ライフサイエンスコーパス: miRNA

1 miRNA profiling in the human brain has revealed miR-132

2 miRNA targeting principles are consistent with observati

3 miRNAs are evolutionarily conserved small noncoding RNAs

4 P value <0.01) were identified, including 13 miRNAs between the NC and CM, 17 between the NC and CS,

5 Within the 16 miRNA signature for the normal SC niche, we found that m

6 Combined expression of the 36 miRNAs was used to create a novel miRNA-based risk class

7 tectures, in the embryonic function of lsy-6 miRNA.

8 We identified 63 miRNAs deregulated in a statistically significant way.

9 th mirSTP, we not only predicted TSSs for 72 miRNAs, but also identified 12 primary miRNAs with signi

10 -irradiation and identified cel-mir-237 as a miRNA which when deleted caused animals to be more resis

11 ort a causal link between dysregulation of a miRNA target and SCZ-related deficits and provide key in

12 We found that changing the 3'UTR of a miRNA-targeted reporter modulates translational repressi

13 e at diagnosis or ethnic variation affecting miRNA epigenetic regulation or sequence of miRNA precurs

14 ng factor influencing multiple steps in AGO2-miRNA-mediated mRNA decay.

15 Unexpectedly, we observe that >40% of all miRNAs tested significantly affect SR time, revealing pe

16 well-known competition and cooperation among miRNAs when attempts to discover target sites.

17 However, ancient miRNAs and their binding sites in target genes are conse

18 obally interrogating miRNA gene function and miRNA-based therapeutic intervention.

19 Global gene and miRNA expression profiles were also determined using pai

20 mRNA and miRNA expression profile frequently performed the best,

21 genome-wide sequencing and analyzed mRNA and miRNA expression, DNA copy number, and DNA methylation i

22 nk between decay of cleaved target mRNAs and miRNA stability in RISC.

23 pliced sequences for splicing signatures and miRNA events.

24 el profiling of methylome, transcriptome and miRNA using Next Generation Sequencing.

25 the specific relationship between Notch1 and miRNAs during muscle development has not been establishe

26 exRNA is enriched in small ncRNAs, such as miRNAs in exosomes, and precisely processed tRNA and Y R

27 ly when incorporated with biomarkers such as miRNAs, quantitative imaging features, and clinical data

28 ts were used to test the association between miRNA and brain lesions (T2 hyperintense lesion volume [

29 study was to provide an interactome between miRNAs and their targetome in Chagas heart disease by in

30 s study shows that modular synergism between miRNAs and neuronal subtype-specific transcription facto

31 However, target mRNAs recognized by both miRNA and AUF1 are less abundant upon AUF1 overexpressio

32 Notably, both miRNAs synergized with the frontline colorectal cancer c

33 ed collateral vessel growth is controlled by miRNAs, among which miR-352 is a novel candidate that ne

34 logy to the regulation of gene expression by miRNAs, we propose that the main function of m(6)A is po

35 nclude the added dimensions of modulation by miRNAs and long-noncoding RNA (lncRNA).

36 nscriptional, oncogenic network regulated by miRNAs.

37 ence of alternative splicing, which bypasses miRNA-mediated down-regulation under drought stress.

38 and an R package, to easily infer candidate miRNA-mRNA target interactions that could be functional

39 We tested several candidate miRNA-deletion mutants post gamma-irradiation and identi

40 Four candidate miRNAs were identified during the initial screening, and

41 e circulating levels of 7 selected candidate miRNAs in 14 LTxR with AR, 7 with BOS, and compared them

42 thod to identify canonical and non-canonical miRNA-binding sites from peaks identified by Ago2 Cross-

43 sed miRNA-like RNAs different from canonical miRNAs from cassava miRNA precursors detected under four

44 egulation and most of the well-known cardiac miRNAs were up-regulated at later stages for suppression

45 different from canonical miRNAs from cassava miRNA precursors detected under four distinct chilling c

46 r alcohol dependence and identify the causal miRNAs for alcohol-dependency in patients which were val

47 Next generation sequencing of Kupffer cell miRNA identified miRNA 181b-3p (miR181b-3p) as sensitive

48 possibility that the pattern of circulating miRNAs may identify recurrence prior to radiological det

49 e implement our framework on a comprehensive miRNA expression data set for alcohol dependence and ide

50 ich demonstrated the dynamic and conditional miRNA regulation during cancer progression.

