ライフサイエンスコーパス: meningitidis

1 causing bacteria, Neisseria meningitidis (N. meningitidis).

2 a, Mycobacterium tuberculosis, and Neisseria meningitidis.

3 amic transmission model of group A Neisseria meningitidis.

4 hracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

5 ations frequently fail to identify Neisseria meningitidis.

6 l disease caused by infection with Neisseria meningitidis.

7 explain adolescent/adult colonization by N. meningitidis.

8 ctedly versatile Cas9 protein from Neisseria meningitidis.

9 ance, and virulence in the human pathogen N. meningitidis.

10 mary human meningothelial cells to Neisseria meningitidis.

11 ence factor and vaccine antigen of Neisseria meningitidis.

12 studies on PglL, the O-OTase from Neisseria meningitidis.

13 GI has also been found in some strains of N. meningitidis.

14 tion caused by the human pathogen, Neisseria meningitidis.

15 interactions between 11 Pil proteins from N. meningitidis.

16 d by three DsbA oxidoreductases in Neisseria meningitidis.

17 bA-catalysed oxidative protein folding in N. meningitidis.

18 revalent outer membrane protein in Neisseria meningitidis.

19 land (GGI), as do a few strains of Neisseria meningitidis.

20 or the lack of -35 consensus sequences in N. meningitidis.

21 ar with current recommendations regarding N. meningitidis.

22 s (phasevarions), have been identified in N. meningitidis.

23 inhibit the CRISPR-Cas9 system of Neisseria meningitidis.

24 loyment and assessment of vaccines against N meningitidis.

25 lactamica prevents colonization by Neisseria meningitidis.

26 tive as conventional methods in detecting N. meningitidis (13.2% versus 5.7%; P < 0.0001).

27 species (58.0%), followed by GBS (18.1%), N. meningitidis (13.9%), H. influenzae (6.7%), and L. monoc

28 86) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus pneumoniae

29 regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7-phosphate

30 umoniae (84% of positive CSF isolates) and N.meningitidis (4%).

31 rofiles to characterize strains of Neisseria meningitidis, a major cause of bacterial meningitis worl

32 mbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum was compl

33 and Prevention for the analysis of Neisseria meningitidis and Bordetella bronchiseptica genomes.

34 PorB is located on the surface of N. meningitidis and can be recognized by receptors of the h

35 obactam, cefepime, and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus influenzae

36 shared function of fHbp in N. cinerea and N. meningitidis and cross-reactive responses elicited by Be

37 resulted in loss of functional traits in N. meningitidis and E. coli Our study indicates that the ex

38 We screened 126 isolates of N. meningitidis and found the GGI in 17.5% of strains, with

39 lysialic acid expressed on the surface of N. meningitidis and in the absence of specific antibody ser

40 ies cellular TNF secretion in response to N. meningitidis and may influence susceptibility to meningo

41 and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the two other or

42 cal isolate described here expresses both N. meningitidis and N. gonorrhoeae 16S rRNA genes, as shown

43 the lipooligosaccharide (LOS) from Neisseria meningitidis and N. gonorrhoeae engages the TLR4-MD-2 co

44 ggest that phosphoryl moieties of LA from N. meningitidis and N. gonorrhoeae LOSs play an important r

45 Comparison of PG fragment release from N. meningitidis and N. gonorrhoeae showed that meningococci

46 P activation on diverse strains of Neisseria meningitidis and N. gonorrhoeae specifically using nativ

47 eisseria contains two pathogenic species (N. meningitidis and N. gonorrhoeae) in addition to a number

48 ons that allow for discrimination between N. meningitidis and N. gonorrhoeae.

