ライフサイエンスコーパス: hydatidiform mole

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1 trations of hCG 6 months after evacuation of hydatidiform mole.

2 concentrations 6 months after evacuation of hydatidiform mole.

3 orms are derived from the precursor lesions, hydatidiform moles.

4 hat these tumors are not related to complete hydatidiform moles.

5 ostic methods for accurate classification of hydatidiform moles.

6 nal-triploidy and the development of partial hydatidiform moles.

7 , NLRP7 and NLRP2, cause familial biparental hydatidiform mole and multilocus imprinting disturbance,

8 e genome-wide restriction maps of a complete hydatidiform mole and three lymphoblast-derived cell lin

9 iform moles (purely androgenetic in complete hydatidiform moles and diandric triploid in partial hyda

10 emalignant disorders of complete and partial hydatidiform mole, and the malignant disorders of invasi

11 Hydatidiform moles are intriguing pathologic entities re

12 haliana, Drosophila melanogaster and a human hydatidiform mole cell line (CHM1) from SMRT sequencing.

13 sed 77 twin pregnancies, comprising complete hydatidiform mole (CHM) and healthy co-twin, to ascertai

14 is based on the use of a homozygous complete hydatidiform mole (CHM) as the reference.

15 for alpha satellite containing reads in the hydatidiform mole (CHM1, haploid-like) genome.

16 used genomic DNA from an essentially haploid hydatidiform mole, CHM1.

17 Complete hydatidiform moles (CHMs) are diploid tumors that result

18 diagnosis, routine morphologic assessment of hydatidiform moles continues to suffer from interobserve

19 Rare familial biparental hydatidiform moles (due to NLRP7 or KHDC3L mutations) sh

20 rations 6 months after uterine evacuation of hydatidiform mole, even when values are falling.

21 Familial biparental hydatidiform mole (FBHM) is the only known pure maternal

22 We used data generated from a haploid hydatidiform mole genome (CHM1) and a diploid human geno

23 76 (<1%) of 13,960 patients with hydatidiform moles had persistently high hCG concentrati

24 Hydatidiform mole (HM) is an abnormal gestation characte

25 development of the conceptus into a complete hydatidiform mole in which extraembryonic trophoblastic

26 istinguishable from an androgenetic complete hydatidiform mole, in which abnormal extra-embryonic tis

27 eotide polymorphism markers in most complete hydatidiform moles indicating that these tumors are not

28 The most frequent type of complete hydatidiform mole is a 46, XX homozygote formed by the f

29 iform moles and diandric triploid in partial hydatidiform moles) is a fundamental genetic event leadi

30 The homozygous nature of these complete hydatidiform moles makes them unique resources for human

31 niparental tissues of germline origin, i.e., hydatidiform moles (paternal origin) and complete ovaria

32 Partial hydatidiform moles (PMs) rarely require chemotherapy and

33 sentation of the paternal genome in sporadic hydatidiform moles (purely androgenetic in complete hyda

34 We leverage a haploid hydatidiform mole to identify highly identical sequences

35 esent evidence of homozygosity of a complete hydatidiform mole using 20 diallelic markers distributed

36 These diseases vary from partial hydatidiform mole, which rarely metastasizes and infrequ

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