ライフサイエンスコーパス: coronary artery

1 fficacy is early reperfusion of the occluded coronary artery.

2 cal assessment has focused on the epicardial coronary artery.

3 ete ligature of the left anterior descending coronary artery.

4 r placed around the left anterior descending coronary artery.

5 ict macroscopic and microscopic responses of coronary arteries.

6 profile compared with BVS in normal porcine coronary arteries.

7 ism for angina in patients with unobstructed coronary arteries.

8 omographical or cross-sectional image of the coronary arteries.

9 on of critical plaque pathophysiology in the coronary arteries.

10 ucers SMAD1/5/8 were activated in developing coronary arteries.

11 e of myocardial ischemia with no obstructive coronary arteries.

12 l part of LAD was the most commonly affected coronary artery (14 cases, 63.3%) in the studied patient

13 no revascularization of non-infarct-related coronary arteries (590 patients).

14 Careful initial management of evolving coronary artery abnormalities is essential, necessitatin

15 dentify patients with chest pain with normal coronary arteries and no clinical events during follow-u

16 red for noninvasive testing will have normal coronary arteries and no long-term clinical events.

17 s beyond the territory subtended by a single coronary artery and recovers within days or weeks.

18 the LAD, left circumflex artery (LCx), right coronary artery, and all three coronary arteries combine

19 Coronary artery anomalies may cause life-threatening car

20 Atherothrombotic events in coronary arteries are most often due to rupture of unsta

21 namically significant stenosis in at least 1 coronary artery as indicated by a fractional flow reserv

22 roximal right coronary artery (RCA) and left coronary artery balloon occlusion at baseline before and

23 nal (3D) microstructural mechanical model of coronary artery based on measured microstructure includi

24 CT, and [15O]H2O PET with examination of all coronary arteries by fractional flow reserve was perform

25 coronary artery disease (LMCAD) treated with coronary artery bypass graft (CABG) or percutaneous coro

26 e (CAD) had improved long-term outcomes with coronary artery bypass graft (CABG) surgery compared wit

27 d improve patients' prognosis after elective coronary artery bypass graft (CABG) surgery.

28 ive, high-risk cardiac surgery (ie, combined coronary artery bypass graft [CABG] surgery and valve re

29 h (SCD) in patients with heart failure after coronary artery bypass graft surgery (CABG) has not been

30 without acute coronary syndromes or previous coronary artery bypass graft surgery in periods before (

31 artery (RA) can improve clinical outcomes in coronary artery bypass graft surgery remains unclear.

32 nts with no acute coronary syndrome/no prior coronary artery bypass graft surgery that were rated as

33 ut lower rates of multivessel disease, prior coronary artery bypass graft surgery, prior MI, and smok

34 myocardial infarction, coronary angioplasty, coronary artery bypass graft surgery, stroke).

35 onary intervention, or previous multi-vessel coronary artery bypass graft surgery.

36 ADBRs included non-coronary artery bypass graft-related Thrombolysis In Myo

37 lectomy (189229 patients at 1876 hospitals), coronary artery bypass grafting (CABG) (218940 patients

38 unclear whether revascularization by either coronary artery bypass grafting (CABG) or percutaneous c

39 Coronary artery bypass grafting (CABG) remains the stand

40 omized trial data support the superiority of coronary artery bypass grafting (CABG) surgery over perc

41 cular (LV) biopsies from patients undergoing coronary artery bypass grafting (CABG), only the activat

42 ty of Thoracic Surgeons composite rating for coronary artery bypass grafting (STS-CABG), and Centers

43 Of 10 633 adults who underwent coronary artery bypass grafting and valve surgery betwee

44 ternal mammary artery (BIMA) conduits during coronary artery bypass grafting have better long-term su

45 ader spectrum of patients who are undergoing coronary artery bypass grafting in routine practice.

46 retrospective analysis of 47 984 consecutive coronary artery bypass grafting surgeries performed from

47 Among the study population, 1482 coronary artery bypass grafting surgeries with BIMA were

48 ndomized clinical trial, patients undergoing coronary artery bypass grafting with an internal mammary

49 th low ejection fraction who were undergoing coronary artery bypass grafting with cardiopulmonary byp

50 o 40% and scheduled for isolated or combined coronary artery bypass grafting with cardiopulmonary byp

51 studies comparing the long-term outcomes of coronary artery bypass grafting with the use of 2-arteri

52 ubgroup analysis of patients undergoing only coronary artery bypass grafting, and results were simila

53 ary intervention, and 2832 (10.7%) underwent coronary artery bypass grafting.