51 Furthermore, we identified conserved miRNA-targets regulations in the control of double flowe

52 xpression, we used Cre recombinase-dependent miRNA tagging and affinity purification in mice.

53 ect to C, with a large number of deregulated miRNAs shared by both neoplastic lesions.

54 In conclusion, blood-derived miRNAs might be promising biomarkers for SZ diagnosis.

55 s shows that PDAC and IPMN have differential miRNA profiles with respect to C, with a large number of

56 ethod to rank, so as to predict, the disease miRNA candidates.

57 rget mRNAs indicate that UPF1 can dissociate miRNAs from their mRNA targets, making the miRNAs suscep

58 ssion profiling tools to define the distinct miRNA expression of MNs, which is likely to enrich futur

59 we identified 14 significantly dysregulated miRNAs.

60 ausing alterations after JQ1 treatment; each miRNA was further validated through BRD4 binding to its

61 More than half of the edited miRNAs showed increased stability, 72.7% of which had De

62 To determine MN-enriched miRNA expression, we used Cre recombinase-dependent miRN

63 Here we show that the brain-enriched miRNAs miR-9/9( *) and miR-124 (miR-9/9( *)-124) trigger

64 We performed a meta-analysis to examine miRNA diagnostic value for SZ and further validated the

65 s validation cohort, plasma derived exosomal miRNA was isolated from 50 early-stage colon cancer pati

66 l decrease in levels of circulating exosomal miRNAs.

67 To assess the value of exosome miRNAs as biomarkers for T1DM, miRNA expression in plasm

68 eport the first characterization of exosomes miRNAs from human synovial fluid.

69 Overall, 330 unique differentially expressed miRNAs (DEMs) were identified in the entire TAA-treatmen

70 Twenty-seven differentially expressed miRNAs (fold change >1.5; P value <0.01) were identified

71 quick search of the differentially expressed miRNAs in certain cancer types.

72 Out of 7 differentially expressed miRNAs, spearman correlation testing highlighted correla

73 rential recruitment of a subset of expressed miRNAs and isoforms of miRNAs (isomiRs) to the miRISC in

74 n binding sites for miR-35 and miR-58 family miRNAs within the egl-1 3' untranslated region (UTR), wh

75 o The microglial exosomes were collected for miRNA microarray analysis, which showed that the express

76 it (25 fM, 0.25 attomol in 10muL sample) for miRNA-21 without any amplification step, a complete disc

77 n could remove more than 40% of the bias for miRNAs.

78 Here we develop an alternative inhibitor for miRNAs, termed 'small RNA zipper'.

79 We found that functional miRNAs are degraded in human cells by the endonuclease T

80 Furthermore, miRNAs that predicted conversion status were found to do

81 ous cancer-driving pathways including global miRNA downregulation, the miR-140-5p/Pin1 axis may play

82 This glomerulocapillary miRNA signature associated with HLA class I-DSA could im

83 oding transcripts, little is known about how miRNAs themselves are degraded.

84 mportant advance in our comprehension of how miRNAs function in the development of higher organisms.

85 sequencing of Kupffer cell miRNA identified miRNA 181b-3p (miR181b-3p) as sensitive to both ethanol

86 ver the past decade, studies have identified miRNAs as playing a role in nearly all aspects of AML di

87 Targets for the identified miRNAs were predicted using five different databases; ge

88 The aim of this study was to identify miRNAs associated with hepatitis B e antigen (HBeAg) sta

89 Here, we aimed to identify miRNAs that are modulated by glucose in mouse pancreatic

90 exposure study, we detected a few important miRNAs that regulated a large number of mRNAs in the con

91 Changes in miRNA activity are common in cancer, but how these chang

92 Here, we also show the changes in miRNA composition of the miRISC in primary human chondro

93 pes resembling those of mutants defective in miRNA biogenesis and activity.

94 iPSC-EVs were enriched in miRNAs and proteins with proangiogenic and cytoprotectiv

95 vidence that small noncoding RNAs, including miRNAs, can be targeted by the CRISPR/Cas9 system despit

96 udies have reported key roles for individual miRNA editing events, but a comprehensive picture of miR

97 ion, the caste-associated role of individual miRNAs is not conserved.

98 The biogenesis of these quiescence-induced miRNAs is independent of Exportin-5 and depends instead

99 accurate TSS prediction for human intergenic miRNAs at a high resolution.

