ライフサイエンスコーパス: cystitis

1 eremia, 75 from urosepsis, and 74 from acute cystitis).

2 biotic-exposed sister with chronic recurrent cystitis.

3 nce of painful bladder syndrome/interstitial cystitis.

4 utic target for the treatment of hemorrhagic cystitis.

5 K, and ERK1/2 in the inflamed bladder during cystitis.

6 r than being attributed to radiation-induced cystitis.

7 ry bladder and in bladder hypertrophy during cystitis.

8 RG with intermediate and chronic CYP-induced cystitis.

9 )-IR was also increased (P < or = 0.01) with cystitis.

10 ders such as overactive bladder syndrome and cystitis.

11 AI contributes to UPEC pathogenesis in acute cystitis.

12 lH significantly protected mice from chronic cystitis.

13 scade in a well-characterized mouse model of cystitis.

14 cord injury, bladder outlet obstruction, or cystitis.

15 diseases, e.g., irritable bowel syndrome or cystitis.

16 al for UPEC virulence in the murine model of cystitis.

17 ry tract infections, clinically described as cystitis.

18 icroenvironment characteristic of high-titer cystitis.

19 emia and was not associated with hemorrhagic cystitis.

20 nd is a potential biomarker for interstitial cystitis.

21 tion of p-CREB-IR cells expressed p-Trk with cystitis.

22 on UPEC virulence in an acute mouse model of cystitis.

23 nic (10 days) cyclophosphamide (CYP)-induced cystitis.

24 YP injection and was maintained with chronic cystitis.

25 cells in the L4-L5 DRG was not altered with cystitis.

26 fold; P < or = 0.05) after acute and chronic cystitis.

27 e resistance in pyelonephritis as well as in cystitis.

28 in L1 DRG with chronic (10 days) CYP-induced cystitis.

29 ed urinary tract plasticity with CYP-induced cystitis.

30 Trk-IR with acute and/or chronic CYP-induced cystitis.

31 nic (10 days) cyclophosphamide (CYP)-induced cystitis.

32 sis, interstitial nephritis, and hemorrhagic cystitis.

33 omarkers and shed light into the etiology of cystitis.

34 lexia in the clinical syndrome, interstitial cystitis.

35 ed infections of the urinary tract including cystitis.

36 single most prevalent clonal group in acute cystitis.

37 tance P and neurokinin-1 (NK-1) receptors in cystitis.

38 ly evaluate the importance of substance P in cystitis.

39 onic model of cyclophosphamide (CYP)-induced cystitis.

40 ladder function observed in animal models of cystitis.

41 the bacterial properties necessary to cause cystitis.

42 amide to reduce the incidence of hemorrhagic cystitis.

43 ot significantly correlated with hemorrhagic cystitis.

44 circuits distant from these rarely produced cystitis.

45 ence factors important in the development of cystitis.

46 requency and decreased bladder capacity with cystitis.

47 in bladder and urine ruled out an infectious cystitis.

48 tion of KGF is protective against CP-induced cystitis.

49 e strain caused bacteremia, whereas 4 caused cystitis.

50 A) strains isolated from patients with acute cystitis.

51 acellular bacterial communities during acute cystitis.

52 d cyclooxygenase (COX)-2 in ketamine-induced cystitis.

53 herichia coli (UPEC) is the leading cause of cystitis.

54 ild-type mice in a well-established model of cystitis.

55 riate first-line therapies for uncomplicated cystitis.

56 ssociated with increasing bacteriuria during cystitis.

57 athogens have complicated treatment of acute cystitis.

58 efractory bladder pain syndrome/interstitial cystitis.

59 paring antimicrobial for acute uncomplicated cystitis.

60 GBS may suppress bladder inflammation during cystitis.

61 complications included transient hemorrhagic cystitis (1 patient), vaginal bleeding (2 patients), gas

62 re: diarrhea (53%), fever (21%), hemorrhagic cystitis (12%), and pneumonitis (11%).

63 age, 6.4% had moderate or severe hemorrhagic cystitis, 2.8% had pulmonary hemorrhage, and 2% had intr

64 of patients with diagnosis of acute urinary cystitis, 294 patients whose urine cultures were positiv

65 mon infections were urinary tract infection (cystitis) (34.9% vs 25.2%), cytomegalovirus viremia (17.