54 , 57,961 patients underwent primary isolated coronary artery bypass grafting.

55 should be considered more frequently during coronary artery bypass grafting.

56 utaneous coronary intervention (28%), urgent coronary artery bypass surgery (27.5%), maternal mortali

57 The EXCEL (Evaluation of Xience Versus Coronary Artery Bypass Surgery for Effectiveness of Left

58 Coronary artery bypass surgery was performed in 44 cases

59 the readmitted patients, 13% had PCI, 2% had coronary artery bypass surgery, and 3% died during the r

60 n cardiac rehabilitation referral after PCI, coronary artery bypass surgery, and valve surgery.

61 ng percutaneous coronary intervention (PCI), coronary artery bypass surgery, or valve surgery be refe

62 Coronary artery calcification (CAC) is highly prevalent

63 Coronary artery calcification (CAC) may impair diagnosti

64 ciation between the polygenic risk score and coronary artery calcification (CARDIA) and carotid arter

65 CI, 1.04-1.68) greater likelihood of having coronary artery calcification and 9.7% higher (95% CI, 2

66 n, computed tomography scans for measures of coronary artery calcification and echocardiographic asse

67 Coronary artery calcification is independently and signi

68 of subclinical atherosclerosis, assessed by coronary artery calcification score >100 AU; (2) ASCV ev

69 levels were positively associated with high coronary artery calcification score (odds ratio, 2.28; 9

70 At baseline, 70 men (50.7%) had a coronary artery calcification score higher than 300 Agat

71 Subclinical atherosclerosis (plaque or coronary artery calcification) was present in 49.7% of C

72 hyperlipidemia) or subclinical CVD measures (coronary artery calcification, early transmitral velocit

73 nfarction and arterial stiffness, as well as coronary artery calcification.

74 , iliofemoral, and abdominal aortic plaques; coronary artery calcification; serum biomarkers; and lif

75 It is not known whether coronary artery calcium (CAC) assessment at baseline in

76 Therefore, we studied whether coronary artery calcium (CAC) can further guide the allo

77 Coronary artery calcium (CAC) is associated with coronar

78 To examine the predictive ability of coronary artery calcium (CAC) score vs age for incident

79 Nonzero coronary artery calcium (CAC) score, abdominal aortic ca

80 sessed the relationships among adult height, coronary artery calcium (CAC) score, incident atheroscle

81 rs for coronary artery disease report higher coronary artery calcium (CAC) scores compared with seden

82 Coronary atherosclerosis was assessed by coronary artery calcium (CAC) scoring done with non-cont

83 re controversies regarding the usefulness of coronary artery calcium score (CACS) for predicting coro

84 Coronary artery calcium score (vs age) had a greater ass

85 Coronary artery calcium score was more likely than age t

86 model (including age, body mass index, sex, coronary artery calcium score, diabetes mellitus, hypert

87 95% CI, 1.74-3.92; P < .001), independent of coronary artery calcium score, visceral adipose tissue,

88 ment of left ventricular hypertrophy by ECG, coronary artery calcium, N-terminal pro B-type natriuret

89 ral flow index in the untreated RCA and left coronary artery changed from 0.071+/-0.082 at baseline t

90 (LCx), right coronary artery, and all three coronary arteries combined were compared with microspher

91 th doses of </=55 Gy, maximal point-doses to coronary arteries could be kept <7Gy, but target conform

92 hat mice lacking endothelial Dach1 had small coronary arteries, decreased endothelial cell polarizati

93 uld benefit from a detailed understanding of coronary artery development during embryogenesis and of

94 OTCH1/VEGFA/VEGFR2 signaling axis is key for coronary artery development.

95 ean dose; hazard ratio, 1.03/Gy; P = .002,), coronary artery disease ( P < .001), and WHO/Internation

96 Prevalence of obstructive coronary artery disease (1%-64%), ACS (1%-44%), downstre

97 rtension (77%), diabetes mellitus (31%), and coronary artery disease (15%).

98 test declining primary cardiac diagnosis was coronary artery disease (32.3%-19.0%; P<0.001).

