ライフサイエンスコーパス: n-3 fatty acid

1 were ameliorated by the presence of dietary n-3 fatty acid.

2 concentrations of long-chain polyunsaturated n-3 fatty acids.

3 studies of AMD risk reduction by long-chain n-3 fatty acids.

4 script levels in the mice fed a diet of high n-3 fatty acids.

5 igh dietary AA but mitigated by high dietary n-3 fatty acids.

6 e not correlated with dietary consumption of n-3 fatty acids.

7 infarction, and stroke in subjects consuming n-3 fatty acids.

8 rapid increase in erythrocyte DHA and total n-3 fatty acids.

9 ifferences based on metabolism of long-chain n-3 fatty acids.

10 uming higher quantities of n-6 or long-chain n-3 fatty acids.

11 se genes are responsible for many effects of n-3 fatty acids.

12 anic is not a guarantee of higher long chain n-3 fatty acids.

13 among women with high intakes of long-chain n-3 fatty acids.

14 ed fish, correlated with plasma phospholipid n-3 fatty acids.

15 the origin of specific volatiles from parent n-3 fatty acids.

16 ing optimal amounts of dietary n-9, n-6, and n-3 fatty acids.

17 may be mediated by an enzymatic affinity for n-3 fatty acids.

18 n of saltwater fish and intake of long-chain n-3 fatty acids.

19 received placebo (adjusted hazard ratio with n-3 fatty acids, 0.97; 95% confidence interval, 0.88 to

20 Patients were randomly assigned to n-3 fatty acids (1 g daily) or placebo (olive oil).

21 Both groups took n-3 fatty acids (2.4 g/d) for 4 wk with the addition of

22 after we adjusted further for marine-derived n-3 fatty acids (22 mum, 95% CI: -1 to 46 mum, p = 0.065

23 eas consumption of the vegetable oil-derived n-3 fatty acid a-linolenic acid is not as effective.

24 were similar for estimated intake of marine n-3 fatty acids: a 1 g/day higher intake was associated

25 domains) in relation to baseline intakes of n-3 fatty acids (absolute and relative to n-6 fatty acid

26 Recent studies have suggested that n-3 fatty acids, abundant in fish oil, protect against h

27 However, whether N-3 fatty acids, active ingredients of fish oil, have di

28 in Barnes maze performance compared with the n-3 fatty acid-adequate rats during the initial training

29 3 fatty acid-deficient rats in comparison to n-3 fatty acid-adequate rats.

30 The mechanisms whereby n-3 fatty acids affect gene expression are complex and i

31 However, the molecular mechanisms by which n-3 fatty acids affect tumor growth remain unknown.

32 re studied in chicken patties, enriched with n-3 fatty acids, after 8days of storage at 4 degrees C,

33 s have evaluated the association between the n-3 fatty acid alpha-linolenic acid (ALA) and the incide

34 Because conversion of the plant-derived n-3 fatty acid alpha-linolenic acid (ALA) to EPA and DHA

35 Intake of the plant-derived omega-3 (n-3) fatty acid alpha-linolenic acid (ALA, 18:3; n-3) ma

36 The use of n-3 fatty acids also had no significant effect on the ra

37 nts enrolled, 6244 were randomly assigned to n-3 fatty acids and 6269 to placebo.

38 %), of whom 733 of 6239 (11.7%) had received n-3 fatty acids and 745 of 6266 (11.9%) had received pla

39 d control subjects in response to intakes of n-3 fatty acids and aspirin.

40 ociation between the ratio of dietary n-6 to n-3 fatty acids and bone mineral density (BMD) in 1532 c

41 d the relation between the dietary intake of n-3 fatty acids and chronic obstructive pulmonary diseas

42 Modulation of specific genes by n-3 fatty acids and cross-talk between these genes are r

43 However, the role of long-chain n-3 fatty acids and fish intake in the development of T2

44 We further analyzed the effects of n-6 and n-3 fatty acids and found that the PUFA-APOA5 interactio

45 ineered fat-1 mice, which can convert n-6 to n-3 fatty acids and have a balanced ratio of n-6 to n-3

46 showed no evidence that supplementation with n-3 fatty acids and instructions to reduce arachidonic a

47 evidenced by an increase in the ratio of n-6/n-3 fatty acids and levels of 4-hydroxynonenal, a marker

48 The results of the comparison between n-3 fatty acids and placebo are reported here.

49 sible to make firm recommendations regarding n-3 fatty acids and the prevention of dementia and AMD.