66 acute pyelonephritis, 38 from patients with cystitis, 49 from patients with catheter-associated bact

67 ns associated with the clinical syndromes of cystitis (82%) and acute pyelonephritis (79%) than in fe

68 syndromes of acute pyelonephritis (79%) and cystitis (82%) than in those associated with catheter-as

69 Among women with uncomplicated cystitis, a 3-day regimen of cefpodoxime compared with c

70 n the United States suffer from interstitial cystitis, a chronic painful urinary bladder disorder cha

71 reemerge to cause some episodes of recurrent cystitis, a familiar clinical scenario in otherwise heal

72 Although cystitis accounts for 95% of visits to physicians for sy

73 premenopausal women with acute uncomplicated cystitis accurately showed evidence of bladder E. coli b

74 o increased (P < or = 0.05) with CYP-induced cystitis (acute, intermediate, and chronic).

75 L2: 8-fold; P < or = 0.05) only with chronic cystitis, although it increased in the L6-S1 DRG with CY

76 l antibiotic use in women with uncomplicated cystitis and 42.6% Escherichia coli fluoroquinolone resi

77 Women with acute cystitis and a history of UTI within the previous year s

78 idence of participation of NK-1 receptors in cystitis and a mandatory participation of these receptor

79 Cystitis and acute pyelonephritis, infection of the blad

80 yoma-BK virus is associated with hemorrhagic cystitis and also with polyomavirus nephropathy (PVN).

81 ptococcus agalactiae causes both symptomatic cystitis and asymptomatic bacteriuria (ABU); however, gr

82 In contrast, cystitis and fecal isolates differed minimally.

83 such conditions as renal colic, interstitial cystitis and inflammatory bowel disease by purinergic ag

84 onization by the parent strain caused severe cystitis and interstitial nephritis as determined by his

85 on the predictability of the agents causing cystitis and knowledge of their antimicrobial susceptibi

86 f clonal and pathotypic similarities between cystitis and NBM isolates of E. coli O18:K1:H7.

87 commonality between O18:K1:H7 isolates from cystitis and NBM suggests common pathogenetic mechanisms

88 ection, control and SCI rats developed acute cystitis and pyelitis without acute differences in histo

89 They respond to both cystitis and pyelonephritis by delivering bacteriostatic

90 athogens: 2 probes were more prevalent among cystitis and pyelonephritis isolates, 2 among pyelonephr

91 n receptor as a UPEC virulence factor during cystitis and pyelonephritis, a fitness factor during bac

92 hogens causing community-acquired UTIs, both cystitis and pyelonephritis, is increasing.

93 at 30 mg/kg of body weight twice a day cured cystitis and renal infection in noncatheterized mice.

94 ered from patients with clinical symptoms of cystitis and that from patients with symptoms of pyelone

95 is isolate used to study the pathogenesis of cystitis and to develop a FimH (type 1 fimbrial adhesin)

96 syndrome and most environmentally similar to cystitis and urethritis, all of which are inflammatory d

97 , significant (P < or = 0.015) predictors of cystitis and/or pyelonephritis (versus fecal) included a

98 al groups were significantly associated with cystitis and/or pyelonephritis.

99 sted), 50 (81%) had symptoms consistent with cystitis, and 12 (19%) had symptoms of pyelonephritis.

100 riuria, 45 complicated UTI, 38 uncomplicated cystitis, and 77 pyelonephritis).

101 Infection progressed to cystitis, and a biofilmlike covering was observed over t

102 tis, pneumonia, gastroenteritis, hemorrhagic cystitis, and keratoconjunctivitis.

103 Irritable bowel syndrome, interstitial cystitis, and other chronic pelvic pain (CPP) disorders

104 s from women with first-episode or recurrent cystitis, and papG genotype was compared with clinical o

105 to gastrointestinal hemorrhage, hemorrhagic cystitis, and pulmonary hemorrhage.

106 ase, acquired hemolytic anemia, interstitial cystitis, and Sjogren's syndrome had higher prevalence r

107 rature on bladder pain syndrome/interstitial cystitis, and the implications for their patients.

108 lar disorder, tension headache, interstitial cystitis, and the postconcussion syndrome.

109 efractory bladder pain syndrome/interstitial cystitis appears promising, larger-scale studies with ad

110 f postinvasion events in the pathogenesis of cystitis are largely undetermined.