99 n DNA sequence variants for association with coronary artery disease (4,831 cases and 115,455 control

100 Main comorbidities included coronary artery disease (51.5%), renal insufficiency (27

101 We also conduct parallel investigations for coronary artery disease (CAD) (viewed as a positive cont

102 The prevalence of coronary artery disease (CAD) among patients with refrac

103 ave identified multiple loci associated with coronary artery disease (CAD) among predominantly Europe

104 lated with QTc prolongation in patients with coronary artery disease (CAD) and investigate the effect

105 to elevated serum calcium levels and risk of coronary artery disease (CAD) and myocardial infarction

106 Genome-wide association studies (GWAS) in coronary artery disease (CAD) had identified 66 loci at

107 ) trial, patients with 3-vessel or left main coronary artery disease (CAD) had improved long-term out

108 asive testing for a diagnosis of significant coronary artery disease (CAD) is ambiguous, but nuclear

109 Coronary artery disease (CAD) is the number one cause of

110 Whether ANGPTL3 deficiency reduces risk of coronary artery disease (CAD) is unknown.

111 oninvasive models to predict the presence of coronary artery disease (CAD) may help reduce the societ

112 tly associated with cardiovascular events in coronary artery disease (CAD) patients and reducing the

113 Coronary artery disease (CAD) severity was quantified in

114 METHODS AND Obstructive coronary artery disease (CAD) was defined as >/=50% sten

115 Comorbid coronary artery disease (CAD) was present in 24.3% of vi

116 rmediate pretest probability for obstructive coronary artery disease (CAD) were randomly assigned to

117 etically higher calcium had a higher risk of coronary artery disease (CAD), myocardial infarction (MI

118 e (MR) perfusion imaging in the detection of coronary artery disease (CAD).

119 olocalize with known susceptibility loci for coronary artery disease (CARDIoGRAMplusC4D) and large ar

120 comes in patients with unprotected left main coronary artery disease (LMCAD) treated with coronary ar

121 (PCI) in diabetic patients with multivessel coronary artery disease (MV-CAD).

122 tion and Meta-analysis [CARDIoGRAM] plus The Coronary Artery Disease [C4D] Genetics; combined total 6

123 Among patients who had multivessel coronary artery disease and acute myocardial infarction

124 Cardiac risk was assessed by noting baseline coronary artery disease and calculating the WHO/Internat

125 e 4-BRS was also associated with severity of coronary artery disease and composite end points.

126 ated loci for Alzheimer's disease, eight for coronary artery disease and five for type 2 diabetes.

127 onal cardiologists to assess the severity of coronary artery disease and guide treatment, coronary an

128 r mixed methods investigation (compared with coronary artery disease and hypertension).

129 Coronary artery disease and its risk factors are some of

130 becoming increasingly popular in the area of coronary artery disease and its risk factors, many of th

131 other or medical treatment in patients with coronary artery disease and left ventricular ejection fr

132 ated to treatment (two in the control group [coronary artery disease and multiorgan failure] and thre

133 he context of a recent GWAS meta-analysis of coronary artery disease and provide a list of targeted e

134 atins have been used for 30 years to prevent coronary artery disease and stroke.

135 robiota-dependent metabolite associated with coronary artery disease and stroke.

136 Dyslipidemia is an important risk factor for coronary artery disease and stroke.

137 we describe GWAB-boosted candidate genes for coronary artery disease and supporting data in the liter

138 Although virtually all coronary artery disease associated single-nucleotide pol

139 ally attributable to an elevated subclinical coronary artery disease burden composed of noncalcified

140 ance imaging, after exclusion of obstructive coronary artery disease by angiography.

141 In a coronary artery disease cohort separate from volunteers

142 pain and had a lower pretest probability of coronary artery disease compared with men.

143 cording to drug-eluting stent generation and coronary artery disease complexity were performed.