49 ld include the beta-proteobacteria Neisseria meningitidis and Neisseria gonnorhoeae, in which the cbb

50 against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and improved activ

51 Both Neisseria meningitidis and Neisseria gonorrhoeae recruit the alter

52 NrrF, a trans-acting sRNA in Neisseria meningitidis and Neisseria gonorrhoeae, has been shown i

53 nts is due largely to interaction between N. meningitidis and other members of the upper respiratory

54 umoniae compared with responses to Neisseria meningitidis and that in each case, the bacterial subcap

55 thogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of commensals that

56 tly increases detection of S. pneumoniae, N. meningitidis, and H. influenzae in CSF, and that applica

57 hat were qPCR positive for S. pneumoniae, N. meningitidis, and H. influenzae, only 10 were culture po

58 against Streptococcus pneumoniae, Neisseria meningitidis, and H. influenzae.

59 athogens Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but the mechan

60 f pathogenic Neisseria gonorrheae, Neisseria meningitidis, and Haemophilus influenzae.

61 qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.

62 against Streptococcus pneumoniae, Neisseria meningitidis, and Hemophilus influenzae type b induce fu

63 er jejuni, Haemophilus influenzae, Neisseria meningitidis, and Pasteurella multocida.

64 f conjugate vaccines against H influenzae, N meningitidis, and S pneumoniae in England.

65 ction with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae causes substa

66 Neisseria gonorrhoeae and Neisseria meningitidis are closely related organisms that cause th

67 r detecting pharyngeal carriage of Neisseria meningitidis are complex.

68 tal colonization and urethritis caused by N. meningitidis are possible across a range of meningococca

69 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation due to alter

70 nses of human tonsil-derived DC to Neisseria meningitidis as a model organism.

71 face outer membrane protein 85 (OMP85) of N. meningitidis as an immobilized selective layer.

72 ate complement-mediated killing of Neisseria meningitidis as they enter the bloodstream from the orop

73 This was particularly relevant for N. meningitidis, as only 1/13 cases was culture positive.

74 a bacterial homologue of ASBT from Neisseria meningitidis (ASBT(NM)) at 2.2 A.

75 tructure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was reported recent

76 isease-associated and carried isolates of N. meningitidis at the level of individual nucleotide varia

77 ation to respond to an outbreak of Neisseria meningitidis B at a U.S. university.

78 cases of meningococcal disease caused by N. meningitidis B were reported among vaccinated students.

79 Neisseria meningitidis binds the complement downregulating protein

80 This isolate was identified as N. meningitidis by biochemical identification methods but g

81 ulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep understan

82 ive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immunization wit

83 Neisseria meningitidis can be transmitted via asymptomatic nasopha

84 heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or Gram-positi

85 At baseline, natural N. meningitidis carriage in the control group was 22.4% (36

86 ures grown overnight doubled the yield of N. meningitidis carriage isolates compared with conventiona

87 show that these proteins bind directly to N. meningitidis Cas9 (NmeCas9) and can be used as potent in

88 Cas9 orthologs (including Neisseria meningitidis Cas9 [NmeCas9]) have also been adopted for

89 Neisseria meningitidis causes 500 000 cases of septicemia and meni

90 Neisseria meningitidis causes bacterial meningitis and septicemia.

91 The pathogen Neisseria meningitidis causes disease amongst infants and adolesce

92 Neisseria meningitidis changes its capsular phenotype through caps

93 Among 25 serogroup B Neisseria meningitidis clinical isolates, we identified four (16%)

94 several lines of supporting evidence that N. meningitidis colonization is correlated with propionic a

95 distinct protospacer adjacent motif, the N. meningitidis CRISPR-Cas machinery increases the sequence

96 real-time PCR assays for detection of (i) N. meningitidis ctrA, H. influenzae hpd, and S. pneumoniae

97 PCR assays have been developed to detect N. meningitidis ctrA, H. influenzae hpd, and S. pneumoniae

98 osphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.

99 ards a promising application in the field of meningitidis diagnosis.

100 rategies for outbreaks of invasive Neisseria meningitidis disease are informed by serogroup assays th

101 sed biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configuration.