100 feasible strategy for globally interrogating miRNA gene function and miRNA-based therapeutic interven

101 our analysis identifies candidate novel key miRNAs regulating networks of significance for normal PC

102 d as putative target mimics of cassava known miRNAs.

103 KSHV miRNAs suppressed STAT3 and STAT5 activation and inhibit

104 Together, we show that KSHV miRNAs suppress a network of targets associated with STA

105 We hypothesized that KSHV miRNAs target human GADD45B to protect cells from conseq

106 To link miRNA identities with downstream targets, transcriptome-

107 Results from experiments using AGO2-loaded miRNAs in duplex with target mRNAs indicate that UPF1 ca

108 Many miRNAs selected by our feature selection algorithm had s

109 We show that the expression of many miRNAs is dramatically regulated during functional matur

110 per-enhancers drive the biogenesis of master miRNAs crucial for cell identity by enhancing both trans

111 ibed to result in increased levels of mature miRNA.

112 Here, we demonstrate that mature miRNA-22 is more abundant in muscle from male mice relat

113 irable biases, it is advantageous to measure miRNA in a crude cell lysate.

114 The hypothesis that microvesicle-mediated miRNA transfer converts noncancer stem cells into cancer

115 Studies have showed that abnormal microRNA (miRNA) expression can affect CRC pathogenesis and develo

116 prehensively profile pediatric AML microRNA (miRNA) samples to identify dysregulated genes and assess

117 s of the neuron, for both mRNA and microRNA (miRNA).

118 ction of a breast cancer biomarker microRNA (miRNA), mir-21 was achieved via electropolymerized polyp

119 The effects of microRNA (miRNA) regulation on the genetic programs underlying beh

120 a sensing scheme for detection of microRNA (miRNA) using electrocatalytic amplification (ECA).

121 We performed microRNA (miRNA) profiling in 318 serum samples from 69 liver tran

122 se-tissue-specific knockout of the microRNA (miRNA)-processing enzyme Dicer (ADicerKO), as well as hu

123 MicroRNAs (miRNA) in cerebrospinal fluid were amplified by quantita

124 MicroRNAs (miRNAs) and heterogeneous nuclear ribonucleoproteins (hn

125 MicroRNAs (miRNAs) are a group of small RNAs that control gene expr

126 MicroRNAs (miRNAs) are important post-transcriptional regulators of

127 MicroRNAs (miRNAs) are involved in the regulation of a number of pr

128 MicroRNAs (miRNAs) are known to play important roles in various cel

129 MicroRNAs (miRNAs) are short, noncoding RNAs involved in the regula

130 MicroRNAs (miRNAs) are small non-coding RNA species that have been

131 MicroRNAs (miRNAs) are small, non-coding RNAs that play critical ro

132 MicroRNAs (miRNAs) exert powerful effects on immunity through coord

133 MicroRNAs (miRNAs) have been known to affect various biological pro

134 MicroRNAs (miRNAs) impinge on the translation and stability of thei

135 MicroRNAs (miRNAs) loss-of-function phenotypes are mainly induced b

136 MicroRNAs (miRNAs) recently have been established as key regulators

137 MicroRNAs (miRNAs) regulate gene expression through interactions wi

138 We found that 15 microRNAs (miRNAs) are regulated by progesterone via PR-A, but not

139 Currently, microRNAs (miRNAs) involved in host resistance to APEC are unknown.

140 estigated the involvement of host microRNAs (miRNAs) in maintaining the viability of C. trachomatis-i

141 ine genome-wide screens for human microRNAs (miRNAs) directly regulating cell-cycle proteins.

142 To identify microRNAs (miRNAs) that were increased in the quadriceps of patient

143 Invertebrate microRNAs (miRNAs) can suppress developmental variability that is c

144 his study aimed to identify novel microRNAs (miRNAs) involved in the regulation of decidual gene expr

145 s have evaluated the potential of microRNAs (miRNAs) as biomarkers for cardiometabolic disease.

146 Some microRNAs (miRNAs) are known to suppress breast cancer.

147 rough down-regulation of specific microRNAs (miRNAs).

148 e cell origin, including specific microRNAs (miRNAs).

