ライフサイエンスコーパス: collaborate in *ing

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1 icate that two different Myb family proteins collaborate in regulating APN gene expression and point

2 53 family as a whole, including p63 and p73, collaborate in controlling autophagy to support tumor su

3 alA and RalB share 85% sequence identity and collaborate in supporting cancer cell proliferation but

4 Both NK cells and CTLs appear to collaborate in restraining growth of Q9-positive tumors.

5 The Mre11 complex and Exo1 collaborate in producing long single-stranded DNA (ssDNA

6 hat genes do not act as individual units but collaborate in overlapping networks, the deregulation of

7 XPB and Bax1 may collaborate in processing nucleic acid in an archaeal-sp

8 Although NOTCH1 and NOTCH3 collaborate in regulating osteoclast formation, NOTCH1 i

9 ying that the two ends of any given vRNA may collaborate in forming specific structures to be recogni

10 These two Cdk inhibitors collaborate in regulating spermatogenesis, helping to en

11 innate immune cells can respond via TLRs and collaborate in promoting Th1 adaptive immune responses t

12 homolog Dlar, suggesting that these proteins collaborate in orchestrating the cytoskeletal events tha

13 ssion, we demonstrate that these two factors collaborate in regulating the Actin57B target gene in vi

14 ined the extent to which these three factors collaborate in regulating the expression of the yeast ge

15 These proteins appeared to collaborate in replacing the iron atom with nonoxidizabl

16 It is possible that they collaborate in stabilizing the transition state.

17 er, our results indicate that HIPK1 and Daxx collaborate in regulating transcription.

18 well established that Ha-ras and c-myc genes collaborate in promoting transformation, tumor progressi

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