ライフサイエンスコーパス: carbamazepine

1 er by the prototypical Na(+) channel blocker carbamazepine.

2 he antiepileptic drug topiramate, but not by carbamazepine.

3 accumulated to a greater degree in bile than carbamazepine.

4 alproate and for lower verbal abilities with carbamazepine.

5 s alleviated by the sodium channel inhibitor carbamazepine.

6 with epilepsy or seizure when compared with carbamazepine.

7 nts compared with topiramate and secondarily carbamazepine.

8 agents, and possibly the antiepileptic agent carbamazepine.

9 non-inferiority of lamotrigine compared with carbamazepine.

10 rotein-induced EAE treated with phenytoin or carbamazepine.

11 c treatments such as lithium, valproate, and carbamazepine.

12 n the third year, they received lithium plus carbamazepine.

13 taking placebo to 3015 minutes while taking carbamazepine.

14 taking placebo to 45.3 minutes while taking carbamazepine.

15 ally relevant thermal stimuli was reduced by carbamazepine.

16 s the best candidate enzyme for metabolizing carbamazepine.

17 intenance period to 136 minutes while taking carbamazepine.

18 enance period and 32 awakenings while taking carbamazepine.

19 ndetectable or significantly lower levels of carbamazepine.

20 d with pain during treatment with placebo or carbamazepine.

21 40s who developed DIHS after treatment with carbamazepine.

22 pharmacological rescue by glibenclamide and carbamazepine.

23 87, 0.85-0.90), and treatment (gabapentin vs carbamazepine, 0.71, 0.59-0.86; topiramate vs carbamazep

24 5, 1.06-1.47), and treatment (lamotrigine vs carbamazepine, 0.76, 0.61-0.95).

25 arbamazepine, 0.71, 0.59-0.86; topiramate vs carbamazepine, 0.81, 0.68-0.98).

26 The anticonvulsant carbamazepine 1 is associated with adverse drug reaction

27 increase was found among children exposed to carbamazepine (1/50) or lamotrigine (2/30).

28 ere validated using metabolites (10,11-epoxy carbamazepine, 10,11-dihydroxy carbamazepine, and acridi

29 uding 10,11-dihydro-10-hydroxycarbamazepine, carbamazepine-10,11-epoxide, acridone-N-carbaldehyde, 4-

30 Four TPs (carbamazepine-10,11-epoxide, metoprolol acid, 1-naphthol

31 o valproate (mean 97, 95% CI 94-101) than to carbamazepine (105, 102-108; p=0.0015), lamotrigine (108

32 of patients were receiving monotherapy with carbamazepine (237 patients), phenobarbitone/primidone (

33 e paralleled by pharmacological coupling, as carbamazepine (30 muM) depolarizes S214T activation, as

34 thoxazole (2-33 days), naproxen (6-19 days), carbamazepine (355-1,624 days), and ciprofloxacin were n

35 e taking placebo to 9.1 minutes while taking carbamazepine (400 mg then 200 mg/day), and a reduction

36 taking placebo to 231.9 minutes while taking carbamazepine (400 mg/day), and a reduction in total TIP

37 European centres and randomly assigned them carbamazepine 600 mg daily (n=230) or vigabatrin 2 g dai

38 93%), indomethacin (98%), timolol (50%), and carbamazepine (70%) were assigned to the high %F region;

39 95% CI 5.5% to 8.3%) compared with 2.6% with carbamazepine (95% CI 1.9% to 3.5%) and 2.3% with lamotr

40 nts lower than the score of those exposed to carbamazepine (95% CI, 0.6 to 12.0; P=0.04).

41 Carbamazepine, a widely used neuroactive pharmaceutical,

42 for degradation of 1,4-dioxane, benzoate and carbamazepine across pH 5.5-8.3.

43 sal was ca. 50%, comparable to the effect of carbamazepine administered orally at 100 mg/kg.

44 ates suggest a non-significant advantage for carbamazepine against lamotrigine (0.91 [0.77-1.09]), to

45 , suggesting that lacosamide, lidocaine, and carbamazepine all bind to the same site.

