ライフサイエンスコーパス: bone marrow chimera

1 etic or nonhematopoietic cells, we generated bone marrow chimeras.

2 tion of Ag-specific IMP CD8(+) T cells using bone marrow chimeras.

3 egress was demonstrated by generating mixed bone marrow chimeras.

4 an anti-tumor effect induced by RLI in mixed bone marrow chimeras.

5 stitute the Treg population in the thymus of bone marrow chimeras.

6 g excretion compared with infected wild-type bone marrow chimeras.

7 and on circulating CD8 T cells in the mixed bone marrow chimeras.

8 hemokine Grobeta was abrogated in CXCR4(-/-) bone marrow chimeras.

9 PLZF(-/-) mice and mixed wild-type:PLZF(-/-) bone marrow chimeras.

10 rant of MHC class I-deficient cells in mixed bone marrow chimeras.

11 orylation and reduced platelet production in bone marrow chimeras.

12 sponses of flagellin-treated MyD88 radiation bone marrow chimeras.

13 of CD83-cKO B cells in GC responses in mixed bone marrow chimeras.

14 ls and intrinsic renal cells, we constructed bone marrow chimeras.

15 leting peripheral CD4 T cells or by creating bone marrow chimeras.

16 elium from that of hemopoietic APCs by using bone marrow chimeras.

17 we performed fate-tracing experiments using bone marrow chimeras.

18 ted HO-2(-/-) and wild-type (WT) mice and in bone marrow chimeras.

19 nd this relationship is recapitulated in the bone marrow chimeras.

20 In comparison with BAFF- or APRIL-sufficient bone marrow chimeras, absence of hematopoietic compartme

21 nal hematopoiesis emerged in Casp9-deficient bone marrow chimeras after alkylator exposure.

22 same observation in PHD2(+/+) --> PHD2(+/-) bone marrow chimeras also suggests organization of a bet

23 ce showed a reduced CHS response to DNFB, in bone marrow chimera and adoptive transfer experiments, w

24 ion after DSS-induced injury, as revealed by bone marrow chimera and dendritic cell-depletion experim

25 models and monocytopenic mice, together with bone marrow chimera and parabiotic models, we found that

26 mune responses to HSV-1 were dissected using bone marrow chimeras and adoptive cell transfer approach

27 In this study, we used bone marrow chimeras and adoptive transfer analysis to i

28 Using mixed bone marrow chimeras and adoptive transfer studies in wh

29 To test this, we created bone marrow chimeras and cell-type-specific knockouts of

30 We then used bone marrow chimeras and fetal liver reconstitutions to

31 identify the relevant APC in K14 mice using bone marrow chimeras and found that radioresistant cells

32 Using mixed bone marrow chimeras and Foxo1-deficient mice, we demons

33 function, and we confirmed that result with bone marrow chimeras and in vitro, where the absence of

34 c role in type 2 responses was explored with bone marrow chimeras and induction of gastrointestinal t

35 Using a combination of bone marrow chimeras and intracellular chemokine stainin

36 We analyzed bone marrow chimeras and mice with a conditional gain-of

37 Analyses using bone marrow chimeras and mutant mice demonstrate that su

38 CD8(+) T-cell response to the VP2 peptide in bone marrow chimeras and mutant mice lacking peripheral

39 rin(+) cells in vivo, together with congenic bone marrow chimeras and parabiotic mice as tools to dif

40 iNKT thymic development in limited-dilution bone marrow chimeras and show that higher TCR avidity co

41 Results from radiation bone marrow chimeras and TCR transgenic mice indicate th

42 nduces a host-versus-graft reaction in mixed bone marrow chimeras and that rejection of donor cells l

43 CNS during EAE development, we created CD24 bone marrow chimeras and transgenic mice in which CD24 e

44 mice was shown to be B cell intrinsic using bone marrow chimeras and was not due to a developmental

45 s, HLA-DR4-transgenic mice, MAIThighHLA-DR4+ bone marrow chimeras, and humanized NOD-scid IL-2Rgamman

46 13 inbred mouse strains, F1 and F2 hybrids, bone marrow chimeras, and neutrophil function assays.