111 tags remained in mice that developed chronic cystitis, arguing that during the acute stages of infect

112 ervoirs (QIRs) to the development of chronic cystitis as defined by persistent bacteriuria.

113 ns of Escherichia coli, the primary cause of cystitis, assemble adhesive surface organelles called ty

114 This report describes two episodes of acute cystitis associated with "mixed flora" in an elderly mal

115 total of 29 isolates from two collections of cystitis-associated E. coli were evaluated by using meth

116 ence genotype that included papG allele III (cystitis-associated P fimbrial adhesin), sfaS (S fimbria

117 Epidemiological studies indicate that cystitis-associated strains appear to differ from pyelon

118 d was derived from the same (meningitis- and cystitis-associated) subclone of E. coli O18:K1:H7 as th

119 ssified as causing acute cystitis, recurrent cystitis, asymptomatic bacteriuria, or pyelonephritis co

120 A/OT-I mice spontaneously develop autoimmune cystitis at 10 wk of age.

121 and inflamed (cyclophosphamide (CYP)-induced cystitis) bladder urothelium and their contribution to l

122 scopic urinalysis for hematuria (hemorrhagic cystitis, bladder cancer), ECG (anthracycline-related co

123 f the micturition reflex in diseases such as cystitis, bladder/sphincter dyssynergia following spinal

124 Interstitial cystitis/bladder pain syndrome (IC) is associated with s

125 Patients with interstitial cystitis/bladder pain syndrome (IC/BPS) suffer from chro

126 l pelvic pain disorders such as interstitial cystitis/bladder pain syndrome (IC/BPS).

127 s/chronic pelvic pain syndrome, interstitial cystitis/bladder pain syndrome and chronic orchialgia ar

128 hronic pelvic pain syndrome and interstitial cystitis/bladder pain syndrome.

129 Conditions covered include acute cystitis (both uncomplicated and complicated), catheter-

130 genic GBS isolated from a patient with acute cystitis bound to human T24 bladder uroepithelial cells

131 ted from cells of patients with interstitial cystitis but not controls.

132 cutaneous hypersensitivity in the context of cystitis, but had no effect in uninjured, naive mice.

133 herichia coli (UPEC) in the initial stage of cystitis, but the bacterial determinants of postinvasion

134 Normal responses following induction of cystitis by cyclophosphamide were also observed in both

135 sses neutrophil migration early in bacterial cystitis by eliciting an IDO-mediated increase in local

136 ated with E. coli but appear to rarely cause cystitis by themselves.

137 ine model of urinary tract infections (UTI), cystitis by uropathogenic Escherichia coli (UPEC) occurs

138 (+/-SD) days of antibiotic therapy for ASB, cystitis, CA-UTI and pyelonephritis were 10.0 (4.5), 11.

139 regulation of p-CREB in DRG, suggesting that cystitis can reveal an altered CREB phosphorylation that

140 Most notably, they are found in up to 50% of cystitis cases in children and 30% of pyelonephritis in

141 ibodies against CNF1 and/or Hly would reduce cystitis caused by CP9.

142 new bladder pain syndrome, ketamine-induced cystitis, characterized by chronic inflammation and urot

143 The dog's cystitis clone was the most extensively recovered clone.

144 aged 18 to 55 years with acute uncomplicated cystitis comparing ciprofloxacin (n = 150) with cefpodox

145 -37), were infected transurethrally with the cystitis-derived uropathogenic Escherichia coli (UPEC) s

146 each of the most virulent pyelonephritis and cystitis E. coli strains in our collection.

147 rthermore, in women with acute uncomplicated cystitis, empirical therapy without a pretherapy urine c

148 ients treated for BKV-associated hemorrhagic cystitis experienced complete resolution of gross hematu

149 rs of recurrence and/or persistence in acute cystitis, extended virulence genotypes were compared wit

150 The O18:K1:H7 cystitis, fecal, and NBM isolates were clonally derived.

151 were reported including vomiting, diarrhoea, cystitis, gastroenteritis, and bleeding.

152 past year the association with interstitial cystitis has been explored, and neurally mediated hypote

153 ave been elucidated, and the murine model of cystitis has generated a new paradigm by which acute and

154 ctory bladder pain syndrome and interstitial cystitis have emerged.

