ライフサイエンスコーパス: mycoplasmal

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1 hod targeting part of the strongly conserved mycoplasmal 16S RNA genes, Mycoplasma pneumoniae was fou

2 (vi) The nonenteric temperate phages with mycoplasmal and mycobacterial hosts are relatively close

3 use strains that differ in susceptibility to mycoplasmal arthritis.

4 surface proteins and the R-M activity of the mycoplasmal cell population.

5 Often, high-frequency changes in the mycoplasmal chromosome are associated with antigenic and

6 The location of Tn916 insertion sites in the mycoplasmal chromosome was random, suggesting that Tn916

7 odeficient (SCID) mice resulted in increased mycoplasmal colonization of spleens and lesions in extra

8 ew outbreak, even at a time of the year when mycoplasmal conjunctivitis is low in free-living birds.

9 Seasonal variation in mycoplasmal conjunctivitis prevalence in house finches i

10 fection with an inflammatory ocular disease, Mycoplasmal conjunctivitis, caused by Mycoplasma gallise

11 That finding was remarkable because mycoplasmal disease is rarely fatal in animals.

12 te, for mycoplasmacidal activity and virtual mycoplasmal elimination.

13 e clostridial sialidase are conserved in the mycoplasmal gene, but the leader sequence necessary for

14 hromosomal form and loss of phiMFV1 from the mycoplasmal genome were documented in a series of clonal

15 Mycoplasmal genomes subjected to this process and transp

16 ts in minimal (approximately 0.6- to 1.4-Mb) mycoplasmal genomes suggest their key role in the adapta

17 Mycoplasmal growth was further enhanced in BacT/ALERT PF

18 cted iNOS(+/+) mice, whereas neither CYP nor mycoplasmal infection altered NOx in iNOS(-/-) mice.

19 ntributors to nitrotyrosine formation during mycoplasmal infection and that treatment with CYP decrea

20 SP-A(-/-) mice were as susceptible to mycoplasmal infection as highly susceptible C3H/He mice,

21 in defense against systemic dissemination of mycoplasmal infection, but cellular immune responses may

22 tory cells in nitrotyrosine formation during mycoplasmal infection.

23 L mice and the susceptibility of C3H mice to mycoplasmal infection.

24 Mycoplasmal infections apparently affected the fidelity

25 smal defense and suggest that differences in mycoplasmal killing by AMs may explain the resistance of

26 yme-linked immunosorbent assay and inhibited mycoplasmal killing by iNOS(+/+) mice in vivo.

27 Mycoplasmal killing was abrogated by 1,000 units/ml of c

28 decreases NO* production by AMs and inhibits mycoplasmal killing.

29 esponses may be important in exacerbation of mycoplasmal lung disease.

30 oper anchoring of cytadhesin proteins in the mycoplasmal membrane at an attachment organelle through

31 Heat-killed mycoplasmas or mycoplasmal membrane preparations alone could support co

32 on assays demonstrated homology to the human mycoplasmal P30 and P32 cytadhesins.

33 -infected pigs with minimal to nondetectable mycoplasmal pneumonia lesions manifested significantly i

34 f serologically confirmed community-acquired mycoplasmal pneumonia.

35 system was constructed to identify potential mycoplasmal promoter-containing regulatory sequences in

36 e results indicate that E. coli can identify mycoplasmal promoters which have transcriptional element

37 VsaA R40 is the first mycoplasmal protein shown to be associated with resistan

38 e presence of TGA codons, the translation of mycoplasmal proteins terminates prematurely when cloned

39 sma pulmonis is a model for studying chronic mycoplasmal respiratory disease.

40 generated products by primer extension with mycoplasmal RNA.

41 A new mechanism expanding mycoplasmal surface diversity is described.

42 s examined for antibody accessibility on the mycoplasmal surface.

43 Unlike the activity of other mycoplasmal TLR2 agonists such as macrophage-activating

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