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1  acute lymphoblastic leukemia, and 1 chronic myelocytic leukemia.
2 hematopoietic cell transplantation and acute myelocytic leukemia.
3 ment of acute childhood leukemia and chronic myelocytic leukemia.
4 ignificant promise in the treatment of acute myelocytic leukemia.
5 ions (DLI) in patients with relapsed chronic myelocytic leukemia after allogeneic bone marrow transpl
6 locytic leukemia (APL) was noted among acute myelocytic leukemia (AML) cases at the Los Angeles Count
7 g ATRA/ATO-based strategies to non-APL acute myelocytic leukemia (AML) is currently lacking.
8 ients who were recently diagnosed with acute myelocytic leukemia (AML) were assigned randomly to stan
9 ) have been evaluated in patients with acute myelocytic leukemia (AML), both as potential priming age
10 elocytic leukemia and in some cases of acute myelocytic leukemia and acute lymphocytic leukemia, the
11 blood cells derived from patients with acute myelocytic leukemia and chronic myelocytic leukemia, whi
12 er survival in groups of patients with acute myelocytic leukemia and epithelial ovarian cancer who ha
13 n successfully administered to patients with myelocytic leukemia and has produced therapeutic effects
14                     In most cases of chronic myelocytic leukemia and in some cases of acute myelocyti
15 a RA-dependent manner in the NB-4, acute pro-myelocytic leukemia, and the MCF-7, breast carcinoma, ce
16                          Most cases of acute myelocytic leukemia are homozygous for the allele with t
17              Previous work showed that acute myelocytic leukemia blasts accumulate less long chain po
18       The HLA class I- and class II-negative myelocytic leukemia cell line K562 was transfected with
19                       These included chronic myelocytic leukemia (CML) blast crisis K562 and HL-60/Bc
20                                      Chronic myelocytic leukemia (CML) is a common neoplasm of hemato
21 ped progressive blast crisis (BC) of chronic myelocytic leukemia (CML) while receiving therapy with i
22                     In patients with chronic myelocytic leukemia (CML), the breakpoint cluster region
23 mportant roles in the progression of chronic myelocytic leukemia (CML), we performed allelotype analy
24 clin A1 overexpression was observed in acute myelocytic leukemias, especially those that were at the
25 s with acute myelocytic leukemia and chronic myelocytic leukemia, which express BCR-Abl, demonstrate
26 toire in four patients with relapsed chronic myelocytic leukemia who achieved a complete remission af

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