47 h the use of adoptively transferred T-cells, bone marrow chimeras, and reconstituted severe combined

48 We generated MD-2(-/-) bone marrow chimeras, and showed that MD-2 expression on

49 Using a bone marrow chimera approach, we further demonstrated th

50 Using a novel minimal myeloablation-bone marrow chimera approach, we visualized priming of e

51 , we used adoptive transfer, transgenic, and bone marrow chimera approaches to show increased infiltr

52 dependent conditional Blimp-1 knockout mixed bone marrow chimera as well as an adoptive transfer appr

53 ed wild-type (wt)-->MHCI-/- or wt-->MHCII-/- bone marrow chimeras as recipients in GVHD experiments.

54 e Foxp3(-) precursors using studies of mixed bone marrow chimeras as well as TCR-specific generation

55 In mixed-bone marrow chimera assays, we found that CCR4-deficient

56 can differentiate successfully in radiation bone marrow chimera bearing normal cells.

57 For dissection between the two, bone marrow chimeras between Cfh-deficient (Cfh(-/-)) an

58 Analysis of bone marrow chimeras between Chop(-/-) and Chop(+/+) mic

59 levant accessory cells in vivo, we generated bone marrow chimeras between either wild-type (WT) and M

60 Radiation bone marrow chimeras between normal and C3(-/-) mice wer

61 T regulatory cells (Tregs), we studied mixed bone marrow chimeras between wild-type and IFN-alpha/bet

62 Reciprocal bone marrow chimeras between wild-type and TLR4 mutant m

63 role of TLR4 in lung protection, using used bone marrow chimeras; cell-specific transgenic modeling;

64 Mixed bone marrow chimeras, conditional knockout mice, and ado

65 Bone marrow chimeras confirmed that the myeloproliferati

66 Irradiated bone marrow chimeras confirmed the protective effects of

67 o failed to inhibit EAE in reciprocal, mixed bone marrow chimeras constructed by transferring IL-10-d

68 beta T cells are poorly established in mixed bone marrow chimeras, contrasting with strong gammadelta

