ライフサイエンスコーパス: dorsal root ganglia

1 iceptive fibres of the somatosensory system (dorsal root ganglia).

2 rum/pylorus, enteric neurones, and vagal and dorsal root ganglia.

3 subclasses of peripheral neurons from mouse dorsal root ganglia.

4 g sympathetic, parasympathetic, enteric, and dorsal root ganglia.

5 eceptors were among the highest expressed in dorsal root ganglia.

6 expressed in nociceptive sensory neurons of dorsal root ganglia.

7 C activity is found associated with TRPV1 in dorsal root ganglia.

8 00 years ago, VZV was carried along in their dorsal root ganglia.

9 fibers in human skin and sensory neurons in dorsal root ganglia.

10 mRNA and protein expression was detected in dorsal root ganglia.

11 nal segments in cocultures with neurons from dorsal root ganglia.

12 nd reduce levels of phosphorylated VEGFR1 in dorsal root ganglia.

13 ently infected neurons in trigeminal but not dorsal root ganglia.

14 dermal innervation and cell-body loss in the dorsal root ganglia.

15 prevalent in distal sural nerves compared to dorsal root ganglia.

16 tained the lowest mean HSV-2 DNA load in the dorsal root ganglia.

17 essed in cardiac sensory neurons in thoracic dorsal root ganglia.

18 f many broadly expressed mRNA transcripts in dorsal root ganglia.

19 ions from nociceptive sensory neurons of rat dorsal root ganglia.

20 , including the injured peripheral nerve and dorsal root ganglia.

21 o reactivate from trigeminal and lumbosacral dorsal root ganglia.

22 4 genes associated with nerve injury in the dorsal root ganglia.

23 essed in cardiac sensory neurons in thoracic dorsal root ganglia.

24 le in maintaining proper P2X3R expression in dorsal root ganglia.

25 ely in sensory neurons in the trigeminal and dorsal root ganglia.

26 an immortalized cell line derived from human dorsal root ganglia.

27 , and groups of somatosensory neurons in the dorsal root ganglia.

28 eno-associated virus transgene expression in dorsal root ganglia.

29 els were greater than Piezo1 channels in rat dorsal root ganglia.

30 nist in BE(2)-C cells, and in trigeminal and dorsal root ganglia.

31 e neurons from cultures of dissociated mouse dorsal-root ganglia.

32 Axotomy of the peripheral branch of adult dorsal root ganglia (a "conditioning" lesion) triggers a

33 resent in mouse and human sensory neurons of dorsal root ganglia, a substantial number of peripheral

34 haracteristic lysosomal storage pathology in dorsal root ganglia affecting neurons, satellite cells (

35 from the 4th lumbar (L4) and 5th lumbar (L5) dorsal root ganglia after L5 spinal nerve ligation (SNL)

36 eripheral nervous system consists of reduced dorsal root ganglia and cranial nerves, and the entire g

37 sections showed intense Na(V)1.8 labeling in dorsal root ganglia and intracardiac ganglia and only mo

38 d genes that is primarily expressed in human dorsal root ganglia and mast cells.

39 al vein results in extensive transduction of dorsal root ganglia and motor neurons throughout the spi

40 lite glia, and no Schwann cell precursors in dorsal root ganglia and peripheral nerves.

41 so commonly develop in other cranial nerves, dorsal root ganglia and peripheral nerves.

42 lar aberrations are apparent in prephenotype dorsal root ganglia and primary sensory neurons from dt

43 Olfm1 is coexpressed with NgR1 in dorsal root ganglia and retinal ganglion cells in embryo

44 to severe degenerative pathologic changes in dorsal root ganglia and sciatic nerve.

45 show that memory CD4 T cells migrate to the dorsal root ganglia and spinal cord in response to infec

46 was detected in peptidergic neurons of mouse dorsal root ganglia and spinal cord that transmit itch a

47 -derived neurotrophic factor in the thoracic dorsal root ganglia and spinal cord, and down-regulated

48 olog of Cbln2 and examined its expression in dorsal root ganglia and spinal cord.

