ライフサイエンスコーパス: coronary

1 good and rapid method for evaluation of the coronary anatomy and for early detection and grading of

2 Hard coronary and cardiovascular events, and total cardiovasc

3 ACb progressed from 1 to 399 to CAC5y>/=400, coronary and total cardiovascular risk were nearly 2-fol

4 ome because patients have generally a normal coronary angiogram and left ventricular dysfunction, whi

5 ercise stress test, computerized tomographic coronary angiogram, and cardiovascular magnetic resonanc

6 ssing creatinine values, no or an incomplete coronary angiogram, or previous dialysis.

7 ients were subjected to CSA procedure during coronary angiography (CSA group), and 25 patients served

8 ulness of screening with computed tomography coronary angiography (CTCA), and assess the safety and e

9 mly assigned to radial or femoral access for coronary angiography and percutaneous intervention, and

10 Subsequent rates of coronary angiography and revascularization after stress

11 severity was quantified in each subject via coronary angiography by calculating a CAD score.

12 trials of routine versus selective invasive coronary angiography have high rates of crossover from c

13 Compared with coronary angiography performed soon after recanalization

14 nting a case of critical hand ischemia after coronary angiography performed through radial access des

15 CT coronary angiography revealed positive coronary artery d

16 raphy by visual estimate and by quantitative coronary angiography when compared with FFR and evaluate

17 y manifestation, with indications to undergo coronary angiography with intent to perform percutaneous

18 ical outcome of patients with ACS undergoing coronary angiography, as compared with patients with sta

19 They underwent coronary angiography, endothelial function testing; meas

20 rospective registry of patients referred for coronary angiography, the goal of this study was to deve

21 Since the first coronary angioplasty on Sept 16, 1977, the field of perc

22 cardiovascular event (myocardial infarction, coronary angioplasty, coronary artery bypass graft surge

23 red for noninvasive testing will have normal coronary arteries and no long-term clinical events.

24 (LCx), right coronary artery, and all three coronary arteries combined were compared with microspher

25 d in EC overlying atherosclerotic plaques in coronary arteries from patients with ischemic heart dise

26 on of critical plaque pathophysiology in the coronary arteries.

27 ucers SMAD1/5/8 were activated in developing coronary arteries.

28 e of myocardial ischemia with no obstructive coronary arteries.

29 ary blood flow and vasodilatory responses of coronary arterioles were evaluated in all groups at the

30 idance when performing unprotected left main coronary artery (LMCA) percutaneous coronary interventio

31 as obtained during a 1-minute proximal right coronary artery (RCA) and left coronary artery balloon o

32 s beyond the territory subtended by a single coronary artery and recovers within days or weeks.

33 Coronary artery anomalies may cause life-threatening car

34 namically significant stenosis in at least 1 coronary artery as indicated by a fractional flow reserv

35 roximal right coronary artery (RCA) and left coronary artery balloon occlusion at baseline before and

36 without acute coronary syndromes or previous coronary artery bypass graft surgery in periods before (

37 artery (RA) can improve clinical outcomes in coronary artery bypass graft surgery remains unclear.

38 nts with no acute coronary syndrome/no prior coronary artery bypass graft surgery that were rated as

39 myocardial infarction, coronary angioplasty, coronary artery bypass graft surgery, stroke).

40 onary intervention, or previous multi-vessel coronary artery bypass graft surgery.

41 lectomy (189229 patients at 1876 hospitals), coronary artery bypass grafting (CABG) (218940 patients

42 unclear whether revascularization by either coronary artery bypass grafting (CABG) or percutaneous c

43 ubgroup analysis of patients undergoing only coronary artery bypass grafting, and results were simila

44 should be considered more frequently during coronary artery bypass grafting.

45 utaneous coronary intervention (28%), urgent coronary artery bypass surgery (27.5%), maternal mortali

46 Coronary artery calcification (CAC) is highly prevalent

47 Coronary artery calcification (CAC) may impair diagnosti

48 ciation between the polygenic risk score and coronary artery calcification (CARDIA) and carotid arter

49 At baseline, 70 men (50.7%) had a coronary artery calcification score higher than 300 Agat

50 , iliofemoral, and abdominal aortic plaques; coronary artery calcification; serum biomarkers; and lif

51 It is not known whether coronary artery calcium (CAC) assessment at baseline in

52 To examine the predictive ability of coronary artery calcium (CAC) score vs age for incident

53 sessed the relationships among adult height, coronary artery calcium (CAC) score, incident atheroscle

54 Coronary artery calcium score was more likely than age t

55 test declining primary cardiac diagnosis was coronary artery disease (32.3%-19.0%; P<0.001).

