ライフサイエンスコーパス: myeloblastosis

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1 Avian myeloblastosis and Moloney murine leukemia virus RTs als

2 Here, we show that BES1 interacts with myeloblastosis family transcription factor-like 2 (MYBL2

3 The myeloblastosis (MYB)-type transcription factors PHOSPHAT

4 Of 19 positional candidate genes, one-the myeloblastosis oncogene (Myb)-stood out.

5 ween GTP-binding elongation factor HBS1L and myeloblastosis oncogene MYB on chromosome 6q that are as

6 Tf encodes a myeloblastosis oncoprotein (MYB)-like transcription fact

7 ow that the Drosophila homologue of the Myb (Myeloblastosis) oncoprotein family is tightly associated

8 on assays while no specific binding to avian myeloblastosis or Moloney murine leukemia RTs was detect

9 FLD (Flowering locus D), GI (GIGANTEA), Myb (Myeloblastosis), SFH3 (SEC14-like 3), bZIP (basic-leucin

10 nt1 as a closely linked gene encoding a R2R3 myeloblastosis transcription factor.

11 tric gliomas in the related gene v-myb avian myeloblastosis viral oncogene homolog (MYB) on 6q23.3.

12 V-myb myeloblastosis viral oncogene homolog (MYB) proteins bel

13 ry gland origin, has a signature v-myb avian myeloblastosis viral oncogene homolog-nuclear factor I/B

14 l stabilities of wild-type recombinant avian myeloblastosis virus (AMV) and Moloney murine leukemia v

15 construction experiments with purified avian myeloblastosis virus (AMV) IN and retrovirus-like donor

16 The v-myb oncogene of the avian myeloblastosis virus (AMV) is unique among known oncogen

17 nuclear protein v-Myb, encoded by the avian myeloblastosis virus (AMV), can induce acute monoblastic

18 the well characterized retroviral RT, avian myeloblastosis virus (AMV).

19 ilarly inhibits the activities of both avian myeloblastosis virus and human immunodeficiency virus ty

20 e and full-site reactions catalyzed by avian myeloblastosis virus and human immunodeficiency virus ty

21 Two acute transforming retroviruses, avian myeloblastosis virus and the E26 leukemia virus, transdu

22 The v-myb oncogene of the avian myeloblastosis virus has led to the discovery of a large

23 ed attachment (att) site sequences and avian myeloblastosis virus IN (4 nm) were as competent for ful

24 ular insights into the organization of avian myeloblastosis virus IN at the viral DNA ends were gaine

25 specific activities of virion-derived avian myeloblastosis virus integrase and bacterial recombinant

26 Avian myeloblastosis virus integrase can efficiently insert th

27 The v-Myb oncoprotein encoded by Avian Myeloblastosis Virus is highly oncogenic, induces leukem

28 tem, extension of the primer by either avian myeloblastosis virus reverse transcriptase (AMV-RT) or h

29 HIV-1 or avian myeloblastosis virus reverse transcriptase (RT) were cap

30 here the deleted cDNA was generated by avian myeloblastosis virus reverse transcriptase from a synthe

31 , DNA polymerase I Klenow fragment and avian myeloblastosis virus reverse transcriptase incorporated

32 The shortened cDNA was generated by avian myeloblastosis virus reverse transcriptase, but not by t

33 v-Myb, the transforming protein of avian myeloblastosis virus, causes acute myeloid leukemia in c

34 ologue of the transforming gene of the avian myeloblastosis virus, is preferentially expressed in all

35 Thus, the v-myb gene encoded by the Avian Myeloblastosis Virus, is truncated at both the 5' and 3'

36 t other reverse transcriptases tested (avian myeloblastosis virus, Moloney murine leukemia virus, and

37 roducts with the ability to combat the avian myeloblastosis virus.

38 2), Moloney murine leukemia virus, and avian myeloblastosis virus.

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