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1 e tuberculous granulation tissue surrounding caseous and liquefied pulmonary foci and cavities, we fo
2                  Echocardiographic images of caseous calcification are often heterogenous due to calc
3                    However the appearance of caseous calcification can imitate that of abscess, tumor
4                                              Caseous calcification of mitral annulus is rather rare e
5 ary sarcoma, 2; myxoma, 4; fibroelastoma, 1; caseous calcification of mitral annulus, 3; and thrombus
6 y large, often with an ulcerated surface and caseous core.
7 g absence of tubercle lesions grossly and of caseous granulomas histologically.
8                                              Caseous granulomas were observed in the lung as early as
9                                         Only caseous granulomas were observed in the lungs at these e
10 r and local tissue environment, resulting in caseous granulomas with incomplete bacterial sterilizati
11 cell alpha-chemoattractant) within solid and caseous granulomas, and there was only minimal expressio
12 lli in low-oxygen microenvironments, such as caseous granulomas, has been hypothesized to have the po
13 and that the highest signal intensity was in caseous granulomas.
14 xin production leads to development of large caseous lesions, and in infective endocarditis, increase
15 s within the nutrient-starved environment of caseous lung granulomas.
16                                              Caseous lymphadenitis is a chronic suppurative disease c
17                                              Caseous lymphadenitis was first reported for goats in th
18 ys a major role in the liquefaction of solid caseous material and in the subsequent cavity formation.
19                                     Although caseous mitral annular calcification is typically an inc
20                 CT brought the suggestion of caseous mitral annular calcification.
21  in tuberculosis immunopathology, leading to caseous necrosis and compromising the immune response, r
22 ion may be a critical initial event, causing caseous necrosis as opposed to resulting from it.
23 from intense granulomatous inflammation with caseous necrosis for infection with type C to minimal in
24 l tenet of tuberculosis pathogenesis is that caseous necrosis leads to extracellular matrix destructi
25 egates of leukocytes and a greater degree of caseous necrosis than those from JH2-2-infected mice.
26 lar to TB in humans, namely, granulomas with caseous necrosis, liquefaction, and cavities.
27 ion of those from inbred rabbits showed more caseous necrosis, more visible bacilli, and fewer mature
28     Zebrafish tuberculous granulomas undergo caseous necrosis, similar to human tuberculous granuloma
29 n of human matrix metalloproteinase 1 causes caseous necrosis, the pathological hallmark of human tub
30 acellular matrix destruction map to areas of caseous necrosis.
31  was done and dilated bile ducts filled with caseous necrotic material were seen intra-operatively.
32 into preexisting granulomas, including their caseous (necrotic) centers, through specific mycobacteri
33 le adduct formation surrounding necrotic and caseous regions of pulmonary granulomas by immunohistoch
34 d by the similar induction of MMP-9 in human caseous tuberculosis (TB) granulomas.
35              Expression of NPC2 was lower in caseous tuberculosis granulomas and M. tuberculosis-infe
36                        Liquefaction of solid caseous tuberculous lesions and the subsequent cavity fo

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