50 ions between fish, shellfish, and long-chain n-3 fatty acids and the risk of T2D in a middle-aged Chi

51 The level of n-3 fatty acids and their metabolite prostaglandin E(3)

52 ever, the relation between dietary intake of n-3 fatty acids and ventricular arrhythmias has not been

53 and spring proved to have a high content of n-3 fatty acids and vitamin D, and hence they serve as a

54 tive was to determine whether walnuts (plant n-3 fatty acid) and fatty fish (marine n-3 fatty acid) h

55 34)), levels of polyunsaturated fatty acids, n-3 fatty acids, and docosahexaoenic acid relative to to

56 ific food components, such as soluble fiber, n-3 fatty acids, and fructose, are discussed.

57 nolenic acid (ALA), a plant-derived omega-3 (n-3) fatty acid, and age-related macular degeneration (A

58 nolenic acid (ALA), a plant-derived omega-3 (n-3) fatty acid, and cardiovascular disease (CVD) risk h

59 uding saturated fatty acids (SFAs), omega-3 (n-3) fatty acids, and refined sugar, with hippocampal-de

60 rticularly lutein and zeaxanthin, as well as n-3 fatty acids, appears beneficial for AMD and possibly

61 alkenyl chain and that of a sn-2-esterified n-3 fatty acid are additive.

62 We sought to examine whether marine-derived n-3 fatty acids are associated with less atherosclerosis

63 n-3 Fatty acids are known to reduce many risk factors as

64 n-3 Fatty acids are purported to have health effects in

65 n-6 fatty acids and concomitant decrease in n-3 fatty acids are thought to be a major driver of the

66 Omega-3 (n-3) fatty acids are essential for proper neuronal funct

67 ioperative immunonutrition (IMN) enriched in n-3 fatty acids, arginine, and nucleotides may improve p

68 ificantly decreased among patients receiving n-3 fatty acids, as compared with those receiving placeb

69 ple, dietary intake of fish and fish-derived n-3 fatty acids, as well as plasma biomarkers of fish in

70 Marine-derived n-3 fatty acids at low levels are cardioprotective throu

71 involved in lipid oxidation, with long-chain n-3 fatty acids being potent activators of PPARA.

72 indicate that syndecan 1 is up-regulated by n-3 fatty acids by a transcriptional pathway involving P

73 ed fatty acids in optimizing the benefits of n-3 fatty acids (C18-C22) on cardiovascular health.

74 genous fatty acids, with a preference toward n-3 fatty acids (C18:3 and C22:6).

75 iet that is high in omega-3 polyunsaturated (n-3) fatty acids can slow disease progression in patient

76 ga-3 fatty acids (omega-3-FAs; also known as n-3 fatty acids) can exert potent anti-inflammatory effe

77 In contrast to their agonistic properties, n-3 fatty acids competitively inhibit the responses of v

78 ffects of varying n-9, n-6, and longer-chain n-3 fatty acid composition on markers of coronary heart

79 ctive was to examine the association between n-3 fatty acid consumption and ventricular ectopy among

80 tigate whether a diet enriched with fish and n-3 fatty acid consumption are associated with changes i

81 Critical time periods for n-3 fatty acid consumption may differ by sex.

82 udies have evaluated the effects of fish and n-3 fatty acid consumption on fatal coronary heart disea

83 se findings extend existing evidence linking n-3 fatty acid consumption to a reduced risk of ventricu

84 ariable adjustment, neither fish nor dietary n-3 fatty acid consumption was significantly associated

85 im was to study the associations of fish and n-3 fatty acid consumption with risk of total major chro

86 problems in evaluating the health effects of n-3 fatty acid consumption, highlighted challenges to re

87 Increased omega-3 (n-3) fatty acid consumption is reported to benefit patie

88 sions based on plant oils differing in their n-3 fatty acid content were compared.

