ライフサイエンスコーパス: medullary

1 1 malignant FNA, 5 oncocytic/Hurthle cell, 2 medullary, 1 follicular, and 4 metastases from underlyin

2 ced V-ATPase apical membrane accumulation in medullary A-ICs but not in cortical A-ICs or other IC su

3 Our study shows that medullary A-ICs respond to luminal adenosine through ADO

4 nce of the NKCC2 transporter responsible for medullary accumulation of ammonium.

5 nsepithelial resistance, and defective renal medullary accumulation of sodium and other osmolytes.

6 -pituitary-adrenal axis, sympathetic adrenal-medullary activation and catecholamine levels, the infla

7 001), cortical ADC (r = 0.43, P = .003), and medullary ADC (r = 0.35, P = .01).

8 Reconstructions reveal that many trans-medullary-afferents (TmAs) connect the eye with each LGM

9 progression, was associated with aggressive medullary and anaplastic carcinomas, and its expression

10 other, more rare subtypes of thyroid cancer-medullary and anaplastic-are ideally treated by physicia

11 or of total renal NKCC2 protein abundance in medullary and cortical thick ascending limb.

12 tic disorders, cyst-dependent obstruction of medullary and cortical tubules initiates a process culmi

13 of the sympathetic nervous system, expanded medullary and extramedullary hematopoiesis.

14 n of SLAMF7-CAR T cells led to resolution of medullary and extramedullary myeloma manifestations in a

15 eatment options for patients with hereditary medullary and nonmedullary thyroid cancers related to mu

16 t due to the accumulation of ammonium in the medullary and papillary interstitium.

17 rons in the medial aspect of the superficial medullary and spinal dorsal horn from the trigeminal sub

18 The latter is reflected in medullary and supramedullary control.

19 -expressing NK cells, including conventional medullary and thymic NK cells, whereas TRAIL(+) Eomes(-)

20 llular scaffolds with vascular, cortical and medullary architecture, a collecting system and ureters.

21 d into the preBotzinger complex (preBotC), a medullary area that is critical for breathing.

22 CR7, how other T cell subsets gain access to medullary areas during their normal development is not c

23 eses that in conditions of MeCP2 deficiency, medullary astrocytes are unable to produce/release appro

24 (ii) MeCP2 deficiency impairs the ability of medullary astrocytes to sense changes in PCO2/[H(+) ].

25 tude of CO2 -induced [Ca(2+) ]i responses in medullary astrocytes was markedly reduced in conditions

26 ice with normal angiopoietin-Tie2 signaling, medullary AVR exhibited an unusual hybrid endothelial ph

27 adrenal cortex, and the sympathetic-adrenal-medullary axis, which controls stress-induced catecholam

28 some also prevented the secondary decline in medullary blood flow and ischemia that develops 2 hours

29 ctive role in renal IR injury by maintaining medullary blood flow and that a genetic deficiency in th

30 Medullary bone (MB), an estrogen-dependent reproductive

31 hat progressively increases from the cortico-medullary boundary to the inner medullary tip.

32 ning REM sleep is located in the pontine and medullary brainstem and includes ascending and descendin

33 c databases revealed ANTXR1 amplification in medullary breast carcinoma and overexpression in estroge

34 atory muscle, and in patients with defective medullary breathing pattern generators.

35 Medullary, but not cortical, ADCT values stayed increase

36 nce of Wnt7b signaling, the periureteric bud medullary capillaries displayed narrower lumens lined wi

37 ing and the ureteric bud epithelium in renal medullary capillary development.

38 ndocrine glands-like C cells of the thyroid (medullary carcinoma), the parasympathetic and sympatheti

39 we test whether these rostral ventrolateral medullary catecholaminergic (RVLM-CA) neurons use glutam

40 which bone tissue progressively invades the medullary cavity in the mid-diaphysis.

41 of the limb bones are solid without an open medullary cavity, for buoyancy control in water.

