ライフサイエンスコーパス: electroretinography

1 tive immunohistochemistry) and functionally (electroretinography).

2 provement of retinal function (assessed with electroretinography).

3 n of Akita and control mice was evaluated by electroretinography.

4 kinetic and static perimetry, and full-field electroretinography.

5 lted in functional deficits as determined by electroretinography.

6 fundus ophthalmoscopy, dark adaptometry, and electroretinography.

7 r degeneration was measured by histology and electroretinography.

8 static contrast sensitivity test; and global electroretinography.

9 cone survival was assessed by histology and electroretinography.

10 Retinal function was assayed by electroretinography.

11 escued photoreceptor function as measured by electroretinography.

12 inal histology up to 24 months of age and by electroretinography.

13 ull-field electroretinography and multifocal electroretinography.

14 Rescue of visual function was confirmed by electroretinography.

15 T) or DKO mice, as assessed by histology and electroretinography.

16 ctrometry, and degeneration by histology and electroretinography.

17 ncluding ophthalmoscopy and full-field flash electroretinography.

18 nohistochemistry, Western blot analysis, and electroretinography.

19 l function was characterized with the use of electroretinography.

20 t hypomorphic and Ret conditional mice using electroretinography.

21 ) measures of central retinal thickness, and electroretinography.

22 isual function in these mice was analyzed by electroretinography.

23 reserves RGC activity as measured by pattern electroretinography.

24 nction of USH2A patients was quantified with electroretinography.

25 Neural function was assessed with electroretinography.

26 t time of the N95 wave on results of pattern electroretinography.

27 es of mRDH11 disruption were investigated by electroretinography.

28 munohistochemistry, electron microscopy, and electroretinography.

29 us photography, fluorescein angiography, and electroretinography.

30 ant visual loss in treated abcr(-/-) mice by electroretinography.

31 promoted functional recovery as assessed by electroretinography.

32 the physiology of the retina was analyzed by electroretinography.

33 Dark adaptation was monitored by electroretinography.

34 Retinal function was evaluated by electroretinography.

35 tion of outer nuclear layer thickness and by electroretinography.

36 Dawley rats, and recovery was measured using electroretinography.

37 optical coherence tomography angiography and electroretinography.

38 glaucoma as well as cone-rod dysfunction on electroretinography.

39 imaging, and visual function was assessed by electroretinography.

40 omography, kinetic visual field testing, and electroretinography.

41 erence tomography, and multifocal or pattern electroretinography.

42 ed peripheral retinal function by multifocal electroretinography.

43 icity was evaluated by clinical findings and electroretinography: 30-Hz flicker responses were compar

44 thanol, and retinal function was assessed by electroretinography 72 hours after the initial dose of m

45 ticularly in areas with decreased multifocal electroretinography amplitude.

46 BEST1 mutations, imaging findings, electroretinography amplitudes, and implicit times.

47 ptor outer nuclear layer (ONL) thickness and electroretinography amplitudes.

48 ty, biomicroscopy, color fundus photography, electroretinography analysis, and visual-evoked potentia

49 tion and visual sensitivity were examined by electroretinography and an active avoidance behavioral t

50 Multifocal electroretinography and chromatic/achromatic contrast se

51 on retinal function and neurodegeneration by electroretinography and detailed morphology.

52 static perimetry, full-field and multifocal electroretinography and electro-oculography.

53 3 expression, mice were analyzed by scotopic electroretinography and fluorescein angiography.

54 Electroretinography and gene expression analyses suggest

55 Full-field electroretinography and histologic examination were used

56 Photoreceptor survival was assessed by electroretinography and histology.

57 rvival time, visual acuity decline rate, and electroretinography and imaging findings.

58 er retinal function as measured, in vivo, by electroretinography and manganese-enhanced MRI.

59 mprovement observed in this case, multifocal electroretinography and microperimetry indicate that sub

60 and cone function was analyzed by full-field electroretinography and multifocal electroretinography.