56 lated with QTc prolongation in patients with coronary artery disease (CAD) and investigate the effect

57 Whether ANGPTL3 deficiency reduces risk of coronary artery disease (CAD) is unknown.

58 tly associated with cardiovascular events in coronary artery disease (CAD) patients and reducing the

59 Coronary artery disease (CAD) severity was quantified in

60 rmediate pretest probability for obstructive coronary artery disease (CAD) were randomly assigned to

61 etically higher calcium had a higher risk of coronary artery disease (CAD), myocardial infarction (MI

62 r mixed methods investigation (compared with coronary artery disease and hypertension).

63 other or medical treatment in patients with coronary artery disease and left ventricular ejection fr

64 ated to treatment (two in the control group [coronary artery disease and multiorgan failure] and thre

65 he context of a recent GWAS meta-analysis of coronary artery disease and provide a list of targeted e

66 In a coronary artery disease cohort separate from volunteers

67 pain and had a lower pretest probability of coronary artery disease compared with men.

68 CT coronary angiography revealed positive coronary artery disease findings in 16 patients; LAD was

69 -wide Replication and Meta-analysis Plus the Coronary Artery Disease Genetics (CardiogramplusC4D) con

70 s (N = up to 61079 individuals) and from the Coronary Artery Disease Genome-wide Replication and Meta

71 Eligible patients with coronary artery disease had to have had a myocardial inf

72 ing process of ACS patients with obstructive coronary artery disease is associated with a high reclas

73 ASA sensitivity with known/suspected stable coronary artery disease or presenting with an acute coro

74 ictors of adverse cardiovascular outcomes in coronary artery disease patients.

75 ic role of elevated WBC across a spectrum of coronary artery disease presentations are warranted.

76 55+/-10 years), mostly with an intermediate coronary artery disease probability, between cardiac CT

77 hemorrhagic shock in swine with preexisting coronary artery disease reduced renal dysfunction and ca

78 e genetic association between rs11556924 and coronary artery disease risk by characterizing its effec

79 e-industrial lifestyle and low prevalence of coronary artery disease risk factors, we examined the Ts

80 similar age, sex, and low Framingham 10-year coronary artery disease risk scores with an echocardiogr

81 ing the total number of loci associated with coronary artery disease to 95 at the time of analysis.

82 reserve with best MT in patients with stable coronary artery disease to assess clinical outcomes and

83 mparison to Functional Testing for Suspected Coronary Artery Disease) prospectively randomized 350 pa

84 gists by 2 1 method required the presence of coronary artery disease, a common interpretation of the

85 outh), BMI, height, systolic blood pressure, coronary artery disease, and type 2 diabetes using data

86 sterol, low-density lipoprotein cholesterol, coronary artery disease, C-reactive protein, HbA1c, heig

87 undergoing initial evaluation for suspected coronary artery disease, coronary CTA was associated wit

88 nfarction in the past 20 years, multi-vessel coronary artery disease, history of stable or unstable a

89 n risk for five vascular diseases, including coronary artery disease, migraine headache, cervical art

90 stable outpatients presenting with suspected coronary artery disease, most patients experiencing clin

91 ion (T2MI2007); and 1 method did not require coronary artery disease, the 2012 universal definition (

92 utpatients (age: 63+/-9 years; 76% men) with coronary artery disease.

93 This paper reports on patients with coronary artery disease.

94 re, cardiac masses, pericardial disease, and coronary artery disease.

95 r against each other in patients with stable coronary artery disease.

96 recludes assessing physiological severity of coronary artery disease.

97 raphy, as compared with patients with stable coronary artery disease.

98 large cohort of patients suspected of having coronary artery disease.

99 stenosis and low to intermediate-complexity coronary artery disease.

100 irin (ASA) desensitization for patients with coronary artery disease.

101 .G202V HAND2 variant associated with CHD and coronary artery diseases found in a large Lebanese famil

102 ake of normal and PE STBEVs by primary human coronary artery endothelial cells (HCAEC) and the effect

103 NFalpha and cigarette smoke extract on human coronary artery endothelial cells under oscillatory, nor

104 wever, developmental mechanisms underpinning coronary artery formation remain ill-defined.

