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1 une-modulatory agents, cytostatic drugs, and biological response modifiers.
2  Interferons (IFNs) and retinoids are potent biological response modifiers.
3 ased therapies, including growth factors and biological response modifiers.
4  Interferons (IFNs) and retinoids are potent biological response modifiers.
5 uct with either leukocyte allo-antibodies or biological response modifiers.
6 eening of anticancer therapeutics, including biological response modifiers.
7  the xenotransplantation subcommittee of the Biological Response Modifiers Advisory Committee to disc
8 onundrum, and recent prospective trials with biological response modifiers aimed at the inflammatory
9 scular beds, as well as through secretion of biological response modifiers and recruitment of immune
10 rotein member of the TGF-beta superfamily of biological-response modifiers, causes regression of the
11                                              Biological response modifiers, cytoprotective drugs, sal
12                                PDT acts as a biological response modifier in addition to directly dam
13  increase in immunogenicity provided by this biological response modifier in an otherwise well-tolera
14         Ingested interferon (IFN)-alpha is a biological response modifier in experimental autoimmune
15 omodulatory properties and can function as a biological response modifier in vivo.
16   Issues concerning the safety of the use of biological response modifiers in inducing sarcoidosis ne
17 syl-(1-3)-beta-glucopyranose (PGG) glucan, a biological-response modifier, in protecting against mort
18                            This new class of biological response modifiers may be applicable to the c
19                    Moreover, the nonspecific biological response modifier monophosphoryl lipid A (MPL
20          However, a combination of these two biological response modifiers significantly suppressed t
21      Interleukin-7 (IL-7) is a proteinaceous biological response modifier that has a bioactive tertia
22 ulation, indicating that Thy-1 is a critical biological response modifier that protects against fibro
23         Moreover, pharmacological agents and biological response modifiers that independently display
24          Mounting evidence suggests that the biological response modifiers, which consistently produc
25 RA) represent a growing class of pleiotropic biological response modifiers with demonstrable efficacy

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