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1 n between FANCD2 and 10 proteins involved in homologous recombination repair.
2 BRCA1 and BRCA2 pathways as they function in homologous recombination repair.
3 Rad51 gene family, thought to be central to homologous recombination repair.
4 s early DNA end resection, the first step of homologous recombination repair.
5 tion with non-homologous end joining but not homologous recombination repair.
6 promoting early ATM checkpoint responses and homologous recombination repair.
7 er of DSB processing that is a requisite for homologous recombination repair.
8 precipitation studies) and together, promote homologous recombination repair.
9 g protein (CTIP)-dependent end resection and homologous recombination repair.
10 WI5 or MEI5 in human cells causes defects in homologous recombination repair.
11 has an evolutionarily conserved function in homologous recombination repair.
12 SG2285 was primarily dependent on ERCC1 and homologous recombination repair.
13 also govern the choice of templates used in homologous recombination repair.
14 ity, defective G2/M checkpoint, and impaired homologous recombination repair.
15 anded DNA, which is critically important for homologous recombination repair.
16 ensive end processing associated with failed homologous recombination repair.
17 se and subsequently recruit Rad51 to promote homologous recombination repair.
18 mpaired Fancd2 foci assembly and a defect in homologous recombination repair.
19 between BRCA1 and PALB2 is important for the homologous recombination repair.
20 sis and the reinitiation of DNA synthesis by homologous recombination repair.
22 f SA2 but not SA1 decreased sister chromatid homologous recombination repair and affected repair path
25 on of BRCA2 and links BRCA1 and BRCA2 in DNA homologous recombination repair and breast cancer suppre
26 ssion of miR-155 decreased the efficiency of homologous recombination repair and enhanced sensitivity
27 polymerase-helicase complex participates in homologous recombination repair and is essential for cel
28 ncodes a DNA helicase proposed to operate in homologous recombination repair and replicational stress
29 nomic instability and, consequently, require homologous recombination repair and the DNA damage check
30 genes, BLM and WRN, play important roles in homologous recombination repair and they have been impli
31 g assays of cellular survival and viability, homologous recombination repair, and genome instability.
32 plays a central role in DNA replication and homologous recombination repair, and is known to be invo
33 tion of Rad51 focus formation, inhibition of homologous recombination repair, and persistent gamma-H2
34 ngle-strand break repair, XRCC3 and RAD51 in homologous recombination repair, and XRCC7 in nonhomolog
35 rate cells, the primary proteins involved in homologous recombination repair are RAD51 and the five R
36 mid containing a sequence that could support homologous recombination repair between the two plasmids
37 uppression of Rad51 expression, required for homologous recombination repair, blocked the ability of
38 nly enhances reparation of DNA lesions (e.g. homologous recombination repair), but also prolongs acti
39 P1 to a DNA double-strand break (DSB) during homologous recombination repair, but a role in DSB repai
41 We propose that not all components of the homologous recombination repair complex can act as cance
44 nt nuclear depletion of BRCA1 and subsequent homologous recombination repair deficit was induced with
47 ed in protecting stressed replication forks: homologous recombination repair, DNA inter-strand cross-
48 ithelium-derived growth factor and the Rad51 homologous recombination repair factor at DNA breaks.
49 is suppressed by concomitant deletion of the homologous recombination repair factor, Rhp51 (Rad51).
50 dependent on the XPF-ERCC1 heterodimer, and homologous recombination repair factors XRCC2 and XRCC3.
51 er DNA cleavage, lesser focal recruitment of homologous recombination repair factors, impaired DNA do
52 d sequence (block size of six, stratified by homologous recombination repair gene mutation status, pr
54 vidence that Ape1 facilitates BRCA1-mediated homologous recombination repair (HR), while counteractin
55 se cDNA or dominant-negative mutant inhibits homologous recombination repair (HRR) and increases sens
56 SB repair pathways exist in mammalian cells: homologous recombination repair (HRR) and nonhomologous
57 e lesions are repaired by BCR/ABL-stimulated homologous recombination repair (HRR) and nonhomologous
58 sion cassette, allowing for the detection of homologous recombination repair (HRR) by GFP expression.
59 tive tumour cell lines are also defective in homologous recombination repair (HRR) induced by DNA dou
60 DSBs and speculate that the contribution of homologous recombination repair (HRR) is at a stage afte
61 urate joining of DNA double-strand breaks by homologous recombination repair (HRR) is critical to the
66 The involvement of BRCA1 and BRCA2 in the homologous recombination repair (HRR) of double-strand b
70 ed the ATM-dependent DNA damage response and homologous recombination repair (HRR) via decreasing DIC
72 for the RAD51 homolog XRCC2 and defective in homologous recombination repair (HRR), displays signific
73 FANCD2 in promoting RAD51 foci formation and homologous recombination repair (HRR), EGFR-mutant cells
74 BL stimulate unfaithful DSB repair pathways, homologous recombination repair (HRR), nonhomologous end
75 t depletion of BRCA1, an important factor of homologous recombination repair (HRR), preferentially se
76 though the SHU genes have been implicated in homologous recombination repair (HRR), their precise rol
77 ocking down Hus1 decreases the efficiency of homologous recombination repair (HRR), which is associat
88 tablishes a previously unidentified role for homologous recombination repair in correct neuronal deve
89 idelity repair of DSBs by not only promoting homologous recombination repair in G2/M phase but also f
90 s (such as with etoposide), up-regulation of homologous recombination repair in response to p53 disru
91 s to repair DNA double-strand breaks through homologous recombination repair, increasing the involvem
92 suggest that G(2) checkpoint abrogation and homologous recombination repair inhibition both contribu
96 t the absence of BRCA2 substantially reduced homologous recombination repair of DNA breaks, whereas t
97 h the RAD51 protein complex is essential for homologous recombination repair of DNA damage and mainta
99 and Nse2 function together with Rhp51 in the homologous recombination repair of DNA double strand bre
100 ced radiation-induced cell death and reduced homologous recombination repair of DNA double-strand bre
101 he Archaea away from the eukaryotic model of homologous recombination repair of DNA double-strand bre
102 (BRCA2), the latter of which is involved in homologous recombination repair of DNA double-strand bre
103 2, and Rad54, plays an important role in the homologous recombination repair of double strand breaks.
104 ing the possibility of hSNM1B involvement in homologous recombination repair of double-strand breaks
105 oreover, we find that SETD2 is necessary for homologous recombination repair of DSBs by promoting the
106 -way junctions are structures present during homologous recombination, repair of double stranded DNA
107 ed for the SOS response and are defective in homologous recombination, repair of UV damaged DNA, doub
108 r sensitivity to nucleotide excision repair, homologous recombination repair, or postreplication repa
109 ts in two BRCA1-mediated DDR events: (i) the homologous recombination repair pathway and (ii) the arr
110 g PPP2R2A thereby impaired the high-fidelity homologous recombination repair pathway and sensitized c
111 necessary host polymerases, proteins of the homologous recombination repair pathway may be considere
112 w DNA repair, especially the RAD51-dependent homologous recombination repair pathway, is executed in
121 T also induces a distinct set of foci of the homologous recombination repair protein Rad51 that are c
123 gnificant reduction in MRN/ATM signaling and homologous recombination repair, suggesting that Thr622
126 on yeast homologue Swi5-Sfr1 is critical for homologous recombination repair, the budding yeast count
127 epair components have been implicated in DNA homologous recombination repair, the exact function of h
128 oteins might function in a common pathway in homologous recombination repair to ensure accurate nucle
129 ionally, POH1 acts independently of 53BP1 in homologous recombination repair to promote RAD51 loading
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