ライフサイエンスコーパス: nA

1 2.1-20.6 mM and a sensitivity of 7.8 +/- 1.0 nA/mM (n = 6).

2 k exhibited high sensitivity (465.9 +/- 48.0 nA/mM), a low detection limit of 1 muM, a linear respons

3 ciliary neurons had large-amplitude (1.5-8.0 nA) EPSCs that could be classified according to the kine

4 ns during GABA diffusion from the pipette (0 nA) and the response quickly progressed to complete sile

5 n potential (AP) generation (0.012 +/- 0.004 nA); (iv) firing of APs throughout a depolarizing pulse

6 dition to a high sensitivity (-0.16 +/- 0.02 nA muM(-1)) and a low limit of detection (0.33 +/- 0.20

7 ectrode is achieved at a sensitivity of 0.02 nA/spot.

8 nM for cysteine with sensitivities of 0.023 nA/microM and 4.71 nA/microM, respectively.

9 evation (from 0.22 +/- 0.04 to 0.30 +/- 0.03 nA, P < .05) of the threshold current amplitude required

10 , increased dramatically from -0.14 +/- 0.04 nA at P1 to -6.71 +/- 0.65 nA at P4 with sharp jumps bet

11 rent amplitudes to pH 6.0 were 0.84 +/- 0.06 nA (oxidative muscle) versus 1.36 +/- 0.07 nA (glycolyti

12 6 nA (oxidative muscle) versus 1.36 +/- 0.07 nA (glycolytic muscle, P < 0.05).

13 range with a resulting sensitivity of 0.096 nA muM(-1).

14 Muller cells (1.3 +/- 0.1 versus 1.2 +/- 0.1 nA at -160 mV) or in inwardly rectifying current-voltage

15 an be focused to less than 3 microm with 0.1 nA currents.

16 ar response above 20 microm with approx. 0.1 nA/microM slope.

17 Nonmass selective currents up to 1.1 nA and mass-selected currents of up to 500 pA have been

18 /- 3 mU/min; and LCL was 4 +/- 1 and 5 +/- 1 nA in steatotic and nonsteatotic hepatocytes, respective

19 g the aptamer surface coverage, was 67 +/- 1 nA muM(-1) cm(-2), and the dopamine LOD was 62 nM.

20 imally activated currents (Imax) of around 1 nA.

21 ourse similar to the PF sEPSP, peaking at -1 nA in 700 ms.

22 GHz, at which the pumping current exceeds 1 nA.

23 -14 mice, light-evoked EPSCs were large (> 1 nA at -70 mV).

24 ted Cl(-) currents in HEK cells that were >1 nA in amplitude.

25 or example, a mean outward K(+) current of 1 nA for 2 s could decrease [K(+)](i) from 10 mM to 3 mM i

26 N cells of several hundred pA to more than 1 nA.

27 ane potential, firing rate in response to +1 nA of injected current, slope of the frequency-current c

28 Lower sensitivities (1.13 x 10(-1) nA/mM glucose) are observed when immobilization method i

29 y increased in amplitude from -0.76 +/- 0.10 nA at 25 degrees C to -1.11 +/- 0.19 nA 35 degrees C.

30 was attenuated by application of Mg2+ (1-10 nA) in sixteen of seventeen neurones or Cd2+ (2-10 nA) i

31 sixteen of seventeen neurones or Cd2+ (2-10 nA) in seven of eight neurones tested.

32 Ionophoresis of AP5 (2-10 nA), at currents which selectively inhibited NMDA-evoked

33 h was evident at low ejection currents (5-10 nA), had relatively short onset (4-12 s) and offset (6-2

34 of approximately 0.1 V and approximately 10 nA cm(-2) .

35 outward potassium current ( approximately 10 nA), decreased muscle input resistance (50-fold), and a

36 samples yielding mass 44 ion currents of 10 nA.