46 Patients who stopped taking carbamazepine also had a 31.2% decline in lipoprotein(a)

47 dition to its effect on channel trafficking, carbamazepine also inhibited KATP channel activity.

48 his study we have investigated the effect of carbamazepine, an anti-convulsant, as it is known to hav

49 This study focuses on carbamazepine, an anticonvulsant drug which is ubiquitou

50 We report that carbamazepine, an anticonvulsant, corrects the trafficki

51 mazepine was only transformed to 10,11-epoxy carbamazepine and 10,11-dihydroxy carbamazepine as a dea

52 ard and feedback inhibition is unaffected by carbamazepine and additional commonly used Na(+) channel

53 by the use-dependent sodium channel blocker carbamazepine and by a blocker of reverse Na-Ca exchange

54 ging preliminary evidence include memantine, carbamazepine and citalopram.

55 All efficacy outcomes favoured carbamazepine and failed to show equivalence between the

56 ies showed complete elimination of (14)C for carbamazepine and fluoxetine treatments and partial elim

57 following short-term laboratory exposures to carbamazepine and fluoxetine.

58 vidence that despite structural differences, carbamazepine and glibenclamide compete for binding to K

59 HLA) associations: abacavir and HLA-B*57:01, carbamazepine and HLA-B*15:02, allo-purinol and HLA-B*58

60 In carbamazepine and HLA-B*15:02, certain T-cell receptor (

61 laimed wastewater-irrigated produce excreted carbamazepine and its metabolites in their urine, while

62 CMs after monotherapy exposure to valproate, carbamazepine and lamotrigine.

63 Aggravation of seizures was reported for carbamazepine and lamotrigine.

64 We conclude that carbamazepine and lidocaine access the sodium channel in

65 endent rapamycin as well as mTOR-independent carbamazepine and minoxidil, markedly attenuated cyst fo

66 uticals acetaminophen, sulfamethoxazole, and carbamazepine and on the pharmaceutical intermediate 5-m

67 The difference between carbamazepine and other drugs was not explained by diffe

68 Because carbamazepine and oxcarbazepine can enhance the sensitiv

69 Carbamazepine and oxcarbazepine were not immunosuppressi

70 s treatment with the sodium channel blockers carbamazepine and oxcarbazepine, which although effectiv

71 targets to antiepileptic drugs (AEDs; i.e., carbamazepine and phenytoin (CBZ, PHT)).

72 treatments with antiepileptic drugs, such as carbamazepine and phenytoin, which are sodium channel bl

73 f sodium channels, in contrast to drugs like carbamazepine and phenytoin, which bind tightly to fast-

74 ole, as well as by the anticonvulsant drugs, carbamazepine and phenytoin.

75 Triazolam, oral midazolam, St. John's Wort, carbamazepine and pimozide, are contraindicated with DAA

76 Prescribing of carbamazepine and sodium valproate have declined since 1

77 escribed in pregnancy over older AEDs namely carbamazepine and sodium valproate.

78 mug L(-1)), and several compounds, including carbamazepine and sulfamethoxazole, were detected throug

79 One of the pharmacological treatments is Carbamazepine and the most prevalent surgical treatments

80 For mood stabilizers, whereas carbamazepine and topiramate continued to produce positi

81 Furthermore, one new form of carbamazepine and two new forms of sulfamethoxazole were

82 Here we examined whether carbamazepine and two other sodium channel inhibitors, r

83 Lithium, carbamazepine and valproic acid are effective mood-stabi

84 thium and certain anticonvulsants, including carbamazepine and valproic acid, are effective antimanic

85 This study investigated the effect of carbamazepine and verapamil (0.005-10 mg/kg) on a range

86 af concentrations of 821.9 and 2.2 mg/kg for carbamazepine and verapamil, respectively.

87 xposed to phenytoin, 98 for those exposed to carbamazepine, and 92 for those exposed to valproate.