155 ts, are associated not only with hemorrhagic cystitis (HC) but also with hepatitis, conjunctivitis, a

156 iation of BK plasma viremia with hemorrhagic cystitis (HC) in hematopoietic cell transplant (HCT) rec

157 Hemorrhagic cystitis (HC) remains a common complication of allogenei

158 otype O18:K1:H7, taken from women with acute cystitis, healthy control patients, and infants with neo

159 This study on interstitial cystitis (IC) aims to identify a unique urine metabolomi

160 atory bladder disorders such as interstitial cystitis (IC) deserve attention since a major problem of

161 Interstitial cystitis (IC) is a debilitating disease that has been ad

162 d in the urine of patients with interstitial cystitis (IC)-a chronic, painful bladder disease of unkn

163 ithelial cells of patients with interstitial cystitis (IC).

164 dder problems (possibly urinary interstitial cystitis [IC]), thyroid disorders, chronic headaches/mig

165 The analysis of 236 episodes of cystitis in 226 women yielded 202 paired specimens of mi

166 These findings suggest that in acute cystitis in adult women, the class III papG allele predo

167 antoin, or fosfomycin is indicated for acute cystitis in adult women.

168 in renal transplant patients or hemorrhagic cystitis in bone marrow transplant recipients.

169 Acute uropathogenic Escherichia coli (UPEC) cystitis in C57BL/6 and C3H/HeN males recapitulated the

170 ly responsible for the signs and symptoms of cystitis in humans infected with toxigenic UPEC.

171 i type 1 pili may have utility in preventing cystitis in humans.

172 unctionally to reveal marked exacerbation of cystitis in IL-10-deficient mice.

173 ome of uropathogenic Escherichia coli (UPEC) cystitis in mice using genome-wide expression profiling

174 e inflammatory signaling of ketamine-induced cystitis in rat urinary bladder.

175 r, ameliorates cyclophosphamide (CP)-induced cystitis in rats, KGF (5 mg/kg) was injected in rats as

176 As with cystitis in reproductive-age women, sexual behaviors and

177 acute (24 h) and chronic (8 day) CYP-induced cystitis in the rat, B1R mRNA was detected throughout th

178 under the name Elmiron for the treatment of cystitis in the United States.

179 Acute uncomplicated cystitis in women can be diagnosed without an office vis

180 ts from experimental infections and cases of cystitis in women suggest that type 1 fimbrial genes are

181 NA-seq) analyses of RNA from E. coli causing cystitis in women that iron acquisition systems, includi

182 es for the management of acute uncomplicated cystitis in women that recommend empirical therapy in pr

183 choice for treatment of acute uncomplicated cystitis in women.

184 ng empirical therapy for acute uncomplicated cystitis in women.

185 Virulence genes, group B2, and having caused cystitis (in the mother or dog) corresponded to coloniza

186 38-51% of co-trimoxazole resistance in acute cystitis), including ten (34%) of 29 isolates that were

187 CYP-induced cystitis increased (P < or = 0.001) p75(NTR) expression

188 These results demonstrated that CYP-induced cystitis increases the expression and phosphorylation of

189 elevated urinary IL-10 in patients with UPEC cystitis, indicating a role for IL-10 in the innate resp

190 in micturition reflex pathways after chronic cystitis induced by cyclophosphamide (CYP).

191 We present a model of neurogenic cystitis induced by viral infection of specific neuronal

192 zing NGF antibody also subsequently reversed cystitis-induced increases in bladder weight.

193 hanges in sensory pathways may contribute to cystitis-induced pain and hyperactivity of the bladder.

194 use the neutralizing NGF antibody attenuated cystitis-induced type I collagen up-regulation in the in

195 Interstitial cystitis is a common disease among women and is frequent

196 Interstitial cystitis is a complex inflammatory condition of the blad

197 Bladder pain syndrome/interstitial cystitis is an important and poorly understood disorder.

198 Hemorrhagic cystitis is an inflammatory and ulcerative bladder condi

199 Cystitis is caused by a relatively small number of bacte

200 and urothelial ATP release with CYP-induced cystitis is decreased with TGF-beta inhibition.

