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1 g adhesive interactions of MM cells with the bone marrow microenvironment.
2 al for survival of leukemic cells within the bone marrow microenvironment.
3 ly strong dependence on interaction with the bone marrow microenvironment.
4 actor on hematopoietic stem cells within the bone marrow microenvironment.
5 us bone formation and the homeostasis of the bone marrow microenvironment.
6 irectly or indirectly through effects on the bone marrow microenvironment.
7 ng that HNK inhibits neovascurization in the bone marrow microenvironment.
8 interactions between myeloma cells and their bone marrow microenvironment.
9 oth soluble factors and cell contacts in the bone marrow microenvironment.
10 suggest that most TSP2 is acquired from the bone marrow microenvironment.
11 y to overcome the stimulatory effects of the bone marrow microenvironment.
12 fficking and localization of MM cells in the bone marrow microenvironment.
13 W654652 acts directly on MM cells and in the bone marrow microenvironment.
14 and maturation of B-cell progenitors in the bone marrow microenvironment.
15 87 acts both directly on MM cells and in the bone marrow microenvironment.
16 stitution in the presence of an intact human bone marrow microenvironment.
17 ng at the level of B cell development in the bone marrow microenvironment.
18 eractions within the cytoarchitecture of the bone marrow microenvironment.
19 anulocytic precursors within fetal liver and bone marrow microenvironment.
20 y for hematopoiesis to be established in the bone marrow microenvironment.
21 between interleukin-5, eosinophils, and the bone marrow microenvironment.
22 the leukemic cells in a leukemia-permissive bone marrow microenvironment.
23 pond appropriately and/or survive within the bone marrow microenvironment.
24 self-renewal and megakaryocytopoiesis in the bone marrow microenvironment.
25 ression and represent a unique target in the bone marrow microenvironment.
26 , immune abnormalities, and disorders of the bone marrow microenvironment.
27 r and molecular impact of osteoblasts on the bone marrow microenvironment.
28 pproach would be effective in the protective bone marrow microenvironment.
29 intained under hypoxic conditions within the bone marrow microenvironment.
30 PN development induced by the Ptpn11-mutated bone marrow microenvironment.
31 ate the expansive cell-cell crosstalk in the bone marrow microenvironment.
32 static factor secreted by osteoblasts in the bone marrow microenvironment.
33 mechanism by which ALL cells modulate their bone marrow microenvironment.
34 is with a particular emphasis on the role of bone marrow microenvironment.
35 he crosstalk between BMSCs and T(reg) in the bone marrow microenvironment.
36 ns between MM cells and stromal cells of the bone marrow microenvironment.
37 rimary MM cells alone and in the presence of bone marrow microenvironment.
38 essive loss of stem cells from the postnatal bone marrow microenvironment.
39 stem cell recruiting agent, TGF-beta1 in the bone marrow microenvironment.
40 d in their respective niches by cells of the bone marrow microenvironment.
41 of myeloid cells and vascularization of the bone marrow microenvironment.
42 endent upon cellular interactions within the bone marrow microenvironment.
43 t, differentiation, and interaction with the bone marrow microenvironment.
44 esumably because of improved survival in the bone marrow microenvironment.
45 ltered ribosome function, and changes in the bone marrow microenvironment.
46 n which tumor cells receive signals from the bone marrow microenvironment.
47 able of providing RA to DC precursors in the bone marrow microenvironment.
48 hich may contribute to independence from the bone marrow microenvironment.
49 argeting both multiple myeloma cells and the bone marrow microenvironment.
50 utrophils and hematopoietic cells within the bone marrow microenvironment.
51 he expression of the chemokine CXCL12 in the bone marrow microenvironment.
52 ng migration and adhesion of WM cells in the bone marrow microenvironment.
53 leukemic cells by targeting their protective bone marrow microenvironment.
54 nitor cell movement and adherence within the bone marrow microenvironment.
55 ukemic stem cells is clonal dominance of the bone marrow microenvironment.
56 al hematopoiesis in fetal liver, spleen, and bone marrow microenvironments.