88 (10,11-epoxy carbamazepine, 10,11-dihydroxy carbamazepine, and acridine) as primary substrates and f

89 nt mood stabilizers, e.g., valproic acid and carbamazepine, and atypical antipsychotic drugs (APDs),

90 Lamotrigine, 10,11,-dihydro-10,11,-dihydroxy-carbamazepine, and carbamazepine were the most persisten

91 ine, olanzapine, ziprasidone, valproic acid, carbamazepine, and citalopram were associated with highe

92 ions of triclosan, triclocarban, miconazole, carbamazepine, and diphenhydramine in plant tissues that

93 All the PCs except acesulfame, carbamazepine, and fluconazole were attenuated along the

94 he tricyclic anticonvulsant drugs phenytoin, carbamazepine, and lamotrigine block neuronal voltage-ga

95 orketamine, O-desmethyltramadol, diclofenac, carbamazepine, and methoxetamine were not substantially

96 Diphenhydramine, diltiazem, carbamazepine, and norfluoxetine were detected in 11 of

97 to treatment with mood stabilizers (lithium, carbamazepine, and sodium valproate) and plasma levels o

98 Finally, rapamycin as well as spermidine, carbamazepine, and tamoxifen could also rescue the motor

99 hree other autophagy activators (spermidine, carbamazepine, and tamoxifen) in a FTLD-U mouse model wi

100 lsant mood stabilizers (AMS), valproic acid, carbamazepine, and zonisamide, but not lithium, also pre

101 ely prescribed anticonvulsants phenytoin and carbamazepine are potent inducers of cytochrome P450 enz

102 mood stabilizers, e.g., sodium valproate and carbamazepine, are human teratogens.

103 0,11-epoxy carbamazepine and 10,11-dihydroxy carbamazepine as a dead-end product.

104 Our findings identify carbamazepine as a novel small molecule corrector that m

105 o significant difference for patients taking carbamazepine at randomization, because of a low rate of

106 sample volume were evaluated for five drugs: carbamazepine, atenolol, sulfamethazine, diazepam, and a

107 odynamic analysis, and functional profiling, carbamazepine attenuated pain in patients with IEM due t

108 onic or uncharged trimethoprim and uncharged carbamazepine, but did not affect sequestration of fast-

109 lizers, valproic acid (VPA), zonisamide, and carbamazepine, but not the typical APD haloperidol, incr

110 Advanced oxidation can efficiently degrade carbamazepine, but the toxicity and persistence of the o

111 We followed the decomposition of carbamazepine by the white-rot fungus Pleurotus ostreatu

112 gate transport of phenytoin, lamotrigine and carbamazepine by using seven in-vitro transport models.

113 In both crops, the nonionic PCs (carbamazepine, caffeine, and lamotrigine) were detected

114 hether the products of advanced oxidation of carbamazepine can be biotransformed and ultimately miner

115 These data suggest that systemic carbamazepine can reduce the level of spontaneous activi

116 lacosamide monotherapy or controlled-release carbamazepine (carbamazepine-CR) twice daily.

117 95% CI and p values for no difference) were: carbamazepine (CBZ) 0.43 (-0.19 to 1.05) p=0.17; lamotri

118 Carbamazepine (CBZ) and oxcarbazepine (OXC) are widely u

119 Administration of carbamazepine (CBZ) causes hypersensitivity reactions cl

120 re we show that the autophagy-enhancing drug carbamazepine (CBZ) decreased the hepatic load of ATZ an

121 Although carbamazepine (CBZ) has a known anti-epileptic mechanism

122 The drug carbamazepine (CBZ) has the opposite effect on sleep; it

123 al sunlight photolysis of the pharmaceutical carbamazepine (CBZ) in the presence of dissolved organic

124 Carbamazepine (CBZ) is a worldwide used antiepileptic dr

125 Carbamazepine (CBZ) is an antiepileptic drug which is pe

126 The antiepileptic drug carbamazepine (CBZ) is one of the most frequently detect

127 ental emission rates of alprazolam (APZ) and carbamazepine (CBZ) through domestic WWTPs (both sludge

128 rated TPs of the recalcitrant pharmaceutical carbamazepine (CBZ) were used for a target screening for

129 It is shown here that carbamazepine (CBZ) would undergo direct photolysis and

130 hlighting this difficulty, it is unclear why carbamazepine (CBZ), a frontline AED with a known molecu