69 In mixed bone marrow chimeras, Crel(-/-)Nfkappab1(-/-)Rela(+/-) n

70 Our studies with bone marrow chimeras demonstrate that PD-L1/PD-L2 expres

71 VitD receptor-deficient (vdr(-/-)) radiation bone marrow chimeras demonstrate that reductions in pulm

72 Bone marrow chimeras demonstrate that this inflammatory

73 Competitive bone marrow chimera demonstrated that intact IRAK4 funct

74 Bone marrow chimera demonstrated that mucosa-derived MMP

75 Mixed bone marrow chimera demonstrated that the increased acti

76 Mixed bone marrow chimeras demonstrated a B cell-intrinsic req

77 Bone marrow chimeras demonstrated retardation of T cell

78 Experiments using bone marrow chimeras demonstrated that AKT3(-/-) mice re

79 Bone marrow chimeras demonstrated that alpha2,6-sialylat

80 Bone marrow chimeras demonstrated that loss of H2-O expr

81 Moreover, bone marrow chimeras demonstrated that LTbR deficiency i

82 Experiments in IL-7Ralpha(-/-) bone marrow chimeras demonstrated that the drainage-enha

83 Finally, mixed bone marrow chimeras demonstrated that the worsening of

84 Mixed bone marrow chimeras demonstrated that these phenotypes

85 Bone marrow chimeras demonstrated that TIMP3 derived fro

86 The use of bone marrow chimeras determined that deletion of caspase

87 S. mansoni-infected Arg I-deficient bone marrow chimeras develop a marked accumulation of wo

88 In mixed bone marrow chimeras, Dpy30-deficient HSCs cannot differ

89 TFH cells developed poorly in a competitive bone marrow chimera environment.

90 Bone marrow chimeras established that CD73 expression in

91 In vivo, ARAP3 PH domain point mutant bone marrow chimeras exhibit reduced neutrophil recruitm

92 Lrrc8a(-/-) mice and Lrrc8a(-/-)-->Rag2(-/-) bone marrow chimeras exhibited a severe cell-intrinsic b

93 Bone marrow chimera experiment demonstrated that T cell-

94 Bone marrow chimera experiments confirmed that microglia

95 Mixed bone marrow chimera experiments demonstrate that IL-27 d

96 Bone marrow chimera experiments demonstrated that CXCR3

97 Bone marrow chimera experiments demonstrated that hemato

98 Finally, bone marrow chimera experiments demonstrated that Ifitm3

99 Competitive bone marrow chimera experiments demonstrated that the de

100 Bone marrow chimera experiments demonstrated that TNF ca

101 Bone marrow chimera experiments determined that maximal

102 Bone marrow chimera experiments indicate that TEC expres

103 Bone marrow chimera experiments indicated that CRAMP der

104 Bone marrow chimera experiments indicated that IRAK-M ex

105 Bone marrow chimera experiments indicated that LRIG1 als

106 Bone marrow chimera experiments indicated that PAD4 in h

107 Bone marrow chimera experiments indicated that the obser

108 Mixed bone marrow chimera experiments reveal a markedly reduce

109 In addition, competitive bone marrow chimera experiments reveal that RasGRP1/3 do

110 Mixed bone marrow chimera experiments reveal that, under homeo

111 Bone marrow chimera experiments revealed a cell-intrinsi

112 Bone marrow chimera experiments revealed a T cell-autono

113 Bone marrow chimera experiments revealed that HMGN1 deri

114 Furthermore, bone marrow chimera experiments revealed that MDA5 expre

115 Red blood cell transfer and bone marrow chimera experiments show that the aging phen

116 However, intrathymic injection and bone marrow chimera experiments showed a saturable incre

117 Finally, mixed bone marrow chimera experiments showed that both the pro

118 Bone marrow chimera experiments showed that hematopoieti

119 Bone marrow chimera experiments showed that resistance i

120 Mixed bone marrow chimera experiments showed this paucity to b

121 prevent autoreactive T cells from deletion; bone marrow chimera experiments suggest that CD24 on rad

122 We show through mixed bone marrow chimera experiments that NKT17 polarization

123 Mixed bone marrow chimera experiments using cells from Flip-de

124 Bone marrow chimera experiments using OVA-treated C57BL/

125 Bone marrow chimera experiments using Rag-1 and beta2-mi

126 Bone marrow chimera experiments verified that bone marro

127 Bone marrow chimera experiments were performed with mice

128 As observed by bone marrow chimera experiments, Mac-1-dependent neutrop

129 In reciprocal bone marrow chimera experiments, the protective phenotyp

130 tion by structural cells was corroborated by bone marrow chimera experiments.

131 -b deficiency in macrophages, as assessed by bone marrow chimera experiments.

132 ressing core protein throughout the body and bone marrow chimeras expressing core protein in either t

133 In experiments with bone marrow chimeras, expression of BCL3 by acinar cells

134 T cells from the STAT6-/- bone marrow chimeras failed to recognize the STAT6(531-5

135 in LPL-deficient hematopoietic cells, using bone marrow chimeras, failed to rescue the phenotype of

136 HNF1A(-/-) bone marrow chimera featured a dramatic defect in B lymp

137 Bone marrow chimeras formed by the transfer of WT bone m

138 Rag2(-/-), but not WT-->IkappaBalpha mutant, bone marrow chimeras formed proper lymphoid organs and d

139 in naive CD8(+) T cells in vivo by creating bone marrow chimera from hematopoietic progenitors trans

140 s to trans present IL-15, we generated mixed bone marrow chimera from IL-15Ralpha- and IL-2/15Rbeta-d

141 Analysis of bone marrow chimera generated from a mixture of wild-typ

142 ied using reciprocal hematopoietic radiation bone marrow chimeras generated between male wild-type an

143 Using a series of bone marrow chimeras generated from 5-lipoxygenase(-/-)

144 The current study used bone marrow chimeras generated from green fluorescent pr

145 mal alpha7nAChRs, as shown by experiments in bone marrow chimeras generated with wild-type and alpha7

146 In competitive bone marrow chimeras, however, LN3alphabeta thymocytes w

147 Sepsis induction in C5aR(+)/C5aR(-) mixed bone marrow chimeras identified cognate engagement of C5

148 Studies using bone marrow chimeras implicate lung resident hematopoiet

149 Next, we used bone marrow chimeras in an experimental colitis model an

150 cause fibrotic susceptibility, we generated bone marrow chimeras in HPS and wild-type mice.