49 to two functionally discrete structures, the dorsal root ganglia and sympathetic ganglia (SGs), are u

50 ith alterations in the central compartments (dorsal root ganglia and the spinal cord) and symptomatic

51 ecapitulates the selective death of sensory (dorsal root ganglia) and autonomic neurons observed in F

52 cluding locus ceruleus, retina, hippocampus, dorsal root ganglia, and adrenal chromaffin cells.

53 s tested, including spleen, paw skin, lumbar dorsal root ganglia, and lumbar spinal cord, postinfecti

54 TRPA1 in dermal sensory nerve fibers, their dorsal root ganglia, and mast cells.

55 a isoform is neuroprotective in hippocampal, dorsal root ganglia, and retinal neurons, but its proper

56 reduced the viral DNA load in the feet, the dorsal root ganglia, and the spinal cord relative to tha

57 (Axolotl) the correct number and spacing of dorsal root ganglia are regenerated.

58 muscle cells, and neurons in trigeminal and dorsal root ganglia, as detected by light and electron m

59 inhibit mammalian NaV channels expressed in dorsal root ganglia at concentrations up to 100 microM.

60 fferentiated motor neurons as well as in the dorsal root ganglia at E12.5.

61 essures up to 65 +/- 30 Pa for 10 min, while dorsal root ganglia axons can resist to pressures up to

62 We found that dorsal root ganglia axons have a 20% lower elastic modul

63 Application of CSPGs to postnatal rat dorsal root ganglia axons results in an increase in the

64 eir associated small-diameter neurons in the dorsal root ganglia (both IB4- and TrkA-positive), expre

65 the vagina and reduced latent viral loads in dorsal root ganglia but induced lower serum neutralizing

66 TRPv1 protein expression in dorsal root ganglia, but not skeletal muscle, was signif

67 bpopulation of neurons in the trigeminal and dorsal root ganglia, but was absent in sympathetic neuro

68 ll lines and in nociceptive neurons of mouse dorsal root ganglia, Cat-S and a decapeptide mimicking t

69 C)) acted as an enhancer in hypothalamic and dorsal root ganglia cells and responded to MAPK activati

70 ignificantly more active in hypothalamic and dorsal root ganglia cells but, significantly, and in con

71 cation transporter 2 (OCT2) is expressed on dorsal root ganglia cells within the nervous system wher

72 nction on specification and morphogenesis of dorsal root ganglia, craniofacial skeleton and melanopho

73 ta opioid receptor-Ca(2+)channel coupling in dorsal root ganglia desensitized by ARM390 and the rate

74 rdiac outflow tract septation and thymic and dorsal root ganglia development.

75 infection or shortly after virus reached the dorsal root ganglia, disease severity was significantly

76 infectious gE2-del virus was recovered from dorsal root ganglia (DRG) after multiple routes of inocu

77 e protease inhibitor, was upregulated in the dorsal root ganglia (DRG) after nerve injury, which was

78 mmunoreactive (-IR) cells in the lumbosacral dorsal root ganglia (DRG) also increased (P < or = 0.05)

79 ce to label neurons retrogradely in both the dorsal root ganglia (DRG) and nodose ganglia (NG).

80 ic and nonpeptidergic nociceptive neurons in dorsal root ganglia (DRG) and on axon terminals in lamin

81 cleared from plasma but all persisted in the dorsal root ganglia (DRG) and sciatic nerve (SN) for up

82 hese effects correlated with degeneration in dorsal root ganglia (DRG) and sciatic nerve and abundanc

83 pes simplex virus 1 (HSV-1) and HSV-2 DNA in dorsal root ganglia (DRG) and spinal cord (SC) were quan

84 icular cartilage and synovium, as well as in dorsal root ganglia (DRG) and spinal cord from a rat mod

85 a glutamate receptor and transporters in the dorsal root ganglia (DRG) and spinal cord.

86 To define which dorsal root ganglia (DRG) are activated by BAT SNS stimu

87 Neurons in the dorsal root ganglia (DRG) are composed of a variety of s

88 Peripheral sensory neurons in the dorsal root ganglia (DRG) are the initial transducers of

89 al-ventral pattern of neuronal identities in dorsal root ganglia (DRG) are unclear.