42 st PSB-0739 in primary cultures of rat inner medullary CD cells potentiated the expression of AQP2 an

43 GPC protects inner medullary cells against the perturbing effects of high N

44 ine release was measured from bovine adrenal medullary chromaffin cell (CC) cultures maintained over

45 ine and norepinephrine released from adrenal medullary chromaffin cells and norepinephrine released l

46 Mice lacking the inner medullary collecting duct (IMCD) urea transporter A1 (UT

47 n regulates NO production in the renal inner medullary collecting duct (IMCD), the segment with the g

48 3D cell culture model that uses mouse inner-medullary collecting duct (mIMCD3) cells to generate epi

49 hibition of p38MAPK activity in murine inner medullary collecting duct 3 (mIMCD3) cells decreased exp

50 (PI3K-C2alpha) in renal tubule-derived inner medullary collecting duct 3 cells and show that PI3K-C2a

51 ar to cells lacking PC2, NEK8-depleted inner medullary collecting duct cells exhibited a defective re

52 genetic knockdown of TRIP13 in murine inner medullary collecting duct cells in the presence of hydro

53 retinal pigmented epithelial and mouse inner medullary collecting duct cells-3.

54 s from the proximal tubule through the inner medullary collecting duct of rat kidneys.

55 ely integrated into the aquaporin 2-positive medullary collecting duct when microinjected into the ki

56 ENaC was appropriately increased in the medullary collecting duct, suggesting a localized inhibi

57 ubule and decreased slightly in the cortical/medullary collecting duct, whereas BK channel abundance

58 nts with the highest expression in the inner medullary collecting duct.

59 We employed proteomic analysis of inner medullary collecting ducts (IMCD) from rats fed with a p

60 n vitro microperfusion of cortical and outer medullary collecting ducts, was unaffected in mutant mic

61 de use of primarily cultured rat renal inner medullary collecting-duct cells and microarray analysis

62 artment kidney phantom (70% cortical and 30% medullary compartment), a sphere, and an ellipsoid of eq

63 TECs and in addition affects the size of the medullary compartment, TEC-specific HIPK2 deletion only

64 t are connected to or separated from a major medullary compartment.

65 ion-mediated cross-talk in generation of the medullary compartment.

66 The microenvironment of the medullary cords is positioned to control these key proce

67 at yellow fluorescent protein(+) ASCs in the medullary cords migrated along myelomonocytic cells and

68 s sensed higher concentrations of S1P in the medullary cords than in the T cell zone and that the S1P

69 h environments were observed in the expanded medullary cords.

70 omplete SCI, we created bilaterally complete medullary corticospinal tract lesions in adult mice, eli

71 utosomal dominant polycystic kidney disease, medullary cystic kidney disease, diabetic nephropathy, o

72 These findings demonstrate that cortical and medullary DCs play specialized roles in their respective

73 he immune defense against pyelonephritis, as medullary DCs were less CX3CR1 dependent than cortical D

74 contrast, Foxp3(+) nTreg cell development is medullary dependent, with mTECs fostering the generation

75 F-kappaB in the thymus and are necessary for medullary development.

76 equirement for cross-talk with thymocytes in medullary development.

77 tricted to interneurons in lamina IIi of the medullary dorsal horn, where they constitute 1/3 of tota

78 ters by projection neurons in the spinal and medullary dorsal horn.

79 Mechanisms promoting thymocyte medullary entry and interactions with APCs are incomplet

80 microscopy to demonstrate that CCR4 promotes medullary entry of the earliest post-positive selection

81 vation with 5-thioglucose stimulated adrenal medullary epinephrine (Epi) release (3,153%) and feeding

82 fluenced the ability of Aire to regulate the medullary epithelial cell transcriptome and so was cruci

83 alysis of Aire-sufficient and Aire-deficient medullary epithelial cells (mTECs).