149 Recent studies have shown that microRNAs (miRNAs) play a pivotal role in vascular development, hom

150 In this study, we aimed to use microRNAs (miRNAs) - which are critical regulators of signaling cas

151 While microRNAs (miRNAs) regulate the vast majority of protein-encoding t

152 Bacterial infection modulates miRNA expression to subvert any innate immune response.

153 e validated increased expression of multiple miRNAs, including miR126, following eCig exposure.

154 developed a sensitive and accurate multiplex miRNA profiling method using modified isothermal EXPAR c

155 ingle-strand (ss) RNAs in vitro; but neither miRNAs nor miRNA*s in vivo.

156 ysomes correlated with the production of new miRNAs that target these mRNAs at sites distal to the st

157 d (ss) RNAs in vitro; but neither miRNAs nor miRNA*s in vivo.

158 urface markers, and gene expression, but not miRNA profiles, were associated with depleted serum cult

159 Here we present a novel miRNA prediction algorithm, miRCat2.

160 of the 36 miRNAs was used to create a novel miRNA-based risk classification scheme (AMLmiR36).

161 Taken together, our data support a novel miRNA-mediated pathway downstream of MeCP2 that influenc

162 t implications regarding the design of novel miRNA-based therapeutic strategies against angiogenesis.

163 -throughput sequencing revealed that a 22 nt miRNA with 3G ('22-3G') comprised <63% of total miR-122

164 tochondrial role in Ld-induced alteration of miRNA activity and stability is further corroborated by

165 se chain reaction (qRT-PCR) amplification of miRNA extracted directly from 500 microL of serum had li

166 It utilizes an sxRNA switch for detection of miRNA-mRNA interactions combined with a fluorophore-bind

167 exposed groups, suggesting the disruption of miRNA activity due to chemical exposure.

168 e, revealing pervasive behavioral effects of miRNA regulation in the early larva.

169 Our work thus reveals pervasive effects of miRNA regulation on a complex innate behavior in Drosoph

170 Together our results reveal modulation of miRNA-mediated repression by characteristics and feature

171 Here, we provide an overview of miRNA biogenesis and function in regulating key genes an

172 iting events, but a comprehensive picture of miRNA editing in human cancers remains largely unexplore

173 e filtered out to improve the reliability of miRNA detection.

174 Results of miRNA profiling demonstrated that miR-30a was markedly d

175 enetic investigations to explore the role of miRNA in early stage BC in young women, including ethnic

176 In this short review, the roles of miRNA in epidermal development, psoriasis, cutaneous squ

177 g miRNA epigenetic regulation or sequence of miRNA precursors.

178 pting two independent and essential steps of miRNA biogenesis.

179 The presence of cleaved transcripts of miRNA-targeted ARFs in C. roseus cells was confirmed by

180 The data also confirmed translation of miRNA target mimics and lncRNAs that produce trans-actin

181 provide a greater level of understanding of miRNA biology and critical insights into the many transl

182 rovides evidence for better understanding of miRNA regulatory roles in the process of fiber developme

183 Ago1 is the predominant carrier of miRNAs in Anopheles gambiae.

184 uced silencing complex (RISC) is composed of miRNAs and AGO proteins.

185 It is clear that the aberrant expression of miRNAs promotes tumor initiation and progression, is lin

186 y a novel cross talk between a key family of miRNAs and proapoptotic Bcl-2 proteins in human pancreat

187 Little is known about the functions of miRNAs in IL-22/IL-17-producing T cells.

188 The identification of miRNAs discriminating CNs from neoplastic nodules may ha

189 n, our findings underscore the importance of miRNAs in posttranscriptional control of the biosynthesi

190 of AUF1 abolishes the global interaction of miRNAs and AGO2.

191 r of broadly comprehensive investigations of miRNAs involved in this process have been conducted.

192 a subset of expressed miRNAs and isoforms of miRNAs (isomiRs) to the miRISC in response to IL-1beta,

193 The sensors can determine relative levels of miRNAs in total RNA extracts with sensitivity similar to

194 so used to estimate the copy number range of miRNAs in total RNA extracts.

195 t-transcriptional regulation and the role of miRNAs in Drosophila circadian rhythms.

196 To clarify the role of miRNAs in RPE cells, we used two different mature RPE ce

197 Different accumulation trends of miRNAs, observed upon drought and in different genotypes

198 dysregulated genes and assess the utility of miRNAs for improved outcome prediction.

199 as the antagonizing effect of RBP binding on miRNA repression.