61 Further investigations included electroretinography and neuroradiologic imaging.

62 Multifocal electroretinography and ocular coherence tomography are

63 We showed using electroretinography and single cell recordings that the

64 tween photoreceptors and ON-BC neurons using electroretinography and single cell recordings.

65 mice were examined by scotopic and photopic electroretinography and then killed for biochemical and

66 phototransduction components was assessed by electroretinography and to couple to vertebrate transduc

67 In vivo electroretinography and transretinal recordings revealed

68 To delineate mechanisms, we used electroretinography and visual evoked potential recordin

69 times after infection, eyes were analyzed by electroretinography and were harvested for quantitation

70 used electrophysiological techniques (i.e., electroretinography) and molecular analyses, this work s

71 se of infection by biomicroscopy, histology, electroretinography, and bacterial and inflammatory cell

72 enetrating electrode arrays, multi-electrode electroretinography, and electromyography, are also viab

73 9 months of age using immunohistochemistry, electroretinography, and fluorescein angiography.

74 A), kinetic and static perimetry, full-field electroretinography, and fundus autofluorescence (FAF).

75 VF), dark-adapted absolute thresholds (DAT), electroretinography, and fundus photography.

76 t lamp examination, indirect ophthalmoscopy, electroretinography, and histologic examination.

77 ded toxicology (clinical examination, serial electroretinography, and histopathology) in normal rabbi

78 al retinal examination, noninvasive imaging, electroretinography, and histopathology/immunohistochemi

79 At P30, retinal function was measured with electroretinography, and morphologic preservation of the

80 Slit lamp examination (SLE), electroretinography, and myeloperoxidase activity were p

81 ields, optical coherence tomography, pattern electroretinography, and neuro-ophthalmic examinations.

82 notype was characterized with psychophysics, electroretinography, and optical coherence tomography.

83 d safety were evaluated with ophthalmoscopy, electroretinography, and pathology.

84 s were analyzed by biomicroscopy, histology, electroretinography, and quantitation of bacteria and in

85 l recovery after ischemia was measured using electroretinography, and retinal histology was examined

86 Retinal function was assessed by electroretinography, and retinal morphological features

87 Retinal function was assessed by electroretinography, and retinal structure by light micr

88 photoreceptor function was assessed through electroretinography, and survival was documented in morp

89 e scanning laser ophthalmoscopy (AF-SLO) and electroretinography, and the extent of laser-induced CNV

90 nical ophthalmic examinations, neuroimaging, electroretinography, and the results of MMACHC mutation

91 -fluorescence, optical coherence tomography, electroretinography, and ultrasound.

92 fundus photography, fluorescein angiography, electroretinography, and visual evoked potentials were o

93 nfocal and electron microscopy, single-flash electroretinography, and whole-cell patch-clamp recordin

94 Visual acuity, retinal features, electroretinography, and whole-exome sequencing.

95 with fundus autofluorescence and multifocal electroretinography as indicated), will greatly minimize

96 Electroretinography assessed functional recovery after i

97 y histopathology, morphometric analysis, and electroretinography at P60.

98 erical equivalent refraction, visual fields, electroretinography B-wave amplitudes, and qualitative i

99 By electroretinography, b-wave amplitude was reduced by 75%

100 Eyes were analyzed by electroretinography, bacterial quantitation, and antibio

101 Patients were also assessed by electroretinography, brain MRI and magnetic resonance sp

102 Electroretinography can be usually be obtained in infant

103 mination, visual acuity (VA), visual fields, electroretinography, color vision testing, and retinal i

104 r initial rate of loss of visual function by electroretinography, compared with eyes without these st

105 patients (aged 10-62 years) with full-field electroretinography-confirmed achromatopsia.