105 ater sensitivity to PGF2alpha in the porcine coronary artery from males.

106 ta support a model in which DACH1 stimulates coronary artery growth by activating Cxcl12 expression a

107 to restore blood flow in an infarct-related coronary artery improves outcomes.

108 B6sv129-mice were subjected to in vivo left coronary artery ligation for 30 minutes followed by 72 h

109 ale C57BL/6 mice were subjected to permanent coronary artery ligation.

110 Adult ANX - A1 (-/-) mice subjected to coronary artery occlusion exhibited increased infarct si

111 e group receiving PCI for an infarct-related coronary artery only.

112 The primary outcome was noncalcified coronary artery plaque volume, as determined by coronary

113 stosterone slows progression of noncalcified coronary artery plaque volume.

114 e been used in clinical research for imaging coronary artery plaque, and ongoing clinical studies are

115 d white men and women from the observational Coronary Artery Risk Development in Young Adults study,

116 and 2 observational cohort studies (CARDIA [Coronary Artery Risk Development in Young Adults] and Bi

117 e ACS that occur without apparent epicardial coronary artery thrombus or stenosis.

118 the LAD, left circumflex artery (LCx), right coronary artery, and all three coronary arteries combine

119 raphy (IVUS) of the left anterior descending coronary artery, within 8 weeks of HT.

120 ence imaging detected nanoparticles in human coronary artery-sized atheroma in vivo (P<0.05 versus re

121 ) is an index used to assess the severity of coronary-artery stenosis.

122 dhood obesity with obesity and complexity of coronary atherosclerosis (SYNTAX score) in a cohort of 3

123 Metformin may protect against coronary atherosclerosis in prediabetes and early diabet

124 CL5 and molecular phenotypes associated with coronary atherosclerosis severity in patients at least 6

125 le in chronic inflammatory diseases, such as coronary atherosclerosis, is not well defined.

126 with myocardial dysfunction and accelerated coronary atherosclerosis.

127 HDL cholesterol concentrations, obesity, and coronary atherosclerosis.

128 fetime AAS dose was strongly associated with coronary atherosclerotic burden (increase [95% confidenc

129 ete ANGPTL3 deficiency showed no evidence of coronary atherosclerotic plaque.

130 Results Mean measured coronary attenuation values +/- standard deviation were

131 actional flow reserve (FFR) in patients with coronary bifurcation lesion.

132 ew possibilities to measure maximal absolute coronary blood flow and minimal microcirculatory resista

133 Coronary blood flow and vasodilatory responses of corona

134 oglycerin causes changes in the systemic and coronary circulation that combine to reduce myocardial o

135 microvasculature but interrogates the entire coronary circulation.

136 The primary study end point was coronary collateral flow index as obtained during a 1-mi

137 ders at 193 North American sites interpreted coronary computed tomographic (CT) angiography as part o

138 Coronary computed tomographic (CT) angiography has emerg

139 bstructive LM CAD was frequently detected on coronary computed tomographic angiography and strongly a

140 onary artery plaque volume, as determined by coronary computed tomographic angiography.

141 tial noninvasive cardiac testing with either coronary computed tomography angiography (CTA) or functi

142 clear stress, or stress echocardiography) or coronary computed tomography angiography (CTA).

143 on (CAC) may impair diagnostic assessment of coronary computed tomography angiography (CTA).

144 They underwent ICA and coronary CT angiography performed with a whole-heart CT

145 n Coronary Opacification and Heart Rhythm in Coronary CT Angiography, or IsoCOR, trial.

146 rocedures, and medications were higher after coronary CTA ($995 vs. $718; p < 0.001).

147 ation for suspected coronary artery disease, coronary CTA was associated with greater use of statins,

148 (HR: 1.95; 95% CI: 1.72 to 2.21), unheralded coronary death (HR: 1.78; 95% CI: 1.51 to 2.10), abdomin

149 beverage intake with risk of mortality from coronary diseases, diabetes, or cancer, but few studies

150 Clarification of the origins of coronary endothelial cells during cardiac repair is esse

151 eract with SES to influence CAC and incident coronary events in a population-based cohort.