90 itry, in particular spinal cord targeting of dorsal root ganglia (DRG) axons and differentiation of n

91 Dorsal root ganglia (DRG) contain somatosensory neurons

92 g the C133W SPT mutant, we found that mutant dorsal root ganglia (DRG) during growth in vitro exhibit

93 We used dorsal root ganglia (DRG) explants and Drosophila larval

94 ransducing peripheral sensory neurons of the dorsal root ganglia (DRG) express kainate receptors (KAR

95 Here we used the ZW-X mouse line to analyze dorsal root ganglia (DRG) for intraganglionic connection

96 N-nitrosourea (ENU)-induced mutation affects dorsal root ganglia (DRG) formation in ouchless mutant z

97 he cell bodies of primary nociceptors within dorsal root ganglia (DRG) has been found to make major c

98 mber of TRPV1(+) neurons is increased in the dorsal root ganglia (DRG) in paclitaxel-treated rats and

99 spartate receptors (NMDARs) expressed in the dorsal root ganglia (DRG) in the inflammatory sensitizat

100 Changes in dorsal root ganglia (DRG) included macrophage infiltrati

101 expressed in most nociceptive neurons in the dorsal root ganglia (DRG) including TRPV1-positive cells

102 ed in primary afferent neurons isolated from dorsal root ganglia (DRG) innervating the lower gastroin

103 Delivering gene constructs into the dorsal root ganglia (DRG) is a powerful but challenging

104 Traffic of activated monocytes into the dorsal root ganglia (DRG) is critical for pathology in H

105 increase in FAAH mRNA and enzyme activity in dorsal root ganglia (DRG) L3-L5 ipsilateral to the affec

106 The sensory neurons of the dorsal root ganglia (DRG) must project accurately to the

107 a(2+)](i)) responses of small diameter adult dorsal root ganglia (DRG) neurones were determined.

108 We have previously shown that dorsal root ganglia (DRG) neurons activate the mammalian

109 itro and in patch-clamp electrophysiology in dorsal root ganglia (DRG) neurons and hippocampal slices

110 ECE-1 are expressed and colocalize in murine dorsal root ganglia (DRG) neurons and human skin nerves.

111 h lipid kinases generate PIP2 in nociceptive dorsal root ganglia (DRG) neurons and if these kinases r

112 hydrolyze extracellular AMP to adenosine in dorsal root ganglia (DRG) neurons and in the dorsal spin

113 Using recombinant systems, mouse-cultured dorsal root ganglia (DRG) neurons and in vivo experiment

114 acutely dissociated small diameter (<27 mum) dorsal root ganglia (DRG) neurons and on miniature (m)EP

115 Dorsal root ganglia (DRG) neurons are integral in transf

116 Dorsal root ganglia (DRG) neurons are known to express n

117 f diabetic neuropathy, increased Na(V)1.7 in dorsal root ganglia (DRG) neurons correlates with the em

118 rvous system revealed that Schwann cells and dorsal root ganglia (DRG) neurons developed abnormally i

119 r in which gain-of-function mutations render dorsal root ganglia (DRG) neurons hyperexcitable.

120 axons.SIGNIFICANCE STATEMENT Small-diameter dorsal root ganglia (DRG) neurons mediating nociception

121 he enhanced excitability of colon projecting dorsal root ganglia (DRG) neurons observed in diabetes i

122 Nociceptors are a subpopulation of dorsal root ganglia (DRG) neurons that detect noxious st

123 nels were examined in guinea-pig dissociated dorsal root ganglia (DRG) neurons using calcium imaging

124 receptor activation was investigated in rat dorsal root ganglia (DRG) neurons using whole cell patch

125 7, also blocked TTx-r sodium currents in rat dorsal root ganglia (DRG) neurons with comparable potenc

126 performed on isolated naive and injured rat dorsal root ganglia (DRG) neurons, and the analgesic eff

127 nd that PKDs were expressed in rat and mouse dorsal root ganglia (DRG) neurons, including nociceptive

128 Dorsal root ganglia (DRG) neurons, including the nocicep

129 lic Ca(2+) signaling is depressed in injured dorsal root ganglia (DRG) neurons, we investigated wheth

130 hyperexcitability and spontaneous firing of dorsal root ganglia (DRG) neurons, whole-cell patch clam

131 neuron populations, including cerebellar and dorsal root ganglia (DRG) neurons.

