ライフサイエンスコーパス: commensal organism

1 ome-wide transcriptional response of an oral commensal organism.

2 gly, the majority of these species were oral commensal organisms.

3 g pathogens while tolerating colonization by commensal organisms.

4 and maintenance of virulence in recombining commensal organisms.

5 have been confounded by these contaminating commensal organisms.

6 arbon receptor, Janus kinase inhibitors, and commensal organisms also in trials for topical applicati

7 the JCI, Lei and colleagues demonstrate that commensal organisms also influence the host response to

8 As GBS has the disposition of both a commensal organism and an invasive pathogen, it is impor

9 iae naturally colonizes the nasopharynx as a commensal organism and sometimes causes infections in re

10 while suppressing inflammatory responses to commensal organisms and food Ags.

11 can be colonized by many bacteria including commensal organisms and potential pathogens, such as Sta

12 Discrimination between beneficial commensal organisms and potentially harmful pathogens is

13 2 (34.3%) grew a pathogen (with or without a commensal organism) and 143 (30.2%) grew commensal bacte

14 onmental organisms, opportunistic pathogens, commensal organisms, and fungi that can actively and exp

15 ant metabolite, produced by host tissues and commensal organisms, and it represents an important pote

16 lth, and it is now clear that the effects of commensal organisms are much broader than previously bel

17 doxically, ZPSs, which are found in numerous commensal organisms, are able to elicit both proinflamma

18 amily of Gram-negative bacteria that include commensal organisms as well as primary and opportunistic

19 n expressed on the surface of S. gordonii, a commensal organism, binds to soluble and immobilized fib

20 Vulvovaginal candidiasis (VVC) caused by the commensal organism Candida albicans remains a significan

21 The bacterium is a commensal organism colonizing the rectum and the gastroi

22 Enterococcus faecalis, a Gram-positive human commensal organism, depends on a two-component signaling

23 n that bacterial pathogens must compete with commensal organisms for the limited territory afforded b

24 t some unique features that distinguish skin commensal organisms from pathogenic microbes.

25 In addition, comparison with oral commensal organisms from the Ann Arbor, Mich., locale co

26 ponses are differentially regulated and that commensal organisms have a role in stimulating mucosal e

27 Escherichia coli O157:H7 is a commensal organism in cattle, but it is a pathogen in hu

28 why C. jejuni is more frequently found as a commensal organism in poultry and as the cause of diseas

29 Because C. albicans is not a commensal organism in rodents and mice are thus naive to

30 rtunistic pathogen Bacteroides fragilis is a commensal organism in the large intestine, where it util

31 obiome, and the dysbalance of pathogenic and commensal organisms in the intestinal microbiome may con

32 Normally abundant gut commensal organisms, including the Ruminococcaceae and L

33 Pathogenic E. coli, but not normal commensal organisms, increase Gal1-R mRNA synthesis and

34 pathogenic Escherichia coli, but not normal commensal organisms, increase galanin-1 receptor express

35 l pathogens as mutants of otherwise harmless commensal organisms is the challenging new feature of ou

36 S2(-/-)) formed massive abscesses containing commensal organisms, mostly enteric bacteria, even when

37 ia meningitidis is also polyploid, while the commensal organism Neisseria lactamica maintains chromos

38 scarring was associated with the presence of commensal organisms (odds ratio [OR], 1.46; 95% confiden

39 used by Candida albicans, a dimorphic fungal commensal organism of the gastrointestinal and lower fem

40 pathogen Chlamydia trachomatis evolved as a commensal organism of the human gastrointestinal (GI) tr

41 Mannheimia haemolytica, a commensal organism of the upper respiratory tract in cat

42 i) is a gram-negative bacterium and a common commensal organism of the upper respiratory tract in hum

43 Enterococci are commensal organisms of the gastrointestinal (GI) tracts

44 Lactobacilli are low-virulence, commensal organisms of the gastrointestinal and genitour

45 They serve as commensal organisms of the gastrointestinal tract and ar

46 Members of the genus Enterobacter are commensal organisms of the gastrointestinal tract and ar

47 cross-reactivity with pathogenic bacteria or commensal organisms of the oropharynx and genital tract,

48 ad to inappropriate immune reactivity toward commensal organisms, perhaps contributing to mucosal inf

49 against pathogens while tolerating harmless commensal organisms remains an important puzzle.

50 Humans are colonized by multitudes of commensal organisms representing members of five of the

51 arkable advances in our understanding of how commensal organisms shape host immunity.

52 To that end, the srtA gene in the human oral commensal organism Streptococcus gordonii was insertiona

53 regulated via TLR2 expression and signaling, commensal organisms such as N. lactamica would benefit f

54 ty toward several oral streptococci or other commensal organisms tested.

55 cellular pathogen Staphylococcus aureus is a commensal organism that can cause severe disease when it

56 le Haemophilus influenzae is a gram-negative commensal organism that is commonly associated with loca

57 Candida albicans is a commensal organism that under certain circumstances can

58 Group B streptococci (GBS) usually behave as commensal organisms that asymptomatically colonize the g

59 Medically important fungi range from commensal organisms that cause opportunistic infections

60 The genus Neisseria comprises pathogenic and commensal organisms that colonize the human nasopharynx.

61 our approach, we used it to identify several commensal organisms that induce intestinal expression of

62 of Lactobacillus spp., which are beneficial commensal organisms that thrive in similar locales as En

63 er surfaces are home to a vast population of commensal organisms that together encode millions of pro

64 may facilitate the transition of GBS from a commensal organism to a virulent meningeal pathogen.

65 The mechanisms used by commensal organisms to circumvent this immune response h

66 into the microbiome suggest that modulating commensal organisms within the gut or lung may also be a

67 gastrointestinal or genitourinary tract as a commensal organism, yet it has the potential to cause li