89 An increase in plasma n-3 fatty acid content, particularly eicosapentaenoic ac

90 Cooking treatments had little effect on n-3 fatty acid content; however, fried treatments genera

91 ivo reduction of DHA by dietary depletion of n-3 fatty acids decreased hippocampal PS and increased n

92 ute to neurological deficits associated with n-3 fatty acid deficiency and support protective effects

93 tudy, the authors demonstrate that rats with n-3 fatty acid deficiency display spatial learning defic

94 Results showed that dietary n-3 fatty acid deficiency elevates the vulnerability to

95 The effect of long-term n-3 fatty acid deficiency on rod and cone phototransduct

96 upplementation, combined with the effects of n-3 fatty acid deficiency, on acute blue-light photochem

97 sapentaenoic acid, which replaces DHA during n-3 fatty acid deficiency, was less effective in accumul

98 The n-3 fatty acid-deficient rats had reduced tissue levels

99 s of brain docosahexaenoic acid (DHA) in the n-3 fatty acid-deficient rats in comparison to n-3 fatty

100 In the reversal learning task, the n-3 fatty acid-deficient rats showed a profound deficit

101 The long-chain omega-3 (n-3) fatty acids derived from fish, eicosapentaenoic aci

102 achieved through overexpressing a C. elegans n-3 fatty acid desaturase gene, mfat-1.

103 norhabditis elegans gene, fat-1, encoding an n-3 fatty acid desaturase.

104 antiinflammatory, and vasodilatory omega-3 (n-3) fatty acids (DHA and EPA) are significantly reduced

105 The addition of aspirin to n-3 fatty acids did not alter any SPMs in either group.

106 ovascular risk factors, daily treatment with n-3 fatty acids did not reduce cardiovascular mortality

107 Daily supplementation with 1 g of n-3 fatty acids did not reduce the rate of cardiovascula

108 2(-/-)/Cx3cr1(-/-) mice that ingested a high n-3 fatty acid diet showed a slower progression of retin

109 Thus, n-3 fatty acids differentially regulate TRPV1 and this f

110 enter addresses is whether botanical n-6 and n-3 fatty acids directly block recognized biochemical pa

111 Together, these results suggest that N-3 fatty acids directly inhibit vascular calcification,

112 esult in altered brain concentrations of the n-3 fatty acid docosahexaenoic acid (DHA) during the per

113 of the transcription factor PPARgamma by the n-3 fatty acid docosahexaenoic acid (DHA) is implicated

114 Our studies showed that the n-3 fatty acid docosahexaenoic acid (DHA) up-regulated s

115 ation of TLR2 with TLR1 was inhibited by the n-3 fatty acid docosahexaenoic acid.

116 ns unresolved, possibly due to low levels of n-3 fatty acids docosahexaenoic acid (DHA) and eicosapen

117 The omega-3 (n-3) fatty acids docosahexaenoic acid and eicosapentaeno

118 One n-3 fatty acid, docosahexaenoic acid, is an important co

119 Dams were deprived of n-3 fatty acids during pregnancy and lactation, and thei

120 r diets with different levels of the various n-3 fatty acids during pregnancy and lactation, and thei

121 d interventional, assessing the influence of n-3 fatty acids during pregnancy or the early postpartum

122 itro experiments showed that addition of the n-3 fatty acid eicosapentaenoic acid or PGE(3) inhibited

123 at consuming fish or fish oil containing the n-3 fatty acids eicosapentaenoic acid (EPA) and docosahe

124 rvings/wk), especially species higher in the n-3 fatty acids eicosapentaenoic acid (EPA) and docosahe

125 attributed to the marine-derived long-chain n-3 fatty acids eicosapentaenoic acid (EPA) and docosahe

126 correlated with the RBC long-chain omega-3 (n-3) fatty acids eicosapentaenoic acid (EPA) and docosah

127 A greater intake of n-3 fatty acids (eicosapentaenoic acid + docosahexaenoic

128 flaxseed addition in the chicken feed (i.e., n-3 fatty acid enrichment), and for the different packag

129 ns, a significant increase in serum omega-3 (n-3) fatty acids (EPA and DHA), and a decrease in serum

130 Total n-3 fatty acids, EPA, and the SI for palmitic to palmito

131 iew current evidence on the relation between n-3 fatty acids, especially docosahexaenoic acid (DHA),

132 n-3 Fatty acids exert important effects on eicosanoid me

133 We questioned if acute administration of n-3 fatty acids (FA) carried in n-3 rich triglyceride (T

134 molecular studies relating to the effects of n-3 fatty acids (FA) on inhibiting atherogenesis.