132 om developing growth cones (GCs) of isolated dorsal root ganglia (DRG) neurons.

133 tential vanilloid-1 (TRPV1)-expressing mouse dorsal root ganglia (DRG) neurons.

134 betic patients result in oxidative injury of dorsal root ganglia (DRG) neurons.

135 acutely dissociated small diameter (<27 mum) dorsal root ganglia (DRG) neurons.

136 rat SCI model depends upon hyperactivity in dorsal root ganglia (DRG) neurons.

137 rtantly, the trivalent vaccine protected the dorsal root ganglia (DRG) of 32/33 (97%) mice between da

138 isoform, Nes-S, was identified in neurons of dorsal root ganglia (DRG) of adult rats.

139 ed the expression of VEGF-A in lumbar (L)4/5 dorsal root ganglia (DRG) of control rats and VZ+434-tre

140 cells (BMDCs) that fuse with neurons in the dorsal root ganglia (DRG) of mice, leading to diabetic n

141 is known to reduce this subpopulation in the dorsal root ganglia (DRG) of neonatal rats.

142 e central nervous system, spinal nerves, and dorsal root ganglia (DRG) of rhesus macaques that were i

143 urrent study investigated MC4R expression in dorsal root ganglia (DRG) of the MC4R-GFP reporter mouse

144 (B(1)) and GABA(B(2)) mRNA, in the L4 and L5 dorsal root ganglia (DRG) of the rat in the absence of i

145 sulfasalazine targets in sciatic nerves and dorsal root ganglia (DRG) of treated animals.

146 n with rAAV8 would result in transduction of dorsal root ganglia (DRG) or trigeminal ganglia (TG), re

147 verning the maintenance and proliferation of dorsal root ganglia (DRG) progenitors are largely unknow

148 However, proliferation within the dorsal root ganglia (DRG) remains to be characterized.

149 ion of non-coding microRNAs (miRs) occurs in dorsal root ganglia (DRG) sensory neurons.

150 It is expressed by neurons in dorsal root ganglia (DRG) that may also express neuropep

151 We found that in rat dorsal root ganglia (DRG) TMEM16C was expressed mainly i

152 used the up-regulation of Ran and RanGAP1 in dorsal root ganglia (DRG) under basal conditions and dur

153 ficient method for in vivo gene silencing in dorsal root ganglia (DRG) using replication-defective he

154 L4-L5 and L6-S2 dorsal root ganglia (DRG) were collected and compared be

155 Dorsal root ganglia (DRG) were examined for histological

156 the nodose ganglia (NG) and L6/S1 and L1/L2 dorsal root ganglia (DRG) were quantified.

157 from the sciatic nerve (SN), the lumbar 4/5 dorsal root ganglia (DRG), and the trigeminal ganglia (T

158 In mice dorsal root ganglia (DRG), some neurons express calciton

159 g COX-2, EP2, EP4) in endometriosis lesions, dorsal root ganglia (DRG), spinal cord, thalamus and for

160 urons, including some sensory neurons of the dorsal root ganglia (DRG), suggesting an extranuclear ro

161 in acutely and latently infected guinea pig dorsal root ganglia (DRG), suggesting that this region i

162 spinal afferents is well known to reside in dorsal root ganglia (DRG), the morphology and location o

163 nt increased the number of T cells in lumbar dorsal root ganglia (DRG), where CD8(+) T cells were the

164 of cultured primary afferent neurons of the dorsal root ganglia (DRG).

165 ncluding the sensory neurons and glia of the dorsal root ganglia (DRG).

166 sensory neurons from rat trigeminal (TG) and dorsal root ganglia (DRG).

167 s and concomitant changes in the innervating dorsal root ganglia (DRG).

168 s, including sensory neurons and glia of the dorsal root ganglia (DRG).

169 (2+) influx in cultured primary neurons from dorsal root ganglia (DRG).

170 NaV1.8 mRNA in the injured nerve but not in dorsal root ganglia (DRG).

171 (NCPs) that give rise to neurons and glia in dorsal root ganglia (DRG).

172 apoptotic corpse removal in developing mouse dorsal root ganglia (DRG).

173 gation of sensory NCC progeny into metameric dorsal root ganglia (DRG).

