ライフサイエンスコーパス: co-treat

コーパス検索結果 (１語後でソート)

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1 served mainly in tubular epithelial cells in CO-treated, but not air-exposed, kidney grafts at 3 hr a

2 rate that SFN reverses the effects of PMI in co-treated cells by reducing the accumulation of p62 in

3 s enhancer by nuclear proteins isolated from CO-treated cells, although HIF-1 protein levels were una

4 gher mineral to matrix ratios of DEX/rhBMP-2 co-treated cultures compared with control.

5 These changes in CO-treated grafts correlated with well-preserved CD31(+)

6 owed Ngb distribution in normal controls and CO treated groups.

7 monstrate interesting activity profiles when co-treated in a panel of metal(II) cations in MIC assays

8 ted females for chlamydial infection, and to co-treat individuals with gonorrhea for chlamydia.

9 d upregulation of HIF-1alpha and VEGF in the CO-treated kidney grafts as early as 1 hr after reperfus

10 In a lethal 85% PHTx, CO-treated mice showed a greater survival rate compared

11 d more then 9 hours earlier in the livers of CO-treated mice.

12 When co-treating normal STBEVs with HbF, the miRNA deposition

13 s to beta-amyloid (Abeta) peptides, we first co-treated primary cultured microglia with a tyrosine ph

14 ding produced by TNF-alpha were abrogated by co-treating the cells with dexamethasone.

15 otably, 10, 1 and 0.1 microM 3Me-H TRH, when co-treated with 500 microM Glu, protected fetal neurons

16 induction of hsp72 mRNA was blocked in cells co-treated with actinomycin D. hsp72 mRNA levels were af

17 Thus, when BFA-treated HepG2 cells were co-treated with ALLN, which protects apoB but does not f

18 g7 or Atg5 prolonged the survival of neurons co-treated with BDNF and rapamycin, suggesting that supp

19 tion of K-Ras was highly resistant even when co-treated with both inhibitors.

20 a dramatic decrease in the survival of cells co-treated with Casodex and ATM inhibitor as compared wi

21 e in contrast to what we saw when cells were co-treated with cisplatin plus an EGFR tyrosine kinase i

22 However, cells co-treated with CPT-11 and Apo2L/TRAIL, or pretreated wi

23 nover as similar findings were seen in cells co-treated with cycloheximide.

24 so were studied in MG-63 cells that had been co-treated with either sterol and transforming growth fa

25 reactive oxygen species (ROS) scavenger, is co-treated with ETO, it inhibits an ETO-induced increase

26 17beta-estradiol (estrogen) for 1 h and then co-treated with glutamate, apoptotic death was significa

27 ellular matrix (ECM) degradation, cells were co-treated with interleukin-1 (IL-1).

28 Fractionation of pol II from cells co-treated with MG132 and cisplatin indicated that the u

29 ted in response to thapsigargin if they were co-treated with PMA, while adherent cells secreted in re

30 M-SMCs co-treated with polyI:C and a polyclonal antibody to Ial

31 iated degradation is reversed when cells are co-treated with TCDD and Erk kinase inhibitors.

32 deficient HL-60/MX2 cells and in HL-60 cells co-treated with the Top II catalytic inhibitor ICRF-193.

33 proviral DNA synthesis as compared with MDMs co-treated with Vpx+ VLP and HIV vector.

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