135 h low dietary omega-6 (n-6) or high omega-3 (n-3) fatty acid (FA) content resulted in reduced body fa

136 The omega-6 (n-6) to omega-3 (n-3) fatty acid (FA) ratio (n-6:n-3 ratio) was previousl

137 and Treatment of Coronary Heart Disease with n-3 Fatty Acids," faculty who presented at the conferenc

138 Studies on the relation between dietary n-3 fatty acids (FAs) and cardiovascular disease vary in

139 n-3 Fatty acids (FAs) have been shown to be beneficial f

140 n-3 Fatty acids (FAs) when used in doses of 3-4 g/d eico

141 The role of omega-3 (n-3) fatty acids (FAs) in the development of type 2 diab

142 the relation of plasma and dietary omega-3 (n-3) fatty acids (FAs) with heart failure (HF) risk have

143 Nevertheless, the most consistent effect of n-3 fatty acids feeding in rats is to decrease lipogenes

144 then recommendations for specific intakes of n-3 fatty acids for different conditions relating to car

145 Alpha-linolenic acid, an intermediate-chain n-3 fatty acid found primarily in plants, may decrease t

146 Intake of long-chain n-3 fatty acids found in fish is low in many countries w

147 ps: n-6 fatty acids found in plant seeds and n-3 fatty acids found in marine vertebrates.

148 Intake of long-chain n-3 fatty acids from fish was not associated with depres

149 fat-1 transgenic pigs produce high levels of n-3 fatty acids from n-6 analogs, and their tissues have

150 Twice-daily enteral supplementation of n-3 fatty acids, gamma-linolenic acid, and antioxidants

151 Twice-daily enteral supplementation of n-3 fatty acids, gamma-linolenic acid, and antioxidants

152 ion of retinal lesions compared with the low n-3 fatty acids group.

153 Long chain n-3 fatty acids had considerably lower concentrations in

154 Some mice that were given high levels of n-3 fatty acids had lesion reversion.

155 Whereas dietary intake of long-chain n-3 fatty acids has been associated with risk of nonfata

156 Very high levels of marine-derived n-3 fatty acids have antiatherogenic properties that are

157 Most of the health benefits observed for n-3 fatty acids have been attributed to the marine-deriv

158 Although n-3 fatty acids have effects that could favorably influe

159 n-3 Fatty acids have important visual, mental, and cardi

160 Omega-3 (n-3) fatty acids have antiinflammatory and antiprolifera

161 plant n-3 fatty acid) and fatty fish (marine n-3 fatty acid) have similar effects on serum lipid mark

162 t Greenland Eskimos, who have a diet high in n-3 fatty acids, have a lower mortality from coronary he

163 W n-6) or a diet with a relative increase in n-3 fatty acid (HIGH n-3) compared with the CTR from pos

164 al cord blood samples (n = 101), we measured n-3 fatty acids, IgE concentrations, and immunologic res

165 ammation in animal models, we tested whether n-3 fatty acids impact SPM profiles in patients with CAD

166 ctable way to increase a specific long-chain n-3 fatty acid in plasma, tissues, or human milk is to s

167 Docosahexaenoic acid (DHA) is the principal n-3 fatty acid in tissues and is particularly abundant i

168 Limited storage of the n-3 fatty acids in adipose tissue suggests that a contin

169 imed to investigate the effect of long-chain n-3 fatty acids in blood on the risk of nonfatal MI.

170 The long-chain n-3 fatty acids in fish have been demonstrated to have a

171 ypothesized to reduce the health benefits of n-3 fatty acids in fish.

