ライフサイエンスコーパス: blood protein

1 sent protein aggregates that contain several blood proteins.

2 ytic activity may influence cross-linking of blood proteins.

3 ed from fragments of extracellular matrix or blood proteins.

4 T cells to peptide-MHC complexes, results in blood protein Ags having a profound effect on thymic T c

5 s in our understanding of control of soluble blood proteins and blood cell receptors by functional di

6 h as extreme dilutions and interactions with blood proteins and cells.

7 whether DNA is either bound to aggregates of blood proteins and lipid micelles or intrinsically assoc

8 ics of this versatile interface, interfering blood proteins and potential interferences associated wi

9 t a >25 million-fold excess of contaminating blood proteins and represents a 4 order of magnitude imp

10 lead to undesired peptide interactions with blood proteins and self-aggregation due to exposed hydro

11 strongly conserved among vitamin K-dependent blood proteins and, in addition to a high relative conte

12 cursors to neuropeptides, synaptic proteins, blood proteins, and transporters.

13 ise the integrity of this barrier, and allow blood proteins as large as albumin to gain access to the

14 previously, including over 200 cleavages of blood proteins by aminopeptidases.

15 A production from glutamic acid derived from blood protein digestion.

16 Spectra indicated that blood proteins displaced the nanotube coating of synthet

17 effect of sample matrix (animal tissues and blood proteins, etc.).

18 ion has been paid to serum albumin, the main blood protein, even though blood disposal is a severe pr

19 Our findings not only suggest that blood protein expression is influenced by polymorphisms

20 , in which brain tissue becomes permeable to blood proteins, extravascular fibrin deposition correlat

21 Here we show that the blood protein fibrinogen induces rapid microglial respon

22 Here we show that the blood protein fibrinogen is an inhibitor of neurite outg

23 Here we show that the blood protein fibrinogen, which leaks into the CNS immed

24 y quenching with acetonitrile to precipitate blood proteins followed by analysis on an LC-electrospra

25 entation of various extracellular matrix and blood proteins generates antiangiogenic substances that

26 s identified in humans involves the abundant blood protein haptoglobin.

27 oach is demonstrated through the assays of a blood protein human alpha-thrombin and an oncoprotein hu

28 arriers, degradation of hemoglobin and other blood proteins, immune evasion, and activation of inflam

29 blood-brain barrier, the potential role for blood proteins in repair processes remains unknown.

30 on of the brain microenvironment by allowing blood proteins into the CNS.

31 and binding.Von Willebrand factor (VWF) is a blood protein involved in clotting and is proposed to be

32 g genes encoded stress response proteins and blood proteins involved in coagulation that were differe

33 Secretion of multiple blood proteins is a major drain on liver energy and nutr

34 However, direct capillary electrophoresis of blood proteins is challenging due to its high content of

35 We show that initial degradation of host blood proteins is ordered, occasionally redundant, and s

36 attachment to extracellular matrices and to blood proteins is regulated from inside the cell.

37 he second data set to simulate variations in blood protein levels.

38 in reversing inactivation by the mixtures of blood proteins, membrane lipids, and fatty acids studied

39 about 70% of circulating Hcy is N-linked to blood proteins, mostly to hemoglobin and albumin.

40 illebrand factor, an ultralarge concatemeric blood protein, must bind to platelet GPIbalpha during bl

41 Basket samples yielded collagen and blood proteins of bovine origin (Bos genus) and a large

42 Using hen egg-white lysozyme, the effect of blood proteins on CD4 thymic cells was examined.

43 the effects of human serum albumin, a major blood protein, on this phase separation.

44 cts from reactions with nucleophilic loci of blood proteins, particularly Cys34 of human serum albumi

45 Measurement of glycated blood proteins, particularly glycated hemoglobin (HbA1c)

46 ib showed a wider spectrum of cardiovascular blood protein reduction, but the protein reduction effec

47 ciated with cardiovascular risks, we studied blood proteins related to inflammation and cardiovascula

48 (SWCNT) and use it against a panel of human blood proteins, revealing a specific corona phase that r

49 lar dynamics simulations to demonstrate that blood proteins such as bovine fibrinogen (BFG) can absor

50 ften observed during the binding of drugs to blood proteins such as human serum albumin (HSA).

51 HNE adduction of blood proteins, such as human serum albumin (HSA), yield

52 Albumin is an abundant blood protein that acts as a transporter of a plethora o

53 of inhibitory proteoglycans is induced by a blood protein that leaks in the CNS after vasculature ru

54 our knowledge, this is the first study with blood proteins to show that, in addition to the aromatic

55 sed of tryptic peptides from human and mouse blood proteins using high-resolution time-of-flight mass

56 ature of the associations between correlated blood proteins utilizing Mendelian randomization.

57 function of the mechanosensitive, multimeric blood protein von Willebrand factor (VWF) is dependent o

58 acid (bis-ANS) to hydrophobic pockets in the blood protein von Willebrand factor (VWF) is enhanced up

59 A total of 157 blood proteins were quantified by a proximity extension

60 SNPs influencing six blood proteins were used to evaluate the nature of the a