201 The cause of acute uncomplicated cystitis is determined on the basis of cultures of voide

202 standing of the pathogenesis of interstitial cystitis is emerging and this will hopefully lead to the

203 h we view bladder pain syndrome/interstitial cystitis is evolving, as is apparent in the literature t

204 The pathophysiology of interstitial cystitis is incompletely understood, although altered ep

205 II(UTI89) during the establishment of acute cystitis is maintenance of wild-type leuX, and not PAI I

206 Acute cystitis is one of the most commonly encountered bacteri

207 tutive expression of type 1 fimbriae in UPEC cystitis isolate F11 and the laboratory strain E. coli K

208 ed mutants of uropathogenic Escherichia coli cystitis isolate F11 were used to assess the role of the

209 eys were challenged with 1 mL of 108 E. coli cystitis isolate NU14.

210 The pathogenesis of the K. pneumoniae cystitis isolate TOP52 was compared to that of the uropa

211 isolate UTI89 and the Klebsiella pneumoniae cystitis isolate TOP52.

212 Escherichia coli NU14, a cystitis isolate used to study the pathogenesis of cysti

213 virulence of two reference UPEC strains, the cystitis isolate UTI89 and the urosepsis isolate CFT073.

214 migration by purified YbcL(UTI), encoded by cystitis isolate UTI89, required the presence of a uroep

215 studies showed that the effects of the UTI89 cystitis isolate were fimH dependent.

216 e factors (VFs) among the 35 sought than did cystitis isolates (n=23).

217 The cystitis isolates and archetypal NBM isolates RS218 and

218 brial adhesin gene) predominates among human cystitis isolates and confers an adherence phenotype res

219 ng pyelonephritis isolates only, and 3 among cystitis isolates only than among fecal isolates.

220 Cystitis isolates present in the urine of mice during th

221 auxotrophs, which are found frequently among cystitis isolates, exhibited normal growth in urine.

222 Compared with fecal and cystitis isolates, pyelonephritis isolates exhibited a g

223 In general, cystitis isolates, unlike pyelonephritis isolates, were

224 om seven UPEC (three pyelonephritis and four cystitis) isolates and three fecal/commensal strains, in

225 iator of bladder dysfunction and pain during cystitis, it is presently unclear if it is also importan

226 Ketamine-induced ulcerative cystitis (KIC) initially damaged the bladder mucosa and

227 tiates bladder pain and, in ketamine-induced cystitis, loss of urothelium from large areas of the bla

228 isplayed a striking predilection for chronic cystitis, manifesting as persistent bacteriuria, high-ti

229 Changes in PACAP expression after cystitis may play a role in altered visceral sensation (

230 Alterations in GAP-43-IR following chronic cystitis may suggest a reorganization of bladder afferen

231 er titers and formed more IBCs in the murine cystitis model than wild type.

232 In a murine cystitis model, UTI89 surA::kan was unable to persist in

233 richia coli (UPEC) isolate UTI89 in a murine cystitis model.

234 ivity has not been delineated in a dedicated cystitis model.

235 both an in vitro model and an in vivo murine cystitis model.

236 States with acute pyelonephritis (n = 170), cystitis (n = 83), or no infection (fecal; n = 76).

237 Cystitis, nausea, vomiting, and diarrhea were also obser

238 Episodes of urinary tract infection (cystitis) occurred more often in the denosumab than in t

239 increased in the L6-S1 DRG with CYP-induced cystitis of acute (2-3-fold; P < or = 0.05) and chronic

240 t in the L2, L4, and L5 DRG with CYP-induced cystitis of acute and chronic duration compared with con

241 The effects of acute cystitis on distal colonic sensory thresholds to CRD and

242 ts who experienced grades 2 to 3 hemorrhagic cystitis, only 1 of whom had received continuous bladder

243 No hemorrhagic cystitis or bone marrow suppression was observed.

244 ntiating Escherichia coli strains that cause cystitis or pyelonephritis from fecal E. coli remain inc

245 Escherichia coli isolates from patients with cystitis or pyelonephritis produce the pore-forming cyto

246 id clinical isolates from cases of pediatric cystitis or pyelonephritis, adult pyelonephritis or uros

247 infections, which can present clinically as cystitis or pyelonephritis.

248 After chronic cystitis, PACAP immunoreactivity in cells in the L1, L2,

249 After chronic, CYP-induced cystitis, PACAP immunoreactivity increased dramatically

250 ive bladder syndrome (OABS) and interstitial cystitis/painful bladder syndrome (IC/PBS) are debilitat

251 ycopeptide urinary biomarker of interstitial cystitis/painful bladder syndrome (IC/PBS), a chronic co

252 secreted by bladder cells from interstitial cystitis patients and is a potent inhibitor of both norm

253 ical sources (acute pyelonephritis patients, cystitis patients, and fecal controls) were tested under

254 the patient was readmitted with hemorrhagic cystitis, persistent thrombocytopenia, and bilateral pul

255 lly relevant subgroups within the "recurrent cystitis" population.