151 Numerous studies of bone marrow chimeras in the human and the mouse point to

152 e T-B interactions, we constructed radiation bone marrow chimeras in which CD4+ T cells would be acti

153 the development of PGIA, we generated mixed bone marrow chimeras in which CD80/CD86 is specifically

154 addressed through analyses of NOD background bone marrow chimeras in which H2(nb1) molecules were sel

155 ized in the transgenic mice and in radiation bone marrow chimeras in which ligand-bearing mice served

156 ing wild-type and DNMAML T cells together in bone marrow chimeras increased accumulation of Notch-dep

157 EAE adoptive transfer experiments and bone marrow chimeras indicated that expression of LIGHT

158 ransgenic CD4 T cells and studies with mixed bone marrow chimeras indicated that indirect effects and

159 Reciprocal bone marrow chimeras indicated that IRF-3 or IRF-7 expre

160 Experiments with bone marrow chimeras indicated that RSV-induced lung pat

161 Studies with mixed bone marrow chimeras indicated that the defect is intrin

162 ellular MZ microenvironment, and analysis of bone marrow chimeras indicated that the MZ B cell develo

163 LTbeta(-/-), LTalpha(+/-)beta(+/-) mice and bone marrow chimeras, indicated that rapid protective Th

164 Analysis of bone marrow chimeras indicates that both forms of HEL ar

165 Mouse bone marrow chimeras lacking Casp9 or its cofactor Apaf1

166 Bone marrow chimeras, luminex, and quantitative reverse

167 In this study we found that in bone marrow chimeras made by reconstituting lethally irr

168 In parallel, Mertk(-/-)/Mfge8(-/-) bone marrow chimeras manifested increased accumulation o

169 Experiments with bone marrow chimera, mast cell-deficient animals, platel

170 tion of CD4(+) T cell adoptive transfers and bone marrow chimera mice in which the presence or absenc

171 autoimmune encephalomyelitis (EAE), we used bone marrow chimera mice that allowed us to distinguish

172 e macrophage inflammatory phenotype by using bone marrow chimera mice with conditional PU.1 knockout.

173 g the use of p47(phox-/-) mice, p47(phox-/-) bone marrow chimera mice, adoptive transfer of macrophag

174 Using bone marrow chimera mice, we demonstrated that CD73 expr

175 nflamed cremaster muscle microcirculation in bone marrow chimera mice.

176 tors can also give rise to CD8alpha(+) DC in bone marrow chimera mice.

177 In addition, IL-1R1(KO) bone marrow-chimera mice showed that IL-1R1 expression w

178 We have used a mixed bone marrow chimera model to generate mice in which the

179 Using a bone marrow chimera model, we determined that IRAK-M's e

180 ze of a differentiating Treg population in a bone marrow chimera model, which correlated with reduced

181 Bone marrow chimera models were used to localize the sit

182 In radiation bone marrow chimeras, NK resistance was significantly di

183 In this study, we used bone marrow chimeras of IFN-alphabetagammaR(-/-) (AG129)

184 Bone marrow chimeras of mixed CCR2+/+ green fluorescent

185 Bone marrow chimeras of MyD88 deficient/wild type and TR

186 Restoring CCR4 on myeloid cells in bone marrow chimeras or intracerebral microinjection of

187 Once niche effects are neutralized in mixed bone marrow chimeras, positive and negative selection ar

188 Bone marrow chimeras produced using Cxcl5(-/-) donors an

189 CD28 wild-type/CD28-deficient mixed bone marrow chimeras provided evidence of both cell-auto

190 In bone marrow chimeras, radioresistant and, likely, nonhem

191 s transferred into wild-type mice, but mixed bone marrow chimeras receiving wild-type and Crel(-/-)Nf

192 emergence of DN PD-1(-/-) 2C cells in H-2(k) bone marrow chimera recipients.

193 examined this possibility using XX versus XY bone marrow chimeras reconstituted with a common immune

194 Moreover, using mixed bone marrow chimeras reconstituted with Bim(+/+) and Bim

195 in neutrophil trafficking using mouse mixed bone marrow chimeras reconstituted with Cxcr2(-/-) and W

196 was abrogated in IL-4R-deficient mice and in bone marrow chimeras reconstituted with myeloid cells th

197 Mixed xenogeneic bone marrow chimeras resulting from the transplantation

198 l populations, and DT injection into zDC-DTR bone marrow chimeras results in cDC depletion.

