ライフサイエンスコーパス: endonuclease G

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1 otic factors, apoptosis inducing factor, and endonuclease G.

2 and apoptosis-inducing factor (AIF), but not endonuclease G.

3 e c, Smac/DIABLO, Omi/HtrA2, and AIF but not endonuclease G.

4 lacking the mitochondrial endonuclease CPS-6/endonuclease G.

5 rocess requiring the mitochondrial nuclease, Endonuclease G.

6 Endonuclease G, a protein historically thought to be inv

7 Data is presented localizing rat liver endonuclease G activity exclusively to the mitochondrial

8 Endonuclease G activity, measured by a plasmid relaxatio

9 Endonuclease G also showed the predicted recombination p

10 Furthermore, we show that endonuclease G, an apoptotic nuclease downstream of Casp

11 Microsequencing showed it to be human endonuclease G, an enzyme with a strong preference for G

12 related factors (apoptosis-inducing factor, endonuclease G, and HtrA2/Omi).

13 6 encodes a homologue of human mitochondrial endonuclease G, and its protein product similarly locali

14 Endonuclease G appears to be the only cellular enzyme th

15 and distinct in its properties from that of endonuclease G can be detected.

16 Additionally, it is shown that endonuclease G can be selectively released from the mito

17 enerated by cleavage of viral a sequences by endonuclease G during genome isomerization.

18 Endonuclease G (endo G) is one of the most abundant nucl

19 and both apoptosis-inducing factor (AIF) and endonuclease G (Endo G) were released from the mitochond

20 ochrome c, Smac/Diablo and HtrA2/Omi but not endonuclease G (EndoG) and apoptosis-inducing factor (AI

21 Using endonuclease G (endoG) as a model IMS protein, we found

22 f cisplatin and found that the expression of endonuclease G (EndoG) increased whereas the expression

23 Endonuclease G (EndoG) is a mitochondrial protein that t

24 Endonuclease G (EndoG) is a nuclear-encoded mitochondria

25 Endonuclease G (endoG) is released from mitochondria dur

26 rome c, apoptosis-inducing factor (AIF), and endonuclease G (EndoG) release.

27 ed with the mitochondrial endonuclease CPS-6/endonuclease G (EndoG) to promote DNA degradation and ap

28 Our earlier studies had suggested that endonuclease G (EndoG), a member of the evolutionarily c

29 This nuclease is endonuclease G (endoG), a mitochondrion-specific nucleas

30 factors, apoptosis-inducing factor (AIF) and endonuclease G (EndoG), through p53-dependent upregulati

31 Here we identified endonuclease G (Endog), which previously was implicated

32 oles for the conserved mitochondrial protein endonuclease G in budding yeast apoptosis and proliferat

33 ation of apoptosis-inducing factor (AIF) and endonuclease G in CNGA3(-/-)/Nrl(-/-) and CNGB3(-/-)/Nrl

34 Based on these findings, we propose that endonuclease G initiates the a sequence-mediated inversi

35 drion, such as apoptosis-inducing factor and endonuclease G, may induce caspase-9-independent apoptos

36 In Endonuclease G mutants, persisting nucleoids are swept o

37 CPS-6, a mitochondrial endonuclease G, serves as a paternal mitochondrial facto

38 but release of apoptosis-inducing factor and endonuclease G was detected only in TK6 cells.

39 lso depends on apoptosis-inducing factor and endonuclease G, which are effectors of caspase-independe

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