37 ials of +/-2 VDC and gains of 1, 10, and 100 nA/V.

38 as highly selective and sensitive (LRS>/=100 nA/mM) for each analyte and within an adequate range for

39 is kept below a certain critical level (<100 nA at positive potential and <25 nA at negative potentia

40 t can be tuned from zero to greater than 100 nA.

41 from approximately 14.5 to approximately 103 nA by combining the pyro-phototronic and piezo-phototron

42 ble, albeit at a decreased sensitivity (0.11 nA muM(-1)).

43 creased Im (-0.249+/-0.038 to -0.571+/-0.111 nA, P<0.05) at -100 mV and reduced Rm (151+/-21 to 77+/-

44 rease in hSlo1 current density from 20 to 12 nA*M ohm.

45 eurones tested, ionophoresis of Mg2+ (10-120 nA) attenuated the PBG-evoked increases in synaptic nois

46 (40 nA) and excitation at high currents (120 nA).

47 of 8.25 nmol L(-1) and a sensitivity of 120 nA L micromol(-1).

48 response time (<4 s), high sensitivity (1200 nA/mM x cm2), low interference from endogenous electroac

49 king electrode yields a sensitivity of 0.127 nA/muM and a limit of quantitation (LOQ) of 9 muM.

50 reased sensitivity to dopamine, at 46 +/- 13 nA/muM, compared to 26 +/- 6 nA/muM for the electrodes c

51 culture medium (pH 7.3), a sensitivity of 13 nA/mM was obtained and the response was linear up to 5 m

52 Three-terminal memory devices produced 14 nA read currents at an operating voltage of 5 V, and ope

53 induced current amplitudes were 2.3 +/- 0.15 nA (oxidative muscle) versus 3.1 +/- 0.21 nA (glycolytic

54 the energy spread of the electron beam at 15 nA is 100 meV or better.

55 4-fold beta-saturated porphynoids are 13-17 nA/T, showing that the inner-cross 18pi [16]annulene pat

56 ch would deny coherent superpositions of 170 nA currents over a approximately 10 ns timescale.

57 /- 0.10 nA at 25 degrees C to -1.11 +/- 0.19 nA 35 degrees C.

58 tegrated NG device reaches up to 3 V and 195 nA under human walking conditions.

59 SP produced an outward current of 3 +/- 0.2 nA (n = 10).

60 8 +/- 0.2 nA in control cells to 0.9 +/- 0.2 nA in cells expressing the epsilon subunit (P < 0.05).

61 de activated by GABA (1 mM) from 1.8 +/- 0.2 nA in control cells to 0.9 +/- 0.2 nA in cells expressin

62 mit of detection for glucose of 19.4 +/- 0.2 nA mM(-1) and 13.1 +/- 0.7 muM, respectively.

63 njected or uninjected) oocytes (-1.0 +/- 0.2 nA); the Na+-dependent histidine transport showed a stoi

64 Na+-independent outward current (11 +/- 1.2 nA) in NBAT-expressing oocytes.

65 ectrodes coated in 200 muM EDOT and 13 +/- 2 nA/muM for an uncoated fiber.

66 10 nM and 220 microM, a sensitivity of 14.2 nA x microM(-1), and good selectivity against ascorbic a

67 pA pF(-1); cutaneous Aalpha/beta LTMs: -2.2 nA, -20 pA pF(-1); Abeta-nociceptors: -2.6 nA, -21 pA pF

68 tailoring of the glucose electrodes for > 2 nA/mM sensitivity; 0-30 mM dynamic range; drift of < or

69 l initiation required currents of at least 2 nA for their generation and never occurred repetitively.

70 te voltages (<1 V) with low gate leakage (<2 nA), highlighting the defect-free and conformal nature o

71 In most cells, short (2 ms), strong (2 nA) current injections elicited a single spike followed

72 Prolonged GABA application (0-20 nA, 2-4 min) reduced basal impulse activity, but was les

73 DNQX (5-20 nA), at currents which selectively inhibited AMPA-evoked

74 ively high AMPH ejection currents (> or = 20 nA).