174 ly, the vaccine strain was not isolated from dorsal root ganglia (DRG).

175 localization in the brain, spinal cord, and dorsal root ganglia (DRG).

176 rategy in vitro and in vivo, specifically to dorsal root ganglia (DRG).

177 eral cortex of the femur and the ipsilateral dorsal root ganglia (DRG).

178 n and inflammatory cells infiltration in the dorsal root ganglia (DRG).

179 anglia neurons--trigeminal ganglia (Vg), and dorsal root ganglia (DRG): C(2), C(5), T(5), L(5)--were

180 oth peripheral and central neuronal tissues (dorsal root ganglia [DRG], spinal cord, and brain), wher

181 d in peripheral somatosensory neurons of the dorsal root ganglia (DRGs) and have been implicated in i

182 with neuropathic and/or inflammatory pain in dorsal root ganglia (DRGs) and spinal cord both during t

183 ressed by specific subsets of neurons in the dorsal root ganglia (DRGs) and spinal cord starting shor

184 tes nerve injury and inflammatory markers in dorsal root ganglia (DRGs) and spinal cord up to 2 wk af

185 induced regulation of NECAB1/NECAB2 in mouse dorsal root ganglia (DRGs) and spinal cord.

186 ced nociception, 5,6-EET levels increased in dorsal root ganglia (DRGs) and the dorsal spinal cord, a

187 L(5) spinal cord segments, the corresponding dorsal root ganglia (DRGs) and the sciatic nerve.

188 ) is expressed by nociceptive neurons of the dorsal root ganglia (DRGs) and trigeminal ganglia, but i

189 Neurons in the mouse dorsal root ganglia (DRGs) are composed of a variety of

190 Dorsal root ganglia (DRGs) arise from trunk neural crest

191 , starting with gene expression profiling of dorsal root ganglia (DRGs) combined with multi-level bio

192 diated depletion of overall GAP-43 mRNA from dorsal root ganglia (DRGs) decreased the length of axons

193 Rapidly adapting (RA) mechanoreceptors in dorsal root ganglia (DRGs) express Ret and the co-recept

194 d range of optical stimulation parameters on dorsal root ganglia (DRGs) expressing channelrhodopsin 2

195 and sensitization responses to capsaicin in dorsal root ganglia (DRGs) following application of supe

196 lpha3 (GFRalpha3) was increased in the L2/L3 dorsal root ganglia (DRGs) following nerve injury.

197 ty on the regeneration of different types of dorsal root ganglia (DRGs) neurons after sciatic nerve i

198 -EpOME (9,10-epoxy-12Z-octadecenoic acid) in dorsal root ganglia (DRGs) of paclitaxel-treated mice as

199 pes, and its gene expression is increased in dorsal root ganglia (DRGs) of paclitaxel-treated rats.

200 here was a 33% neuronal loss in the lumbar 5 dorsal root ganglia (DRGs) of the db(-)/db(-) mouse vers

201 ansfer to the primary sensory neurons of the dorsal root ganglia (DRGs) with self-complementary recom

202 types of nociceptors have been described in dorsal root ganglia (DRGs).

203 stimulated axonal growth from chicken or rat dorsal root ganglia (DRGs).

204 l afferent neurons, whose cell bodies lie in dorsal root ganglia (DRGs).

205 Dorsal root ganglia Epac1 mRNA levels increase during ne

206 The dorsal root ganglia from the TrkAP782S knock-in mice dis

207 e surgical procedure for extraction of human dorsal root ganglia (hDRG) and the necessary modificatio

208 edding of HSV-2 DNA, and latent infection of dorsal root ganglia in guinea pigs.

209 ormed gD-2 alone, particularly in protecting dorsal root ganglia in mice and reducing recurrent vagin

210 ells, in vitro and is impaired for infecting dorsal root ganglia in mice.

211 by proprioceptive sensory neurons (pSNs) in dorsal root ganglia in mice.

212 Dorsal root ganglia in Zfp36l2 knock-out mice, or wild-t

213 Using embryonic sensory neurons (rat dorsal root ganglia) in a growth cone turning assay, we

214 not substance P-positive cells in the lumbar dorsal root ganglia; increased labeling was not affected

215 er proportion of nociceptors compared to the dorsal root ganglia innervating the rest of the body.