199 Adoptive transfer experiments and radiation bone marrow chimera reveal that a Txb21(+/+) environment

200 Analyses of reciprocal bone marrow chimeras reveal that the magnitude and produ

201 .L2 WT and n3.L2 PKCtheta(-/-) thymocytes in bone marrow chimeras revealed a more dramatic defect, wi

202 Moreover, bone marrow chimeras revealed a role for type I IFN sign

203 Bone marrow chimeras revealed a specific reduction in AT

204 Mixed bone marrow chimeras revealed an alteration of immune re

205 The analysis of mixed bone marrow chimeras revealed that cGVHD resistance was

206 Other experiments with bone marrow chimeras revealed that inflammation was not

207 Analyzing mixed bone marrow chimeras revealed that intact Zap70-dependen

208 Moreover, bone marrow chimeras revealed that lymphocyte survival r

209 However, analysis of mixed bone marrow chimeras revealed that Nfkb1(SSAA/SSAA) FM B

210 Mixed radiation bone marrow chimeras revealed that RORgamma functions in

211 Mixed bone marrow chimeras revealed that the aforementioned gp

212 Reciprocal bone marrow chimeras revealed that the aged microenviron

213 Bone marrow chimeras revealed that TLR4 expression on he

214 Experiments using bone-marrow chimeras revealed that it is the lack of A(2

215 Experiments with bone marrow chimeras (S1pr2(+/+) --> S1pr2(+/+), S1pr2(+

216 Bone marrow chimeras showed that bone marrow-derived cel

217 Experiments using bone marrow chimeras showed that FcRn expression in thes

218 Bone marrow chimeras showed that Mif expression in bone

219 Neutrophil transmigration studies and bone marrow chimeras showed that neutrophil MMP-9 is nei

220 Bone marrow chimeras showed that platelet, not endotheli

221 Flow cytometry and GFP bone marrow chimeras showed that spinal cord microglia w

222 Bone marrow chimeras showed that the anti-inflammatory r

223 Rather, mixed bone marrow chimeras showed that the factor(s) responsib

224 Evidence from mixed bone marrow chimeras shows that the ability of Egr1 to c

225 Bone marrow chimera studies demonstrate that mTORC1 cont

226 Bone marrow chimera studies revealed that STAT6 expresse

227 Consistent with this, bone marrow chimera studies show that aberrant Pkhd1 mus

228 Bone marrow chimera studies showed reduced lesions in ap

229 Bone marrow chimera studies suggest that pulmonary paren

230 In bone marrow chimera studies, recipient irradiated Ifnar(

231 From adoptive reconstitution and mixed bone-marrow chimera studies in B cell-deficient (microMT

232 nflammatory responses, and lipid uptake, and bone marrow chimeras suggest that hematopoietic EphA2 de

233 tion of Ly6-G(+) cells and use of C5aR1(-/-) bone marrow chimeras suggested an essential role of C5aR

234 sing from bone marrow precursors in neonatal bone marrow chimeras suggested that Foxp3(+) DP cells ar

235 e with wild-type T cell progenitors in mixed bone marrow chimeras, suggesting that in normal mice, th

236 ed iNKT cell frequencies in Myb heterozygous bone marrow chimeras suggests that miR-150 differentiall

237 In bone marrow chimeras, synthesis of C3 by radioresistant

238 rim24 deficiency to T cells by using a mixed bone marrow chimera system and found that T-cell-intrins

239 kely to contribute to disease, we utilized a bone marrow chimera system to distinguish between these

240 using IFN-gamma receptor knockout mice in a bone marrow chimera system, we show that the IFN-gamma p

241 ow-density lipoprotein-deficient (Ldlr(-/-)) bone marrow chimeras that express a transgene containing

242 ment of systemic autoimmune disease by using bone marrow chimeras that lacked expression of major his

243 Intriguingly, in mixed bone marrow chimeras the phenotype conferred by DTR-expr

244 In bone marrow chimeras, the development of DC cancer could

245 Using TLR4-deficient bone marrow chimeras, the priming phenotype was restrict

246 In these studies, we first used bone marrow chimeras to demonstrate a requirement for My

247 viduals and, more importantly, use radiation bone marrow chimeras to demonstrate that restriction of

248 ing sepsis, we used CXCL1-deficient mice and bone marrow chimeras to demonstrate the importance of th

249 We used bone marrow chimeras to determine if SAMP ileitis result

250 In this study, we use bone marrow chimeras to show that clinical and histologi

251 use mixed G-CSF receptor (G-CSFR)-deficient bone marrow chimeras to show that G-CSF-induced mobiliza

252 In mixed bone marrow chimeras, Tpl2 was shown to play a T cell-in

253 ly inhibited in Selplg(-/-) mice or Syk(-/-) bone-marrow chimeras treated with pertussis toxin.