106 , and reduced scotopic responses observed on electroretinography consistent with the CRD phenotype of

107 Full-field electroretinography demonstrated a reduced rod response

108 Multifocal electroretinography demonstrated normal amplitude and im

109 Electroretinography demonstrated that cone responses wer

110 also remain functional, albeit with reduced electroretinography (ERG) amplitudes typical of RetGC1-/

111 To determine toxicity, electroretinography (ERG) amplitudes were measured in re

112 tinal tissue as indicated by TUNEL assay and electroretinography (ERG) analysis.

113 Electroretinography (ERG) and both chemical and histolog

114 Functional retinal changes were evaluated by electroretinography (ERG) and by morphologic and ultrast

115 me course of retinal dysfunction by scotopic electroretinography (ERG) and by quantitative morphology

116 rimetry, fluorescein angiography, full-field electroretinography (ERG) and electro-oculography (EOG),

117 Electroretinography (ERG) and intraocular pressure (IOP)

118 Electroretinography (ERG) and light microscopy were used

119 ete ophthalmic examination, full-field flash electroretinography (ERG) and multifocal ERG, light-adap

120 gating their visual capabilities by means of electroretinography (ERG) and patterned visual evoked po

121 Baseline electroretinography (ERG) and preretinal oxygen (Po(2))

122 Function was assessed with perimetry and electroretinography (ERG) and retinal structure with opt

123 Finally, anesthetized mice were studied by electroretinography (ERG) at different times after expos

124 Spectral electroretinography (ERG) demonstrated significant impro

125 ship among vector dose, visual function, and electroretinography (ERG) findings.

126 The mice were analyzed by full-field electroretinography (ERG) for light response.

127 microvilli area and an abnormal response on electroretinography (ERG) in UBE3D(+/-) heterozygous mic

128 chanism of confocal IOS, comparative IOS and electroretinography (ERG) measurements were made using n

129 icity was evaluated by clinical findings and electroretinography (ERG) on 244 evaluable injections in

130 munoblots, and cone function was analyzed by electroretinography (ERG) recordings.

131 Scotopic electroretinography (ERG) showed a diminished c-wave amp

132 At 7 days after exposure to light electroretinography (ERG) showed that minocycline signif

133 We used electroretinography (ERG) to evaluate retinal function a

134 cy of optical coherence tomography (OCT) and electroretinography (ERG) to monitor pathological and fu

135 Electroretinography (ERG) was performed before surgery a

136 Among mice followed for 8 months, electroretinography (ERG) was performed on both eyes bef

137 Electroretinography (ERG) was performed to evaluate reti

138 Electroretinography (ERG) was used to assess rod and con

139 Electroretinography (ERG) was used to evaluate the recov

140 Strobe flash electroretinography (ERG) was used to examine outer reti

141 Computerized pupillometry and electroretinography (ERG) were performed to assess optic

142 tions, blood screenings, vision testing, and electroretinography (ERG) were performed.

143 transmission electron microscopy (TEM), and electroretinography (ERG) were used to analyze 6 genotyp

144 testing, dark adaptation testing, full-field electroretinography (ERG), and electro-oculography (EOG)

145 handheld tonometer, indirect ophthalmoscopy, electroretinography (ERG), and histology.

146 of bacteria, organ function as determined by electroretinography (ERG), and histopathologic changes w

147 on were documented using fundus photography, electroretinography (ERG), and histopathology.

148 ry, fundus-guided microperimetry, full-field electroretinography (ERG), and multifocal ERG.

149 ry, fundus-guided microperimetry, full-field electroretinography (ERG), and multifocal ERG.

150 Functional abnormalities were assessed by electroretinography (ERG), and neurodegeneration was ass

151 netic perimetry, chromatic static perimetry, electroretinography (ERG), and optical coherence tomogra

152 Retinal function was measured with electroretinography (ERG), and relative content of selec

153 sponses to light were measured by full-field electroretinography (ERG), and retinal tissues were exam

154 Visual acuity (VA), electroretinography (ERG), and spectral-domain optical c

155 nd electron microscopy, immunocytochemistry, electroretinography (ERG), and spectrophotometry.

