ライフサイエンスコーパス: genome-wide association study

1 i for fiber-quality-related traits through a genome-wide association study.

2 -nucleotide polymorphisms were identified by genome-wide association study.

3 oaches that can be used to test for GxE in a genome-wide association study.

4 ffect estimates (odds ratios) extracted from genome-wide association studies.

5 n content (PC) using single- and multi-locus genome-wide association studies.

6 netic variants is a promising alternative to genome-wide association studies.

7 el locus missed by conventional single-trait genome-wide association studies.

8 on in complex human diseases, especially via genome-wide association studies.

9 P (rs964184) for triglycerides identified by genome-wide association studies.

10 were associated with 100 genetic loci using genome-wide association studies.

11 AAA risk loci using data from all available genome-wide association studies.

12 case-control status have been replicated by genome-wide association studies.

13 tives to the standard univariate analysis in genome-wide association studies.

14 ntial heritability not explained by previous genome-wide association studies.

15 Among the participants in the genome-wide association studies, 21 loci jointly influen

16 Omics data included those from historical genome-wide association studies (23,000 children) and DN

17 nducting a meta-analysis of worldwide asthma genome-wide association studies (23,948 asthma cases, 11

18 A genome-wide association study across all VNB isolates re

19 Genome-wide association studies allow us to understand t

20 genetics to CKD progression, we performed a genome-wide association study among participants in the

21 Genome-wide association study analyses have found loci a

22 te at Bonferroni significance in an external genome-wide association study analysis of 37,930 individ

23 This genome-wide association study analyzed criterion counts

24 Genome wide association studies and candidate gene scree

25 etic predictors of inflammatory markers from genome-wide association studies and applied them to a la

26 Here we hypothesized that performing genome-wide association studies and exome array analyses

27 proved mmLD method will be useful for future genome-wide association studies and genetic association

28 ivariate methods has often been neglected in genome-wide association studies and if used, replication

29 ted in autoimmune diseases in humans through genome-wide association studies and in mice using cell t

30 th the continued emergence of risk loci from Genome-Wide Association studies and variants of uncertai

31 with genes near loci identified by psoriasis genome-wide association studies and were enriched for ge

32 Here, the authors perform a genome-wide association study and identify an associatio

33 ETATION: Despite the small sample size for a genome-wide association study, and acknowledging the pot

34 proximately 4200 common variants reported in genome-wide association studies, approximately 1000 de n

35 Genome-wide association studies are identifying multiple

36 s (SNPs), the most common genetic markers in genome-wide association studies, are usually in linkage

37 polymorphisms previously identified through genome-wide association studies as robustly associated w

38 gene implicated in human type 2 diabetes by genome-wide association studies but without a clear conn

39 mputation can enhance the discovery power of genome-wide association studies by assessing previously

40 NPs) on the panel from previous studies, and genome-wide association studies by using FarmCPU R packa

41 QT interval at baseline, identified through genome-wide association studies, can predict individual

42 d from these sequences into new and existing genome-wide association study cohorts and tested for ass

43 erived from summary statistics of 2 separate genome-wide association studies conducted from 2007 to 2

44 ata set consisting of summary results from 4 genome-wide association studies conducted from 2007 to 2

45 ythematosus (SLE) susceptibility by a recent genome-wide association study conducted in Europeans.

46 lymorphisms for schizophrenia in the largest genome-wide association study conducted to date suggeste

47 tionship between ALS and schizophrenia using genome-wide association study data from over 100,000 uni

48 Genome-wide association study data has showed that sever

49 Finally, integrating our data with human genome-wide association study data implicates two previo

50 We report analyses of novel genome-wide association study data of 1,234 ALS cases an

51 We conducted meta-analyses of genome-wide association study data on 2080 cannabis-depe

52 cleotide polymorphisms deriving from a large genome-wide association study data set.

53 METHODS AND Through a meta-analysis of 6 genome-wide association study data sets and a validation

54 Genome-wide association study data were available for 46

55 -sample MR approach and publically available genome-wide association study data.MR results indicated

56 Here we perform genome-wide association studies for anti-PEG IgM and IgG

57 Several consortia have pursued genome-wide association studies for identifying novel ge

58 Interpretation of results from genome-wide association studies for T2D is challenging.

59 el and colleagues perform a meta-analysis of genome-wide association studies for whole body lean body

60 We performed the first genome-wide association study for molar shape and used 3

61 Here, we perform a genome-wide association study for NFT pathologic burden

62 We conducted a genome-wide association study for plasma catestatin, the

63 In a genome-wide association study for variants associated wi

64 We performed a genome-wide association study for VTE with approximately

65 Combining data from genome-wide association studies from multiple phenotypes

66 d then validated significant SNPs in another genome-wide association study from Harvard University.