75 ones ionophoretic application of PBG (10-200 nA) depolarized the membrane and increased firing rate w

76 ound to be generated at a current of 150-200 nA in detectable quantities: with a yield of 0.5-1 H2O2

77 ug/mL was electrosprayed at a current of 200 nA.

78 15 nA (oxidative muscle) versus 3.1 +/- 0.21 nA (glycolytic muscle, P < 0.05).

79 and 100 muM, sensitivity of 275, 500 and 217 nA muM(-1) cm(-2), and detection limits of 0.4, 0.2 and

80 sitivities were determined to be 205 and 222 nA/microM, respectively.

81 mic range, 6 nM-0.4 microM; sensitivity, 225 nA microM(-1); detection limit (k = 3 criterion), 2 nM.

82 11.7, with average NO sensitivities of 1.24 nA/muM and a limit of detection (LOD) of <1 nM.

83 the chlorins and bacteriochlorins are 19-24 nA/T depending on whether the ring current is forced to

84 level (<100 nA at positive potential and <25 nA at negative potential for 96% ethanol; < 40 nA at pos

85 An electron beam of 250 nA could be obtained through the 0.45-mm diameter openin

86 nA, -7.6 pA pF(-1); and C-nociceptors: -0.26 nA, -5 pApF(-1).

87 microm)-stimulated K+ currents of 308 +/-26 nA and 298 +/-29 nA, respectively, which were both Ba2+-

88 ns have the strongest ring current of ca. 27 nA/T among the investigated porphynoids.

89 ed K+ currents of 308 +/-26 nA and 298 +/-29 nA, respectively, which were both Ba2+- and pertussis to

90 Current was injected (+/-0.1-3 nA) at site 1 while recording membrane potential (V(m))

91 biosensor shows a sensitivity of 4.7 +/- 1.3 nA mM(-1)mm(-2) and a detection limit of 1.4mM (S/N = 3s

92 (-1); both Adelta-LTMs and nociceptors: -1.3 nA, approximately -14 pA pF(-1); C-LTMs: -0.4 nA, -7.6 p

93 sensitivity of the biosensor was 110 +/- 1.3 nA/(mM mm(2)) with the apparent Michaelis-Menten constan

94 100 nm gave the highest sensitivity of 19.3 nA mL (pg IL-6)(-1) cm(-2) and the best detection limit

95 0.1 to 100 muM, and high sensitivity of 76.3 nA muM(-)(1) were achieved for the detection of methylgl

96 Prolonged AMPH iontophoresis (2-3 min; 5-30 nA) inhibited both spontaneous impulse activity and Glu-

97 active neurons known to respond to ACh (5-30 nA) when the animals rested quietly with no overt moveme

98 10+/-9 microM), and 3-deazauridine (506+/-30 nA, 50.8+/-9.90 microM).

99 sivity of 0.31 A/W), 3 GHz bandwidth, and 30 nA dark current at a reverse bias of 30 V.

100 deposition at 0 V led to a sensitivity of 34 nA nM(-1) min(-1), a 4-fold improvement over previous me

101 r produced an inward current of 1.6 +/- 0.35 nA (n = 27) in approximately 80% of the neurones.

102 red the current by 29% versus WT (243 +/- 35 nA, n=13, p<0.05).

103 ction limit of 6.4 muM and sensitivity of 36 nA mM(-1).

104 nsitivity were calculated as 0.229 mM, 42.37 nA, 3.3 x 10(-4)nM and 6.4 nA/mM cm(2), respectively.