216 een functional subtypes of sensory neuron in dorsal root ganglia is distorted by Gars mutations, lead

217 inant highly Ca(2+)-sensitive PLC isoform in dorsal root ganglia is PLCdelta4.

218 miR-I is also expressed in human sacral dorsal root ganglia latently infected with HSV-2.

219 virus-mediated transfer of erythropoietin to dorsal root ganglia may prove useful in treatment of dia

220 tivation, and ramp currents that can produce dorsal root ganglia neuron hyperexcitability that underl

221 se neurons represent a small fraction of the dorsal-root ganglia neuronal population, we were able to

222 ditioning lesion to the peripheral branch of dorsal root ganglia neurons (DRGs).

223 ation of AC from an AKAP150-TRPV1 complex in dorsal root ganglia neurons abolishes sensitization of T

224 NP)]Ts1, we were able to optically stimulate dorsal root ganglia neurons and generate action potentia

225 model as well as in vitro effects of HOCl on dorsal root ganglia neurons and mouse bone marrow-derive

226 nd -3 are the major ASIC subunits in cardiac dorsal root ganglia neurons and provide potential molecu

227 lective agonist induced hyperexcitability of dorsal root ganglia neurons and stimulated the release o

228 al manifestations ensue from primary loss of dorsal root ganglia neurons and their associated axons a

229 LIF also induced neuronal plasticity in dorsal root ganglia neurons by increasing the number of

230 Analysis of co-cultures with rat dorsal root ganglia neurons confirmed that OPCs were mor

231 that in small-diameter, capsaicin-sensitive dorsal root ganglia neurons corresponding to nociceptors

232 impaired response to several pruritogens in dorsal root ganglia neurons excised from NC/Nga mice aft

233 Both human and mouse dorsal root ganglia neurons express IL-31RA, largely in

234 ASIC-like current properties of the cardiac dorsal root ganglia neurons from wild-type mice most clo

235 Consistently, GINIP-deficient dorsal root ganglia neurons had lower baclofen-evoked in

236 neurons maintains the position of later born dorsal root ganglia neurons in an activity-dependent man

237 t of the trigeminal (V), superior (IX/X), or dorsal root ganglia neurons in which it is expressed, bu

238 Olfm1 inhibited the growth cone collapse of dorsal root ganglia neurons induced by myelin-associated

239 The number of dorsal root ganglia neurons is not affected by the mutat

240 uces or prevents, respectively, migration of dorsal root ganglia neurons out of the ganglion to locat

241 Transcriptional profiling of IL-31-activated dorsal root ganglia neurons revealed enrichment for gene

242 , the growth cones of primary small-diameter dorsal root ganglia neurons showed abundant IL-31 recept

243 several tissues, and knockdown of Piezo2 in dorsal root ganglia neurons specifically reduced rapidly

244 detected both KCNQ2 and KCNQ3 in a subset of dorsal root ganglia neurons that correspond to D-hair Ad

245 s; (iii) suppresses proinflammatory state of dorsal root ganglia neurons to decrease pelvic pain; (iv

246 , AP-7 olfactory neuronal cells, and primary dorsal root ganglia neurons triggered prolonged Stat-1 T

247 els, we determined that the treatment of rat dorsal root ganglia neurons with E2 increased mRNA conce

248 d sensory neurons, which account for >40% of dorsal root ganglia neurons, display resistance to rabie

249 previous reports of ASIC3 mRNA expression in dorsal root ganglia neurons, we found that the ASIC3 ant

250 and membrane expression of TRPV1 protein in dorsal root ganglia neurons.

251 ore activated, form of cJun, a JNK target in dorsal root ganglia neurons.

252 of nonpeptidergic nociceptors and myelinated dorsal root ganglia neurons.

253 for temperature and pressure sensitivity in dorsal root ganglia neurons.

254 a(2+) currents in rat T-rich nociceptor-like dorsal root ganglia neurons.

255 skin increases TRPV1 and TRPA1 expression in dorsal root ganglia neurons.

256 nhibits ASIC-like currents in naked mole-rat dorsal root ganglia neurons.

257 inhibited the activity of TRPV1 channels in dorsal root ganglia neurons.