102 or exhibits a linear response towards target meningitidis DNA over the concentration range from 10 to

103 idence of laboratory-confirmed serogroup A N meningitidis dropped significantly to 0.01 per 100 000 i

104 Neisseria meningitidis employs redundant heme acquisition mechanis

105 Neisseria meningitidis encodes up to five TpsA proteins that are s

106 c nature and the high diversity of Neisseria meningitidis, epidemiological surveillance incorporating

107 nts with Streptococcus pneumoniae, Neisseria meningitidis, Escherichia coli, and Pseudomonas aerugino

108 raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) antigen wer

109 Recombinant native N. meningitidis FBA was purified and used in a coupled enzy

110 a are at odds with this proposal and that N. meningitidis fits the criteria that we have proposed for

111 In the human pathogen Neisseria meningitidis for example, 23 proteins are dedicated to T

112 keleton linker, were more pronounced when N. meningitidis formed larger colonies on HBMEC under physi

113 to hospital and identification of Neisseria meningitidis from a sterile site.

114 (fHbp), a virulence factor which protects N. meningitidis from innate immunity by binding the human c

115 ty in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical proteins.

116 e class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, thereby auton

117 itis incidence and carriage due to Neisseria meningitidis group A (MenA).

118 s launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-tetanus toxoid

119 ning the N-propionyl derivative of Neisseria meningitidis group B (MenB) capsular polysaccharide (NPr

120 sular polysaccharides from E. coli K1 and N. meningitidis group B and the heparosan-like capsular pol

121 ng protein, are essential for survival of N. meningitidis group B strain H44/76 in normal human serum

122 is (Haemophilus influenzae type b, Neisseria meningitidis group C and seven serotypes of Streptococcu

123 Vaccines against Neisseria meningitidis group C are based on its alpha-2,9-linked p

124 s that can be protective against MenB and N. meningitidis group C strains.

125 athogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Pasteurella mu

126 Neisseria meningitidis, Haemophilus influenzae, and Streptococcus

127 ingitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus

128 ections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsut

129 Group A Neisseria meningitidis has been a major cause of bacterial meningi

130 N. meningitidis has been considered a paradigmatic case of

131 Neisseria meningitidis has several strategies to evade complement-

132 eria, including the human pathogen Neisseria meningitidis, have evolved means to preferentially take

133 protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the bacterium

134 e, GBS, Listeria monocytogenes, or Neisseria meningitidis in cerebrospinal fluid or other normally st

135 ach for fast instrument-free diagnosis of N. meningitidis in resource-limited settings.

136 k so far and shows the continued threat of N meningitidis in sub-Saharan Africa.

137 p genes provides a metabolic advantage to N. meningitidis in the adult oral cavity, which is rich in

138 vel aspects of the methylcitrate cycle in N. meningitidis include a propionate kinase which was purif

139 vity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bactericidal anti

140 The incidence of Neisseria meningitidis infection decreased from 0.721 per 100 000

141 understanding the epidemiology of Neisseria meningitidis infection.

142 Neisseria meningitidis inhibits the alternative pathway (AP) of co

143 ake via GltT-GltM plays multiple roles in N. meningitidis internalization into HBMEC.

144 We previously reported that Neisseria meningitidis internalization into human brain microvasoc

145 Neisseria meningitidis is a commensal microbe that colonizes the h

146 Neisseria meningitidis is a commensal of humans that can colonize

147 Neisseria meningitidis is a frequent colonizer of the human nasoph

148 Neisseria meningitidis is a human commensal that can also cause li

149 Neisseria meningitidis is a human pathogen causing bacterial menin

150 Neisseria meningitidis is a human-specific bacterium that varies i

151 Neisseria meningitidis is a human-specific pathogen and leading ca

152 GNA2091 of Neisseria meningitidis is a lipoprotein of unknown function that i

153 Neisseria meningitidis is a major cause of bacterial meningitis an

154 Neisseria meningitidis is a major cause of bacterial meningitis wo

155 Neisseria meningitidis is a major global pathogen causing invasive

156 The capsular polysaccharide surrounding N. meningitidis is a major virulence factor.

157 Neisseria meningitidis is a strict human pathogen that closely int

158 The limit of detection of N. meningitidis is about 3 copies per LAMP zone within 45 m

159 that the closely related bacterium Neisseria meningitidis is also polyploid, while the commensal orga