172 d, and because of limited interconversion of n-3 fatty acids in humans, ALA supplementation does not

173 What are the roles of n-3 fatty acids in hypertriglyceridemia, in the metaboli

174 rized databases were searched for studies of n-3 fatty acids in immune-mediated diseases from 1966 to

175 The adverse effects of n-3 fatty acids in men merit confirmation.

176 lt in appreciable accumulation of long-chain n-3 fatty acids in plasma.

177 What are the roles of n-3 fatty acids in primary versus secondary prevention o

178 tribute to the chemopreventive properties of n-3 fatty acids in prostate cancer.

179 odents caused by an inadequate amount of the n-3 fatty acids in the diet.

180 vitro studies indicates a beneficial role of n-3 fatty acids in the prevention and management of card

181 ty acids and have a balanced ratio of n-6 to n-3 fatty acids in their tissues and organs independent

182 Triglyceride synthesis could be reduced by n-3 fatty acids in three general ways: reduced substrate

183 d activation and provides specificity toward n-3 fatty acids in which the corresponding n-3 acyl-CoAs

184 acid (DHA, 22:6n-3), the principal omega-3 (n-3) fatty acid in brain gray matter, positively regulat

185 moderators of clinical response to omega-3 (n-3) fatty acids in subjects with major depressive disor

186 sociation of fish, shellfish, and long-chain n-3 fatty acid (in g/d) with risk of T2D.

187 suggest that the use of fish oil (containing n-3 fatty acids) in a variety of disorders such as cysti

188 Dietary recommendations have been made for n-3 fatty acids, including alpha-linolenic acid (ALA), e

189 Dietary n-3 fatty acids, independent of the reciprocal changes i

190 wide diversity of dietary intakes of n-6 and n-3 fatty acids influences tissue compositions of n-3 lo

191 n-3 fatty acids inhibit neuronal excitability and reduce

192 Results showed that N-3 fatty acids inhibited alkaline phosphatase (ALP) act

193 fatty acid intake (in whites) and long-chain n-3 fatty acid intake (in African Americans) such that p

194 Of these, four had low n-3 fatty acid intake and four had adequate intakes.

195 imilar association was found between seafood n-3 fatty acid intake and prostate cancer mortality (RR(

196 Our results support a protective effects of n-3 fatty acid intake and the n-7 saturation index again

197 d 0.025) remained negatively associated with n-3 fatty acid intake in women and men, and large HDLs (

198 in both) remained positively associated with n-3 fatty acid intake in women and men.

199 Significant genotype-by-long-chain n-3 fatty acid intake interactions were observed only in

200 Dietary long-chain n-3 fatty acid intake was also inversely associated with

201 Control subjects had typical L, Z, and n-3 fatty acid intake.

202 e was to determine whether habitual omega-3 (n-3) fatty acid intake is associated with the rate of in

203 n-3 Fatty acid intakes (both in terms of absolute amount

204 t is reasonable to hypothesize that maternal n-3 fatty acid intakes might have significant effects on

205 Fish, shellfish, and long-chain n-3 fatty acid intakes were inversely associated with T2

206 We examined the relation of fish and seafood n-3 fatty acid intakes with prostate cancer incidence an

207 PUFA intake, specifically n-6 and long-chain n-3 fatty acid intakes, and multiple lipid measures in t

208 ch as carbohydrates and protein, leucine and n-3 fatty acid intakes, such as of EPA, may be important

209 Here we show that n-3 fatty acids interact directly with TRPV1, an ion cha

210 e trafficking of stable isotopically labeled n-3 fatty acids into phosphatidylcholine and phosphatidy

211 Docosahexaenoic acid (DHA), an n-3 fatty acid, is the major polyunsaturate in rod outer

212 The role of long-chain omega-3 (n-3) fatty acids (LCFAs) in the development of T2DM rema

213 blue light-induced damage, whereas adequate n-3 fatty acid levels reduced the damage in the parafove

214 the human situation in which 1.2% energy as n-3 fatty acids lowers serum triglyceride levels.