254 n murine T cell function, we generated mixed bone marrow chimeras using bone marrow from NIK knockout

255 ulation in the intestines of Arg I-deficient bone marrow chimeras was associated with intestinal hemo

256 Furthermore, using bone marrow chimeras we show that deletion of AhR in the

257 Using mixed bone marrow chimeras we then determined that MyD88 expre

258 phosphate (NADPH) oxidase 2 (NOX2) and their bone marrow chimera, we demonstrated that NOX2 from both

259 Using MHC class II (MHC II) mixed bone marrow chimeras, we compared the bacterial burdens

260 Using mixed bone marrow chimeras, we compared wild-type and cytokine

261 Using reaggregated thymic organ culture and bone marrow chimeras, we demonstrate that positive selec

262 In addition, using bone marrow chimeras, we demonstrate that PVM are the ce

263 al microscopy with DREAM-null mice and their bone marrow chimeras, we demonstrated that both hematopo

264 First, using mixed bone marrow chimeras, we established that Ag-targeted, b

265 Using mixed bone marrow chimeras, we evaluated the effect of TSSP de

266 Using mixed bone marrow chimeras, we found that activating Fc recept

267 horiomeningitis virus-mediated hepatitis and bone marrow chimeras, we found that Bim has a dual role

268 forms of the serine/threonine kinase AKT and bone marrow chimeras, we found that hematopoietic cell-a

269 , PLZF reporter/fate mapping mice, and mixed bone marrow chimeras, we identified two distinct populat

270 Using retroviral vectors and bone marrow chimeras, we observed similar activity with

271 Using mixed bone marrow chimeras, we observed that IL-6 enhances ASC

272 receptor (TCR)-transgenic T cells and mixed bone marrow chimeras, we show that activation of naive p

273 udy, using adoptive transfer experiments and bone marrow chimeras, we show that at least some of thes

274 Using conditional mutants and bone marrow chimeras, we show that intravascular activat

275 Using bone marrow chimeras, we show that lymphohematopoietic c

276 sing reciprocal adoptive transfer models and bone marrow chimeras, we show that Myd88(-/-) CD8 T cell

277 Finally, by generating mixed bone marrow chimeras, we showed that the effect of Sle1b

278 Using bone-marrow chimeras, we found that the signaling molecu

279 Bone marrow chimeras were constructed to test the effect

280 Bone marrow chimeras were constructed with TLR4 only on

281 Bone marrow chimeras were generated and similarly treate

282 esident cells contribute to ischemic injury, bone marrow chimeras were generated by transplanting bon

283 Bone marrow chimeras were generated by transplanting ICO

284 Mixed bone marrow chimeras were generated to assess the effect

285 Bone marrow chimeras were generated using MKK-3(-/-) and

286 in T cells under noncompetitive conditions, bone marrow chimeras were generated, in which the T cell

287 endothelium or other nonhematopoietic cells, bone marrow chimeras were generated.

288 protein receptor-deficient, S100A9-deficient bone marrow chimeras were generated.

289 sangial cells in pathogenesis, (NZB x NZW)F1 bone marrow chimeras were generated.

290 Bone marrow chimeras were performed between young and ag

291 Bone marrow chimeras were performed using bone marrow ce

292 F(-/-) mice, wild-type littermates, or mixed bone marrow chimeras were treated with the protease alle

293 ss accumulation in the inflamed CNS in mixed bone marrow chimeras, when compared with CCR2(+/+) DCs;

294 ock-in mice as well as Sap(R78A) competitive bone marrow chimeras, which retained NKT cells but at si

295 lls (TECs) in Treg selection, we constructed bone marrow chimeras with disrupted CD28/B7 signaling in

296 To clarify the latter, we generated murine bone marrow chimeras with enforced Erg expression in eng

297 a by differential CD45 staining and by using bone marrow chimeras with fluorescent leukocytes.

298 endothelium-specific deficiency in JAM-A and bone marrow chimeras with JAM-A-deficient leukocytes.

299 Using IL-15(WT/WT) and IL-15(-/-) mice, bone marrow chimeras with normal cellularity, and a sele

300 lation on wild-type bystander cells in mixed bone-marrow chimeras with KLF2-deficient cells.