell pole where SpoIIE is protected from proteolysis and activated.

230 Most circulating SAA is protected from proteolysis and misfolding by binding

231 etically deficient in PTP-alpha (Ptpra(-/-)) were protected from pulmonary fibrosis induced by intrat

232 mice had increased macrophage apoptosis and were protected from pulmonary fibrosis.

233 importance, mice that were deficient in MCU were protected from pulmonary fibrosis.

234 n contrast, SMPDL3b overexpressing podocytes were protected from radiation-induced cytoskeletal remod

235 All BCV-treated mice that survived infection were protected from rechallenge without additional treat

236 Ddah1(PT-/-) mice were protected from reduced kidney tissue mass, collagen

237 ogen receptor alpha (ERalpha)-deficient mice are protected from renal disease and have prolonged surv

238 express only talin1(L325R) in myeloid cells were protected from renal ischemia-reperfusion injury.

239 Ksp-CreERT2(+) mice crossed to p62(-/-) mice were protected from renal tumor development.

240 with decoy IL-21 receptor Fc fusion protein are protected from reperfusion injury.

241 Bacterial DNA is protected from restriction endonuclease cleavage by m

242 develop high intraocular pressure (IOP) but are protected from retinal ganglion cell (RGC) dysfuncti

243 rains, we have determined whether cattle can be protected from rinderpest by inoculation with vaccine

244 wever, only the animals given wild-type PPRV were protected from RPV challenge.

245 NSAID users were protected from SAB (OR = 0.78, 95% CI 0.56-1.10), w

246 ingle knockout IL-4(-/-) or IL-13(-/-) mice, were protected from Schistosoma-induced PH, with decreas

247 Interestingly, miR-155(-/-) mice were protected from SEB-mediated inflammation and lung i

248 g for more than 40 weeks, and these macaques were protected from several infectious challenges with S

249 he baboons in the P. knowlesi-only group and were protected from severe anemia.

250 contrast, animals vaccinated with MVA-H7-Sh2 were protected from severe disease.

251 an inoculum containing Nod1 x 2(-/-) T cells were protected from severe graft versus host disease.

252 C3H mice expressing WT Gusb as a transgene were protected from severe Lyme arthritis.

253 ozygotes and alpha(+)thalassemia homozygotes were protected from severe malaria (odds ratio [OR], 0.1

254 Baboons vaccinated with aP were protected from severe pertussis-associated symptoms

255 of three MHC-congenic strains, of which two were protected from severe PIA.

256 le stress fibers, whereas contractile fibers are protected from severing.

257 IL-20R1-deficient mice were protected from short-term and long-term liver injur

258 le explores the question of how the active X is protected from silencing by its own Xist locus, and t

259 d that mice lacking ephrin-B2 in fibroblasts are protected from skin and lung fibrosis and that a dis

260 n of Cosmc in 50% of crypts (IEC-Cosmc(+/-)) were protected from spontaneous inflammation and partial

261 hich also shows reduced muscle strength, but is protected from stretch-induced eccentric damage with

262 erm clearance of tumor after TLR7/RT therapy are protected from subsequent tumor rechallenge by the g

263 Vmac239 challenge, whereas only four animals were protected from subsequent intravenous SIVmac239 cha

264 ptible to WNV NS4B-P38G mutant infection but were protected from subsequent lethal wild-type WNV chal

265 ns were significantly higher in children who were protected from symptomatic malaria compared with th

266 ll-dependent antiviral immune responses, and are protected from T cell-mediated autoimmunity and allo

267 t neuronal cultures treated with Tat protein are protected from Tat-mediated cytotoxicity when treate

268 ned as "metabolically abnormal obese" (MAO), are protected from the adverse metabolic effects of weig

269 graphite die and the graphite punches, which are protected from the alumina fiber film by a graphite

270 In these dry films, the particle aggregates are protected from the environment during storage and ar

271 beta-HSD1 KO mice with circulating GC excess are protected from the glucose intolerance, hyperinsulin

272 caspases promote proliferation and how cells are protected from the potentially harmful action of apo

273 he lumen of beta-barrel OM proteins where it is protected from the hydrophobic membrane interior.

274 cetylated STAT3 emerged upon HDAC inhibition was protected from the proteasome-mediated degradation o

275 Intriguingly, Spns2(-/-) mice were protected from the development of experimental auto

276 e, OVA323-339, mice that received MPO409-428 were protected from the development of humoral and cell-

277 that lack HMGB1 in the intestinal epithelium were protected from the development of lung injury, conf

278 receptor Fcgamma receptor IIB (FcgammaRIIB) were protected from the disorder.

279 IFNAR1(-/-) mice were protected from the effects of hypoxia on the right

280 As expected, HCR rats were protected from the HFD.

281 Mice lacking RANKL in osteocytes were protected from the increase in osteoclast number an

282 Mice transfected with nucleolin were protected from the lethal effects of agonistic anti

283 nd mice lacking the endogenous receptor CD36 were protected from the neuroinflammatory and BBB permea

284 imilar organism burdens, but MyD88(-/-) mice were protected from the PcP-related respiratory impairme

285 Here we discover how the embryo is protected from these genotoxins.

286 e and C5aR were pharmacologically inhibited, were protected from these adverse effects and consequent

287 Gpx1 Tg mice were protected from this aging-related enhanced suscepti

288 ptosis in vitro, and Ripk1(D138N/D138N) mice are protected from TNF-induced shock in vivo.

289 Instead, the HPRT locus appeared to be protected from transposon integration.

290 dramatically altered: encapsulated peptides are protected from trypsin hydrolysis, whereas physicoch

291 nfection, genitally infected IL-10(-/-) mice were protected from tubal pathologies and infertility, w

292 ice lacking nCDase treated with azoxymethane were protected from tumor formation.

293 This data suggests that hESC-EC may be protected from unwanted TLR4-mediated vascular inflam

294 Because limbs are protected from use during a painful condition, this

295 Surprisingly, DNase-accessible euchromatin is protected from UV, while lamina-associated heterochro

296 Cancer cells are protected from VC-mediated cell death when co-cultur

297 Medullary hyperosmolarity is protected from washout by countercurrent exchange and

298 ate the mechanism(s) by which Pemt(-/-) mice are protected from weight gain and insulin resistance.

299 carrying the gene for the human PV receptor are protected from wild-type PV when immunized with the

300 deficiency in complement C3 or C3a receptor were protected from WNV-induced synaptic terminal loss.