160 Neisseria meningitidis is an encapsulated pathogen, and antibodies

161 Neisseria meningitidis is an important cause of invasive bacterial

162 Neisseria meningitidis is an important human pathogen that is capa

163 Neisseria meningitidis is an obligate human nasopharyngeal commens

164 However, most of the time N. meningitidis is carried as a commensal not associated wi

165 or factor H (fH) to the surface of Neisseria meningitidis is critical for evasion of innate host defe

166 lthough the opportunistic pathogen Neisseria meningitidis is enriched for colonization in the throat,

167 Although Neisseria meningitidis is naturally competent and porB genetic mos

168 Neisseria meningitidis is one of the main agents of bacterial meni

169 stems of Neisseria gonorrhoeae and Neisseria meningitidis is presented.

170 An apparently rare Neisseria meningitidis isolate containing one copy of a Neisseria

171 The N. meningitidis isolate described must have obtained N. gon

172 Probe Aptima assays cross-react with this N. meningitidis isolate.

173 re, we analyzed the genomes of 39 diverse N. meningitidis isolates associated with urethritis, collec

174 Almost all invasive Neisseria meningitidis isolates express capsular polysaccharide.

175 report a 1.44 A crystal structure of the N. meningitidis major pilin PilE and a approximately 6 A cr

176 isease-associated and carried isolates of N. meningitidis may provide critical insight into mechanism

177 N. meningitidis MC58 NMB0419 encodes a Sel1-like repeat (SL

178 h glyco-conjugate capsular group C Neisseria meningitidis (Men C) vaccines in infancy.

179 vel protection against serogroup A Neisseria meningitidis (MenA) are unknown.

180 Serogroup B Neisseria meningitidis (MenB) is a major cause of severe sepsis an

181 an effective vaccine, serogroup B Neisseria meningitidis (MenB) remains a major cause of invasive di

182 Neisseria meningitidis (meningococcus) is a symbiont of the human

183 entify the recognition site for three key N. meningitidis methyltransferases: ModA11 (exemplified by

184 y, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, recently su

185 lipopolysaccharide-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most recently i

186 neumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacterium tuber

187 on pathogen detected (n = 17) followed by N. meningitidis (n = 13).

188 Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae

189 main meningitis-causing bacteria, Neisseria meningitidis (N. meningitidis).

190 Last, we revisit the species status of N. meningitidis, N. gonorrheae, and N. lactamica in the lig

191 within the obligate human pathogen Neisseria meningitidis, NApe and NExo, are important for survival

192 The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisseria lactam

193 Neisseria meningitidis (Nm) clonal complex 11 (cc11) lineage is a

194 an important survival strategy of Neisseria meningitidis (Nm) during colonization and infection.

195 Neisseria meningitidis (Nm) is a Gram-negative diplococcus that no

196 The human pathogen Neisseria meningitidis (Nm) is a leading cause of bacterial mening

197 Neisseria meningitidis (Nm) is a leading cause of bacterial mening

198 The human pathogen Neisseria meningitidis (Nm) is known to possess several mechanisms

199 Neisseria meningitidis (Nm) strains infecting these patients are p

200 due to a large epidemic of group A Neisseria meningitidis (NmA) meningitis.