131 Here, we show that carbamazepine (CBZ), an autophagy-stimulating drug that

132 Three CECs (carbamazepine (CBZ), flumequine (FLU), and thiabendazole

133 ed the HLA-B *1502 genetic predisposition to carbamazepine (CBZ)-induced Stevens-Johnson syndrome (SJ

134 Canadian family in which pain is relieved by carbamazepine (CBZ).

135 sing preliminary evidence include memantine, carbamazepine, citalopram, and prazosin, but none of the

136 tine, paroxetine, sertraline, norfluoxetine, carbamazepine, clozapine, flurbiprofen, and sulfobromoph

137 ve metabolites of acetaminophen, diclofenac, carbamazepine, clozapine, p-cresol, 4-ethylphenol, and 3

138 esults show that the oxidation byproducts of carbamazepine containing a hydroxyl or carbonyl group ca

139 ically distinct compounds, glibenclamide and carbamazepine, correct biogenesis defects in ATP-sensiti

140 ths was 90% taking lacosamide and 91% taking carbamazepine-CR (absolute treatment-difference: -1.3%,

141 in the lacosamide group and 308 (70%) in the carbamazepine-CR group completed 6 months of treatment w

142 tients taking lacosamide and 43 (10%) taking carbamazepine-CR had serious treatment-emergent adverse

143 44 patients taking lacosamide and 442 taking carbamazepine-CR were included in the full analysis set

144 ay for lacosamide and 800 or 1200 mg/day for carbamazepine-CR) over 2 weeks with a 1-week stabilisati

145 therapy or controlled-release carbamazepine (carbamazepine-CR) twice daily.

146 doses of 100 mg/day lacosamide or 200 mg/day carbamazepine-CR, uptitration to the first target level

147 receiving lacosamide and 332 (75%) receiving carbamazepine-CR.

148 non-inferiority criteria when compared with carbamazepine-CR.

149 non-inferiority criteria when compared with carbamazepine-CR.

150 A detailed study of carbamazepine crystal growth in four different bis(urea)

151 ), anti-anxiety diazepam (DZP), anti-seizure carbamazepine (CZP) drugs and their metabolites in groce

152 ncluding an antiepileptic drug (phenytoin or carbamazepine), dexamethasone, and ranitidine.

153 sensitivity was observed with amitriptyline, carbamazepine, diazepam and gabapentin.

154 tigated the fate and uptake of (14)C-labeled carbamazepine, diclofenac, fluoxetine, and orlistat in s

155 In patients who achieved therapeutic levels, carbamazepine did not alter the clinical or electromyogr

156 r several other protein misfolding diseases, carbamazepine did not correct KATP channel trafficking d

157 Furthermore, although carbamazepine did not selectively target resurgent curre

158 ding caffeine, meprobamate, primidone, DEET, carbamazepine, dilantin, naproxen, and triclosan.

159 n in these soils can retard trimethoprim and carbamazepine dissipation.

160 erbal domains and negatively associated with carbamazepine dose for verbal performance.

161 transformation products of the (14)C-labeled carbamazepine during UV/hydrogen peroxide advanced oxida

162 ons of widely monitored HHCB, triclosan, and carbamazepine explained 71-82% of the variability in the

163 ile fish bioconcentrated both fluoxetine and carbamazepine from exposure water, fluoxetine accumulate

164 Patients were randomly assigned to receive carbamazepine, gabapentin, lamotrigine, oxcarbazepine, o

165 , and they were randomly assigned to receive carbamazepine, gabapentin, lamotrigine, oxcarbazepine, o

166 iprofloxacin, trimethoprim, propranolol, and carbamazepine (>80%) was achieved within 3 h of electrol

167 nnel inhibitors, including sulfonylureas and carbamazepine, have been shown to correct channel traffi

168 luding clopidogrel, pegylated-interferon and carbamazepine, have led to the identification of specifi

169 e, lamotrigine was significantly better than carbamazepine (hazard ratio [HR] 0.78 [95% CI 0.63-0.97]

170 Upon subsequent removal of carbamazepine, however, the function of rescued channels

171 Carbamazepine in a modified cyclodextrin (hydroxypropyl-

172 interaction was observed for lamotrigine or carbamazepine in any of the seven validated in-vitro tra

173 harmacogenetic testing for HLA-B*1502 before carbamazepine in patients of certain Asian ethnicities a

174 e-Carlo based pharmacokinetic simulations of Carbamazepine in treatment of Trigeminal Neuralgia.