144 CT coronary angiography revealed positive coronary artery disease findings in 16 patients; LAD was

145 uidelines-Stroke and Get With The Guidelines-Coronary Artery Disease from 2006 to 2009 and treating >

146 -wide Replication and Meta-analysis Plus the Coronary Artery Disease Genetics (CardiogramplusC4D) con

147 t disease (CHD) data from CARDIoGRAMplusC4D (Coronary Artery Disease Genome-wide Replication and Meta

148 s (N = up to 61079 individuals) and from the Coronary Artery Disease Genome-wide Replication and Meta

149 Eligible patients with coronary artery disease had to have had a myocardial inf

150 In vitro, MSCs from individuals with coronary artery disease have reduced ability to suppress

151 uccessful treatment of unstable, multivessel coronary artery disease in a child with PCI with BRS imp

152 ant implications for the future treatment of coronary artery disease in children and warrants further

153 ters in epicardial AT of human patients with coronary artery disease in comparison with controls with

154 Few studies have assessed the prevalence of coronary artery disease in masters athletes with a low a

155 Coronary artery disease is a major cause of morbidity an

156 ing process of ACS patients with obstructive coronary artery disease is associated with a high reclas

157 ment in the delivery of OMT to patients with coronary artery disease is one possible step to help the

158 al noninvasive testing to evaluate suspected coronary artery disease might affect subsequent clinical

159 factors, have the lowest reported levels of coronary artery disease of any population recorded to da

160 PCI between 2005 and 2014 because of stable coronary artery disease or acute coronary syndrome were

161 F or at risk of HF, such as in patients with coronary artery disease or hypertension.

162 ASA sensitivity with known/suspected stable coronary artery disease or presenting with an acute coro

163 th out-of-hospital cardiac arrest have shown coronary artery disease or symptoms during the hour befo

164 ictors of adverse cardiovascular outcomes in coronary artery disease patients.

165 ic role of elevated WBC across a spectrum of coronary artery disease presentations are warranted.

166 55+/-10 years), mostly with an intermediate coronary artery disease probability, between cardiac CT

167 larization) at 1 year in 2,008 patients with coronary artery disease randomized to BVS versus cobalt-

168 hemorrhagic shock in swine with preexisting coronary artery disease reduced renal dysfunction and ca

169 table, symptomatic outpatients without known coronary artery disease referred for noninvasive testing

170 hletes with atherosclerotic risk factors for coronary artery disease report higher coronary artery ca

171 Although much less frequent than in adults, coronary artery disease requiring revascularization may

172 s familial hypercholesterolemia and unstable coronary artery disease requiring revascularization.

173 e genetic association between rs11556924 and coronary artery disease risk by characterizing its effec

174 Conventional coronary artery disease risk factors might potentially e

175 t population of the Bolivian Amazon with few coronary artery disease risk factors, have the lowest re

176 e-industrial lifestyle and low prevalence of coronary artery disease risk factors, we examined the Ts

177 similar age, sex, and low Framingham 10-year coronary artery disease risk scores with an echocardiogr

178 s of inflammation may translate into reduced coronary artery disease risk.

179 en genome-wide significantly associated with coronary artery disease risk.

180 ing the total number of loci associated with coronary artery disease to 95 at the time of analysis.

181 reserve with best MT in patients with stable coronary artery disease to assess clinical outcomes and

182 le patients (n = 4,057) without a history of coronary artery disease underwent CZT SPECT MPI.

183 a prospective study, 85 patients with stable coronary artery disease underwent percutaneous coronary

184 history of statin intolerance, diabetes, or coronary artery disease were not eligible.

185 ls (90% on statins) with no prior history of coronary artery disease who had a screening CACS >/=300

186 cantly from those of trials with multivessel coronary artery disease without left main LMCA stenosis.

187 antially reduce morbidity and mortality from coronary artery disease worldwide.

188 mparison to Functional Testing for Suspected Coronary Artery Disease) prospectively randomized 350 pa

189 istory of coronary artery disease, and known coronary artery disease), each gram increase of posterio

190 gists by 2 1 method required the presence of coronary artery disease, a common interpretation of the

191 e kidney transplantation commonly focuses on coronary artery disease, a comprehensive pretransplant c

192 ion, hypercholesterolemia, family history of coronary artery disease, and known coronary artery disea

193 outh), BMI, height, systolic blood pressure, coronary artery disease, and type 2 diabetes using data

194 lead AAA single nucleotide polymorphisms and coronary artery disease, blood pressure, lipids, or diab

195 ated with lower risks of type 2 diabetes and coronary artery disease, but their relations with interm

196 sterol, low-density lipoprotein cholesterol, coronary artery disease, C-reactive protein, HbA1c, heig

197 ension, hyperlipidemia, cardiac arrhythmias, coronary artery disease, congestive heart failure, diabe