105 nA, 59.7+/-17.5 microM), ribavirin (546+/-37 nA, 61.0+/-13.2 microM), AZT (420+/-4 nA, 310+/-9 microM

106 ielded sensitivities of 0.38, 0.41, and 0.38 nA/ppmv for measurements at 9%, 33%, and 76% humidity, r

107 The sensitivity of 2.38 nA ppb(-1) obtained in the present work for As(III) quan

108 roM (five points) had slopes of 35.2 +/- 0.4 nA microM(-1) and correlation coefficients of 0.999.

109 A, approximately -14 pA pF(-1); C-LTMs: -0.4 nA, -7.6 pA pF(-1); and C-nociceptors: -0.26 nA, -5 pApF

110 ith sensitivities of 9 +/- 9 and 1.2 +/- 0.4 nA/muM, respectively.

111 /mug mL(-1), A = 92.9 nA/mug mL(-1), T = 1.4 nA/mug mL(-1), and C = 15.1 9 nA/mug mL(-1)), low limit

112 buffer solutions with a sensitivity of 26.4 nA/mM and a linear range of 0.45 to 9.0 mM.

113 6+/-37 nA, 61.0+/-13.2 microM), AZT (420+/-4 nA, 310+/-9 microM), and 3-deazauridine (506+/-30 nA, 50

114 s 0.229 mM, 42.37 nA, 3.3 x 10(-4)nM and 6.4 nA/mM cm(2), respectively.

115 accelerate a deuteron beam (> 100 keV and >4 nA), which, upon striking a deuterated target, produces

116 single large input, typically larger than 4 nA, emerged from P3-P4.

117 tracellular recording experiments, PBG (0-40 nA) increased the firing rate of thirty-five of the thir

118 Iontophoretically applied GLU (0-40 nA, 20 s) excited all spontaneously active neurons in do

119 neurons were highly sensitive to GABA (0-40 nA, 20 s); most showed short-latency inhibitions during

120 AMPH dose-dependently (5-40 nA) inhibited the vast majority of spontaneously active

121 nded to cause inhibition at low currents (40 nA) and excitation at high currents (120 nA).

122 ut relatively high AA ejection currents (>40 nA) often inhibited fast-firing units.

123 at negative potential for 96% ethanol; < 40 nA at positive potential for water).

124 on limit of 2.8 nM and a sensitivity of 9.46 nA microM-1.

125 reatment from 0.55 +/- 0.32 to 3.25 +/- 0.47 nA (n=6, P < .01).

126 e and excellent selectivity to glucose (0.47 nA/mM) against interferants such as ascorbic acid, uric

127 tion of the M3 receptor produced 2382 +/-478 nA of current which was insensitive to Ba2+ and pertussi

128 le L-type currents were measured (469 +/- 48 nA in 10 mM Ba2+).

129 The sensitivity is 49 nA nM(-1) min(-1) with 30 s deposition time and the LOD

130 round 5 nA (: 40-50 ms) at P10/11 to 0.3-0.5 nA (: 10-15 ms) by P18.

131 ons had smaller-amplitude responses (0.2-1.5 nA when all inputs were activated) that appeared to cont

132 1 mM) evoked current (I(His) = -14.7 +/- 1.5 nA) in NBAT-expressing oocytes compared with native (wat

133 ude (and dominant decay ) fell from around 5 nA (: 40-50 ms) at P10/11 to 0.3-0.5 nA (: 10-15 ms) by

134 iostat with a large dynamic current range (5 nA to 1.2 mA) and short conversion time (10 ms) were fab

135 intracellular current injection (+/-0.5 to 5 nA, 20 s pulses) while V(m) changed linearly between app

136 Microiontophoresis of ACh (500 ms, 500 nA) onto the distal end of a feed artery evoked hyperpol

137 of the SAMN-BMIM-PF6-CP electrode was 206.51 nA muM(-1)cm(-2), with a detection limit (S/N=3) of 0.8

138 76 nA (mean +/- SEM), n=13 versus 342 +/- 55 nA in WT, n=13), while the co-expression of 1/2 WT+1/2 R

139 t of 200 nM and sensitivity of 84.5 +/- 1.56 nA microM(-1)cm(-2).