84 with increased frequencies of AIRE-positive medullary epithelial cells and CD11c-positive dendritic

85 expression of a large set of genes in thymic medullary epithelial cells, thereby controlling the repe

86 d CD40-CD40L pathways is required for normal medullary epithelium and for maintenance of self-toleran

87 -) and that transitioned into a multilaminar medullary epithelium forming neurotubules with adluminal

88 ssion of tissue-specific genes in the thymic medullary epithelium.

89 al source for the tumor and its multilaminar medullary epithelium.

90 ng youth were maintained as a result of less medullary expansion and cortical trabecularization.

91 youth were gradually lost because of greater medullary expansion and cortical trabecularization.

92 Here we examine the medullary expression of sst2a with particular reference

93 was significant correlation between eGFR and medullary FA (r = 0.65, P < .001), cortical ADC (r = 0.4

94 In group B, mean medullary FA was significantly lower in patients whose r

95 owed a 14-24 fold increase from baseline for medullary GRP78, sXBP-1, and CHOP.

96 Medullary hyperosmolarity is protected from washout by c

97 Medullary hypoxia due to intrarenal blood flow redistrib

98 Medullary hypoxia-inducible factor gene expression decre

99 ll mice was markedly increased, resulting in medullary hypoxia.

100 ependent protein phosphorylation in purified medullary ICs that were isolated from mice.

101 hat adenosine activates V-ATPase in isolated medullary ICs.

102 cal deficit that was attributable to lateral medullary infarction.

103 Proliferating, medullary, interstitial myofibroblasts strongly expresse

104 e mice, an effect accompanied by a hypotonic medullary interstitium and impaired countercurrent multi

105 The hyperosmolality in the renal medullary interstitium is of major importance as a drivi

106 al reabsorption of water into the hypertonic medullary interstitium mediated by collecting ducts.

107 rapid accumulation of fluid and cysts in the medullary interstitium, loss of medullary vascular bundl

108 s canonical Wnt signaling in the surrounding medullary interstitium, where the capillaries reside.

109 ith predominant involvement of glomeruli and medullary interstitium.

110 /-) mice exhibit only ~20 small, unconnected medullary islets.

111 ormally induces clearance of the renal outer medullary K(+) channel (ROMK) from the cell surface.

112 ne-independent activation of the renal outer medullary K(+) channel and ENaC, to which angiotensin II

113 iets induced upregulation of the renal outer medullary K(+) channel in AS(-/-) mice, whereas upregula

114 +) and NeuN(+) cells within the multilayered medullary layer approximate expression patterns similar

115 All patients showed a new medullary lesion on brain magnetic resonance imaging.

116 ystagmus had cerebellar peduncle and lateral medullary lesions.

117 of their role has been limited to spinal and medullary loci.

118 s to positions adjacent to both cortical and medullary lymphatic sinuses where the T cells exhibited

119 ered a program of PGE that is common between medullary (m) and cortical TEC, further elaborated in mT

120 vant MF59 localizes in subcapsular sinus and medullary macrophage compartments of mouse draining LNs,

121 CD169(+) SIGNR1(+) subcapsular medullary macrophages are the primary cells to take up G

122 the development and functional importance of medullary microenvironments during self-tolerant T-cell

123 thymus, interactions with both cortical and medullary microenvironments regulate the development of

124 of thymocyte precursors through cortical and medullary microenvironments, enabling specialized stroma

125 ire are involved in the regulation of thymus medullary microenvironments.

126 indicate that a functional compromise of the medullary mTEC(high) compartment may link alloimmunity t

127 chromophobe (n = 5), papillary (n = 5), and medullary (n = 2) RCC and unclassified RCC (uRCC, n = 23

128 mmunoblotting revealed downregulation of the medullary Na(+)-K(+)-2Cl(-) cotransporter NKCC2 in these

129 ation between chronic hyperaldosteronism and medullary nephrocalcinosis has rarely been made, with on

130 a long- standing history in whom associated medullary nephrocalcinosis was established.