156 lowing injection by slit lamp biomicroscopy, electroretinography (ERG), bacterial and inflammatory ce

157 The outcome after ischemia was examined with electroretinography (ERG), by measuring retinal cell lay

158 tical coherence tomography (OCT), full-field electroretinography (ERG), electro-oculography (EOG), an

159 for 8 wk and evaluated by AGE fluorescence, electroretinography (ERG), electron microscopy, and micr

160 -LCA (n = 30; ages, 4-55) were studied using electroretinography (ERG), full-field stimulus testing (

161 d reduced retinal thickness were found using electroretinography (ERG), fundus photography (FP), fund

162 Clinical examinations, electroretinography (ERG), histology, and bacterial quan

163 Electroretinography (ERG), histology, light microscopy,

164 ogy, transmission electron microscopy (TEM), electroretinography (ERG), immunohistochemistry, Western

165 rn electroretinography (PERG) and full-field electroretinography (ERG), incorporating international s

166 -) and wild-type (WT) mice were evaluated by electroretinography (ERG), lectin cytochemistry, and cor

167 ctural, and biochemical assessments included electroretinography (ERG), light and electron microscopi

168 age-matched control puppies were studied by electroretinography (ERG), light and electron microscopy

169 of the gene targeted mice were evaluated by electroretinography (ERG), light, and electron microscop

170 almic examinations were performed, including electroretinography (ERG), multifocal ERG (mfERG), perim

171 s autofluorescence (FAF) imaging, full-field electroretinography (ERG), multifocal ERG, and central v

172 -induced retinal degeneration using scotopic electroretinography (ERG), optical coherence tomography

173 n were determined at multiple time points by electroretinography (ERG), optical coherence tomography

174 ional examinations were performed, including electroretinography (ERG), optical coherence tomography

175 Phenotype was assessed with electroretinography (ERG), optical coherence tomography,

176 almic examinations were performed, including electroretinography (ERG), perimetry, optical coherence

177 bset of the detected RP1 heterozygotes using electroretinography (ERG), psychophysics, and optical co

178 resonance imaging, full-field and multifocal electroretinography (ERG), visual evoked potentials (VEP

179 s measured by optokinetic tracking (OKT) and electroretinography (ERG).

180 ically, and retinal function was analysed by electroretinography (ERG).

181 of CSNB2 and RP were confirmed by full-field electroretinography (ERG).

182 tudied with optical coherence tomography and electroretinography (ERG).

183 Cone function was determined by electroretinography (ERG).

184 A), visual field sensitivity, and full-field electroretinography (ERG).

185 utant Ush1c, were analyzed by microscopy and electroretinography (ERG).

186 ve impact on visual function, as assessed by electroretinography (ERG).

187 er retinal layers as well as functionally by electroretinography (ERG).

188 e analyzed by optokinetic response (OKR) and electroretinography (ERG).

189 electron microscopy, immunofluorescence, and electroretinography (ERG).

190 ron microscopy, and single-flash and flicker electroretinography (ERG).

191 c perimetry, static chromatic perimetry, and electroretinography (ERG).

192 he function of the retina was evaluated with electroretinography (ERG).

193 to examine the function of this rhodopsin by electroretinography (ERG).

194 e rate of dark adaptation was analyzed using electroretinography (ERG).

195 etinal function was assessed by dark-adapted electroretinography (ERG).

196 AF), optical coherence tomography (OCT), and electroretinography (ERG).

197 oldmann visual fields (GVFs), and full-field electroretinography (ERG).

198 inal function was assessed by three forms of electroretinography (ERG): slow-sequence multifocal (mf)

199 ed microperimetry, full-field and multifocal electroretinography (ffERG and mfERG), spectral-domain o

200 retinal function was studied with full-field electroretinography (ffERG) from 3 months through 2 year

201 hthalmoscopy, fundus photography, full-field electroretinography (ffERG), Goldmann visual fields (VFs

202 almoscopy, Goldmann visual field, full-field electroretinography (ffERG), OCT, and FAF photography.