175 including human leukocyte antigen loci with carbamazepine-induced dermatologic outcome and MC4R with

176 ated the relationship between HLA-B*1502 and carbamazepine-induced SJS and TEN and that reported suff

177 lationship between the HLA-B*1502 allele and carbamazepine-induced SJS and TEN in Han-Chinese, Thai,

178 for the relationship between HLA-B*1502 and carbamazepine-induced SJS and TEN was 79.84 (95% CI, 28.

179 The primary outcome was carbamazepine-induced SJS and TEN.

180 bacavir hypersensitivity and HLA-B*57:01 and carbamazepine-induced Stevens-Johnson syndrome and HLA-B

181 lysis, the degree and timing of lithium- and carbamazepine-induced thyroid changes and their subseque

182 However, carbamazepine inhibited sodium channels effectively and

183 In the whole-cell configuration, carbamazepine inhibited sodium current within seconds wh

184 Carbamazepine is one of the most persistent pharmaceutic

185 he widely used anticonvulsant pharmaceutical carbamazepine is recalcitrant in many environmental nich

186 Carbamazepine is widely accepted as a drug of first choi

187 achieving a 12-month remission (lamotrigine-carbamazepine) is 0 (-8 to 7) and 5 (-3 to 12), suggesti

188 ebo were significantly better tolerated than carbamazepine (lamotrigine, RR 5.24, 1.07-26.32; placebo

189 Parent compounds (e.g., buproprion, carbamazepine, lamotrigine) generally were more persiste

190 ntiepileptics approved for bipolar disorder (carbamazepine, lamotrigine, and valproate) are associate

191 ed to maternal IQs among children exposed to carbamazepine, lamotrigine, or phenytoin but not among t

192 ified as either exposed to an antiepileptic (carbamazepine, lamotrigine, or valproate) or not exposed

193 fects exist across four commonly used drugs (carbamazepine, lamotrigine, phenytoin and valproate).

194 epilepsy on antiepileptic drug monotherapy (carbamazepine, lamotrigine, phenytoin, or valproate) bet

195 ho were taking a single antiepileptic agent (carbamazepine, lamotrigine, phenytoin, or valproate) in

196 men with epilepsy receiving monotherapy (ie, carbamazepine, lamotrigine, phenytoin, or valproate) wer

197 Withdrawal of carbamazepine led to acute worsening of EAE symptoms, in

198 ion of the KATP channel opener diazoxide and carbamazepine led to enhanced mutant channel function wi

199 Four units ceased activity with carbamazepine levels above the therapeutic range (15.4-1

200 ption coefficients increased in the order of carbamazepine < diclofenac < fluoxetine < orlistat.

201 elimination of (14)C, increased in the order carbamazepine < diclofenac < fluoxetine and orlistat.

202 Carbamazepine may reduce the dose-limiting neurotoxicity

203 These findings suggest that phenytoin and carbamazepine may substantially increase the risk for ca

204 contraceptive steroids and the inhibition of carbamazepine metabolism by certain macrolide antibiotic

205 During solid-state fermentation, carbamazepine metabolism resulted in the generation of a

206 We also report that the carbamazepine metabolite pattern at this low exposure le

207 Carbamazepine metabolites were found mainly in the leave

208 Carbamazepine metabolites were identified using liquid c

209 re exposed to valproate monotherapy, 1718 to carbamazepine monotherapy and 2198 to lamotrigine monoth

210 mal fields; Group III, 14 patients receiving carbamazepine monotherapy; Group IV, 20 normal individua

211 cated agents were lamotrigine, azithromycin, carbamazepine, moxifloxacin, cephalexin, diclofenac, and

212 polar patients using lithium, valproate, and carbamazepine (N = 718) and then examined the patient de

213 similar irrespective of the system, and with carbamazepine no significant degradation was obtained.