131 increased risk of kidney stone formation or medullary nephrocalcinosis, namely 46% compared with 6%

132 s a causal factor in the etiopathogenesis of medullary nephrocalcinosis.

133 identification of a discrete JAG1(+) thymic medullary niche enriched for DC-lineage cells expressing

134 in multiple cortical areas, and thalamic and medullary nuclei.

135 6-fold specifically in these plastic, caudal medullary nuclei.

136 urn, the PAG has strong access to the caudal medullary nucleus retroambiguus (NRA).

137 to mammalian water balance and depends on a medullary osmotic gradient generated by a countercurrent

138 cal oxygenation and perfusion, but decreased medullary oxygenation and perfusion.

139 3 to 36.3 +/- 3.5 mm Hg (p < 0.001), whereas medullary oxygenation decreased from 29.6 +/- 4.7 to 13.

140 though sleep-active GABAergic neurons in the medullary parafacial zone (PZ) are needed for normal SWS

141 Stenotic kidney cortical/medullary perfusion and RBF were measured using contrast

142 perfusion was not significantly changed, but medullary perfusion decreased (671 BPU [500-900 BPU] to

143 Medullary perfusion in atherosclerotic RAS patients was

144 Single kidney cortical, medullary perfusion, and renal blood flow were measured

145 mic accumulation of mature thymocytes within medullary perivascular spaces and reduced numbers of rec

146 n AC6(-/-) mice seem to be restricted to the medullary portion of the thick ascending limb.

147 ition of the potassium-excretory renal outer medullary potassium (ROMK) channel have also been implic

148 me is caused by mutations in the renal outer medullary potassium (ROMK) channel, but the molecular me

149 elial sodium channel (ENaC), the renal outer medullary potassium channel (ROMK), and other transport

150 The renal outer medullary potassium channel (ROMK, or Kir1.1, encoded by

151 A, SUR1B, SUR2A, SUR2B, or ROMK (renal outer medullary potassium channel).

152 epithelial sodium channel-alpha, renal outer medullary potassium channel, and Na(+), K(+)-ATPase in t

153 on functional expression of the renal outer medullary potassium channel.

154 at changes in the electrical excitability of medullary pre-sympathetic neurones are the main causal m

155 tability of respiratory rhythm recorded from medullary preparations decreased with age but was higher

156 on of both AQP2 and pAQP2 in the renal inner medullary principal cells appeared more dispersed, and t

157 cement of electrodes less than 5 mm from the medullary pyramids resulted in treatment effect arcing i

158 as well as axonal loss, at the level of the medullary pyramids.

159 ventional regional estimates of cortical and medullary R2* levels were measured.

160 ional tissue hypoxia rather than cortical or medullary R2* values used to assess renal BOLD MR imagin

161 ing the nucleus of the solitary tract (NTS), medullary raphe and retrotrapezoid nucleus (RTN).

162 ons of ATP or a P2-receptor blocker into the medullary raphe had no effect on cardiorespiratory activ

163 lamic nucleus [PaMP]) and autonomic (rostral medullary raphe pallidus [RPa]) responses were targeted

164 if CO2/H(+)-sensitive neurons in the NTS and medullary raphe respond to ATP, and whether purinergic s

165 inase drivers targeted different portions of medullary raphe serotonergic, tryptophan hydroxylase 2 (

166 Medullary raphe transcriptome comparisons revealed lower

167 m several respiratory centres, including the medullary raphe.

168 (vertical phloem and inter-connected radial medullary rays [MR]).

169 g in the pre-Botzinger complex (pre-BotC), a medullary region generating the inspiratory phase of bre

170 These mice lacked a thymic medullary region, exhibited thymocyte retention, had a p

171 cluding the rostral and caudal ventrolateral medullary regions (RVLM and CVLM, respectively).