198 undergoing initial evaluation for suspected coronary artery disease, coronary CTA was associated wit

199 prognostic ability of these risk markers in coronary artery disease, heart failure, and atrial fibri

200 spital size, teaching hospital status, known coronary artery disease, heart failure, diabetes mellitu

201 nfarction in the past 20 years, multi-vessel coronary artery disease, history of stable or unstable a

202 n risk for five vascular diseases, including coronary artery disease, migraine headache, cervical art

203 stable outpatients presenting with suspected coronary artery disease, most patients experiencing clin

204 dies suggest that among patients with stable coronary artery disease, patients with diabetes mellitus

205 etes, chronic obstructive pulmonary disease, coronary artery disease, peripheral arterial disease, hy

206 ion (T2MI2007); and 1 method did not require coronary artery disease, the 2012 universal definition (

207 gulating protein] gene increases the risk of coronary artery disease, the leading cause of death worl

208 redicted lower BMI, systolic blood pressure, coronary artery disease, type 2 diabetes, and taller sta

209 s for diabetic patients who have multivessel coronary artery disease, we combine the results of the F

210 diagnostic strategy for women with suspected coronary artery disease, with potential benefits in term

211 ain at least 90% of the attributable risk of coronary artery disease.

212 cibility of FFRangio in patients with stable coronary artery disease.

213 disease in comparison with controls without coronary artery disease.

214 relevance for patients with risk factors or coronary artery disease.

215 stenosis and low to intermediate-complexity coronary artery disease.

216 irin (ASA) desensitization for patients with coronary artery disease.

217 n of patients with known or suspected stable coronary artery disease.

218 mpared with MT alone in patients with stable coronary artery disease.

219 the potent P2Y12 inhibitors in patients with coronary artery disease.

220 cluding hypertension, diabetes mellitus, and coronary artery disease.

221 phoma Kinase) is linked to a protection from coronary artery disease.

222 links to BMI, adiposity-related traits, and coronary artery disease.

223 utpatients (age: 63+/-9 years; 76% men) with coronary artery disease.

224 This paper reports on patients with coronary artery disease.

225 re, cardiac masses, pericardial disease, and coronary artery disease.

226 r against each other in patients with stable coronary artery disease.

227 recludes assessing physiological severity of coronary artery disease.

228 raphy, as compared with patients with stable coronary artery disease.

229 large cohort of patients suspected of having coronary artery disease.

230 Cs) are promising therapeutic strategies for coronary artery disease; however, donor-related variabil

231 .G202V HAND2 variant associated with CHD and coronary artery diseases found in a large Lebanese famil

232 nts because of persistent or new spontaneous coronary artery dissection are common during long-term f

233 ontemporary pregnancy-associated spontaneous coronary artery dissection cases in an attempt to define

234 Pregnancy-associated spontaneous coronary artery dissection is commonly associated with l

235 or cases of pregnancy-associated spontaneous coronary artery dissection reported between 2000 and 201

236 deletion of Smad4 increased the diameter of coronary arteries during mouse embryonic development, a

237 ity, was induced by the passaging of porcine coronary artery ECs from passage P1 to P4, and was assoc

238 ake of normal and PE STBEVs by primary human coronary artery endothelial cells (HCAEC) and the effect

239 tibacter actinomycetemcomitans (Aa) in human coronary artery endothelial cells (HCAECs).

240 NFalpha and cigarette smoke extract on human coronary artery endothelial cells under oscillatory, nor

241 overage and 230-microm spatial resolution at coronary artery evaluation in patients with atrial fibri

242 al contractile function (myocardial strain), coronary artery flow, and myocardial perfusion.

243 ft anterior descending (LAD) ligation of the coronary artery followed by intramyocardial PBS injectio

244 ved angiogenic precursor cell population for coronary artery formation in mice and show that a DLL4/N

245 wever, developmental mechanisms underpinning coronary artery formation remain ill-defined.

246 d in EC overlying atherosclerotic plaques in coronary arteries from patients with ischemic heart dise

247 ater sensitivity to PGF2alpha in the porcine coronary artery from males.

248 ta support a model in which DACH1 stimulates coronary artery growth by activating Cxcl12 expression a

249 ete revascularization of non-infarct-related coronary arteries guided by fractional flow reserve (FFR

250 s (Global Use of Strategies to Open Occluded Coronary Arteries [GUSTO], Thrombolysis in Myocardial In

251 to restore blood flow in an infarct-related coronary artery improves outcomes.