214 Treatment with carbamazepine normalized the abnormalities in transcallo

215 s further evident at a cellular level, where carbamazepine normalized the hyperexcitability of dorsal

216 is usually resistant to pharmacotherapy, but carbamazepine normalizes activation of Na(V)1.7-V400M mu

217 technology was used to assess the effect of carbamazepine on firing of DRG neurons carrying S241T mu

218 The corrector effect of carbamazepine on mutant KATP channels was also demonstra

219 of refractory epilepsy through transport of carbamazepine or lamotrigine.

220 Only lithium (but not carbamazepine or other antiepileptics) had a higher repo

221 We recruited 34 epilepsy patients taking carbamazepine or phenytoin in monotherapy whose physicia

222 g epilepsy patients from the enzyme-inducers carbamazepine or phenytoin to the noninducing drugs leve

223 functions compared with lamotrigine (but not carbamazepine or phenytoin).

224 er calcium concentrations in subjects taking carbamazepine or valproate compared with those taking ot

225 notherapy, patients may have received either carbamazepine or valproate in combination with lithium f

226 h anticonvulsants (phenytoin, phenobarbital, carbamazepine, or a combination) at the same time as ant

227 erphenazine) and a mood stabilizer (lithium, carbamazepine, or valproate), 37 patients were randomly

228 gned to 1 year of lithium and then 1 year of carbamazepine, or vice versa; in the third year, they re

229 nic use of many antiepileptic drugs, such as carbamazepine, oxcarbazepine, and topiramate, enhances h

230 tments of BD such as valproate, lamotrigine, carbamazepine, oxcarbazepine, antipsychotics, electrocon

231 her MCM risk than lamotrigine (p=0.0001) and carbamazepine (p=0.0001) monotherapy.

232 ffect was seen with valproate (p=0.0006) and carbamazepine (p=0.03) exposed pregnancies.

233 metabolized via acridine and 10,11-dihydroxy carbamazepine pathways.

234 Addition of carbamazepine permitted dose escalation to 160 mg/m2 wit

235 During the carbamazepine phase, there was an inverse relationship b

236 NTDs related to exposure to FAAs (including carbamazepine, phenobarbital, phenytoin, primidone, sulf

237 cytochrome P450 enzyme (cP450) inducing AED (carbamazepine, phenytoin, and phenobarbital), a cP450 in

238 ere significantly less in subjects receiving carbamazepine, phenytoin, and valproate than in those re

239 esults of the one-step CEDIA for three AEDs (carbamazepine, phenytoin, and valproic acid), in the pre

240 Among old-generation AEDs, carbamazepine, phenytoin, phenobarbital, and primidone a

241 Among old generation AEDs, carbamazepine, phenytoin, phenobarbital, and primidone i

242 h common drugs as trimethoprim, triamterene, carbamazepine, phenytoin, phenobarbital, and primidone,

243 f bone mass in women, and both phenytoin and carbamazepine produce increases in serum lipids and C-re

244 sy patients, switch from either phenytoin or carbamazepine produced significant declines in total cho

245 ndices and mood stability during lithium and carbamazepine prophylaxis for bipolar disorder.

246 sing measured photolysis rates for atenolol, carbamazepine, propranolol, and sulfamethoxazole in wetl

247 ed in 13 units with a serum concentration of carbamazepine ranging from 3.5 to 8.4 mg/l, which was wi

248 actory solid tumors and to determine whether carbamazepine reduces oxaliplatin-induced neurotoxicity.

249 this study, a mechanism is proposed by which carbamazepine resists biodegradation, and a previously u

250 levels for diclofenac, sulfamethoxazole and carbamazepine, respectively.

251 1.7-V400M mutant channels from a family with carbamazepine-responsive inherited erythromelalgia.

252 sensitivity was observed with amitriptyline, carbamazepine, rofecoxib, and diazepam.

253 .62, 95% credible interval [CrI] 0.38-1.03), carbamazepine (RR 0.68, 0.44-1.06), imipramine (RR 0.95,

254 Caffeine and carbamazepine showed correlations with florescence index

255 Both phenytoin and carbamazepine significantly improved the clinical course

256 n identity test for monoclonal antibodies to carbamazepine, sirolimus, tacrolimus, cyclosporine, cort

257 6.9 (95% confidence interval: 1.9, 25.7) for carbamazepine (six exposed cases).