203 oherence tomography (SD-OCT), and full-field electroretinography (ffERG).

204 Electroretinography findings support the more severe cli

205 Global electroretinography findings were normal.

206 ed on visual acuity, fundus photography, and electroretinography findings.

207 in the retina and choroid were evaluated by electroretinography, fluorescein angiography, light micr

208 Electroretinography from an additional group of normal m

209 t underwent a complete clinical examination, electroretinography (full field and pattern), visual evo

210 dus appearance, visual field, and full-field electroretinography, fundus autofluorescence, and optica

211 phthalmic examinations, including full-field electroretinography, fundus photography, fundus autofluo

212 Multifocal electroretinography had the greatest proportion of posit

213 went complete ocular examination, full-field electroretinography, handheld spectral-domain optical co

214 of endophthalmitis was graded by slit lamp, electroretinography, histological examinations, and dete

215 riologically and by slit lamp biomicroscopy, electroretinography, histology, and inflammatory cell en

216 infection, the EBE incidence was assessed by electroretinography, histology, bacterial counts, and my

217 d a phototransduction deficit as assessed by electroretinography; however, their photoreceptor struct

218 Histology, electroretinography, immunohistochemistry, Western blot

219 dysfunction was provided through multi-focal electroretinography in a subset of such patients.

220 nd maintained visual function as assessed by electroretinography in C57BL/6 mice.

221 unostaining, and measured light responses by electroretinography in mice with targeted disruptions of

222 The role of electroretinography in pediatric practice, and to offer

223 d retinal ganglion cell function by means of electroretinography in three patients with cerebral hemi

224 nction because DMOG normalizes the b-wave on electroretinography in wild-type mice.

225 l loss occurred in adult Mfsd2a KO mice, but electroretinography indicated visual function was normal

226 RKL mutations were studied clinically and by electroretinography, kinetic, and chromatic static perim

227 nifest retinal degeneration, as evidenced by electroretinography, light microscopy and pupillometry r

228 trauma and assessed by clinical examination, electroretinography, light microscopy, electron microsco

229 ed using ophthalmoscopy, fundus photography, electroretinography, light microscopy, immunocytochemist

230 Multifocal electroretinography may be effective in detecting functi

231 the N95 implicit time on results of pattern electroretinography (median, 98.6 milliseconds [95% CI,

232 With appropriate technique, electroretinography methods could be made more widely av

233 coherence tomography (SD-OCT) and multifocal electroretinography (mfERG) along with visual fields.

234 Functional studies by multifocal electroretinography (mfERG) evaluated neurodysfunction,

235 l coherence tomography (OCT), and multifocal electroretinography (mfERG) were performed at baseline a

236 oherence tomography (SD-OCT), and multifocal electroretinography (mfERG) were performed at various in

237 ests consisting of visual fields, multifocal electroretinography (mfERG), and contrast sensitivity we

238 gnosis were followed up including multifocal electroretinography (mfERG), spectral-domain optical coh

239 llary RNFLT, RNFL retardance, and multifocal electroretinography (mfERG).

240 fundus autofluorescence (FAF), or multifocal electroretinography (mfERG).

241 y visual field (HVF) testing, and multifocal electroretinography (mfERG).

242 nt routine examination, including full-field electroretinography, microperimetry, and optical coheren

243 ons included electro-oculography, full-field electroretinography, multifocal electroretinography, spe

244 vision (ie, dark adaptometry, pupillometry, electroretinography, nystagmus, and ambulatory behaviour

245 In vivo electroretinography of Panx1 knockout mice indicated an

246 P measurements were collected daily, whereas electroretinography, optical coherence tomography, and w

247 hy, fundus autofluorescence imaging, sedated electroretinography, optical coherence tomography, genet

248 ce of drug toxicity by clinical examination, electroretinography, or histologic examination.