252 Similar alterations were seen in coronary arteries in vivo Thus, endothelial cells percei

253 undergone primary PCI of an infarct-related coronary artery in a 1:2 ratio to undergo complete revas

254 Calcification of coronary arteries is a prominent feature in both groups

255 in combination with nonobstructed epicardial coronary arteries is the prerequisite of normal myocardi

256 vors where initial assessment reveals normal coronary arteries, left ventricular function, and restin

257 eatment of the vulnerable plaque, that is, a coronary artery lesion with a high likelihood of rupture

258 Kyoto rats (WKY) were subjected to 45 min of coronary artery ligation and reperfusion for 12 weeks.

259 feration and heart functional recovery after coronary artery ligation both correlated with pre-injury

260 B6sv129-mice were subjected to in vivo left coronary artery ligation for 30 minutes followed by 72 h

261 ZYZ-168 was administered to rats with coronary artery ligation over a period of six weeks.

262 jected to permanent left anterior descending coronary artery ligation.

263 lusters were visualized in mice subjected to coronary artery ligation.

264 ale C57BL/6 mice were subjected to permanent coronary artery ligation.

265 Left main coronary artery (LMCA) compression is increasingly recog

266 idance when performing unprotected left main coronary artery (LMCA) percutaneous coronary interventio

267 erm management is based primarily on maximal coronary artery luminal dimensions, normalized as Z scor

268 Myocardial infarction with nonobstructive coronary arteries (MINOCA) occurs in 5% to 10% of all pa

269 Adult ANX - A1 (-/-) mice subjected to coronary artery occlusion exhibited increased infarct si

270 e group receiving PCI for an infarct-related coronary artery only.

271 ion velocity), and coronary atherosclerosis (coronary artery plaque volume).

272 The primary outcome was noncalcified coronary artery plaque volume, as determined by coronary

273 stosterone slows progression of noncalcified coronary artery plaque volume.

274 e been used in clinical research for imaging coronary artery plaque, and ongoing clinical studies are

275 thromboxane and PGF2alpha are released from coronary artery PVAT from pigs.

276 as obtained during a 1-minute proximal right coronary artery (RCA) and left coronary artery balloon o

277 eart Association guidelines, the new AUC for coronary artery revascularization were separated into 2

278 definitions in a population-based study, the Coronary Artery Risk Development in Young Adults (CARDIA

279 ce of 4 established definitions of DD in the Coronary Artery Risk Development in Young Adults (CARDIA

280 mericans (56% women, 18-30 years old) in the Coronary Artery Risk Development in Young Adults (CARDIA

281 Using data from the Coronary Artery Risk Development in Young Adults (CARDIA

282 The Coronary Artery Risk Development in Young Adults (CARDIA

283 d white men and women from the observational Coronary Artery Risk Development in Young Adults study,

284 llow-up evaluations in 977 subjects from the Coronary Artery Risk Development in Young Adults study.

285 and 2 observational cohort studies (CARDIA [Coronary Artery Risk Development in Young Adults] and Bi

286 23; p = 0.008) and high-risk from lower-risk coronary arteries (ROC AUC: 0.76; 95% CI: 0.62 to 0.91;

287 The validated microstructural model of coronary artery sheds light on vascular biomechanics and

288 ence imaging detected nanoparticles in human coronary artery-sized atheroma in vivo (P<0.05 versus re

289 ause patients with diabetes have more severe coronary artery stenosis, we hypothesized that graft pat

290 y artery calcium score (CACS) for predicting coronary artery stenosis.

291 etween patients with different severities of coronary artery stenosis.

292 rmal value, and is independent of epicardial coronary artery stenosis.

293 ) is an index used to assess the severity of coronary-artery stenosis.

294 igned patients who were scheduled to undergo coronary-artery surgery and were at risk for perioperati

295 Coronary artery thrombosis caused by plaque erosion may

296 We propose segmenting coronary artery thrombosis caused by plaque rupture into

297 e ACS that occur without apparent epicardial coronary artery thrombus or stenosis.

298 Doses to the coronary arteries were optimized.

299 clusion Whole-heart CT enables evaluation of coronary arteries with high image quality, low radiation

300 raphy (IVUS) of the left anterior descending coronary artery, within 8 weeks of HT.