57 nstrates an alternative pathway in which the bone marrow microenvironment also supports the different
58 o study consisted of cells isolated from the bone marrow microenvironment and cultured on feeder laye
59 roliferative disorders involve targeting the bone marrow microenvironment and DNA hypermethylation.
60 Multiple myeloma (MM) cells reside in the bone marrow microenvironment and form complicated intera
62 veal a new role of NPY as a regulator of the bone marrow microenvironment and highlight the potential
63 complement each other by targeting both the bone marrow microenvironment and hypomethylating action
64 he extrinsic components are generated by the bone marrow microenvironment and include chemokine recep
65 -alpha (TNF-alpha) is present locally in the bone marrow microenvironment and induces NF-kappaB-depen
66 tezomib also decreased VEGF secretion in the bone marrow microenvironment and inhibited VEGF-triggere
69 the regulation of HSC/P retention within the bone marrow microenvironment and migration between the b
70 nd survival of multiple myeloma cells in the bone marrow microenvironment and strongly support novel
71 nxa2 regulates HSC homing and binding to the bone marrow microenvironment and suggest that Anxa2 is c
72 lls exert an aberrant systemic effect on the bone marrow microenvironment and VEGF-A/VEGFR targeting
73 ) cells, inhibits binding of MM cells in the bone marrow microenvironment, and inhibits cytokines med
74 as predominantly negative regulators of the bone-marrow microenvironment, and indicate that antagoni
75 nic cytokines and related alterations of the bone marrow microenvironment are commonly found in SM.
76 ultiple myeloma and stromal cells within the bone marrow microenvironment are essential for myeloma c
77 progenitor cell (HSPC) interactions with the bone marrow microenvironment are important for maintaini
78 st that inhibitory cytokines produced by the bone marrow microenvironment are likely to be involved i
80 presumed to reside in specific niches in the bone marrow microenvironment (BMM) and may be the cause
84 aintaining immunological homoeostasis in the bone marrow microenvironment, both in physiological cond
85 ogenic when stimulated by FGF ligands in the bone marrow microenvironment, but is also a target for a
86 to respond to extracellular signals from the bone marrow microenvironment, but the critical intracell
87 tosis and altered adhesive properties to the bone marrow microenvironment by BCR/ABL might contribute
88 mors can achieve increased autonomy from the bone marrow microenvironment by mutations that activate
89 peutic strategy to eradicate leukemia in the bone marrow microenvironment by simultaneous targeting o
90 seous sites, increasing Wnt signaling in the bone marrow microenvironment can prevent the development
91 vidence for a multitude of ways in which the bone marrow microenvironment can promote disease pathoge
92 f acute myeloid leukemia (AML) blasts in the bone marrow microenvironment confers protection from che
94 not rescued by transplantation into a young bone marrow microenvironment, demonstrating cell-autonom
95 e brain, the suprachiasmatic nucleus, to the bone marrow microenvironment, directing circadian oscill
98 be altered by genetical manipulation of the bone marrow microenvironment: Extramedullary bone marrow
99 l that T-ALL cells do not depend on specific bone marrow microenvironments for propagation of disease
100 granular lymphocyte (LGL) leukemia, but the bone marrow microenvironment has not been systematically
101 stem cells (HSCs) in culture outside of the bone marrow microenvironment has severely limited their
102 clude that despite the hypoxic nature of the bone marrow microenvironment, Hif-1alpha is dispensable
103 ressing AML cells directly in the SDF-1-rich bone marrow microenvironment if the survival cues of the
104 he triad of laboratory models that mimic the bone marrow microenvironment, immunodeficiency diseases
105 eoblasts but was a consequence of an altered bone marrow microenvironment imposed by Gsalpha deficien
106 (PAH) to induce B cell apoptosis within the bone marrow microenvironment in a clonally nonspecific w
107 ible to recreate all cellular aspects of the bone marrow microenvironment in an in vitro system, and
108 nd/or Bcl-xL We investigated the role of the bone marrow microenvironment in determining Bcl-2 family
109 been accumulating to support the role of the bone marrow microenvironment in hematologic malignancies
110 nsights into the the instructive role of the bone marrow microenvironment in hematological malignanci
111 ng evidence supporting the importance of the bone marrow microenvironment in LSC survival and conside