172 of spindle-shaped cells in both cortical and medullary regions of the kidney.

173 ) has previously been identified as an extra-medullary reservoir for normal hematopoietic stem cells

174 nimals with MeCP2 removed from specific pons medullary respiratory circuits, we performed whole-body

175 nctional connection between the amygdala and medullary respiratory network in humans.

176 esults indicate that MeCP2 expression in the medullary respiratory network is sufficient for normal r

177 e from lumbar locomotor CPG circuitry to the medullary respiratory networks that is able to depolariz

178 lterations in the electrical excitability of medullary respiratory neurones and their central modulat

179 eir spinal projections, several parts of the medullary reticular formation as well as the spinally pr

180 Lhx3-positive medullary reticular formation neurons express Fos follow

181 In the medullary reticular formation of the mouse, we identifie

182 CTb injection into the medullary RF labels cells and axonal endings predominant

183 lox) kidneys showed more cell death in outer medullary S3 segments at 2 hours but less tubular damage

184 ressive increase of neuronal excitability in medullary slices containing the pre-BotC produces mixed-

185 nd hypoglossal (XII) nerve motor activity in medullary slices from neonatal mice in conditions where

186 holographic photolysis of caged glutamate in medullary slices from neonatal mice.

187 We used rhythmically active medullary slices from wild-type (WT) and Lmx1b(f/f/p) ne

188 providing evidence for an independence from medullary support by the earliest stages after positive

189 ein, we evaluated the involvement of ventral medullary sympatho-excitatory catecholaminergic C1 neuro

190 heart rate variability (sympathetic adrenal medullary system), EEG event-related potentials (nocicep

191 s of p53 primarily disrupts the integrity of medullary TEC (mTEC) niche, a defect that spreads to the

192 a crucial transcription factor implicated in medullary TEC function.

193 ression of thymotropic chemokines, decreased medullary TEC to cortical TEC ratios, and altered thymic

194 In mature medullary TEC, AIRE-driven pGE upregulates non-TRA codin

195 adult mice, we found that both cortical and medullary TECs (cTECs and mTECs) proliferated more activ

196 el antigen specifically expressed in Aire(+) medullary TECs (mTECs) induced efficient deletion via di

197 antigens or presenting a neo-self-antigen by medullary TECs, displaying decreased negative selection-

198 The proximal convoluted tubule (PCT) and the medullary thick ascending limb (mTAL) in the kidney cons

199 tion of BMAL1 expression was observed in the medullary thick ascending limb.

200 (2+)]i oscillations were markedly reduced in medullary thick ascending limbs isolated from P2Y2 recep

201 thymocytes, and expression of its ligands by medullary thymic dendritic cells (DCs).

202 tical role in T cell tolerance by regulating medullary thymic epithelial cell (mTEC) development.

203 o-step terminal differentiation model of the medullary thymic epithelial cell (mTEC) lineage from imm

204 hough the molecular mediators that stimulate medullary thymic epithelial cell (mTEC) maturation are p

205 sting of a branching structure that contains medullary thymic epithelial cell (mTEC) networks to supp

206 reduction of autoimmune receptor-expressing medullary thymic epithelial cells (Aire1 mTEC) and a dec

207 ism, we have previously reported that mature medullary thymic epithelial cells (mTEC(high)) expressin

208 eral self-antigens (TRA), which is in mature medullary thymic epithelial cells (mTEC(high)) partly co

209 ize the developmental pathways that generate medullary thymic epithelial cells (mTEC) from their imma

210 In the thymus, medullary thymic epithelial cells (mTEC) regulate T cell

211 in developing T cells is highly dependent on medullary thymic epithelial cells (mTEC), and mTEC devel

212 CD70 was expressed on Aire(-) and Aire(+) medullary thymic epithelial cells (mTECs) and on dendrit

213 ulate transcription of a battery of genes in medullary thymic epithelial cells (mTECs) and, consequen