201 jugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first introduced in

202 The Cas9 RNA-guided endonuclease from N. meningitidis (NmCas9) recognizes a 5'-NNNNGATT-3' protos

203 e HO from the pathogenic bacterium Neisseria meningitidis (NmHO) possesses a crystallographically und

204 enase (HO), from the pathogenic bacterium N. meningitidis(NmHO), which secures host iron, shares many

205 e HO from the pathogenic bacterium Neisseria meningitidis, NmHO, possesses C-terminal His207, Arg208,

206 Serogroup X N. meningitidis (NmX) dominated in both vaccinated and unva

207 nactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhibited the

208 amica is not associated with disease, but N. meningitidis occasionally invades the host, causing meni

209 of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of adaptive immu

210 lonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acquisition o

211 properdin do not bind directly to either N. meningitidis or N. gonorrhoeae but play a crucial role i

212 th S. pneumoniae, S. agalactiae, E. coli, N. meningitidis, or H. influenzae in combination with cefot

213 mmatory responses of lower magnitude than N. meningitidis organisms and N. meningitidis PorB (publish

214 Carriage for Neisseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.0

215 Neisseria meningitidis phasevarions regulate genes including virul

216 nitude than N. meningitidis organisms and N. meningitidis PorB (published elsewhere as Nme PorB).

217 en switched between N. lactamica PorB and N. meningitidis PorB, we identified residues in loop 5 and

218 eriments demonstrate that the E. coli and N. meningitidis protein homologs are functionally conserved

219 erial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Escherichia col

220 urified polysialyltransferase from Neisseria meningitidis (PST(Nm)) to the extracellular environment.

221 We found that N. meningitidis recycles PG fragments via the selective per

222 of comparative sequence analyses against N. meningitidis reference genome sequences of known serogro

223 st time, that PorB2-expressing strains of N. meningitidis regulate the AP of baby rabbits and rats.

224 The decreased release of PG monomers by N. meningitidis relative to N. gonorrhoeae is partly due to

225 us diseases over the past century, Neisseria meningitidis remains a major causative agent of meningit

226 ion of a few DNA copies per LAMP zone for N. meningitidis, S. pneumoniae and Hib were achieved within

227 alciparum infections, and virulent Neisseria meningitidis samples.

228 te sequences were obtained; 92 (76%) were N. meningitidis sequences, and 29 (24%) were N. gonorrhoeae

229 l correlates of protection against Neisseria meningitidis serogroup A (NmA) in Burkina Faso before th

230 To combat Neisseria meningitidis serogroup A epidemics in the meningitis bel

231 An affordable, highly immunogenic Neisseria meningitidis serogroup A meningococcal conjugate vaccine

232 Neisseria meningitidis serogroup B (MnB) is a leading cause of bac

233 recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend partly on dis

234 neumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, and P. bergh

235 luding 1147 (71.5%) that were positive for N meningitidis serogroup C (NmC).

236 Because the N. meningitidis serogroup L capsule polymer consists of a t

237 yze the synthesis of the complex trimeric N. meningitidis serogroup L capsule polymer repeating unit.

238 of the pathophysiologically less relevant N. meningitidis serogroup L, is one of the smallest known S

239 South Africa, and Israel caused by Neisseria meningitidis serogroup Y (NmY) was greater than the worl

240 ingococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Europe, especi

241 i.p.-injected intact, heat-killed Neisseria meningitidis, serogroup C (MenC), a gram-negative bacter

242 ombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA) were chara

243 Neisseria meningitidis serogroups A and X are among the leading ca

244 consists of polyhexosamine phosphates in N. meningitidis serogroups A and X.

245 group-specific genes in the cap locus for N. meningitidis serogroups A, B, C, W135, X, and Y.

246 and S. pneumoniae lytA (NHS assay); (ii) N. meningitidis serogroups A, W135, and X (AWX assay); and

247 ups A, W135, and X (AWX assay); and (iii) N. meningitidis serogroups B, C, and Y (BCY assay).

248 nes are available to protect against four N. meningitidis serogroups, there is currently no commercia

249 ce determinants of disease causing Neisseria meningitidis species are their extracellular polysacchar

250 ynF showed 100% specificity for detecting N. meningitidis species, with high sensitivity (serogroup B

251 e resolution the primary transcriptome of N. meningitidis strain 8013.