256 a (range, 0-1.0 x 10 copies/mL), hemorrhagic cystitis (present/absent), and data on kidney function.

257 vances in both in vitro and animal models of cystitis promise to provide insights into the bacterial

258 from 18 to 49 years of age with symptoms of cystitis provided specimens of midstream urine, after wh

259 c human clinical isolates from patients with cystitis, pyelonephritis, bacteremia, or meningitis, inc

260 ine of women and classified as causing acute cystitis, recurrent cystitis, asymptomatic bacteriuria,

261 ppropriate diagnostic tests for interstitial cystitis remain uncertain.

262 gnificantly among uropathogens causing acute cystitis, resistance to nitrofurantoin and ciprofloxacin

263 en expression in the inflamed bladder during cystitis revealed that Akt, JNK, and ERK1/2 activities w

264 We report that during cystitis, shedding of infected bladder epithelial cells

265 voiding abnormalities presenting with acute cystitis should be treated similarly to women without di

266 ted here that cyclophosphamide (CYP)-induced cystitis significantly increased the production of type

267 pared the impact of ybtPQ on a model E. coli cystitis strain during in vitro culture and experimental

268 ce, an attribute not exhibited by a clinical cystitis strain.

269 There was no significant correlation between cystitis strains (n = 20) and the pic determinant.

270 The cystitis strains also suppressed epithelial cytokine res

271 Cystitis strains colonize the bladder in higher numbers,

272 The results indicate that cystitis strains colonize the bladder more rapidly than

273 These results provide evidence that cystitis strains differ from pyelonephritis strains in t

274 The cystitis strains induced significantly less IL-6 than di

275 onclude that, in this in vitro model system, cystitis strains of UPEC have genes encoding factors tha

276 sion of type 1 fimbriae is more critical for cystitis strains than for pyelonephritis strains in the

277 otoxicity of hemolytic, CNF1(+), and CNF1(-) cystitis strains toward human T24 bladder epithelial cel

278 nses of bladder epithelial cell lines to the cystitis strains UTI89 and NU14.

279 ful for the study of the uropathogenicity of cystitis strains, and that it would be unwise to use pye

280 on, a higher number of IBCs precedes chronic cystitis than precedes QIR formation.

281 nized mice also exhibited significantly less cystitis than sham-immunized mice.

282 t only resulted in the development of strong cystitis that persisted significantly longer than in mic

283 t study we used established animal models of cystitis to determine the time course of bladder inflamm

284 may be lower among women with uncomplicated cystitis treated with TMP-SMX when the infecting pathoge

285 In a murine model of cystitis, type 1 pili-mediated bladder epithelial invasi

286 infection was developed using the prototypic cystitis UPEC isolate NU14 harboring a plasmid encoding

287 During the establishment of bacterial cystitis, UPEC suppresses innate responses via multiple

288 During murine cystitis, uropathogenic E. coli (UPEC) utilizes type 1 p

289 During acute cystitis, uropathogenic Escherichia coli (UPEC) induce b

290 During cystitis, uropathogenic Escherichia coli (UPEC) subvert

291 rain 807, isolated from a patient with acute cystitis, using metabolic arrays of >2,500 substrates an

292 al titers nor a reduction in the severity of cystitis versus that of sham-immunized mice.

293 in vitro and in bladder tissue during murine cystitis via a noncanonical, interferon-independent path

294 olonization of the urine or kidney; however, cystitis was reduced significantly in mice treated with

295 t in urine samples collected from women with cystitis were tested for type 1 fimbria switch orientati

296 strains in the urine of a patient with acute cystitis, where half of the isolates carried a glycine-t

297 provided a fitness advantage during chronic cystitis, which is manifested as persistent bacteriuria,

298 nal cohort study, the outpatients with acute cystitis with isolation of E. coli in their urine cultur

299 -beta signalling in cyclophosphamide-induced cystitis with TbetaR-1 inhibition decreased afferent ner

300 entation, or bladder sympathectomy prevented cystitis without affecting the CNS disease, indicating a