214 Medullary thymic epithelial cells (mTECs) are critical i

215 In this study, we reveal a role for medullary thymic epithelial cells (mTECs) during iNKT ce

216 Medullary thymic epithelial cells (mTECs) eliminate self

217 of tissue-restricted self antigens (TRAs) in medullary thymic epithelial cells (mTECs) is essential f

218 roles for bone marrow (BM)-derived APCs and medullary thymic epithelial cells (mTECs) on the convent

219 We show that bone marrow (BM) APCs and medullary thymic epithelial cells (mTECs) played nonover

220 plethora of tissue-restricted Ags (TRAs) by medullary thymic epithelial cells (mTECs) plays an essen

221 In medullary thymic epithelial cells (mTECs), the Autoimmun

222 neages--cortical thymic epithelial cells and medullary thymic epithelial cells (mTECs)--are yet to be

223 g-presenting, autoimmune regulator (AIRE)(+) medullary thymic epithelial cells (mTECs).

224 Autoimmunity is largely prevented by medullary thymic epithelial cells (TECs) through their e

225 (+) T cells preferentially damaged recipient medullary thymic epithelial cells and impaired negative

226 First, we increase medullary thymic epithelial cells by using mice lacking

227 on is enriched, particularly in neonates, in medullary thymic epithelial cells expressing the autoimm

228 lls that colocalize with dendritic cells and medullary thymic epithelial cells in the thymic medulla.

229 r of surface MHC-self peptide complexes from medullary thymic epithelial cells to thymic DC.

230 ng T cells and autoimmune regulator-positive medullary thymic epithelial cells, a key process for cen

231 nity by promoting self-antigen expression in medullary thymic epithelial cells, such that developing

232 Unlike medullary thymic epithelial cells, which express and pre

233 ngly autoimmune regulator-expressing, mature medullary thymic epithelial cells, which play a pivotal

234 a transcription factor which is expressed in medullary thymic epithelial cells.

235 xpression, particularly in keratinocytes and medullary thymic epithelial cells.

236 ssues of patients with APECED, especially in medullary thymic epithelial cells.

237 ssion of MPO mRNA predominantly localized to medullary thymic epithelial cells.

238 cells, cortical thymic epithelial cells, and medullary thymic epithelial cells.

239 n of Bim during early/cortical, but not late/medullary, thymic development controls the agonist selec

240 s, as well as efficient interactions between medullary thymocytes and DCs.

241 ative selection and functional maturation of medullary thymocytes as well as the production of regula

242 ytes exhibit a stochastic migration, whereas medullary thymocytes show confined migratory behavior.

243 were located in the medulla and conditioned medullary thymocytes.

244 Familial medullary thyroid cancer (MTC) and its precursor, C cell

245 Medullary thyroid cancer (MTC) can be caused by germline

246 ity of nonendocrine manifestations preceding medullary thyroid cancer (MTC) for early diagnosis of mu

247 onstrated clinical activity in patients with medullary thyroid cancer (MTC) in phase I.

248 tory differentiated thyroid cancer (DTC) and medullary thyroid cancer (MTC) in the past 10 years.

249 oring the prospects of a cure for persistent medullary thyroid cancer (MTC) stratified by basal calci

250 tumors including small cell lung cancer and medullary thyroid cancer (MTC).

251 ttee on Cancer (AJCC) TNM staging system for medullary thyroid cancer (MTC).

252 -degree relatives with histologically proven medullary thyroid cancer and phaeochromocytoma.

253 Medullary thyroid cancer arises from calcitonin-producin

254 their antiproliferative efficacy against the medullary thyroid cancer cell line TT.