200 can be modulated by the expression of other miRNA targets acting as competing endogenous RNAs (ceRNA

201 n (self-righting, SR), suggesting that other miRNAs might also be involved in behavioral control.

202 qRT-PCR of pathogen miRNAs isolated from extracellular vesicles in sera from

203 We performed miRNA and mRNA profiling using high throughput stem-loop

204 In the periodontium, miRNAs play key roles in development and periodontal hom

205 small-RNA species of phased and half-phased miRNA-like RNAs different from canonical miRNAs from cas

206 target genes of those phased and half-phased miRNA-like RNAs function in process of cell growth metab

207 Among 38 plasma miRNAs that were elevated following ischemia, eight were

208 nosines mis-paired with cytosines on the pre-miRNA structure.

209 facilitate Drosha/DGCR8 recruitment and pri-miRNA processing to boost cell-specific miRNA production

210 uper-enhancer-directed cotranscriptional pri-miRNA processing.

211 and Drosha/DGCR8-mediated primary miRNA (pri-miRNA) processing.

212 n SR protein family member that promotes pri-miRNA processing.

213 However, rules that distinguish pri-miRNAs from other hairpin-containing transcripts in the

214 ith a large subset of primary microRNAs (pri-miRNAs) in mammalian cells.

215 nscription and Drosha/DGCR8-mediated primary miRNA (pri-miRNA) processing.

216 or 72 miRNAs, but also identified 12 primary miRNAs with significant RNA polymerase pausing alteratio

217 were found to be enriched with proangiogenic miRNAs such as miR-126-3p.

218 of Karolinska Endarterectomies), we profiled miRNA expression in patients with stable versus unstable

219 gests a Ca(2+)-dependent process to regulate miRNA activity in neurons in response to the induction o

220 2 as one of the most severely down-regulated miRNAs at the intermediate and late Braak stages of Alzh

221 an accompanying reduction in Lin28-regulated miRNAs, downstream of brain-derived neurotrophic factor

222 Our results suggest that, in C. reinhardtii, miRNAs might be subject to relatively fast evolution and

223 First, we compiled CRC-related miRNAs, CRC-related genes, and human TFs from multiple d

224 2-5p and miR-142-3p, as inflammation-related miRNAs with potential roles in wound healing.

225 pposite expression profiles than the related miRNAs.

226 miR-500a-5p as an oxidative stress response miRNA whose activity may define breast cancer progressio

227 et (649) of novel, evolutionarily restricted miRNAs.

228 Taken together, the results reveal miRNA signatures in PA useful to identify potential biom

229 , cell proliferation and disease, micro-RNA (miRNA) biology is of great importance and a potential th

230 critical region 8 (Dgcr8), thus removing RPE miRNA regulatory activity in mice by disrupting two inde

231 ranging from a reduction in the schistosomal miRNA level to full recovery from disease.

232 hich is required for the sorting of selected miRNAs into exosomes, plays a role in the sorting of hig

233 e used 304 high-quality microRNA sequencing (miRNA-seq) datasets from NCBI-SRA and calculated express

234 use models of colitis, we identified a serum miRNA signature that indicated the development of coliti

235 We quantified serum miRNA at clinically indicated and/or protocol biopsy eve

236 Fifty-seven miRNAs were detected as modulated by cytokines.

237 Children who outgrew CMA showed miRNA-193a-5p level, and its related targets expression,

238 This is an elegant example of how a single miRNA can control an entire process by acting on a cruci

239 n Drosophila shows that mutation of a single miRNA locus (miR-iab4/iab8) affects the capacity of the

240 Six miRNAs were significantly elevated and 12 diminished in

241 At least six miRNAs show cycling expression levels within the pigment

242 pri-miRNA processing to boost cell-specific miRNA production.

243 ther, our findings identify context-specific miRNA-regulated checkpoints that control myelinogenesis

244 tiple isomiRs and tRFs arising from specific miRNA loci (e.g., miR-200c, miR-21, the miR-17/92 cluste

245 22, an abundant and conserved liver-specific miRNA, regulates hepatic metabolism and functions as a t

246 We propose that exosomal muscle-specific miRNAs may be useful molecular biomarkers for monitoring

247 in vitro, implying roles for these specific miRNAs.

248 ing and provides strategies for subnanomolar miRNA quantification with bilayer-embedded porins.