215 entre randomised trial of low-dose fish oil (n-3 fatty acids <1080 mg/day) in drug resistant epilepsy

216 hythmias by showing that a greater intake of n-3 fatty acids may be associated with low ventricular e

217 ndicate that the cardioprotective effects of n-3 fatty acids may be attributed to a number of distinc

218 d and SCD supports the hypothesis that these n-3 fatty acids may have antiarrhythmic properties.

219 The use of n-3 fatty acids may prevent cardiovascular events in pat

220 Fish and seafood n-3 fatty acids may prevent or delay the progression of

221 A high dietary intake of n-3 fatty acids may protect cigarette smokers against CO

222 nthophylls lutein (L) and zeaxanthin (Z) and n-3 fatty acids may reduce this damage and lower the ris

223 Although dietary omega-3 (n-3) fatty acids may confer some cardiovascular benefits

224 T1A, was strongly associated with markers of n-3 fatty acid metabolism, including degree of unsaturat

225 Therefore, we hypothesized that these n-3 fatty acids might prevent potentially fatal ventricu

226 cids on clinical outcomes in IBD and whether n-3 fatty acids modify the effects of or need for treatm

227 her concentrations of both n-6 (omega-6) and n-3 fatty acids of higher elongation and desaturation le

228 Erythrocyte n-3 fatty acids of marine origin and trans fatty acid co

229 ar consensus exists regarding the effects of n-3 fatty acids on any of these outcomes.

230 were identified that assessed the effects of n-3 fatty acids on any single outcome of clinical, endos

231 Our results support a protective effect of n-3 fatty acids on breast cancer risk and provide additi

232 ence to determine estimates of the effect of n-3 fatty acids on clinical outcomes in IBD and whether

233 ent to draw conclusions about the effects of n-3 fatty acids on clinical, endoscopic, or histologic s

234 ntrolled trials that assessed the effects of n-3 fatty acids on clinical, sigmoidoscopic, or histolog

235 spective, double-blind trials, the effect of n-3 fatty acids on depression during pregnancy or the ea

236 low-dose supplementation with B vitamins or n-3 fatty acids on depressive symptoms in cardiovascular

237 ules of either an ethyl ester concentrate of n-3 fatty acids or olive oil daily for 12 mo.

238 east 900 mg (90% or more) of ethyl esters of n-3 fatty acids or placebo daily and to receive either i

239 per liter]) or standard care and to receive n-3 fatty acids or placebo with the use of a 2-by-2 fact

240 -long-chain polyunsaturated fatty acids, the n-3 fatty acids (or omega-3), have distinct and importan

241 DHA (600 mg) in a 2:1 ratio; B vitamins and n-3 fatty acids; or a placebo.

242 risk factors and RBC saturated and omega-3 (n-3) fatty acids, ORs for CAD associated with each SD in

243 es have a significantly reduced ratio of n-6/n-3 fatty acids (P < 0.001).

244 n-3 Fatty acids, particularly docosahexaenoic acid (DHA)

245 Dietary n-3 fatty acids, particularly eicosapentaenoic acid and

246 and gelled double emulsion, for delivery of n-3 fatty acids (perilla oil at 300g/kg) and hydroxytyro

247 ether the ratio of omega-6 (n-6) to omega-3 (n-3) fatty acids plays a role in tumorigenesis remains t

248 forcement of the antioxidant system, through n-3 fatty acids plus antioxidant vitamin supplementation

249 children had high ratios, a higher intake of n-3 fatty acids predicted a better performance on the pl

250 rse association between intake of long chain n-3 fatty acids [primarily eicosapentaenoic acid (EPA) a

251 These data support the hypothesis that n-3 fatty acids protect from high-fat diet-induced hepat

252 useful model for elucidating the role of n-6/n-3 fatty acid ratio in tumorigenesis.

253 Fish were fed diets containing different n-6/n-3 fatty acid ratios (V0.5 or V1) and sesamin contents

254 Oxidized n-3 fatty acids reacted directly with the negative regul

255 The n-3 fatty acid recommendation to achieve nutritional ade

256 istent across 3 studies was the finding that n-3 fatty acids reduce corticosteroid requirements, alth

257 Fish oils rich in n-3 fatty acids reduce serum triglyceride levels.

258 Several lines of evidence suggest that n-3 fatty acids reduce the risk of some chronic diseases

259 idered tentative, and the mechanism by which n-3 fatty acids reduce triglyceride levels in humans rem

260 As examples, n-3 fatty acids regulate 2 groups of transcription facto

261 ogram produced 11 reports on a wide range of n-3 fatty acid-related topics.