67 Using data from a published genome-wide association study from The University of Tex

68 disease-associated genetic variants through genome wide association studies (GWAS), genetic risk pre

69 As part of a recent genome wide association study (GWAS) to identify novel l

70 Trans-ethnic meta-analysis of genome-wide association studies (GWAS) across diverse po

71 Network-based genome-wide association studies (GWAS) aim to identify f

72 Recent genome-wide association studies (GWAS) and pathway analy

73 In the era of genome-wide association studies (GWAS) and personalized

74 Genome-wide association studies (GWAS) and proteomic stu

75 First, the conventional genome-wide association studies (GWAS) and the distance

76 ingle-nucleotide polymorphisms identified by genome-wide association studies (GWAS) are in noncoding

77 trafficking receptor that was identified by genome-wide association studies (GWAS) as a novel regula

78 Genome-wide association studies (GWAS) can serve as stro

79 disease susceptibility loci annotated in the genome-wide association studies (GWAS) catalog.

80 Applying this approach, we mined Caucasian genome-wide association studies (GWAS) data from two of

81 Genome-wide association studies (GWAS) do not distinguis

82 We calculated polygenic scores from genome-wide association studies (GWAS) for 127 traits an

83 Genome-wide association studies (GWAS) for BMI, waist-to

84 We conducted the largest meta-analysis of genome-wide association studies (GWAS) for psoriasis to

85 Genome-wide association studies (GWAS) for schizophrenia

86 Genetic and genomic studies, including genome-wide association studies (GWAS) have accelerated

87 Multilocus genome-wide association studies (GWAS) have become the s

88 Genome-wide association studies (GWAS) have been success

89 During the past decade, genome-wide association studies (GWAS) have been used to

90 Recent genome-wide association studies (GWAS) have confirmed kn

91 Candidate-gene approaches and recent genome-wide association studies (GWAS) have enabled a de

92 Over the last decade, genome-wide association studies (GWAS) have generated va

93 Genome-wide association studies (GWAS) have identified >

94 Large-scale meta-analyses of genome-wide association studies (GWAS) have identified >

95 Previous genome-wide association studies (GWAS) have identified 1

96 Recent genome-wide association studies (GWAS) have identified m

97 Genome-wide association studies (GWAS) have identified m

98 Previous genome-wide association studies (GWAS) have identified s

99 Genome-wide association studies (GWAS) have identified t

100 Genome-wide association studies (GWAS) have not identifi

101 Genome-wide association studies (GWAS) have played an im

102 Accumulating data from genome-wide association studies (GWAS) have provided a c

103 vances in genome sequencing technologies and genome-wide association studies (GWAS) have provided unp

104 Previous genome-wide association studies (GWAS) have reported sev

105 Genome-wide association studies (GWAS) have transformed

106 Genome-wide association studies (GWAS) have yielded vari

107 Recently, genome-wide association studies (GWAS) identified single

108 Genome-wide association studies (GWAS) identify 5311 exp

109 We performed an exome-chip based genome-wide association studies (GWAS) in 2158 cases fro

110 Genome-wide association studies (GWAS) in coronary arter

111 We developed a Bayesian-based method for genome-wide association studies (GWAS) in which RNA-seq-

112 The genome-wide association studies (GWAS) included 17,931 i

113 loci from the largest body mass index (BMI) genome-wide association studies (GWAS) meta-analysis wit

114 risk by using summary data of six published genome-wide association studies (GWAS) of 12,160 cases a

115 orts and the underlying trait heterogeneity, genome-wide association studies (GWAS) of chronic period

116 bility variants have been identified through genome-wide association studies (GWAS) of predominantly

117 Most genome-wide association studies (GWAS) of QT were perfor

118 Genome-wide association studies (GWAS) of single nucleot

119 Few genome-wide association studies (GWAS) of type 2 diabete

120 While genome-wide association studies (GWAS) on branched-chain

121 Genome-wide association studies (GWAS) on FTD identified

122 Genome-wide association studies (GWAS) previously identi

123 tic loci that are identified using data from Genome-Wide Association Studies (GWAS) provide insights