140 Enhanced sensitivity (up to 1.56 nA muM(-1)) and lowered theoretical detection limits (do

141 ucleoside analog drugs gemcitabine (638+/-58 nA, 59.7+/-17.5 microM), ribavirin (546+/-37 nA, 61.0+/-

142 in AMPA-mediated current amplitude (0.3-0.6 nA) in the range of CA1 apical dendrites that receive a

143 iration a large magnitude (approximately 0.6 nA) outward current generated by Na(+)/K(+) ATPase that

144 from -9 +/- 0.8 nA at pH 7.5 to -19 +/- 2.6 nA at pH 6.5, at which histidine is predominantly cation

145 2 nA, -20 pA pF(-1); Abeta-nociceptors: -2.6 nA, -21 pA pF(-1); both Adelta-LTMs and nociceptors: -1.

146 e, at 46 +/- 13 nA/muM, compared to 26 +/- 6 nA/muM for the electrodes coated in 200 muM EDOT and 13

147 ectively:muscle spindle afferents(MSAs):-4.6 nA,-33 pA pF(-1); cutaneous Aalpha/beta LTMs: -2.2 nA, -

148 n large inward 'AD current' (approximately 6 nA) which was largely prevented by blocking AMPA recepto

149 iezoelectric generator that produces up to 6 nA of current and 400 mV of potential and use it to oper

150 from 0.2 to 3.0 nM (R (2) = 0.9947) and 7.60 nA x microM (-1) from 0.5 to 4.0 microM ( R (2) = 0.9999

151 that of CDPG (maximal amplitude, 272 +/- 62 nA).

152 <1 s, and the sensitivity of analysis was 62 nA muM(-1) cm(-2).

153 om -0.14 +/- 0.04 nA at P1 to -6.71 +/- 0.65 nA at P4 with sharp jumps between P2 and P4.

154 opores have unusually high sensitivity (0.65 nA/A) to extremely small changes in the translocating mo

155 een 5 and 200 nM, and a sensitivity of 83.65 nA x microM(-1) were recorded.

156 can be detected with the sensitivity of 0.7 nA muM(-1) and the limit of detection of 0.5 muM (3 sB/m

157 mit of 50 nM and sensitivity of 98.5 +/- 1.7 nA microM(-1)cm(-2).

158 GLU-induced excitations (mean threshold 19.7 nA) were dose-dependent, inversely correlated with rate

159 osensor exhibits high sensitivity (1.9x10(7) nA(-1)), low limit of detection (1.7x10(-7) M), high sto

160 th sensitivities of 0.023 nA/microM and 4.71 nA/microM, respectively.

161 age sensitivity of the microsensors was 1.72 nA/muM and the limit of detection was 25 nM.

162 00 ng/muL with improved sensitivity of 36.72 nA/ng/cm(2), faster response time of 3s and high stabili

163 reased from -3.14 +/- 0.59 to -4.15 +/- 0.73 nA with the fast decay time constant accelerating from 0

164 rrent were similar (approximately 365 +/- 75 nA at 1 microM CDPG) to those of InsP3.

165 uced a current similar to the WT (369 +/- 76 nA (mean +/- SEM), n=13 versus 342 +/- 55 nA in WT, n=13

166 ward 0.1 mM I(His) increased from -9 +/- 0.8 nA at pH 7.5 to -19 +/- 2.6 nA at pH 6.5, at which histi

167 In a reciprocal manner, +0.8 nA caused depolarization ( approximately 2 mV) of SMCs w

168 Injection of -0.8 nA into an EC caused hyperpolarization ( approximately 5

169 3) was 8-130 muM and the sensitivity was 1.8 nA muM(-1) (RSD=5.0%) for substrate of Baeyer-Villiger o

170 performance with a sensitivity of 2.4+/-1.8 nA/microM, a response time of 20+/-13 s and a lower dete

171 r exhibited excellent sensitivity (G = 178.8 nA/mug mL(-1), A = 92.9 nA/mug mL(-1), T = 1.4 nA/mug mL

172 te biosensor the sensitivity is 12.2 +/- 3.8 nA mM(-1)mm(-2) and the detection limit is 0.3mM.