152 t fluctuation is a risk factor for death and coronary events in patients without cardiovascular disea

153 Incident coronary events were ascertained over a median follow-up

154 genetic effects on loge(CAC+1) and incident coronary events.

155 value of noninvasively assessing indices of coronary flow for diagnosing coronary microvascular dysf

156 rements of fractional flow reserve (FFR) and coronary flow reserve (CFR) and the index of microcircul

157 idated for absolute myocardial perfusion and coronary flow reserve (CFR) by positron emission tomogra

158 Doppler wire-derived coronary flow reserve has been applied in research studi

159 using speckle-tracking echocardiography, (2) coronary flow reserve, (3) pulse wave velocity and augme

160 rmine the long-term risks of acute and fatal coronary heart disease (CHD) events after sepsis hospita

161 telomere length (TL) to be a risk factor for coronary heart disease (CHD), and recently the associati

162 differences of incident heart failure (HF), coronary heart disease (CHD), and stroke in participants

163 consumption to blood lipid levels and hence coronary heart disease (CHD).

164 , 58.5 years; 39% women) with 62240 cases of coronary heart disease (CHD).

165 accepted model to predict outcomes in stable coronary heart disease (CHD).

166 HDL defined by apoC-III are associated with coronary heart disease (CHD).

167 has been associated with type 2 diabetes and coronary heart disease (CHD).

168 .99 to 1.32), but higher risks of death from coronary heart disease (HR: 1.45; 95% CI: 1.21 to 1.74),

169 PHDL-C=0.008 and Ptriglycerides=0.00003) and coronary heart disease (P=0.0007).

170 ntration with first-ever CVD outcomes (i.e., coronary heart disease [CHD], stroke, or the combination

171 hat together enrolled 4726 participants with coronary heart disease and 3529 controls.

172 recognized as an independent risk factor for coronary heart disease and cardiovascular mortality.

173 Chinese patients with coronary heart disease and impaired glucose tolerance we

174 INTERPRETATION: In Chinese patients with coronary heart disease and impaired glucose tolerance, a

175 century, then the decline in mortality from coronary heart disease and stroke has been the success s

176 Outcomes included cumulative CVD (coronary heart disease and stroke) deaths prevented or p

177 /=190 mg/dL, pravastatin reduced the risk of coronary heart disease by 27% (P=0.033) and major advers

178 rs), 1203 incident CVD events, including 916 coronary heart disease cases, were reported.

179 ring the initial trial phase and the risk of coronary heart disease death, cardiovascular death, and

180 d nongenetic effects on the indexing (first) coronary heart disease event.

181 easure to predict progression to death after coronary heart disease is established.

182 the effect of the genetic score on decreased coronary heart disease risk extended beyond its effect o

183 mellitus and prediabetes remain at increased coronary heart disease risk.

184 tive cohorts, carriers of CHIP had a risk of coronary heart disease that was 1.9 times as great as in

185 lood cells and associated such presence with coronary heart disease using samples from four case-cont

186 hat the CETP inhibitor anacetrapib decreased coronary heart disease when added to statin therapy.

187 Despite the reduced incidence of coronary heart disease with intensive risk factor manage

188 ariants at the CXCR4 locus with the risk for coronary heart disease, along with CXCR4 transcript expr

189 ad suffered from CVD, 4.9% had suffered from coronary heart disease, and 2.6% had experienced a strok

190 dent myocardial infarction or death owing to coronary heart disease, and stroke, defined as the first

191 recommendations for patients with prevalent coronary heart disease, and we offer recommendations, wh

192 Incident CVD, which combines coronary heart disease, defined as the first incident my

193 The hazard ratios for incident coronary heart disease, stroke, and CVD associated with

194 are independent and causal risk factors for coronary heart disease.

195 y of APOC3 has been shown to protect against coronary heart disease; we identified APOC3 homozygous p

196 nfirmed that (68)Ga-DOTATATE offers superior coronary imaging, excellent macrophage specificity, and

197 ent in patients with angina and unobstructed coronaries in a European population.

198 comprised CVD death, myocardial infarction, coronary insufficiency, index admission for heart failur

199 echanical support (28%), urgent percutaneous coronary intervention (28%), urgent coronary artery bypa

200 s with STEMI undergoing primary percutaneous coronary intervention (n=1604; mean age, 61+/-12 years;

201 rs of its improvement after CTO percutaneous coronary intervention (PCI) are unknown.