124 functional variants responsible for observed genome-wide association studies (GWAS) signals is one of

125 y marker arrays and Immunochips have powered genome-wide association studies (GWAS) that have mapped

126 among loci previously linked to IBD through genome-wide association studies (GWAS) using functional

127 quantitative trait locus (QTL) analysis and genome-wide association studies (GWAS) using the MAGIC p

128 Analysis of genome-wide association studies (GWAS) with "time to eve

129 Genome-wide association studies (GWAS) with intermediate

130 The ENIGMA consortium conducted genome-wide association studies (GWAS) with standard mul

131 g single nucleotide polymorphism (SNP)-based genome-wide association studies (GWAS) would identify gr

132 In comparison to genome-wide association studies (GWAS), there has been p

133 analysis of 11 loci previously identified in genome-wide association studies (GWAS).

134 sex is a crucial quality control measure in genome-wide association studies (GWAS).

135 nisms, enabling the application of microbial genome-wide association studies (GWAS).

136 ation of promising associations suggested by genome-wide association studies (GWAS).

137 ly during the past decade with the advent of genome-wide association studies (GWAS).

138 es at an unprecedented level of detail using genome-wide association studies (GWAS).

139 ombined with existing data for an aggregated genome-wide association study (GWAS) analysis of lung ca

140 Genome-wide association study (GWAS) has become a widely

141 The initial genome-wide association study (GWAS) included 174 Finnis

142 We performed a comprehensive genome-wide association study (GWAS) including low-frequ

143 Among an approximately 680 known genome-wide association study (GWAS) loci for cardio-met

144 ed in conjunction with data from a recent CF genome-wide association study (GWAS) meta-analysis to de

145 ther fields in terms of gene discovery using genome-wide association study (GWAS) methods.

146 s of hippocampal structure here we perform a genome-wide association study (GWAS) of 33,536 individua

147 on and biology underlying AF, we undertook a genome-wide association study (GWAS) of 6,337 AF individ

148 We conducted a genome-wide association study (GWAS) of alcohol consumpt

149 We performed a genome-wide association study (GWAS) of blood pressure (

150 Biobank sample (N=108 976), we carried out a genome-wide association study (GWAS) of responses to the

151 We performed a genome-wide association study (GWAS) of self-reported al

152 er requiring transplantation, we conducted a genome-wide association study (GWAS) on 1,404 FECD cases

153 Based on our initial genome-wide association study (GWAS) on esophageal squam

154 enetics of Alcoholism (COGA), we performed a genome-wide association study (GWAS) on resting-state fa

155 nt of this vasculitis we performed the first genome-wide association study (GWAS) on this condition.

156 A genome-wide association study (GWAS) predicted additiona

157 Burgeoning availability of genome-wide association study (GWAS) results and nationa

158 The majority of genome-wide association study (GWAS) risk variants resid

159 t cell types by looking for an enrichment of genome-wide association study (GWAS) signal within funct

160 We used genome-wide association study (GWAS) summary statistics

161 n in the Japanese population, we performed a genome-wide association study (GWAS) that included 8,180

162 sceptibility loci for MacTel, we performed a genome-wide association study (GWAS) with 476 cases and

163 We conducted a genome-wide association study (GWAS) with replication in

164 In contrast to genome-wide association study (GWAS), genomic prediction

165 luded a single nucleotide polymorphism (SNP) genome-wide association study (GWAS), the selection of r

166 In this case-control genome-wide association study (GWAS), we did a genome-wi

167 ed QT prolongation in a multi-racial/ethnic, genome-wide association study (GWAS).

168 leotide polymorphisms (SNPs) at 34 loci in a genome-wide association study (GWAS).

169 dentify shared risk variants, we performed a genome-wide association study (GWAS; n = 360,838) of a b

170 -ASM), have become a major focus in the post-genome-wide-association-study (GWAS) era.

