ライフサイエンスコーパス: hydatidiform

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1 sed 77 twin pregnancies, comprising complete hydatidiform mole (CHM) and healthy co-twin, to ascertai

2 is based on the use of a homozygous complete hydatidiform mole (CHM) as the reference.

3 for alpha satellite containing reads in the hydatidiform mole (CHM1, haploid-like) genome.

4 Familial biparental hydatidiform mole (FBHM) is the only known pure maternal

5 Hydatidiform mole (HM) is an abnormal gestation characte

6 , NLRP7 and NLRP2, cause familial biparental hydatidiform mole and multilocus imprinting disturbance,

7 e genome-wide restriction maps of a complete hydatidiform mole and three lymphoblast-derived cell lin

8 haliana, Drosophila melanogaster and a human hydatidiform mole cell line (CHM1) from SMRT sequencing.

9 We used data generated from a haploid hydatidiform mole genome (CHM1) and a diploid human geno

10 development of the conceptus into a complete hydatidiform mole in which extraembryonic trophoblastic

11 The most frequent type of complete hydatidiform mole is a 46, XX homozygote formed by the f

12 We leverage a haploid hydatidiform mole to identify highly identical sequences

13 esent evidence of homozygosity of a complete hydatidiform mole using 20 diallelic markers distributed

14 emalignant disorders of complete and partial hydatidiform mole, and the malignant disorders of invasi

15 used genomic DNA from an essentially haploid hydatidiform mole, CHM1.

16 rations 6 months after uterine evacuation of hydatidiform mole, even when values are falling.

17 istinguishable from an androgenetic complete hydatidiform mole, in which abnormal extra-embryonic tis

18 These diseases vary from partial hydatidiform mole, which rarely metastasizes and infrequ

19 trations of hCG 6 months after evacuation of hydatidiform mole.

20 concentrations 6 months after evacuation of hydatidiform mole.

21 Complete hydatidiform moles (CHMs) are diploid tumors that result

22 Rare familial biparental hydatidiform moles (due to NLRP7 or KHDC3L mutations) sh

23 niparental tissues of germline origin, i.e., hydatidiform moles (paternal origin) and complete ovaria

24 Partial hydatidiform moles (PMs) rarely require chemotherapy and

25 sentation of the paternal genome in sporadic hydatidiform moles (purely androgenetic in complete hyda

26 iform moles (purely androgenetic in complete hydatidiform moles and diandric triploid in partial hyda

27 Hydatidiform moles are intriguing pathologic entities re

28 diagnosis, routine morphologic assessment of hydatidiform moles continues to suffer from interobserve

29 76 (<1%) of 13,960 patients with hydatidiform moles had persistently high hCG concentrati

30 eotide polymorphism markers in most complete hydatidiform moles indicating that these tumors are not

31 The homozygous nature of these complete hydatidiform moles makes them unique resources for human

32 iform moles and diandric triploid in partial hydatidiform moles) is a fundamental genetic event leadi

33 orms are derived from the precursor lesions, hydatidiform moles.

34 hat these tumors are not related to complete hydatidiform moles.

35 ostic methods for accurate classification of hydatidiform moles.

36 nal-triploidy and the development of partial hydatidiform moles.

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