249 r the expressions of these tumor suppressive miRNAs translate to patient survival were not investigat

250 ue of exosome miRNAs as biomarkers for T1DM, miRNA expression in plasma-derived exosomes was measured

251 d to assess the oncogenic role of the target miRNA.

252 o2 have a single GW182-binding site and that miRNA binding increases hAgo's affinity to GW182.

253 We found that miRNA-122 (miR-122) is down-regulated in liver biopsy sp

254 Recent research has shown that miRNA-based regulation of the tumor suppressor gene PTEN

255 ence of the miRNA sensors, demonstrated that miRNAs induce translational repression depending on thei

256 Moreover, we found little evidence that miRNAs previously shown to be associated with caste in A

257 We propose that miRNAs compensate for lineage-specific differences in eg

258 Recently, emerging evidence suggest that miRNAs have crucial roles in control of EMT and EMT-asso

259 t into the regulation of VM and suggest that miRNAs repressed during EMT, in addition to suppressing

260 ate behavior in Drosophila and suggests that miRNAs may be core components of the genetic programs un

261 hat WIG1 governs the miRNA-dependent and the miRNA-independent recruitment of AGO2 to lower the stabi

262 her, our data indicate that WIG1 governs the miRNA-dependent and the miRNA-independent recruitment of

263 Moreover, the miRNA signature identified in mice predicted ulcerative

264 GFbeta signalling controls expression of the miRNA genes comprising an erlotinib response signature i

265 that ALG-5 defines a distinct branch of the miRNA pathway affecting the expression of genes involved

266 SND1, a subunit of the miRNA regulatory complex RISC, has been implicated as an

267 he decay kinetics of the fluorescence of the miRNA sensors, demonstrated that miRNAs induce translati

268 e identify two bulge-depleted regions on the miRNA stem, located approximately 16-21 nt and approxima

269 4 circulating exosomal sequences within the miRNA category were differentially expressed in RRMS pat

270 In conclusion, our work identified the miRNAs specifically released by different human CD4(+) T

271 miR-124-3p increased most apparently in the miRNAs.

272 e miRNAs from their mRNA targets, making the miRNAs susceptible to TumiD.

273 Many of the miRNAs were regulated in a tissue/organ-specific manner.

274 normal rats segregated on the basis of their miRNA expression profiles, and a similar finding was obs

275 particles as delivery vectors of therapeutic miRNA capable of simultaneously targeting dysregulated p

276 These miRNAs were found to regulate the expression of the proa

277 tential pathways that are regulated by these miRNAs.

278 c hepatitis B (CHB) , and to assess if these miRNAs are actively secreted by hepatoma cells.

279 HCV subverts the antiviral actions of these miRNAs by dampening their expression in cell culture mod

280 In vitro secretion of these miRNAs by peripheral blood mononuclear cells was also si

281 Together, our results indicate that these miRNAs are consistent markers of treatment response and

282 hed, highlighting the possibility that these miRNAs could modulate key renal tubular functions in a p

283 d to elucidate the mechanisms by which these miRNAs modify cervical epithelial cell function.

284 This miRNA-based regulatory network enables a robust yet time

285 titutive ablation of all six members of this miRNA family causes derepression of multiple cell cycle-

286 Of this miRNA panel, miR-26a was the most effective in reducing

287 ntial deleterious oncogenic activity of this miRNA.

288 hat ilp7 and ilp8 are direct targets of this miRNA.

289 Fourth, sequences aligned to miRNAs with mismatches are remapped to a reference genom

290 lls demonstrating its potential for tracking miRNA expression and localization in vivo.

291 Here, we demonstrated that these two miRNAs are prominently expressed in wound-infiltrated ne

292 These two miRNAs were also found dysregulated in childhood and adu

293 /212 family being one of the top upregulated miRNAs.

294 ects was hybridized onto Nanostring human v2 miRNA microarray array and expression data were analyzed

295 We further validated miRNA data using AD postmortem brains, amyloid precursor

296 ystems approach of integrating a genome-wide miRNA screen with patient-derived phospho-proteomic sign

297 Functional analysis with miRNA mimics in cellular models confirmed these findings

298 Co-expression network analysis of mRNA with miRNA, lncRNA and virus genes identified key elements wi

299 ncoding RNAs and likely do not interact with miRNAs.

300 ancestral wheat to form evolutionarily young miRNA genes that act to repress the glaucous trait.