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1 er was developed to identify inflammation in coronary atheroma.
2 re caused by acute disruption of an unstable coronary atheroma.
3  to decrease the mechanical stability of the coronary atheroma.
4 mulated at ruptured or eroded sites of human coronary atheroma.
5        Lipoprotein(a) is ubiquitous in human coronary atheroma.
6 d intimal medial thickness and the volume of coronary atheroma also can be reduced by LDL cholesterol
7                          Statins can regress coronary atheroma and lower clinical events.
8  both vascular and cardiac tissue (including coronary atheroma) and effectively constricts isolated a
9 Pl(A) polymorphisms in relation to extent of coronary atheroma as characterized by angiography and a
10 s were compared with regard to the extent of coronary atheroma at baseline and subsequent change in r
11 vastatin on Intravascular Ultrasound-Derived Coronary Atheroma Burden (ASTEROID) assessed whether ros
12 vastatin on Intravascular Ultrasound-Derived Coronary Atheroma Burden [ASTEROID]) was performed at 53
13 A motorized IVUS pullback was used to assess coronary atheroma burden at baseline and after 24 months
14 risk factors with volumetric measurements of coronary atheroma burden in patients with coronary arter
15 omparing the effect of ETC-216 or placebo on coronary atheroma burden measured by intravascular ultra
16 pecifically whether the locus contributes to coronary atheroma burden or plaque instability.
17  Chlamydia pneumoniae has been identified in coronary atheroma, but concomitant serum antibody titers
18                                 The Study of Coronary Atheroma by Intravascular Ultrasound: Effect of
19 , it remains unclear if statins can modulate coronary atheroma calcification in vivo.
20 r plaque-regressive effects, statins promote coronary atheroma calcification.
21                     The TF activity of human coronary atheroma has not been reported.
22 cidence of acute thrombotic complications of coronary atheroma in clinical trials remains unknown.
23                            We analyzed human coronary atheroma in de novo and restenotic disease to i
24  was identified by nonculture methods within coronary atheromas obtained at autopsy.
25 associated with cardiovascular risk markers, coronary atheroma, or CHD.
26                              We investigated coronary atheroma progression in patients with low level
27                                              Coronary atheroma progression was evaluated by serial in
28 etes mellitus is associated with accelerated coronary atheroma progression.
29  evaluate the effect of medical therapies on coronary atheroma progression.
30 tion of C. pneumoniae from human carotid and coronary atheromas provides additional support for a rol
31                                  We examined coronary atheroma removed from 72 patients with stable o
32 bridization studies using specimens of human coronary atheromas showed expression of IL-8 mRNA in a m
33               This study compared changes in coronary atheroma volume and calcium indices (CaI) in pa
34  serial intravascular ultrasound measures of coronary atheroma volume in patients treated with rosuva
35                                              Coronary atheroma was quantified by means of electron be
36 mydia pneumoniae and coronary heart disease, coronary atheromas were collected from patients undergoi

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