249 angiography evidence of active choroiditis, electroretinography parameters indicative of stable or w

250 Finally, electroretinography performed on mice deficient for one

251 Pattern electroretinography (PERG) and full-field electroretinog

252 analyses included transient pattern-reversal electroretinography (PERG) and full-field flash ERG, wit

253 Macular function was assessed with pattern electroretinography (PERG) to checkerboard stimuli of di

254 Patients underwent pattern electroretinography (PERG), optical coherence tomography

255 One month later, pattern electroretinography (PERG), rate of ATP synthesis, gene

256 Electroretinography phenotype (cone-rod vs rod-cone dysf

257 Objective functional tests, such as electroretinography, provide an alternative to subjectiv

258 ice, and to offer suggestions for successful electroretinography recordings in children.

259 ischemia by histologic analyses and scotopic electroretinography, respectively.

260 d b-wave amplitudes of scotopic and photopic electroretinography responses 4 months after diabetes in

261 The mean change in electroretinography responses was not significantly diff

262 mice, their scotopic, maximal, and photopic electroretinography responses were comparable to those o

263 Full-field electroretinography responses were extinguished in 50% o

264 l coherence tomography, and severely reduced electroretinography responses.

265 clinical symptom of night blindness and the electroretinography results suggest a primary rod dysfun

266 nction as assessed by VA, visual fields, and electroretinography results; and retinal structural chan

267 omain optical coherence tomography (SD-OCT), electroretinography, retinal morphology, and visual reti

268 -/-) and Oa1(-/-) mice had normal results on electroretinography, retrograde labeling showed a signif

269 Electroretinography revealed a cone-rod pattern of dysfu

270 Electroretinography revealed a rod-cone pattern of dysfu

271 However, twin flash electroretinography revealed a slight delay in recovery

272 xons project to the superior colliculus, and electroretinography revealed no defect of adult visual f

273 Retinal function was evaluated using electroretinography (scotopic, photopic, and pattern).

274 Ganzfeld electroretinography showed faster recovery of retinal fu

275 ailed neuro-ophthalmic examination including electroretinography showed him to have a typical retinal

276 Electroretinography showed mutant mouse eyes had a selec

277 Histology and electroretinography showed no cre-mediated RPE toxicity.

278 Electroretinography showed that scotopic and photopic re

279 Rod function (measured by electroretinography) showed modest but statistically sig

280 , full-field electroretinography, multifocal electroretinography, spectral-domain optical coherence t

281 Electroretinography studies demonstrated a strong correl

282 proportion to the clinical exams, prompting electroretinography testing that revealed an electronega

283 arent to clinical examination and full-field electroretinography testing.

284 At P28, animals were evaluated by electroretinography; tissues were then harvested for bio

285 Here, we used electroretinography to examine the functional role of BK

286 y screens (such as histological analysis and electroretinography) to identify the subset of fish with

287 ography, fundus autofluorescence, multifocal electroretinography, visual fields) and classification o

288 The high temporal frequency electroretinography was determined mainly by the luminan

289 Simultaneous bilateral dark-adapted electroretinography was performed 2 weeks and 12 weeks a

290 Full-field electroretinography was performed binocularly, using DTL

291 dioactive microsphere blood flow method, and electroretinography was performed during the first 120 m

292 After exposure, electroretinography was performed on mice dark adapted f

293 was performed to assess for cell death, and electroretinography was performed to assess function.

294 Multifocal electroretinography was shown to have a high sensitivity

295 n levels of several phototransduction genes; electroretinography was used to assess quantitatively th

296 -retinylidene-N-retinylethanolamine content; electroretinography was used to measure phototransductio

297 Fundus photography, and electroretinography were performed in 12 patients, and o

298 ure (IOP) tonometry, fundus photography, and electroretinography were performed over multiple time po

299 Microscopy and electroretinography were used to characterize transgenic

300 us photography, fluorescein angiography, and electroretinography were used to evaluate retinal anatom