173 Current injection (up to 8 nA) into a single CM cell elicited electrotonic potentia

174 nnels with 0.33 +/- 0.14 versus 2.5 +/- 0.80 nA of amiloridesensitive inward current at -80 mV.

175 onophoretic application of ATP (0.2 M, 20-80 nA for 40 s) increased the activity of approximately 80

176 Brief applications of AA (20 s, 5-80 nA) elicited few changes in either basal activity or act

177 vity toward the oxidation of oxalic acid (80 nA/nM) achieved by the amperometry method.

178 the P2 receptor blocker suramin (0.02 M, 80 nA), which also reduced the baseline firing in some expi

179 at the sensitivity increased (1840 to 25 800 nA muM(-1)) and the limit of detection decreased (11.10

180 ose, characterized by a sensitivity of 45.85 nA muM(-1)cm(-2) and a detection limit (S/N=3) of 0.9 mu

181 L(-1), T = 1.4 nA/mug mL(-1), and C = 15.1 9 nA/mug mL(-1)), low limit of detection (G, A = 0.5 mug m

182 equivalent current dipole source was 38+/-9 nA-m (n=17).

183 nsitivity (G = 178.8 nA/mug mL(-1), A = 92.9 nA/mug mL(-1), T = 1.4 nA/mug mL(-1), and C = 15.1 9 nA/

184 Sensitivities in the range of 9.97 nA/mM glucose are observed when the enzyme is immobilize

185 imum amplitude of reduction peak current (A, nA), a reduction peak area (S, nA x V), and a peak poten

186 ary A-T couple consisting of 2-aminoadenine (nA) and 2-thiothymine (sT) bases.

187 one that could be used with 2-aminoadenine (nA) and 2-thiothymine (sT) to generate structure-free DN

188 nucleoside triphosphates of 2-aminoadenine (nA) and 2-thiouracil (sU) are taken up by T7 RNA polymer

189 Bicoid (Bcd) morphogen gradient's amplitude nA.

190 In contrast, 300 microgram ml-1 Co nA (a specific kainate receptor desensitization blocker)

191 DNA-RNA hybrids that contained nA and sU in the RNA strand exhibited enhanced specifici

192 RNA hairpins that contained nA and sU were able to hybridize to DNA probes under con

193 r protein heterointeractions of the type D + nA<-->DA(n).

194 implied by classic nucleation theory and its nA right harpoon over left harpoon An, "critical nucleus

195 These results suggest that incorporation of nA and sU during in vitro transcription is a promising s

196 The high stability of nA-T and A-sU base pairs in DNA-RNA hybrids, combined wi

197 life cycle, stemming from a finite value of nA~3, underscores a key feature of developmental systems

198 n permitted high gain (1 V/nA) to measure pA-nA level currents in the detection cell.

199 k current (A, nA), a reduction peak area (S, nA x V), and a peak potential (P, V), were measured for

200 The nA-sT couple is a mismatch even though nA-T and A-sT are

201 ombined with the destabilizing effect of the nA-sU couple in RNA targets, accounts for the improved h

202 The nA-sT couple is a mismatch even though nA-T and A-sT are stable base pairs.

203 No A(U)nA mRNA instability motifs were present.

204 dance configuration permitted high gain (1 V/nA) to measure pA-nA level currents in the detection cel

205 yl or ethyl) can be used in conjunction with nA and sT to render DNA largely structure-free and pseud

206 Either analog could be used with nA and sT to generate DNA that was nearly structure-free