258 of the electrophysiology dynamics in single dorsal root ganglia neurons.

259 myelinating cocultures established from the dorsal root ganglia of embryonic rats.

260 ency of wild-type HSV-1 viral DNA present in dorsal root ganglia of immunized animals was significant

261 10 transduces neurons in the spinal cord and dorsal root ganglia of immunodeficient mice with higher

262 we isolated neurons with attached SGCs from dorsal root ganglia of mice.

263 ased concentrations in the sciatic nerve and dorsal root ganglia of oxaliplatin treated mice.

264 ncreased activity of the Epac target Rap1 in dorsal root ganglia of WT, but not of Epac1(-/-), mice.

265 gated Na(+), K(+) and Ca(2+) channels in rat dorsal root ganglia or VGSC forms individually expressed

266 ed the EP4 receptor protein in the L4 and L5 dorsal root ganglia over their freely perfused counterpa

267 Different types of sensory neurons in the dorsal root ganglia project axons to the spinal cord to

268 After 12 weeks, axons from C6-C7 dorsal root ganglia regenerated through the tenascin-C-r

269 rcentage of cardiac sensory neurons from the dorsal root ganglia respond to acidic pH and generated l

270 ochemistry was used to detect TRPV4 in human dorsal root ganglia samples (from the National Disease R

271 brafish larvae that first differentiate into dorsal root ganglia sensory neurons but later acquire a

272 Survival and maturation of dorsal root ganglia sensory neurons during development d

273 NCSC-derived human Schwann cells with rodent dorsal root ganglia showed interaction of the Schwann ce

274 ession G-protein-coupled receptors in murine dorsal root ganglia showed that both receptors were amon

275 A, protein, and histological analysis of the dorsal root ganglia, spinal cord, and cerebellum.

276 neuronal and non-neuronal tissues, including dorsal root ganglia, spinal cord, and keratinocytes.

277 Despite on-target activity in small-diameter dorsal root ganglia, spinal slices, and in a mouse model

278 97%) animals had no evidence of infection of dorsal root ganglia, suggesting that the vaccine may pre

279 f thrombospondin-4 (TSP4) in spinal cord and dorsal root ganglia that contributes to neuropathic pain

280 Here, we show in neurons of murine dorsal root ganglia that pro-nociceptive TRPM3 channels,

281 xclusively in trigeminal ganglia, and not in dorsal root ganglia, thereby maintaining a role for TRPV

282 nnel isoforms were natively expressed in rat dorsal root ganglia tissue.

283 Instead, HSV-1 spread via the dorsal root ganglia to the autonomic ganglia of the ente

284 ores, and a reduction in latent HSV-2 DNA in dorsal root ganglia to undetectable levels.

285 show here that P2X7Rs in satellite cells of dorsal root ganglia tonically inhibit the expression of

286 In human dorsal root ganglia, TPV4 was expressed by 35% of neuron

287 anscription factor to sensory neurons of the dorsal root ganglia using a gene therapy approach and fo

288 Early development of the trigeminal and dorsal root ganglia was grossly normal in the absence of

289 from SNE-injured and contralateral L4 and L5 dorsal root ganglia were cultured in a compartmentalized

290 the TRPV1 expressing neurons in the rostral dorsal root ganglia were more similar to jugular than no

291 on of injury-related genes due to SNI in the dorsal root ganglia were not affected by SDLs.

292 Lumbosacral dorsal root ganglia were positive for HSV-2 DNA and late

293 r treatment alone when dissociated embryonic dorsal root ganglia were seeded onto inhibitory substrat

294 Neuronal kainate receptors in dorsal root ganglia were sensitive to galectin modulatio

295 Induction of inflammation within dorsal root ganglia, when combined with other treatments

296 as development progresses in sympathetic and dorsal root ganglia, whereas levels in ciliary ganglia b

297 subset of sensory neurons in the cranial and dorsal root ganglia which does not correspond to any spe

298 mns and striking neuronal cell loss from the dorsal root ganglia, which was accompanied by severe mit

299 ed within muscle spindle afferent neurons in dorsal root ganglia with a higher proportion at cervical

300 -regulated K(v)1.1 messenger RNA in thoracic dorsal root ganglia without affecting the expression of