202 rtery bypass grafting (CABG) or percutaneous coronary intervention (PCI) carries benefits or risks in

203 and white patients treated with percutaneous coronary intervention (PCI) in the Veterans Affairs (VA)

204 Primary percutaneous coronary intervention (PCI) may therefore be less benefi

205 Percutaneous coronary intervention (PCI) of saphenous vein grafts (SV

206 infarction (STEMI), the use of percutaneous coronary intervention (PCI) to restore blood flow in an

207 eft main coronary artery (LMCA) percutaneous coronary intervention (PCI), but the overall picture rem

208 d minorities after contemporary percutaneous coronary intervention (PCI).

209 Therapy in Subjects Who Require Percutaneous Coronary Intervention [PCI] [CHAMPION PHOENIX] [CHAMPION

210 Percutaneous coronary intervention and CABG show comparable safety in

211 among 1919 patients undergoing percutaneous coronary intervention at 76 centers.

212 relief of coronary obstruction, percutaneous coronary intervention has become a standard-of-care proc

213 w marking its 40th anniversary, percutaneous coronary intervention has become one of the most common

214 on Sept 16, 1977, the field of percutaneous coronary intervention has evolved rapidly.

215 pulation of patients undergoing percutaneous coronary intervention in the contemporary era.

216 y), 4222 patients who underwent percutaneous coronary intervention in the United States and Europe be

217 e acute stage, intended primary percutaneous coronary intervention increased from 12% (1995) to 76% (

218 ciated with changes in rates of percutaneous coronary intervention or in-hospital mortality in New Yo

219 ETHODS AND We analyzed 1253 CTO percutaneous coronary intervention procedures performed according to

220 ly has led to worse outcomes in percutaneous coronary intervention procedures performed through the t

221 ral complexity, shorter primary percutaneous coronary intervention time was associated with an increa

222 Patients (n=15 003) underwent percutaneous coronary intervention to SVG in England and Wales during

223 Percutaneous coronary intervention was performed in 235 patients (71%

224 ction (MI) treated with primary percutaneous coronary intervention were randomized to prasugrel or ti

225 ete revascularization following percutaneous coronary intervention were randomized to ranolazine vers

226 rates of ischemic events during percutaneous coronary intervention without an increase in severe blee

227 ariables (male sex and previous percutaneous coronary intervention) and 4 biomarkers (midkine, adipon

228 omplete Revascularization after Percutaneous Coronary Intervention) trial, a clinical trial in which

229 s and additionally into primary percutaneous coronary intervention, fibrinolysis, or no reperfusion.

230 e angina, previous multi-vessel percutaneous coronary intervention, or previous multi-vessel coronary

231 y survive the first month after percutaneous coronary intervention, their prognosis is comparable to

232 30 patients were transported to percutaneous coronary intervention-capable hospitals, including 974 i

233 tcomes of patients treated with percutaneous coronary intervention.

234 iography with intent to perform percutaneous coronary intervention.

235 rted mortality statistics after percutaneous coronary intervention.

236 yocardial infarction undergoing percutaneous coronary intervention.

237 metal in-stent restenosis after percutaneous coronary intervention.

238 sess the safety and efficacy of percutaneous coronary interventions (PCIs).

239 n patients treated with primary percutaneous coronary interventions.

240 nfarction in the era of primary percutaneous coronary interventions.

241 MDCT angiography of the coronaries is a good and rapid method for evaluation of

242 atomy and for early detection and grading of coronary lesions in non-diabetic patients.

243 er to discriminate high-risk versus low-risk coronary lesions than [(18)F]FDG.

244 nt interventional tool for heavily calcified coronary lesions.

245 A subgroup of these patients also has coronary microvascular dysfunction and evidence of infla

246 sing indices of coronary flow for diagnosing coronary microvascular dysfunction; in certain diseases,

247 function; in certain diseases, the degree of coronary microvascular impairment carries important prog

248 For relief of coronary obstruction, percutaneous coronary intervention

249 ythmias (VAs) were subjected to percutaneous coronary occlusion to induce myocardial infarction.

250 and angina pectoris during the same 1-minute coronary occlusion.