252 N. meningitidis strain 8047 was subjected to serial passage

253 ive lipooligosaccharide (LOS) from Neisseria meningitidis strain 89I was analyzed by matrix-assisted

254 ect of purified PorB in vitro, a chimeric N. meningitidis strain expressing N. lactamica PorB induces

255 Neisseria meningitidis strain H44/76 was modified by expression of

256 All four opa genes from N. meningitidis strain H44/76 were sequentially disrupted t

257 the transcriptome of adherent serogroup B N. meningitidis strain MC58 was determined at intervals dur

258 much lower than that within the wild-type N. meningitidis strain only upon HBMEC infection and was co

259 ratio of species-specific sequences, the N. meningitidis strain seems to have replaced one of its fo

260 tion against antigenically diverse Neisseria meningitidis strains and to compare this protection to a

261 to construct all possible combinations of N. meningitidis strains deficient in one, two, three, or al

262 allelic patterns in urethritis-associated N. meningitidis strains may reflect genetic diversity in th

263 Laboratory data on 4,735 Neisseria meningitidis strains was collected and reported by the N

264 s-like and distinguishing them from other N. meningitidis strains.

265 henotypes and hence invasive potential of N. meningitidis strains.

266 Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae (S. pneumoniae),

267 nfluenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus ag

268 E6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray crystallogra

269 N. meningitidis subverts immune responses by hijacking a ho

270 s how the important human pathogen Neisseria meningitidis subverts immune responses by mimicking the

271 rleukin 8 (IL-8) secretion than wild-type N. meningitidis, suggesting a role for PorB in induction of

272 In all infected mice, N. meningitidis targeted the human vasculature, leading to

273 These data demonstrate that N. meningitidis targets human endothelial cells in vivo and

274 uenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine (Hib-MenC-

275 genomic island (the prp gene cluster) in N. meningitidis that enables this species to utilize propio

276 study of the AP endonuclease from Neisseria meningitidis that has allowed us to capture structural i

277 land is absent from the close relative of N. meningitidis, the commensal Neisseria lactamica, which c

278 In E. coli and N. meningitidis, the predominant lipid is a lysophosphatidy

279 on has been studied extensively in Neisseria meningitidis, the specific subset of genes that CrgA tar

280 ely related opportunistic pathogen Neisseria meningitidis, there is an absence of adaptive cell-media

281 complement attack achieved by circulating N. meningitidis therefore depends on the relative levels of

282 ilable vaccine for serogroup B strains of N. meningitidis, this kind capsule-switching event could ha

283 ty of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends on surviv

284 ease-associated and 4 carried isolates of N. meningitidis to search for SNPs that show mutually exclu

285 Neisseria meningitidis, typically a resident of the oro- or nasoph

286 damage, are not required for survival of N. meningitidis under oxidative stress.

287 Neisseria meningitidis use Type IV pili (T4P) to adhere to endothe

288 Neisseria meningitidis utilizes capsular polysaccharide, lipooligo

289 r the direct quantification of two Neisseria meningitidis vaccine antigens, in mono- and multivalent

290 Carriage of N. meningitidis was investigated by using three different m

291 ture of an ADEP-ClpP complex derived from N. meningitidis was solved.

292 thogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation of compleme

293 a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeting of an e

294 c variation (Av) of two strains of Neisseria meningitidis were determined using an unbiased DNA seque

295 95.7% and 85.7%, and L. monocytogenes and N. meningitidis were not observed in the study.

296 number of microorganisms including Neisseria meningitidis, which can lead to permanent neurological d

297 ted genomes from the 4 carried genomes of N. meningitidis, which is far more than can be expected by

298 Hyperinvasive lineages of Neisseria meningitidis, which persist despite extensive horizontal

299 hexaacylated endotoxin (LOS) from Neisseria meningitidis with [(13)C]acetate allowed the use of NMR

300 s and 25 isolates from carriers of Neisseria meningitidis without disease.