113 These observations suggest a role for the bone marrow microenvironment in modulating the response
115 whether increasing Wnt signaling within the bone marrow microenvironment in myeloma counteracts deve
116 population of cells and on cells within the bone marrow microenvironment in normal and incisors abse
117 to determine if transplantation of an intact bone marrow microenvironment in the form of a bone graft
119 amic analysis of T-ALL interactions with the bone marrow microenvironment in vivo, supported by evide
120 py to target early survival signals from the bone marrow microenvironment, in particular FGF2, to imp
121 olecules required for the function of normal bone marrow microenvironments, in this case through the
126 exosomes in developing the AML niche of the bone marrow microenvironment, investigating their biogen
127 The interaction of stem cells with their bone marrow microenvironment is a critical process in ma
130 to recapitulate the conditions by which the bone marrow microenvironment is formed and establish com
131 between acute myeloid leukemia (AML) and the bone marrow microenvironment is known to control disease
134 P-BEZ235 targeted WM cells in the context of bone marrow microenvironment, leading to significant inh
137 between human prostate cancer cells and the bone marrow microenvironment mediate bone metastasis dur
138 rovide evidence that gp130 expression in the bone marrow microenvironment, most likely in endothelial
140 ta suggest that increased IL-3 levels in the bone marrow microenvironment of MM patients with imbalan
141 ls, and mounting evidence indicates that the bone marrow microenvironment of tumour cells has a pivot
142 contributes to the favorable effects of the bone marrow microenvironment on megakaryocyte developmen
143 gnancy due, in part, to the influence of the bone marrow microenvironment on survival and drug respon
144 mplex bidirectional interactions between the bone marrow microenvironment (or niche) and hematopoieti
145 he interaction between myeloma cells and the bone marrow microenvironment, point to similar interacti
146 hat Ptpn11 activating mutations in the mouse bone marrow microenvironment promote the development and
147 n myeloma as a model to demonstrate that the bone marrow microenvironment provides a sanctuary agains
151 ause the migration of polyploid MKs from the bone marrow microenvironment requires remodeling of the
152 mation and bone resorption, and altering the bone marrow microenvironment, results in an indirect ant
153 rowth and survival signals elaborated by the bone marrow microenvironment's interaction with myeloma
154 enchymal stromal cells (MSCs) present in the bone marrow microenvironment secrete cytokines and angio
155 Overall, our data indicate that IL-21 in the bone marrow microenvironment significantly affects the b
157 ibronectin, another major constituent of the bone marrow microenvironment, stay alive and growth-arre
160 between breast cancer cells and MSCs in the bone marrow microenvironment that facilitate adaptation
161 t result from a general defect in HSC or the bone marrow microenvironment that impairs development in
162 c (in the tumor clone) and extrinsic (in the bone marrow microenvironment) that regulate tumor progre
164 inhibits MM cell growth but also acts in the bone marrow microenvironment to block angiogenesis and o
165 ical contribution of Ptpn11 mutations in the bone marrow microenvironment to leukaemogenesis and iden
166 l stem cells, may enhance the ability of the bone marrow microenvironment to support hematopoiesis af
167 Multiple myeloma is highly dependent on the bone marrow microenvironment until progressing to very a
169 ultiple myeloma cells and is abundant in the bone marrow microenvironment where it promotes tumor gro
170 alone, potentially mimicking exposure in the bone marrow microenvironment where PG concentrations are
171 hoblastic leukemia (ALL) cells reside in the bone marrow microenvironment which nurtures and protects
172 sults define novel epigenetic changes in the bone marrow microenvironment, which lead to beta-catenin
173 owth- and survival-supporting lymph node and bone marrow microenvironment, which results in clinicall
174 ied in the bone matrix are released into the bone marrow microenvironment, which results in recruitme
175 zomib, which target the myeloma cell and the bone-marrow microenvironment, which plays a crucial part
176 pting the protective signals provided by the bone marrow microenvironment will be critical for more e
177 eraction of human preneoplastic cells in the bone marrow microenvironment with non-malignant cells.
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