262 olic and cardiovascular benefits of omega-3 (n-3) fatty acids, several studies have suggested an adde

263 In the parafovea, animals low in n-3 fatty acids showed greater sensitivity to damage tha

264 a mechanism where membrane alteration by the n-3 fatty acid status affects Akt signaling, impacting n

265 es support a direct association between poor n-3 fatty acid status and increased risk of maternal dep

266 upplementation successfully increased plasma n-3 fatty acid status.

267 Omega-3 (n-3) fatty acids stimulate protein anabolism in animals

268 on trials, that examine the relation between n-3 fatty acids such as DHA and depression and child men

269 both groups, and the addition of aspirin to n-3 fatty acid supplementation did not alter any of the

270 Diet and n-3 fatty acid supplementation dramatically reduced seru

271 ins were increased to a similar extent after n-3 fatty acid supplementation in both groups, and the a

272 ecursors 18-HEPE, 17-HDHA, and 14-HDHA after n-3 fatty acid supplementation that was significantly at

273 ased to a similar extent in the groups after n-3 fatty acid supplementation, and the D-series resolvi

274 solvins as well as of 14-HDHA in response to n-3 fatty acid supplementation.

275 change of serum metabolomics in response to n-3 fatty acid supplements in Chinese patients with type

276 mol per liter) more among patients receiving n-3 fatty acids than among those receiving placebo (P<0.

277 e had 2-fold higher levels of marine-derived n-3 fatty acids than whites and Japanese Americans in th

278 SDA-enriched soybean oil is a land-based n-3 fatty acid that is a sustainable approach to increas

279 te that DHA, but not EPA, is the most potent n-3 fatty acid that suppresses glomerulonephritis and ex

280 ional studies utilizing tissue biomarkers of n-3 fatty acids that more accurately measure dietary con

281 Omega-3 polyunsaturated fatty acids (PUFA, n-3 fatty acids), the key components of fish and flaxsee

282 Amongst the n-3 fatty acids, the concentrations of alpha-linolenic a

283 and other fatty acids, including long-chain n-3 fatty acids, the intake of alpha-linolenic acid was

284 studies fed unphysiologically high doses of n-3 fatty acids, these findings in rats must be consider

285 trate an anticancer (antimelanoma) effect of n-3 fatty acids through, at least in part, activation of

286 ake variations of omega-6 (n-6) and omega-3 (n-3) fatty acids ultimately determine cell membrane inco

287 r prevention, including lycopene, long-chain n-3 fatty acids, vitamin D, vitamin E, and selenium, wer

288 An increasing ratio of total dietary n-6 to n-3 fatty acids was also significantly and independently

289 Dietary consumption of seafood and n-3 fatty acids was annually assessed by a food frequenc

290 However, the allocation to receive n-3 fatty acids was positively associated with depressiv

291 percentage of energy from fat, the intake of n-3 fatty acids was significantly associated with fewer

292 tored hen eggs enriched with very long-chain n-3 fatty acids, was investigated.

293 The proportions of plasma long-chain n-3 fatty acids were not significantly affected by the d

294 ted seafood intake and estimated or measured n-3 fatty acids, whereas associations with self-reported

295 Meat products are generally low in omega-3 (n-3) fatty acids, which are beneficial to human health.

296 icant inverse associations of marine-derived n-3 fatty acids with IMT and CAC prevalence, respectivel

297 is a lack of research on the association of n-3 fatty acids with risk of benign fibrocystic breast c

298 that dietary supplementation with selective n-3 fatty acids would be most beneficial for the treatme

299 ed that children with lower ratios of n-6 to n-3 fatty acids would perform better on tests of plannin

300 phospholipid measures of long-chain omega-3 (n-3) fatty acids would be positively associated with lar