258 Specifically, carbamazepine (SMD, -1.57 [CrI, -2.83 to -0.31]), venlaf

259 peroning mechanism wherein glibenclamide and carbamazepine stabilize the heteromeric subunit interfac

260 xaliplatin); regimen B (to determine whether carbamazepine starting 24 hours before and ending 48 hou

261 ment with 300 microM lidocaine or 300 microM carbamazepine, suggesting that lacosamide, lidocaine, an

262 nd that the wastewater borne MPs diclofenac, carbamazepine, sulfamethoxazole, acesulfame, sucralose,

263 Lamotrigine is clinically better than carbamazepine, the standard drug treatment, for time to

264 r the HLA-B*1502 allele before initiation of carbamazepine therapy in patients of Asian ancestry, but

265 HLA-B*1502 screening in patients requiring carbamazepine therapy is warranted.

266 Carbamazepine therapy was tried in 12 additional patient

267 he lake system ranged from 780-5700 days for carbamazepine to <1-2 days for ketoprofen.

268 the first 6 weeks from randomisation, showed carbamazepine to be significantly more effective than vi

269 at the observed uptake in the fluoxetine and carbamazepine treatments was due to the parent compounds

270 Increased carbamazepine uptake by zucchini resulted in a decrease

271 users (36.5%), valproate users (42.4%), and carbamazepine users (42.2%) with bipolar disorder diagno

272 Carbamazepine users who were male or in the 30-49-year a

273 ) and toxic epidermal necrolysis (TEN) among carbamazepine users, especially in some racial/ethnic po

274 g antiepileptic drug monotherapy (phenytoin, carbamazepine, valproate, and lamotrigine).

275 ineffective, while valproate, gabapentin and carbamazepine varied in their potencies, with only the l

276 Neither lamotrigine nor carbamazepine was a substrate for P-gp in any of the mod

277 on treatment with low doses of rapamycin and carbamazepine was able to attenuate cyst formation as ef

278 Carbamazepine was associated with rash (22 [10%] vs seve

279 sformation of trimethoprim, propranolol, and carbamazepine was attributed to direct electron transfer

280 Arm A included patients for whom carbamazepine was considered the first-line treatment, m

281 Arm A recruited 1721 patients for whom carbamazepine was deemed to be standard treatment, and t

282 icking rescue by reversible sulfonylureas or carbamazepine was facilitated by the KATP channel opener

283 In contrast, the antiepileptic drug carbamazepine was found to inhibit voltage-gated sodium

284 Pharmacoresponsiveness of S241T to carbamazepine was further evident at a cellular level, w

285 d-state-fermentation conditions, 10,11-epoxy carbamazepine was further metabolized via acridine and 1

286 In liquid culture, carbamazepine was only transformed to 10,11-epoxy carbam

287 ore slowly (t1/2 approximately 1.5-2 d), and carbamazepine was recalcitrant.

288 For time to 12-month remission carbamazepine was significantly better than gabapentin (

289 When (14)C-carbonyl-labeled carbamazepine was used as the substrate, (14)C-CO2 relea

290 and functional analysis to be responsive to carbamazepine, was assessed in a double-blind, placebo-c

291 Oxcarbazepine, a keto-analogue of carbamazepine, was recently approved in the United State

292 to enhanced mutant channel function without carbamazepine washout.

293 hium, quetiapine, olanzapine, bupropion, and carbamazepine were associated with high morbidity indice

294 -dihydro-10,11,-dihydroxy-carbamazepine, and carbamazepine were the most persistent (t1/2 = 12 +/- 2.

295 ants such as sucralose, sulfamethoxazole and carbamazepine, which are typical wastewater markers in r

296 taking placebo to 274.1 minutes while taking carbamazepine, while patient 2 reported a reduction of t

297 dwater and revealed persistence for the drug carbamazepine, while the herbicide MCPA (2-methyl-4-chlo

298 Vigabatrin was better tolerated than carbamazepine with fewer withdrawals, but was more frequ

299 attenuation of all compounds studied except carbamazepine within an area similar to that typical of

300 As with valproic acid and carbamazepine, zonisamide (12.5 and 25 mg/kg) increased