251 the Effect of Iso-osmolar Contrast Medium on Coronary Opacification and Heart Rhythm in Coronary CT A

252 single-center cohort of patients undergoing coronary or peripheral angiography with or without inter

253 djustments for age, sex, study site, primary coronary percutaneous intervention (PCI), and norepineph

254 patients with psoriasis had increased total coronary plaque burden (1.22+/-0.31 versus 1.04+/-0.22,

255 Throughout exercise, distal coronary pressure and flow velocity and central aortic p

256 th statin therapy (WOSCOPS [West of Scotland Coronary Prevention Study]; n=4910) and 2 observational

257 ores in the mid-range (4 to 6) indicate that coronary revascularization may be appropriate for the cl

258 Recurrent MI, CHD events (recurrent MI or a coronary revascularization procedure), and mortality wer

259 ular outcomes (death, myocardial infarction, coronary revascularization, or cerebrovascular events) i

260 nary syndrome without myocardial infarction, coronary revascularization, or CHD death.

261 he primary end point was freedom from repeat coronary revascularization.

262 linking common variants in this region with coronary risk is not known.

263 Image quality, coronary segment interpretability, effective dose (ED),

264 treated due to an inability or impediment to coronary sinus (CS) lead implantation.

265 es additional epicardial ablation within the coronary sinus.

266 tivation and subsequent differentiation into coronary smooth muscle, and restores Wt1 activity upon M

267 rrelation between angiographic assessment of coronary stenoses and fractional flow reserve (FFR) is w

268 FR), an index of the hemodynamic severity of coronary stenoses, is derived from invasive measurements

269 characteristic curve of 0.87 (p < 0.001) for coronary stenosis >/=70%.

270 Individuals who underwent coronary stenting and completed 12 months of thienopyrid

271 s older than 18 years with an indication for coronary stenting were enrolled, and 11648 (mean age, 61

272 to millions of patients worldwide following coronary stenting.

273 Bioresorbable coronary stents have been introduced into clinical pract

274 tiplatelet and anticoagulant agents, and new coronary stents will continue the journey to achieve thi

275 the incidence of AKI in patients with acute coronary syndrome (ACS) enrolled in the MATRIX-Access (M

276 Of the 310 subjects, 138 had an acute coronary syndrome (ACS), 101 of whom underwent desensiti

277 s nonculprit arteries in patients with acute coronary syndrome (median difference: 0.69; interquartil

278 patients with non-ST-segment elevation acute coronary syndrome (NSTE-ACS) and an elevated cardiac tro

279 of Treatment Patterns and Events After Acute Coronary Syndrome (TRANSLATE-ACS) study between April 1,

280 between WBC and MACE was consistent in acute coronary syndrome and non-acute coronary syndrome presen

281 Patients admitted for acute coronary syndrome and/or revascularization, with >/=1 LR

282 TTS, however, differs from an acute coronary syndrome because patients have generally a norm

283 centrations in patients with suspected acute coronary syndrome in which the diagnosis was adjudicated

284 rt-term mortality and complications in acute coronary syndrome patients treated with extracorporeal c

285 ent in acute coronary syndrome and non-acute coronary syndrome presentations (interaction P=0.15).

286 tal or nonfatal myocardial infarction, acute coronary syndrome without myocardial infarction, coronar

287 mong patients with stable angina or an acute coronary syndrome, an iFR-guided revascularization strat

288 heart failure, and hospitalization for acute coronary syndrome, and the incidence of acute pancreatit

289 abilized individuals within 10 days of acute coronary syndrome, combination therapy seemed to be more

290 In suspected acute coronary syndrome, high-sensitivity cardiac troponin ide

291 y artery disease or presenting with an acute coronary syndrome, including ST-segment-elevation myocar

292 , with or without ST-segment elevation acute coronary syndrome, were randomly assigned to radial or f

293 n therapy in patients stabilized after acute coronary syndrome.

294 tal number of PCIs in patients with no acute coronary syndrome/no prior coronary artery bypass graft

295 formed in New York in patients without acute coronary syndromes or previous coronary artery bypass gr

296 zed participants with non-ST-elevation acute coronary syndromes or stable angina and to evaluate long

297 ve Versus Conservative Treatment in Unstable Coronary Syndromes) trial compared early invasive strate

298 tes mellitus, and 39.7% presented with acute coronary syndromes.

299 temperature through a dedicated catheter for coronary thermodilution induces steady-state maximal hyp

300 ctors of >/=70% stenosis in at least 1 major coronary vessel were identified from >200 candidate vari