171 In this review, we discuss how genome-wide association studies (GWASs) and recent devel

172 associated polygenic variation identified by genome-wide association studies (GWASs) and specific env

173 Genome-wide association studies (GWASs) bring us closer

174 Recent Genome-wide Association Studies (GWASs) for eye diseases

175 Genome-wide association studies (GWASs) have been perfor

176 Genome-wide association studies (GWASs) have identified

177 Genome-wide association studies (GWASs) have identified

178 Recent genome-wide association studies (GWASs) have used microb

179 A meta-analysis of genome-wide association studies (GWASs) identified multi

180 Genome-wide association studies (GWASs) implicate the PH

181 oid cancer (DTC) were identified by previous genome-wide association studies (GWASs) in Europeans onl

182 Application of the experimental design of genome-wide association studies (GWASs) is now 10 years

183 Recent successes in genome-wide association studies (GWASs) make it possible

184 Most genetic variants identified in genome-wide association studies (GWASs) of complex trait

185 by using the summary data from six published genome-wide association studies (GWASs) of Transdiscipli

186 Despite the success of large-scale genome-wide association studies (GWASs) on complex trait

187 Methods that increase the power of genome-wide association studies (GWASs) to identify risk

188 In recent years, as a secondary analysis in genome-wide association studies (GWASs), conditional and

189 trait-associated DNA variants discovered in genome-wide association studies (GWASs), is to estimate

190 lysis is essential to combine the results of genome-wide association studies (GWASs).

191 s of effect on traits remains a challenge in genome-wide association studies (GWASs).

192 Genome-wide association studies have demonstrated associ

193 Genome-wide association studies have demonstrated the im

194 Genome-wide association studies have enabled identificat

195 Genome-wide association studies have identified 200 infl

196 Genome-wide association studies have identified a great

197 Genome-wide association studies have identified a signal

198 Genome-wide association studies have identified an allel

199 Genome-wide association studies have identified at least

200 Recent genome-wide association studies have identified genetic

201 Genome-wide association studies have identified genetic

202 Genome-wide association studies have identified hundreds

203 Genome-wide association studies have identified loci inf

204 Although candidate gene and genome-wide association studies have identified promisin

205 Genome-wide association studies have implicated the ANK3

206 Genome-wide association studies have implied the associa

207 Genome-wide association studies have recently identified

208 Genome-wide association studies have revealed an associa

209 Genome-wide association studies identified 245 significa

210 Genome-wide association studies identified numerous dise

211 Recent genome-wide association studies identified over 100 gene

212 The identity of this locus is unknown, but genome-wide association studies identified potentially c

213 A genome-wide association study identified LMO1, which enc

214 isms remain unknown for most type 2 diabetes genome-wide association study identified loci.

215 This multicohort genome-wide association study identified new genomic loc

216 Here we perform a meta-analysis of six genome-wide association studies, imputed using a merged

217 pes and risk for heart defects, we conducted genome-wide association studies in 5 cohorts.

218 Genome-wide association studies in frontotemporal dement

219 particularly on genetic approaches including genome-wide association studies in humans and mice, -omi

220 and provide a behavioral paradigm for future genome-wide association studies in populations with incr

221 We performed a genome-wide association study in 1001 healthy participan

222 weighted genetic risk score study based on a genome-wide association study in KD.

223 onses to an acellular pertussis vaccine by a genome-wide association study in mice.

224 We used a two-stage approach: a genome-wide association study in patients with IPF of Eu

225 NPs) related to fetuin-A concentrations by a genome-wide association study in six population-based st

226 We performed a genome-wide association study in UK Biobank testing appr

227 es with the DR3-DQ2.5 haplotype and a recent genome-wide association study indicating that B*08 and B

228 support analyses involving complexity beyond genome-wide association studies is not standardized or c

229 and trait-associated variants identified in genome-wide association studies largely cluster at regul

230 tes that genetic variants identified through genome-wide association studies may contribute significa

231 We conducted a cross-disorder genome-wide association study meta-analysis to identify

232 ociation studies and applied them to a large genome wide association study of BMD.

233 Here, we report a multivariate genome-wide association studies of 23 immunoglobulin G (

234 riants that have been identified in previous genome-wide association studies of 25(OH)D concentration

235 We explored genome-wide association studies of chronic lymphocytic l

236 193 CpGs with an enrichment of signals from genome-wide association studies of lipid levels (PTC=0.0

237 r Consortium (TECAC) combined five published genome-wide association studies of testicular germ cell