255 The prevention of medullary thyroid cancer in patients with multiple endoc

256 ycaemia, pancreatitis, pancreatic cancer, or medullary thyroid cancer reported between GLP-1 receptor

257 The discovery of a locally advanced medullary thyroid cancer that is not amenable to surgery

258 ment and treatment of patients with advanced medullary thyroid cancer with emphasis on current target

259 s cabozantinib (FDA-approved for progressive medullary thyroid cancer) and PF-04217903 block their ac

260 from a genome-wide association study of non-medullary thyroid cancer, including in total 3,001 patie

261 Knockdown of RET by shRNA in medullary thyroid cancer-derived cells stimulated expres

262 ctivation of ATF4 during the pathogenesis of medullary thyroid cancer.

263 eted medical therapies are now available for medullary thyroid cancer.

264 tient (0.6%) died from incidentally detected medullary thyroid cancer.

265 MET in addition to VEGFR and is approved for medullary thyroid cancer.

266 nts that are approved for differentiated and medullary thyroid cancers have prolonged progression-fre

267 t breast cancers, non-Hodgkin lymphomas, and medullary thyroid cancers represent novel indications fo

268 One of 5 medullary thyroid cancers was positive with the agonist,

269 t frequently mutated malignant subtypes were medullary thyroid carcinoma (9/12, 75%) and PTC (14/30,

270 nase, are associated with the development of medullary thyroid carcinoma (MTC) and pathogenesis of mu

271 Medullary thyroid carcinoma (MTC) is a neuroendocrine ca

272 Medullary thyroid carcinoma (MTC) is a neuroendocrine tu

273 efines these 2 phenotypes is the presence of medullary thyroid carcinoma (MTC).

274 d peptides labeled with (111)In or (131)I in medullary thyroid carcinoma (MTC).

275 (4q32A>C) in a large pedigree displaying non-medullary thyroid carcinoma (NMTC).

276 Medullary thyroid carcinoma accounts for 2% to 5% of thy

277 explain the increased expression of CAIX in medullary thyroid carcinoma and provide a rationale for

278 ilar expression pattern was recapitulated in medullary thyroid carcinoma cells in vivo, consistent wi

279 Medullary thyroid carcinoma is a relatively rare tumor w

280 onal activation of mutated RET gene in human medullary thyroid carcinoma TT cells.

281 ce of acute pancreatitis, pancreatic cancer, medullary thyroid carcinoma, and serious adverse events

282 ogene and virtually all of them will develop medullary thyroid carcinoma.

283 ne in the TT cell line, derived from a human medullary thyroid carcinoma.

284 ic carcinomas, and its expression pattern in medullary thyroid carcinomas suggested contribution of b

285 ressed in various human cancers (e.g., lung, medullary thyroid, pancreatic, colon, and gastrointestin

286 the cortico-medullary boundary to the inner medullary tip.

287 normal sheep, resting levels of cortical and medullary tissue PO2 were 29.5 +/- 4.4 and 29.1 +/- 4.3

288 In one model, approximately one-third of medullary Tph2(+) neurons are silenced by postnatal (P)

289 in the other, approximately three-fourths of medullary Tph2(+) neurons, also with some Tph2(low or ne

290 the mechanisms underlying the modulation of medullary trigeminovascular (Sp5C) neurons have not been

291 ng autophagic defects in epithelial cells of medullary tubules.

292 t majority of thyroid cancers are of the non-medullary type.

293 cysts in the medullary interstitium, loss of medullary vascular bundles, and decreased urine concentr

294 exity of the tidal volume signal (related to medullary ventilatory command), (3) autonomic function,

295 We measured cortical and medullary volumes, perfusion, and RBF using multidetecto

296 ogenous adrenal gland hormones (cortical and medullary) was associated with 3- to 10-fold higher conc

297 ation of the prohemocytes located within the medullary zone and the secondary lobes of the lymph glan

298 The central, medullary zone which contain undifferentiated hematopoie

299 ich normally consists of an immature center (medullary zone) where cells remain undifferentiated, and

300 known to be required to maintain an immature medullary zone.