238 Most genome-wide association studies of this trait have been

239 Genome-wide association studies of tumor samples have id

240 From a genome-wide association study of 16,596 individuals of E

241 In a genome-wide association study of 19,219 individuals, we

242 We did a genome-wide association study of 297 P falciparum isolat

243 We performed a genome-wide association study of African Americans with

244 The authors conducted a genome-wide association study of anorexia nervosa and ca

245 Here we report the results of a genome-wide association study of breast cancer in 122,97

246 In this study, we performed a two-stage genome-wide association study of executive inhibition in

247 Here, we conduct a genome-wide association study of IOP in 69,756 untreated

248 ci underlying LBM, we performed a gene-based genome-wide association study of lean mass index (LMI) i

249 Here, we performed a genome-wide association study of lobe attachment in a mu

250 Retirement Study (HRS) to perform the first genome-wide association study of loneliness.

251 We performed a genome-wide association study of melanin levels in 285 H

252 Here we report findings from a genome-wide association study of non-medullary thyroid c

253 METHODS AND We performed a genome-wide association study of serum MMP-8 levels in 2

254 We performed a genome-wide association study of serum PTH concentration

255 g a polygenic score derived from a published genome-wide association study of subjective well-being.

256 f 43 SCZ risk genes identified by the recent genome-wide association study of the Schizophrenia Worki

257 We performed a genome-wide association study of total WBC and different

258 om a case and control sample, we conducted a genome-wide association study of usual daily methadone d

259 We meta-analyzed 2 genome-wide association studies on self-reported allergy

260 ined a search for protein quantity loci with genome-wide association studies on the abundance of 7S a

261 DNA markers in these regions, we conducted a genome-wide association study on a set of 185 U.S. winte

262 diastolic blood pressure measurements for a genome-wide association study on long-term average systo

263 2, Tyrobp) defined as risk factors for AD by genome-wide association study or identified as genetic n

264 omal K(+) channel, is centered under a major genome-wide association studies peak for PD, making it a

265 Whole-genome sequencing and a genome-wide association study permitted identification o

266 f P-wave morphology through meta-analysis of genome-wide association study results for P-wave duratio

267 We also conducted meta-analyses using the genome-wide association study results from the CTD cohor

268 lation of polygenic risk scores was based on genome-wide association study results generated by the P

269 Recent microbiome genome-wide association studies reveal that variants in

270 ntified 14,059 segregating polymorphisms and genome-wide association studies revealed 28 GWAS hits in

271 hrenia risk, including several schizophrenia genome-wide association study risk genes (e.g., calcium

272 In the individual genome-wide association studies, several genome-wide sig

273 a subset of these polymorphisms is linked to genome-wide association study signals of complex traits

274 WAS Catalog has provided data from published genome-wide association studies since 2008.

275 methylation is associated in cis with known genome-wide association study single nucleotide polymorp

276 Compellingly, recent genome-wide association studies suggest repeat polymorph

277 A genome-wide association study suggested that VO2 max tra

278 s applied to two large Caucasian and Chinese genome-wide association study summary datasets of bone m

279 Meta-analysis with published genome-wide association study summary statistics replica

280 genetics of depression has been explored in genome-wide association studies that focused on either m

281 Here, we report a genome-wide association study that included 1,122 PBC ca

282 entifying response-associated genes in large genome-wide association studies, the results have been f

283 ingle-nucleotide polymorphisms identified by genome-wide association studies to be associated with BM

284 tistics from the largest Alzheimer's disease genome-wide association study to date.

285 We have conducted a genome-wide association study to identify genomic varian

286 We conducted an exploratory genome-wide association study to identify single-nucleot

287 tide polymorphisms (SNPs) were analyzed in a genome-wide association study to identify susceptibility

288 For the same maize data, we also conducted genome-wide association study, transcriptome-wide associ

289 We report a genotypic variability-based genome-wide association study (vGWAS) of six cohorts of

290 ymorphisms associated with BMI from previous genome-wide association studies was constructed, and the

291 mmary association results from 7 large-scale genome-wide association studies, we examined the effect

292 Through interrogation of a genome-wide association study, we examined the associati

293 In a genome-wide association study, we identified a naturally

294 Through a genome-wide association study, we report the identificat

295 ng information for any diagnostic criterion, genome-wide association studies were performed on 2 samp

296 Discovery case-control genome-wide association studies were performed where cas

297 METHODS AND Summary statistics from several genome-wide association studies were used in a 2-sample

298 Genotyping, imputation, and genome-wide association study were performed at each stu

299 We therefore performed a genome-wide association study with a dimensional, PD/AG-

300 In the first step (n = 979), we performed a genome-wide association study with a predefined suggesti